ライフサイエンスコーパス: hyperplastic

1 ere non-neoplastic lesions (of which 62 were hyperplastic).

2 the images of the test set as neoplastic or hyperplastic.

3 In two of these individuals, the polyps were hyperplastic.

4 lveoli and the mesenchyme remained thick and hyperplastic.

5 A total of 273 colorectal glands (126 hyperplastic, 147 adenomatous) were isolated via laser c

6 On this basis, we predicted that hyperplastic aberrant crypt foci (ACF), a putative preca

7 een BRAF mutations and serrated histology in hyperplastic ACF supports the idea that these lesions ar

8 ver, that the lesions formerly classified as hyperplastic actually represent a heterogeneous group of

9 Furthermore, MUFA-HFD-fed mice displayed hyperplastic adipose tissue, with enhanced adipogenic po

10 bnormal production of corticotropin in these hyperplastic adrenal glands.

11 er RNA (mRNA) was detected in all samples of hyperplastic adrenal tissue.

12 h local production of the peptide within the hyperplastic adrenals.

13 subpopulation of steroidogenic cells in the hyperplastic adrenals.

14 and alpha-cells, with high expression in the hyperplastic alpha-cells of Gcgr(-/-) mice.

15 DCK was elevated in hyperplastic alveolar epithelial cells of patients with

16 l heterogeneity of IPF, we hypothesized that hyperplastic alveolar epithelial cells regulate the fibr

17 Interrogating the D2-HAN (hyperplastic alveolar nodule) model of breast cancer dor

18 helial cell proliferation and development of hyperplastic alveolar nodules and increased expression l

19 creased the percentage of mice with multiple hyperplastic alveolar nodules.

20 A test set of images (96 hyperplastic and 188 neoplastic polyps, smaller than 5 m

21 icant reduction in vascular surface areas in hyperplastic and angiogenic lesions in pancreatic islets

22 of the K5 promoter led to the development of hyperplastic and dysplastic epithelial lesions and carci

23 d that Stat3-deficient mice initially formed hyperplastic and early adenoma-like lesions that later c

24 epidermal keratinocytes become progressively hyperplastic and eventually produce tumors.

25 cardial cushions in FOG-2 deficient mice are hyperplastic and fail to remodel appropriately, suggesti

26 ly, mammary glands from transgenic mice were hyperplastic and highly vascularized.

27 In addition, the hyperplastic and inflammatory responses to topical phorb

28 transcriptional activity contributes to the hyperplastic and invasive phenotype of synoviocytes that

29 nts leading to the transition from normal to hyperplastic and neoplastic endometrial epithelium.

30 utation that elevates Dpp signaling was also hyperplastic and rescued.

31 whereas diameter decreased significantly in hyperplastic and SCC lesions.

32 -associated CS, especially in the context of hyperplastic and/or adenomatous polyps.

33 e terms risk or risk factor, and serrated or hyperplastic, and polyps or adenomas, and colorectal (or

34 The isolation of additional loci that affect hyperplastic as well as neoplastic growth indicates the

35 orneal wound-healing defect characterized by hyperplastic basal progenitor cells (some of which under

36 r cells in the cardinal veins, together with hyperplastic, blood filled lymph sacs and hyperplastic d

37 Histologic features include hyperplastic bone consisting of mature cortical and trab

38 eatures suggestive of a latent form based on hyperplastic bone marrow and erythroid progenitor cell c

39 Cxcr4 and identify a molecular signature in hyperplastic breast lesions that signifies inappropriate

40 body mass and whole body energy expenditure, hyperplastic brown/white adipose tissues and larger hype

41 mTORC1 was also activated in the hyperplastic but not involuted follicles of HIV-associat

42 during goitrogenic stimulation abrogates the hyperplastic but not the hypertrophic thyrocyte response

43 mice, the mutant ICC stem cells were grossly hyperplastic but remained imatinib-resistant.

