ライフサイエンスコーパス: hyperplasia

1 "Castleman's disease" and "giant lymph node hyperplasia".

2 sed differentiation of adipocyte precursors (hyperplasia).

3 HRL histologic results (eg, atypical ductal hyperplasia).

4 ration of submucosal gastrin-expressing cell hyperplasia.

5 to be patent with nonobstructive neointimal hyperplasia.

6 elivering rapamycin inhibit patch neointimal hyperplasia.

7 esses that result in dramatic beta-like cell hyperplasia.

8 risks of primary macronodular adrenal gland hyperplasia.

9 H-HCA, IHCA, b-HCA, UHCA, and focal nodular hyperplasia.

10 s (TART) in patients with congenital adrenal hyperplasia.

11 ins (HSP27 and CREB) associated with intimal hyperplasia.

12 th regenerative glandular changes and neural hyperplasia.

13 develop novel therapies for benign prostatic hyperplasia.

14 , IL-5, and IL-13) and decreased goblet cell hyperplasia.

15 (PTEN) experience autoimmunity and lymphoid hyperplasia.

16 One CFLD patient had nodular regenerative hyperplasia.

17 dilatation and carotid artery intima-medial hyperplasia.

18 increased NFkappaB activation and neointimal hyperplasia.

19 flammation in atherosclerosis and neointimal hyperplasia.

20 on in liver and muscle, and pancreatic islet hyperplasia.

21 enesis, plaque stabilization, and neointimal hyperplasia.

22 n did patients with RAEB-1 without erythroid hyperplasia.

23 entiate prostate cancer and benign prostatic hyperplasia.

24 solid nodular malignancies as well as cystic hyperplasia.

25 d thrombocytosis due to a megakaryocyte (MK) hyperplasia.

26 tosis, increased hemoglobin, and bone marrow hyperplasia.

27 tory sensitivity to hypoxia and carotid body hyperplasia.

28 gnostic differentiation of benign and malign hyperplasia.

29 ctive autophagy in IL-17A-mediated epidermal hyperplasia.

30 s due to increased adipocyte hypertrophy and hyperplasia.

31 ficient propagation of SSCs derived from the hyperplasia.

32 cgr(-/-) mice as a consequence of alpha-cell hyperplasia.

33 nts targeted to inhibit localized neointimal hyperplasia.

34 ation, and its inhibition leads to stem cell hyperplasia.

35 e recruitment of eosinophils and goblet cell hyperplasia.

36 ompanied by a reduction in venous neointimal hyperplasia.

37 death on a ventilator and type II pneumocyte hyperplasia.

38 tional PAF expression induces mammary ductal hyperplasia.

39 is repositioned in both prostate cancer and hyperplasia.

40 ent to cause skin inflammation and epidermal hyperplasia.

41 athologically proven parathyroid adenomas or hyperplasia.

42 eight months of age as a result of myofiber hyperplasia.

43 mice, indicated by flattened villi and crypt hyperplasia.

44 show severe fibrosis and display significant hyperplasia.

45 in the preventive action of E2 on neointimal hyperplasia.

46 nfection leading to biliary inflammation and hyperplasia.

47 -induced airway inflammation and goblet cell hyperplasia.

48 nesis and results in lumen-occlusive intimal hyperplasia.

49 ed by vasoproliferation and endothelial cell hyperplasia.

50 increased cell proliferation and epithelial hyperplasia.

51 ME bacterial clearance and prolonged mucosal hyperplasia.

52 ink glucagon receptor blockage to alpha-cell hyperplasia.

53 beta-cell tumours (insulinomas) or beta-cell hyperplasia.

54 l pool to cause hyperproliferation and gland hyperplasia.

55 ion factor, which contributes to goblet cell hyperplasia.

56 accompanied by a decrease in UVR-induced (1) hyperplasia, (2) Hsp90beta-PKCvarepsilon interaction, an

57 (f/f) mice also revealed no complex atypical hyperplasia, a well-established EAC precursor.