44 This is accompanied by hyperplastic changes in cells and desmoplastic alteratio

45 specific vivo-morpholino sequences inhibited hyperplastic cholangiocyte proliferation, liver damage,

46 atory epithelial cells of adult lung induced hyperplastic clusters of cells expressing increased leve

47 sion of DDB2 and RNF43 was observed in human hyperplastic colonic foci.

48 tem called DNN-CAD to identify neoplastic or hyperplastic colorectal polyps less than 5 mm.

49 ormal terminal duct lobular units (TDLUs) by hyperplastic columnar epithelial cells is one of the mos

50 luate the significance of PKD1 in normal and hyperplastic conditions.

51 ced inflammation and dramatically attenuated hyperplastic cryptal response to infection in this group

52 The hyperplastic-deficient esophagus has a distinct expressi

53 th hyperplastic, blood filled lymph sacs and hyperplastic dermal lymphatic vessels.

54 yonic stem (ES) cells, we identify mammalian hyperplastic discs protein EDD, a known E3 ubiquitin lig

55 TTA indicated that the architecture of human hyperplastic discs protein is preserved.

56 t the DDMI protocol, we redesigned the human hyperplastic discs protein to bind to the kinase domain

57 yroidectomy, 100 to 300 mg of adenomatous or hyperplastic diseased parathyroid tissue was prepared an

58 s to determine which cells gave rise to both hyperplastic ductal cells and duct-associated insulin(+)

59 herefore, we tested the hypothesis that both hyperplastic ductal cells and their associated insulin(+

60 We found that hyperplastic ductal cells arose largely from acinar cell

61 tructures, corresponding to normal or mildly hyperplastic ductal epithelium.

62 t islet endocrine cells can intercalate into hyperplastic ducts as they develop.

63 srupts mammary gland involution and leads to hyperplastic ducts associated with reduced numbers of ap

64 Some of the hyperplastic ducts have acinar structures, indicative of

65 del that develops insulin(+) cell-containing hyperplastic ducts in response to the growth factor TGFa

66 ct cells in the epithelium that makes up the hyperplastic ducts of both chronic pancreatitis and panc

67 that this is responsible for the presence of hyperplastic ducts.

68 In addition, the lymphatic vessels in hyperplastic/dysplastic lesions were partly compressed a

69 l fluid pressure was already elevated in the hyperplastic/dysplastic stage.

70 ase in the frequency of medulloblastomas and hyperplastic EGL lesions, formed by preneoplastic GCPs.

71 It is not expressed in normal or hyperplastic endometrial.

72 These hyperplastic enlarged lobular units (HELUs) are importan

73 however, AERD was distinguished from chronic hyperplastic eosinophilic sinusitis by the prominent exp

74 y has a severe course accompanied by chronic hyperplastic eosinophilic sinusitis with nasal polyps, b

75 from control subjects, patients with chronic hyperplastic eosinophilic sinusitis, and patients with A

76 at NAFs can inhibit the growth of normal and hyperplastic epi-thelium but are less able to regulate t

77 ML3 is a useful marker for normal and benign hyperplastic epidermal development, whereas the loss of

78 Here, we report that DNMAML1 mice display hyperplastic epidermis and spontaneously develop cutaneo

79 of hair shafts with increased apoptosis, and hyperplastic epidermis during this time.

80 ated ATM and other markers of the DDR in the hyperplastic epidermis of transgenic mice expressing E2F

81 ion was localized to the upper region of the hyperplastic epidermis.

82 AB2IP(-/-) mice, the prostate gland exhibits hyperplastic epithelia, in which AR becomes more active.

83 d by abnormal tissue repair characterized by hyperplastic epithelial cell formation, is a pathogenic

84 alysis revealed a novel mediator, MMP-19, in hyperplastic epithelial cells adjacent to fibrotic regio

85 Bik reduces hyperplastic epithelial cells by releasing calcium from

86 rmore, after prolonged exposure to allergen, hyperplastic epithelial cells persist longer, and nuclea

87 in IPF lungs and colocalized with MMP-19 in hyperplastic epithelial cells.

88 We show that MMP3 overexpression promotes hyperplastic epithelial growth, surprisingly, in a nonpr

89 nasal epithelium when compared to normal or hyperplastic epithelium.