58 l displayed features of atypical adenomatous hyperplasia, adenocarcinoma in situ, and invasive adenoc

59 e rate of underestimation of atypical ductal hyperplasia (ADH) and ductal carcinoma in situ (DCIS) at

60 Atypical ductal hyperplasia (ADH) is a known risk factor for breast canc

61 ease, such as lung eosinophilia, goblet cell hyperplasia, Ag-specific Th2 responses, and IgE producti

62 including airway hyperresponsiveness, mucus hyperplasia, airway eosinophilia, and type 2 pulmonary i

63 chemia time developed severe TV with intimal hyperplasia (alpha-smooth muscle actin positive cells in

64 d it rely upon hyperinsulinemia or beta-cell hyperplasia, although PKA activity was protective for be

65 ol mice, with a greater extent of epithelial hyperplasia and a larger tumor burden.

66 eveloping mouse pituitary, results in severe hyperplasia and abnormal morphogenesis of the gland by t

67 Alveolar adenomatous hyperplasia and adenocarcinoma were significantly increa

68 ar inflammatory disorders such as neointimal hyperplasia and atherosclerosis.

69 omplications resulting from benign prostatic hyperplasia and bladder outlet obstruction continue to b

70 eproductive disorders, including endometrial hyperplasia and breast cancer.

71 in mice using a cre/lox strategy induces EC hyperplasia and causes blood vessels to coalesce into la

72 1 (TRAP1) in the prostate develop epithelial hyperplasia and cellular atypia.

73 n and p53-dependent parietal epithelial cell hyperplasia and crescent formation.

74 Gdf5-lineage cells prevents synovial lining hyperplasia and decreases contribution of Gdf5-lineage c

75 remalignant gastric disorder with epithelial hyperplasia and enhanced EGFR signalling.

76 n of Ptch1 in RP progenitors leads to severe hyperplasia and enlargement of the Sox2(+) stem cell com

77 Fallot associated with nodular regenerative hyperplasia and focal nodular hyperplasia (FNH), which f

78 Trp53 loss was sufficient to drive both ICC hyperplasia and formation of multifocal GIST-like tumors

79 d to differentiate PCa from benign prostatic hyperplasia and have high false positive rates.

80 e liver, Hippo pathway deregulation promotes hyperplasia and hepatocellular carcinoma primarily throu

81 in vitro isoproterenol-induced cardiomyocyte hyperplasia and hypertrophy.

82 meotic)-like (Drosophila)] develop epidermal hyperplasia and impaired epidermal stratification upon a

83 subepithelial fibrosis, airway smooth muscle hyperplasia and increased angiogenesis.

84 PHKCs and immunocytes abolished psoriasiform hyperplasia and inflammation in vivo.

85 are due to the presence of basal melanocyte hyperplasia and intraepidermal Langerhans cells.

86 n obesity may result from hypertrophy and/or hyperplasia and is frequently associated with adipose ti

87 target that is activated in ASCs during WAT hyperplasia and is functionally required for dermal ASC

88 uces an IL-22 response that drives epidermal hyperplasia and keratinocyte proliferation in a mouse mo

89 xposure to complete darkness reduces biliary hyperplasia and liver fibrosis in bile-duct-ligated (BDL

90 synthase (iNOS), a gene linked to bile duct hyperplasia and liver fibrosis.

91 stological types: normal cases, usual ductal hyperplasia and low/high grade ductal carcinoma in situ

92 ischemic organ damage through rapid intimal hyperplasia and luminal occlusion.

93 cular damage, with markedly increased medial hyperplasia and neointimal growth, and evidence of highe

94 h Amelogenesis Imperfecta (AI) with gingival hyperplasia and nephrocalcinosis, while FAM20C mutations

95 acterized by thyrocyte epithelial cell (TEC) hyperplasia and proliferation (H/P).

96 s associated with FBP loss; besides cerebral hyperplasia and pulmonary hypoplasia, pale livers, hypop

97 s to a vascular segment to reduce neointimal hyperplasia and restenosis.

98 ctive in prevention of UVR-induced epidermal hyperplasia and SCC.

99 -dependent network that constrains epidermal hyperplasia and squamous tumorigenesis.

100 (particularly clobetasol) restored epidermal hyperplasia and terminal differentiation versus minimal

101 yrosine kinases Src42A and Src64B to promote hyperplasia and tissue disorganization, which results in

102 po3 fusion events are sufficient to initiate hyperplasia and tumour development in vivo, without addi

103 elial eosinophilia in addition to basal cell hyperplasia and vascular remodeling.

104 with coordinated sprouting, thereby causing hyperplasia and vessel enlargement.

105 promotes MC dedifferentiation, motility, and hyperplasia and, in an autochthonous melanoma model, res

106 Loss of this regulation results in GALT hyperplasia and, in some animals, mucosa-associated B ce

107 strophy and non classical congenital adrenal hyperplasia, and an essential splice site mutation in a

108 ological indices of eosinophilia, epithelial hyperplasia, and angiogenesis by immunohistochemistry an

109 polyarticular arthritis, dermatitis, myeloid hyperplasia, and autoimmunity.

110 ctable from birth to adulthood, is driven by hyperplasia, and coincides with excess neurons at birth

111 ts, mucosa-associated lymphoid tissue (MALT) hyperplasia, and dysplasia.

112 attern of organ growth, pancreatic beta cell hyperplasia, and elevated plasma insulin and leptin conc

113 inished intestinal smooth muscle hypertrophy/hyperplasia, and impaired worm expulsion.

114 nti-Pneumocystis IgE production, goblet cell hyperplasia, and increased airway resistance.

115 t mice had reduced iNOS induction, bile duct hyperplasia, and liver fibrosis.

116 69 years of age, those with benign prostatic hyperplasia, and those with a family history of prostate

117 by eosinophil infiltration, epithelial cell hyperplasia, and tissue remodeling.

118 role in wound healing, phorbol ester-induced hyperplasia, and tumor development.

119 istologic assessment demonstrated neointimal hyperplasia around the IVC filter within 2 weeks after I

120 Goblet cell hyperplasia, as indicated by PAS staining, was not diffe

121 vascular restenosis by preventing neointimal hyperplasia at the early stage via suppression of the MM

122 Optical coherence tomography showed RPE hyperplasia at the perimeters of annular lesions with lo

123 re includes thrombotic occlusion and intimal hyperplasia at the site of vascular anastomosis.

124 prone to thrombosis, infection, and intimal hyperplasia at the venous anastomosis.

125 roducing adenoma (APA) and bilateral adrenal hyperplasia (BAH), remains a matter of debate.

126 cterized by epithelial shedding, goblet cell hyperplasia, basement membrane thickening, subepithelial

127 Benign prostatic hyperplasia (BPH) and associated lower urinary tract sym

128 sparing undesired effects of benign prostate hyperplasia (BPH) and liver-mediated decrease in HDL-C.

129 ronic prostatitis (CPr) and benign prostatic hyperplasia (BPH) are complex inflammatory conditions fo

130 The etiology of benign prostatic hyperplasia (BPH) is multifactorial, and chronic inflamm

131 (VOIs) for prostate tumors, benign prostatic hyperplasia (BPH) nodules, prostatitis, and healthy tiss

132 cotherapies for symptomatic benign prostatic hyperplasia (BPH), an androgen receptor-driven, inflamma

133 ct symptoms associated with benign prostatic hyperplasia (BPH), the etiology of which is not well und

134 uggested as an etiology for benign prostatic hyperplasia (BPH).

135 h normal prostate tissue and benign prostate hyperplasia (BPH).

136 patients with prostatitis or prostate benign hyperplasia (BPH).

137 ous disease categories (eg, benign prostatic hyperplasia [BPH], prostatic intraepithelial neoplasia [

138 nevus (492 [61%]), benign reactive lymphoid hyperplasia (BRLH) (38 [5%]), nodular conjunctivitis (31

139 600E) activation was sufficient to drive ICC hyperplasia but not GIST tumorigenesis.