90 The hyperplastic field of normal-looking cells surrounding t

91 In the absence of SMAD4 and PTEN, hyperplastic foci emerge exclusively from bile ducts of

92 All carcinomas and hyperplastic foci on MR images were localized on the bas

93 sion yielded enhanced mammary gland density, hyperplastic foci, cellular dysplasia, and mammary carci

94 ted a ductal thickening accompanied by small hyperplastic foci.

95 ed classification system for differentiating hyperplastic from adenomatous polyps.

96 aging expertise is required to differentiate hyperplastic from neoplastic polyps with high levels of

97 A total of 119 polyps (81 neoplastic, 38 hyperplastic) from 75 patients was assessed.

98 ed expression of 32,381 murine genes in nine hyperplastic ganglia harvested at three time points and

99 T) lymphoma, as well as hyperplastic polyps, hyperplastic gastropathy, postendoscopic resection for g

100 utonomous growth, leading to a homogeneously hyperplastic gland and to the development of follicular

101 MP-3, MMP-9, MMP-13, and MT1-MMP (MMP-14) in hyperplastic glands and in mammary tumors of MMTV-Wnt1 t

102 nsive centrosome amplification in tumors and hyperplastic glands prior to tumor formation suggests th

103 of PTEN or activation of AKT in regenerated hyperplastic glands that were surrounded by wild-type st

104 t had four-gland hyperplasia, of which three hyperplastic glands were not localized.

105 lands (9 adenomas, 1 atypical adenoma, and 9 hyperplastic glands) were removed from 17 patients, and

106 on in parathyroid chief cells in adenoma and hyperplastic glands, and also in normal parathyroid by i

107 in SH, EGFR activation is the cause of both hyperplastic growth and VDR reduction and therefore infl

108 of the vertebrate embryonic heart occurs by hyperplastic growth as well as the incorporation of cell

109 age-independent growth and conferred benign, hyperplastic growth in a skin-like environment.

110 into new adipocytes during the formation and hyperplastic growth of white adipose tissue.

111 proliferation and plays an important role in hyperplastic growth, which has a positive effect on meat

112 Similar p53 activity occurred in hyperplastic growths that were triggered either by the R

113 astic brown/white adipose tissues and larger hyperplastic hearts.

114 genes, myocardial genes, and development of hyperplastic hearts.

115 ce endoscopic interpretations of adenoma and hyperplastic histology for polyps 5 mm and smaller in si

116 High-confidence predictions of adenoma and hyperplastic histology were correct for 91% and 95%, res

117 Endoscopic predictions of adenoma and hyperplastic histology were made with high confidence fo

118 pic features associated with adenomatous and hyperplastic histology.

119 ophthalmia, failed lens differentiation, and hyperplastic hyaloid vessels.

120 ogenitors are true stem cells for normal and hyperplastic ICC and give rise to GIST.

121 ivity and postjunctional neural responses in hyperplastic ICC tissues appeared normal but were up-reg

122 DNN-CAD classified polyps as neoplastic or hyperplastic in 0.45 +/- 0.07 seconds-shorter than the t

123 Bone marrow was hyperplastic in 16 of 41 subjects (12/16 JAK2V617F posit

124 bing depth and plaque, papilla bleeding, and hyperplastic index scores were recorded.

125 est that Glis1 expression is associated with hyperplastic, inflammatory epidermis.

126 beta-catenin, treatment shifted cells within hyperplastic intestinal crypts from a stem cell to a tra

127 VS28 mice showed a hyperplastic intestinal epithelium and, strikingly, deve

128 ovascular angiogenesis, reduced outgrowth of hyperplastic intima, and minimized CD4(+)CD103(+) tissue

129 Within the hyperplastic left ventricle, increased numbers of prolif

130 It is a concentric vascular intimal hyperplastic lesion composed of smooth muscle-like cells

131 ute to chronic inflammation, and its role in hyperplastic lesion development requires further examina

132 oped renal injury with loss of podocytes and hyperplastic lesion formation; beta-arrestin-1 expressio

133 transformation of benign renal cysts into a hyperplastic lesion, suggesting that fumarate-induced se

134 of undifferentiated nestin(+) cells in both hyperplastic lesions and primary tumors that may serve a

135 ransgenic mice (WAP-HA-14-3-3zeta) developed hyperplastic lesions and showed increased susceptibility

136 d tissues correlates with the development of hyperplastic lesions and the presence of PthA4.