140 arette smoke promotes ductal epithelial cell hyperplasia by stimulating stromal endothelial cell prol

141 or basic protein immunostaining, goblet cell hyperplasia by using periodic acid-Schiff staining, and

142 mainstay of treatment for congenital adrenal hyperplasia (CAH) but has a narrow therapeutic index and

143 e genital virilization is congenital adrenal hyperplasia (CAH), in which excess androgen production d

144 Congenital adrenal hyperplasia (CAH), resulting from mutations in CYP11B1,

145 1A2 variants give rise to congenital adrenal hyperplasia (CAH).

146 ely treated patients with congenital adrenal hyperplasia (CAH).

147 Regardless of the presence of erythroid hyperplasia, calculating the percentage of BM blasts fro

148 Parathyroid adenomas and hyperplasia can be grouped into three relative enhanceme

149 though early stent thrombosis and neointimal hyperplasia causing vessel renarrowing were key limitati

150 yroid cancer (MTC) and its precursor, C cell hyperplasia (CCH), is associated with germline RET mutat

151 lture conditions, using zebrafish testicular hyperplasia cells that accumulate early stage spermatogo

152 Furthermore, goblet cell hyperplasia, collagen deposition, and airway hyperrespon

153 arteries showed a 20% increase in neointimal hyperplasia compared with similarly injured wild-type co

154 by a p27 mutation, develop pancreatic islet hyperplasia containing elevated numbers of ghrelin-produ

155 epine to diminish Notch-driven colonic crypt hyperplasia curtailed the fitness advantage conferred by

156 kin-13 (IL-13) reconstituted the goblet cell hyperplasia, cytokine hypersecretion and decreased cilia

157 lated with epithelial remodeling (basal zone hyperplasia, desquamation, and dilated intercellular spa

158 firmed by CLE), and basal cell and papillary hyperplasia developed without surface erosions.

159 ne if fat/PGZ depots could decrease vascular hyperplasia development in a porcine model of hemodialys

160 Stenosis due to intimal hyperplasia development often occurs after placement of

161 assess effects of PGZ/fat depots on vascular hyperplasia development.

162 Atypical ductal hyperplasia diagnosed via excisional biopsy was associat

163 Female mutant mice develop endometrial hyperplasia due to aberrant phosphorylation of signal tr

164 d closely paralleled by marked reductions in hyperplasia, dysplasia and vascularization.

165 ardiomyocyte dedifferentiation, overt muscle hyperplasia, epicardial activation, increased vasculariz

166 ture psoriatic plaques by inducing epidermal hyperplasia, epidermal cell proliferation, and recruitme

167 ated cells (TNCs), unless there is erythroid-hyperplasia (erythroblasts >/= 50%), calculated from non

168 nsive tissue remodeling including basal zone hyperplasia, fibrosis, and angiogenesis.

169 PAI-1 expression correlated with basal zone hyperplasia, fibrosis, and markers of esophageal remodel

170 MCs promote hyperplasia, fibrosis, and vascular cell activation.

171 Follicular nodular hyperplasia (FNH) is a common benign liver tumor for whi

172 r regenerative hyperplasia and focal nodular hyperplasia (FNH), which finally evolved to a giant hepa

173 -fold) and strongly upregulated in sebaceous hyperplasia (fold change > 4, 54.1-fold).

174 served that miR-31 induction results in lung hyperplasia, followed by adenoma formation and later ade

175 esity-related adipose tissue hypertrophy and hyperplasia for health, critical pathways and mechanisms

176 high accuracies in discriminating normal and hyperplasia from high-grade lesions (AUC > 0.94).

177 PD-1KO mice showed severe epidermal hyperplasia, greater neutrophilic infiltration, and high

178 mooth muscle cell, collagen, and goblet cell hyperplasia; hyper IgE syndrome; mucus plugging; and ext

179 al hair cycle, epidermal and sebaceous gland hyperplasia, hyperkeratosis, and increased epidermal thi

180 red during the postacute phase for astrocyte hyperplasia, hypertrophy, and glial scar formation.