137 uitary, the p27CK- protein dominantly caused hyperplastic lesions and tumors in multiple organs, incl

138 We did not observe a detectable DDR in early hyperplastic lesions arising in transgenic mice expressi

139 These results suggest that glomerular hyperplastic lesions derive from the proliferation and m

140 a significant inhibition in the formation of hyperplastic lesions during tumor progression.

141 ession of IGF-1(des) was sufficient to cause hyperplastic lesions in all mice, however the well-diffe

142 astoma development in these mice begins with hyperplastic lesions in early postnatal sympathetic gang

143 drogenase (ALDH) as well as by generation of hyperplastic lesions in humanized fat pads of NOD (nucle

144 opmental defect of the prostate and prostate hyperplastic lesions in mouse.

145 Prostate hyperplastic lesions in Mp(+/-) mice also induced stroma

146 gammaH2AX expression was elevated in hyperplastic lesions in the mammary fat pads of BALB-neu

147 The cellular composition of the resulting hyperplastic lesions is controversial, although a popula

148 LA-exposed SHHF rats developed atypical hyperplastic lesions of bronchiolar epithelial cell orig

149 teristically develop severe, diffuse intimal hyperplastic lesions that eventually compromise luminal

150 -regulated during multistep progression from hyperplastic lesions to islet tumors.

151 When hyperplastic lesions were further classified into serrat

152 levels of IGF-1 facilitate the emergence of hyperplastic lesions while imposing a strong IGF-1-depen

153 an prevented podocyte loss, formation of the hyperplastic lesions, and normalized expression of glome

154 ut mice leads to the development of prostate hyperplastic lesions, and this effect was mediated throu

155 ogen receptor (AR) does not give rise to any hyperplastic lesions, but when combined with high levels

156 eolar type II (ATII) cells were able to form hyperplastic lesions, some of which progressed to adenom

157 as a reduction in the number and severity of hyperplastic lesions, the endometrial phenotype persiste

158 ll GCPs and, remarkably, reduces the area of hyperplastic lesions.

159 colonoscopy, and obtained tissue blocks for hyperplastic lesions.

160 , and CD4(+) and gammadelta T cells in mouse hyperplastic lesions.

161 Hyperplastic livers and tumors from YAP(S127A) mice had

162 pport a novel role for the CaR in preventing hyperplastic lung disease in utero.

163 We obtained specimens of hyperplastic macronodular adrenal tissue from 30 patient

164 Tg (c-neu) contains hyperplastic mammary ducts with high proliferative index

165 s an oncogene, giving rise to a premalignant hyperplastic mammary phenotype as well as to a high inci

166 rrow reference cells) recapitulate the known hyperplastic mechanism of weight gain with pioglitazone

167 Bmp9-knockout (KO) pups presented hyperplastic mesenteric collecting vessels that maintain

168 Second, affected organs showed variable hyperplastic, metaplastic, and connective tissue changes

169 ugh Kras/KRAS mutation promotes serrated and hyperplastic morphologic features in colon epithelium, i

170 Plxnd1 deficiency in zebrafish induced hyperplastic morphology in visceral adipose tissue (VAT)

171 ment of the typical celiac lesion (the crypt hyperplastic mucosal atrophy) as it generally unfolds: t

172 evels in both human papilloma virus-positive hyperplastic murine skin and a subset of human squamous

173 ident; persistent expression of N-myc in the hyperplastic myocardium and attenuated expression of the

174 ed expansion and oncogenic transformation of hyperplastic neuroblasts, thus promoting progression to

175 plastic) or reactive (nonmalignant, lymphoid hyperplastic) nodes.

176 recedes loss of liver cells and regenerative hyperplastic nodules that do not express HBsAg.

177 d the percentage of mice exhibiting multiple hyperplastic nodules, invasive mammary cancers and cance

178 If HZ disruption is severe, hyperplastic OB-ISCs expand across the interorgan bounda

179 The hyperplastic ocular phenotypes were largely suppressed b

180 for patients whose colonoscopies identified hyperplastic (odds ratio [OR] = 3.1; 95% CI, 1.7-5.5) or

181 common and accurate endoscopic prediction of hyperplastic or adenomatous polyp histology could reduce

182 45%-52% after colonoscopies that identified hyperplastic or adenomatous polyps.

183 5 or more GI polyps, 1 or more of which was hyperplastic or hamartomatous (N = 397), were prospectiv

184 Other hyperplastic or neoplastic mutations did not rescue RpL3

185 at seen in normal (nonmalignant, nonlymphoid hyperplastic) or reactive (nonmalignant, lymphoid hyperp

186 scs (lgd) induce strong Notch activation and hyperplastic overgrowth of Drosophila imaginal discs.

187 s lacking the tumor suppressor PTEN to cause hyperplastic overgrowth.

188 ge and reduced the incidence of diabetes and hyperplastic pancreatic islets at 6 months of age.

189 rmoid carcinoma cell line A431, which mimics hyperplastic parathyroid cells.

190 CAAT enhancer binding protein-beta, in human hyperplastic parathyroid glands and in the human epiderm

191 e parathyroid adenomas in 6 patients, and 10 hyperplastic parathyroid glands in 3 patients).

192 posite characteristics, the degenerative and hyperplastic pathologies of aging are at least partly li

193 ea that it also drives both degenerative and hyperplastic pathologies, most likely by promoting chron

194 ially among vertebrates, aging also promotes hyperplastic pathologies, the most deadly of which is ca

195 n in later life, leading to hypertrophic and hyperplastic pathologies.

196 ly elevated in mammary glands and leads to a hyperplastic phenotype by 12 weeks of age.

197 Ovariectomized Mig-6(d/d) mice exhibit this hyperplastic phenotype in the presence of E2 and P4 but

198 type genome, and upon stratification, a more hyperplastic phenotype, with an increase in the number o

199 100A8/A9 and 27 other genes and reversed the hyperplastic phenotype.

200 study were a total of 688 adenoma cases, 210 hyperplastic polyp cases, and 1306 polyp-free controls f

201 ty-one percent of respondents would survey a hyperplastic polyp in 5 years or less, 71% would survey

202 and the association between the report of a hyperplastic polyp in the baseline CC report and the pro

203 d be in part attributed to the presence of a hyperplastic polyp in the baseline CC.

204 idual crypts from six colonic adenomas and a hyperplastic polyp were microdissected and characterized

205 he most advanced lesion present: no polyp, a hyperplastic polyp, a tubular adenoma less than 1 cm in

206 xclusive categories, including normal colon, hyperplastic polyp, nonadvanced adenoma, and advanced ad

207 l polyp were recognized: the adenoma and the hyperplastic polyp.

208 nd 1.30 in the 55 cases vs 235 controls with hyperplastic polyps (95% CI, 0.96-1.77).

209 regions containing normal epithelium (NE) or hyperplastic polyps (HP) to be distinguished from region

210 ecursors to colorectal cancer (CRC), whereas hyperplastic polyps (HPPs) have low risk of progression

211 444 cases with adenomas only, 662 cases with hyperplastic polyps (HPPs) only, and 437 cases with sync

212 re phosphorylated in serrated areas of human hyperplastic polyps (HPPs), sessile serrated adenomas, a

213 6 of 372) of serrated lesions, respectively; hyperplastic polyps (HPs) accounted for 58.9% (219 of 37

214 a (odds ratio: 1.60; 95% CI: 1.12, 2.29) and hyperplastic polyps (odds ratio: 1.85; 95% CI: 1.09, 3.1

215 itivity 0.94; 95% CI 0.90-0.97) and 55 of 62 hyperplastic polyps (specificity 0.89; 0.78-0.95), with

216 d adenomas (TSAs) are now distinguished from hyperplastic polyps and recognized as precursors to colo

217 The family of serrated lesions includes hyperplastic polyps and sessile serrated adenomas withou

218 an increased risk of colorectal adenomas and hyperplastic polyps as well as colorectal cancer.