181 Intimal hyperplasia (IH) remains a major cause of poor patient o

182 pro-restenotic pathologies including intimal hyperplasia (IH), endothelium impairment, and vessel shr

183 pancreatic beta-cell mass, leading to islet hyperplasia in aged mice.

184 s are alternate initiating events, fostering hyperplasia in gastrointestinal stromal tumours (GISTs),

185 We noted significantly greater intimal hyperplasia in HET versus WT allografts, indicating aggr

186 that IL-17A gene transfer induces epidermal hyperplasia in Il23r(-/-)Rag1(-/-)- and Tcrdelta-deficie

187 has been strongly associated with epidermal hyperplasia in many cutaneous disorders.

188 gen- and cigarette smoke-induced mucous cell hyperplasia in mice and in differentiated primary human

189 retion system (T3SS) to induce colonic crypt hyperplasia in mice, thereby gaining an edge during its

190 These mice also develop adrenal gland hyperplasia in old age.

191 bition of Ras-MAPK pathway impeded epidermal hyperplasia in Pten animals.

192 pha requires interaction with Ras to promote hyperplasia in Pten-deficient skin, thus demonstrating a

193 brafish have been shown to exhibit beta-cell hyperplasia in response to high fat feeding or periphera

194 , bronchial hyperreactivity, and goblet cell hyperplasia in the airways.

195 g adiponectin stimulates beta cell viability/hyperplasia in the context of PCOS.

196 mpartments, and the loss of mouse Nf2 causes hyperplasia in these compartments, replicating the ocula

197 solenopsin derivatives normalized cutaneous hyperplasia in this model, decreased T cell infiltration

198 eatures of RSV lung disease, including mucus hyperplasia, in murine lungs and that HIS mice can be us

199 enges in the treatment of congenital adrenal hyperplasia include avoidance of glucocorticoid overtrea

200 dilated cardiomyopathy, coupled with myocyte hyperplasia (increased cell number), as observed in Mybp

201 chthyosis samples showed increased epidermal hyperplasia (increased thickness and keratin 16 expressi

202 mited, CRS phenotypes demonstrate epithelial hyperplasia, increased matrix deposition and degradation

203 , alveolar space consolidation, and lymphoid hyperplasia; indicating lagged unfavorable effects of ea

204 pendent VSM cell function and neointimal VSM hyperplasia induced by vascular injury.

205 TSS improvement correlated with changes in hyperplasia, infiltrates, and differentiation markers.

206 flammatory disease characterized by synovial hyperplasia, inflammatory cell infiltration, irreversibl

207 By serially transplanting hyperplasias into immunodeficient rag1 mutant zebrafish,

208 ed airway hyperresponsiveness or goblet cell hyperplasia, irrespective of the presence or absence of

209 Congenital adrenal hyperplasia is a group of autosomal recessive disorders

210 epithelial goblet cell metaplasia (GCM) with hyperplasia is a prominent feature of asthma, but the ef

211 responses to helminth infection; goblet cell hyperplasia is abrogated and worm expulsion is compromis

212 liver disease, although its role in biliary hyperplasia is unknown.

213 , the most common type of congenital adrenal hyperplasia, is in place in many countries, however cosy

214 ern blotting revealed reduction in epidermal hyperplasia (Ki67) and in the dermal infiltration of inf

215 livering rapamycin developed less neointimal hyperplasia, less smooth muscle cell proliferation, and

216 ausing a significant reduction in neointimal hyperplasia, lipid burden, cholesterol clefts, and overa

217 histological staining exhibited goblet cell hyperplasia, lung inflammation, thickening of smooth mus

218 ischemia/reperfusion model and a neointimal hyperplasia model of the femoral artery.

219 , 0.05, and 0.06 mm, whereas mean neointimal hyperplasia obstruction was 4.5+/-2.4%, 5.2+/-3.4%, and

220 generative changes of mature neutrophils and hyperplasia of bone marrow myeloid cells.