219 ns with no polyps at baseline and those with hyperplastic polyps at baseline (1.1% [12 of 1057] and 2

220 These serrated polyps include not only hyperplastic polyps but also traditional serrated adenom

221 eported that colon carcinomas, adenomas, and hyperplastic polyps exhibiting a serrated histology were

222 low risk adenoma in 158 cases (11.3 %), and hyperplastic polyps in 119 cases (8.5 %).

223 lying conditions were hemorrhoids in 7 (6%), hyperplastic polyps in 4 (3.5%), adenomatous polyps in 2

224 UFAs were not associated with adenomatous or hyperplastic polyps in either men or women.

225 cid was associated with an increased risk of hyperplastic polyps in men (P-trend = 0.03), which was n

226 tion between the outcome and the presence of hyperplastic polyps in the baseline CC, showed a statist

227 d be considered a mixed polyp syndrome, with hyperplastic polyps most prevalent, with a risk of early

228 ases with adenomatous polyps only (n = 639), hyperplastic polyps only (n = 294), and both types of po

229 valuation in 5-10 years, whereas people with hyperplastic polyps only should have a 10-year follow-up

230 for adenoma only, 0.2 (95% CI: 0.1, 0.3) for hyperplastic polyps only, and 0.2 (95% CI: 0.2, 0.4) for

231 tous polyps only, 5.0 (95% CI: 3.3, 7.3) for hyperplastic polyps only, and 6.9 (95% CI: 4.4, 11.1) fo

232 s of cigarette smoking and were stronger for hyperplastic polyps than for adenoma.

233 aging system in the identification of distal hyperplastic polyps that do not need resection, as well

234 the precursor to colorectal cancer, whereas hyperplastic polyps were viewed as innocuous lesions wit

235 st set, the DNN-CAD identified neoplastic or hyperplastic polyps with 96.3% sensitivity, 78.1% specif

236 ly unimportant lesions (diminutive adenomas, hyperplastic polyps).

237 No SNP was significantly associated with hyperplastic polyps, and only rs6983267 was significantl

238 31st 2005, without reported lesions or with hyperplastic polyps, based on secondary data extracted f

239 lymphoid tissue (MALT) lymphoma, as well as hyperplastic polyps, hyperplastic gastropathy, postendos

240 stology, they were wild type for BRAF; among hyperplastic polyps, KRAS mutations were found mainly in

241 mages of neoplastic polyps and 681 images of hyperplastic polyps, obtained from the picture archiving

242 The specificity in individuals with normal, hyperplastic polyps, or nonadvanced adenomas was 82.0% (

243 SPs comprise hyperplastic polyps, sessile serrated adenomas/polyps (S

244 radic high-grade dysplastic adenomas, and 19 hyperplastic polyps--and tissue derived from patients wi

245 intake on risk of colorectal adenomatous and hyperplastic polyps.

246 er for sessile serrated adenomas/polyps than hyperplastic polyps.

247 Of the 64, half had hyperplastic polyps.

248 g intake in relation to both adenomatous and hyperplastic polyps.

249 ith an elevated risk of both adenomatous and hyperplastic polyps.

250 An inverse association trend was found for hyperplastic polyps.

251 ize and was not engendered by nonneoplastic (hyperplastic) polyps.

252 were found to accelerate the development of hyperplastic precancerous lesions.

253 urred in microphthalmic eyes with persistent hyperplastic primary vitreous (more recently termed pers

254 ting in eye disease mimicking the persistent hyperplastic primary vitreous (PHPV) found in Arf-null m

255 Persistent hyperplastic primary vitreous (PHPV) is a developmental

256 logical process resembling severe persistent hyperplastic primary vitreous (PHPV), a human eye diseas

257 ilar to that of human eye disease persistent hyperplastic primary vitreous (PHPV).