221 641E/+) mice, which have genetically induced hyperplasia of ICCs, also had accelerated GE.

222 prising the two diseases neuroendocrine cell hyperplasia of infancy (NEHI) and pulmonary interstitial

223 dermal junction (DEJ) by RCM correlated with hyperplasia of melanocytes in hematoxylin-eosin sections

224 ound changes in tissue physiology, including hyperplasia of mucus-secreting goblet cells and smooth m

225 uscle of transgenic mice features a dramatic hyperplasia of muscle stem cells (i.e. satellite cells,

226 , seminiferous tubule diameter reduction and hyperplasia of prostate epithelium, were detected, altho

227 Earlier reports showed that hyperplasia of sympathoadrenal cell precursors during em

228 By contrast, there was no hyperplasia of the intestine after VSG, but the intestin

229 ped microvascular neoangiogenesis as well as hyperplasia of the intimal layer, implicating T cells in

230 ation of these signaling pathways can induce hyperplasia of the MD stroma, which could play a signifi

231 One patient had four-gland hyperplasia, of which three hyperplastic glands were not

232 control patches developed robust neointimal hyperplasia on the patch luminal surface characterized b

233 , conversely, were sufficient to induce mild hyperplasia on their own.

234 To avoid hyperplasia or atrophy, organ turnover demands strict eq

235 re uncoupled, and the organ developed either hyperplasia or atrophy.

236 s of normal-appearing dendritic melanocytes (hyperplasia or early neoplasia) that generally remain co

237 in Manchester, UK, with atypical endometrial hyperplasia or endometrial endometrioid adenocarcinoma.

238 igenesis in Pten(+/-) mice without affecting hyperplasia or prostatic intraepithelial neoplasia.

239 was seen in normal prostate, benign prostate hyperplasia or prostatitis indicating that Runx2 S319 ph

240 uctal inflammatory cell accumulation, ductal hyperplasia, or dysplastic lesions/pancreatic intraepith

241 exhibited a significantly reduced epidermal hyperplasia, oxidative skin damage, and photocarcinogene

242 temic innate immune response that promotes a hyperplasia phenotype in the midgut.

243 simultaneously does not induce the adipocyte hyperplasia phenotype, confirming that SIRT1 controls ad

244 n discovered in primary macronodular adrenal hyperplasia (PMAH), a cause of Cushing syndrome.

245 Diffuse gastrin cell hyperplasia precedes the appearance of MEN1 gastrinomas,

246 98 tumor biopsies including complex atypical hyperplasias, primary tumors and paired abdominopelvic m

247 ology similar to pulmonary reactive lymphoid hyperplasia (PRLH), a condition associated with EBV in H

248 ntium infection, manifested by reduced crypt hyperplasia, reduced epithelial expression of IL-6 and T

249 n cardiovascular surgery, however neointimal hyperplasia remains a significant concern, especially un

250 zation of glands in patients with four-gland hyperplasia remains to be investigated.

251 he molecular mechanism that drives adipocyte hyperplasia remains unknown.

252 In this study, we demonstrate that skin hyperplasia requires FGF receptor adaptor protein Frs2al

253 n of CK1alpha and IFNAR1 leads to intestinal hyperplasia, robust attenuation of apoptosis, and rapid

254 ion and is mainly characterized by epidermal hyperplasia, scaling, and erythema.

255 e biomarkers with SCORAD and with a combined hyperplasia score [thickness and keratin 16 (K16) mRNA]

256 ons were obtained between biomarkers and the hyperplasia score.