258 evelop eye disease reminiscent of persistent hyperplastic primary vitreous (PHPV).

259 mutants strongly resembles human persistent hyperplastic primary vitreous cases and may provide insi

260 suggest that autosomal recessive persistent hyperplastic primary vitreous is caused by N46H and is e

261 syndrome closely resembles human persistent hyperplastic primary vitreus (PHPV), attributed to faile

262 RP782 uptake tracks the hyperplastic process in vascular remodeling and provides

263 and loss of RB is proposed to facilitate the hyperplastic proliferation associated with tumorigenesis

264 wever, we have previously shown that SCs are hyperplastic, rather than neoplastic, and able to induce

265 n disease pathogenesis, become activated and hyperplastic, releasing proinflammatory factors and tiss

266 damaged blood-brain barrier and a subsequent hyperplastic response that results in a dense scar that

267 mount an appropriate compensatory beta-cell hyperplastic response to maintain normoglycemia.

268 exhibited a markedly exaggerated neointimal hyperplastic response to wire injury of the femoral arte

269 d with the inhibition of the UVR-induced (i) hyperplastic response, (ii) Hsp90beta-PKCvarepsilon inte

270 eby down-regulating Pim1 and suppressing the hyperplastic response.

271 We further demonstrate that hyperplastic SCs accumulate non-cell-autonomously via si

272 kin show follicles in prolonged telogen with hyperplastic sebaceous glands.

273 olyps, including at least 1 hamartomatous or hyperplastic/serrated polyp.

274 es blood vessels to coalesce into large flat hyperplastic sinuses with no distinctive hierarchical or

275 aser-capture microdissection of normal skin, hyperplastic skin and papillomas showed that amplificati

276 -treated HNSCC lines, human HNSCC tumors and hyperplastic squamous epithelia of transgenic mice overe

277 hibition of NF-kappaB increases apoptosis in hyperplastic stages of tumor development and decreases p

278 This persistent hyperplastic state appeared to result from an equilibriu

279 umor cells and their transformation beyond a hyperplastic state.

280 elopmentally-timed apoptotic response in the hyperplastic sympathoadrenal cells.

281 s with MYCN to increase the proliferation of hyperplastic sympathoadrenal precursor cells, leading to

282 ining, and the formation and behavior of the hyperplastic synovial lining.

283 nced cell proliferation, consistent with the hyperplastic synovial tissue observed in MPS patients.

284 endent cell proliferation and development of hyperplastic three-dimensional acini without affecting a

285 Distinguishing hyperplastic thymus from tumors was better with SII sign

286 changes in the timing of the transition from hyperplastic to hypertrophic growth of cardiomyocytes co

287 Mammalian cardiomyocytes undergo a critical hyperplastic-to-hypertrophic growth transition at early

288 ative activity and is likely associated with hyperplastic-to-hypertrophic growth transition.

289 RNAs may have important functions in cardiac hyperplastic-to-hypertrophic growth transition.

290 rointestinal lymphoid hyperplasia, extensive hyperplastic tonsils, thymus hyperplasia, autoimmune lym

291 Mutations in the expanded gene act as hyperplastic tumor suppressors, interfere with cell comp

292 Mutations in the hyperplastic tumor-suppressor genes result in increased

293 els, including loss-of-function mutations in hyperplastic tumour suppressors in the Hippo-Salvador-Wa

294 EoE), in which basal progenitor cells become hyperplastic upon proinflammatory stimulation.

295 f cytokeratin 14-positive basal cells in the hyperplastic urothelial mucosa in male Foxa1 knockout mi

296 ysis AV graft hyperplasia and possibly other hyperplastic vascular disorders.

297 we report that differentiated adipocytes are hyperplastic when SIRT1 is knocked down stably in mouse

298 Klf5CN palpebral epidermis was hyperplastic with 7-9 layers of keratinocytes, compared

299 Nkx2-5 mutants died shortly after birth with hyperplastic working myocytes and conduction system incl

300 weak PSCA staining on tumor cells present in hyperplastic zones and in areas that contained focal and