257 reatment After Grading Stent-Induced Intimal Hyperplasia Study (PRODIGY) trial assessed unselected pa

258 anges in epithelial basal cell and papillary hyperplasia, surface erosions, intercellular space width

259 athway activation in urothelial cells causes hyperplasia that neither progresses to frank carcinoma n

260 y explain the mechanism underlying adipocyte hyperplasia that occurs much later than adipocyte hypert

261 re, we show that by triggering colonic crypt hyperplasia, the C. rodentium T3SS induced an excessive

262 The median neointimal hyperplasia thickness was 0.04, 0.05, and 0.06 mm, where

263 ldren showed comparable or greater epidermal hyperplasia (thickness and keratin 16) and cellular infi

264 nse to vascular injury that leads to intimal hyperplasia, this study investigated the in vivo efficac

265 pe, confirming that SIRT1 controls adipocyte hyperplasia through c-Myc regulation.

266 injury, Gdf5-lineage cells underpin synovial hyperplasia through proliferation, are recruited to a Ne

267 icantly higher than that in benign prostatic hyperplasia tissues, and PRMT5 expression correlates pos

268 Usual ductal hyperplasia (UDH) of the breast is generally regarded as

269 breast lesions as either benign usual ductal hyperplasia (UDH) or malignant ductal carcinoma in situ

270 tersection of PP-downregulated and sebaceous hyperplasia-upregulated gene lists generated a gene expr

271 ing killer (Bik) decreases airway epithelial hyperplasia via apoptosis mediated by calcium release fr

272 Venous neointimal hyperplasia (VNH) at the outflow vein of hemodialysis AV

273 Comparison of benign reactive lymphoid hyperplasia vs lymphoma revealed lymphoma with greater m

274 intravascular ultrasound percent neointimal hyperplasia was 8.10+/-5.81 and 8.85+/-7.77, respectivel

275 In some cases, stromal hyperplasia was admixed with epithelial metaplasia, some

276 However, lymph node hyperplasia was clearly visible postviremia but was asso

277 Biliary hyperplasia was induced in rats via bile duct ligation (

278 Indeed, the Notch-induced hyperplasia was reduced by mutations affecting two of th

279 e disease-causing gene in congenital adrenal hyperplasia, we now provide a full structural explanatio

280 Trabeculation and valvular hyperplasia were diminished in hearts of embryonic mice

281 l/Forty-one patients with congenital adrenal hyperplasia were evaluated by gray-scale and color Doppl

282 Autoimmunity and peripheral lymphoid hyperplasia were found in 43% of 79 patients with PHTS.

283 roblast content, angiogenesis, and epidermal hyperplasia were markedly reduced following irradiation.

284 OS expression, liver fibrosis, and bile duct hyperplasia were significantly reduced in WT mice admini

285 te cancer than in normal tissue and prostate hyperplasia, whereas MMP2 is repositioned in both prosta

286 mer is pathophysiologic resulting in intimal hyperplasia, whereas the latter is physiological leading

287 endothelial cells (ECs) to cause neointimal hyperplasia, which correlated with the high echogenicity

288 in the formation and development of intimal hyperplasia, which is the main cause of graft failure.

289 ce, beta-cell-specific loss of Cdkn2a causes hyperplasia, while overexpression leads to diabetes, hig

290 ing of the molecular mechanisms of adipocyte hyperplasia will open new avenues toward understanding o

291 ll division and a distinctive form of ductal hyperplasia with 'squamoid' ghost cell nodules in young

292 Megakaryocytic hyperplasia with abnormal features are characteristic, a

293 The Stk11(CKO) mice develop prostatic hyperplasia with bladder outlet obstruction, most likely

294 ymph node of a patient with an angiolymphoid hyperplasia with eosinophilia or Kimura disease.

295 atypical lymphoid infiltrates, Kupffer cell hyperplasia with erythrophagocytosis, and an inconstant

296 e), pancreatic swelling is due to alpha-cell hyperplasia with gross hypersecretion of glucagon, which

297 kins (IL) IL-4 and IL-13, induce goblet cell hyperplasia with mucus production, ultimately resulting

298 tery embolization (PAE) for benign prostatic hyperplasia with spherical particle polyvinyl alcohol (s

299 However, the development of hyperplasia within the arteriovenous synthetic grafts wa

300 al inflammation and basal cell and papillary hyperplasia without loss of surface cells.