ライフサイエンスコーパス: hyperplastic polyp

1 ly unimportant lesions (diminutive adenomas, hyperplastic polyps).

2 l polyp were recognized: the adenoma and the hyperplastic polyp.

3 5%) of surgeons recommend surveillance for a hyperplastic polyp.

4 intake on risk of colorectal adenomatous and hyperplastic polyps.

5 Of the 64, half had hyperplastic polyps.

6 g intake in relation to both adenomatous and hyperplastic polyps.

7 er for sessile serrated adenomas/polyps than hyperplastic polyps.

8 ith an elevated risk of both adenomatous and hyperplastic polyps.

9 An inverse association trend was found for hyperplastic polyps.

10 ll adenoma, and follow-up is not advised for hyperplastic polyps.

11 ng were associated with an increased risk of hyperplastic polyps.

12 1 (5%) of 20 Ashkenazi Jewish patients with hyperplastic polyps.

13 No FDG accumulation was noted in 35 hyperplastic polyps.

14 style factors and the presence of colorectal hyperplastic polyps.

15 ize and was not engendered by nonneoplastic (hyperplastic) polyps.

16 serrated adenomas had a higher frequency of hyperplastic polyps (1.3 +/- 1.6) as compared to patient

17 ge and sex, was 2.6 for patients with distal hyperplastic polyps, 4.0 for those with distal tubular a

18 nt had no detected lesions, 10.0 percent had hyperplastic polyps, 8.7 percent had tubular adenomas, a

19 nd 1.30 in the 55 cases vs 235 controls with hyperplastic polyps (95% CI, 0.96-1.77).

20 he most advanced lesion present: no polyp, a hyperplastic polyp, a tubular adenoma less than 1 cm in

21 smic staining occurred in about one third of hyperplastic polyps, adenomas, and adenocarcinomas and i

22 carcinoma was compared with classic isolated hyperplastic polyps, adenomas, and solitary serrated ade

23 adenoma was collected among 81 subjects with hyperplastic polyps and 480 controls.

24 nd in excess of guidelines, particularly for hyperplastic polyps and low-risk lesions such as a small

25 nce of endocrine cells compared with classic hyperplastic polyps and normal colon and similar immunoh

26 d adenomas (TSAs) are now distinguished from hyperplastic polyps and recognized as precursors to colo

27 The family of serrated lesions includes hyperplastic polyps and sessile serrated adenomas withou

28 ch more similar to serrated adenomas than to hyperplastic polyps and were characterized by large size

29 r gene hMLH1 is diminished or absent in some hyperplastic polyps, and it has been suggested that HPP1

30 No SNP was significantly associated with hyperplastic polyps, and only rs6983267 was significantl

31 radic high-grade dysplastic adenomas, and 19 hyperplastic polyps--and tissue derived from patients wi

32 Three hundred ninety-one patients had hyperplastic polyps as the worst lesion found at colonos

33 an increased risk of colorectal adenomas and hyperplastic polyps as well as colorectal cancer.

34 ns with no polyps at baseline and those with hyperplastic polyps at baseline (1.1% [12 of 1057] and 2

35 Hyperplastic polyps at the initial examination did not p

36 31st 2005, without reported lesions or with hyperplastic polyps, based on secondary data extracted f

37 These serrated polyps include not only hyperplastic polyps but also traditional serrated adenom

38 study were a total of 688 adenoma cases, 210 hyperplastic polyp cases, and 1306 polyp-free controls f

39 ry analysis examined risk factors for having hyperplastic polyps compared with having no polyps and c

40 A hyperplastic polyp/dysplasia-to-adenocarcinoma sequence

41 eported that colon carcinomas, adenomas, and hyperplastic polyps exhibiting a serrated histology were

42 he multivariate-adjusted odds ratio (OR) for hyperplastic polyps for individuals in the upper vs. the

43 Hyperplastic polyps had a characteristic surface "pit pa

44 regions containing normal epithelium (NE) or hyperplastic polyps (HP) to be distinguished from region

45 ecursors to colorectal cancer (CRC), whereas hyperplastic polyps (HPPs) have low risk of progression

46 444 cases with adenomas only, 662 cases with hyperplastic polyps (HPPs) only, and 437 cases with sync

47 re phosphorylated in serrated areas of human hyperplastic polyps (HPPs), sessile serrated adenomas, a

48 6 of 372) of serrated lesions, respectively; hyperplastic polyps (HPs) accounted for 58.9% (219 of 37

49 s are reported to have multiple and/or large hyperplastic polyps (HPs) and an increased risk of color

50 ts with hyperplastic polyposis have multiple hyperplastic polyps (HPs) and increased risk of colorect

51 We studied CIMP in hyperplastic polyps (HPs), with emphasis on patients wit

52 lymphoid tissue (MALT) lymphoma, as well as hyperplastic polyps, hyperplastic gastropathy, postendos

53 ty-one percent of respondents would survey a hyperplastic polyp in 5 years or less, 71% would survey

54 and the association between the report of a hyperplastic polyp in the baseline CC report and the pro

55 d be in part attributed to the presence of a hyperplastic polyp in the baseline CC.

56 low risk adenoma in 158 cases (11.3 %), and hyperplastic polyps in 119 cases (8.5 %).

57 lying conditions were hemorrhoids in 7 (6%), hyperplastic polyps in 4 (3.5%), adenomatous polyps in 2

58 patterns appeared: gastric ulcers in 32% and hyperplastic polyps in 68% of gerbils.

59 UFAs were not associated with adenomatous or hyperplastic polyps in either men or women.

60 cid was associated with an increased risk of hyperplastic polyps in men (P-trend = 0.03), which was n

61 tion between the outcome and the presence of hyperplastic polyps in the baseline CC, showed a statist

62 6 serrated adenomas with multiple (6 to 10) hyperplastic polyps, including 5 with admixed hyperplast

63 stology, they were wild type for BRAF; among hyperplastic polyps, KRAS mutations were found mainly in

64 Despite the high prevalence of the hyperplastic polyp, little is known about its etiology.

65 er in gerbils with ulcers than in those with hyperplastic polyps (median IFN-gamma/glyceraldehyde-3-p

66 that the different outcomes (e.g., ulcers or hyperplastic polyps) might relate to imbalances among cy

67 d be considered a mixed polyp syndrome, with hyperplastic polyps most prevalent, with a risk of early

68 with familial adenomatous polyposis (n = 7), hyperplastic polyps (n = 3), dysplasias arising in ulcer

69 acent to adenomas or adenocarcinomas (n=41), hyperplastic polyps (n=8), adenomatous polyps (=35), and

70 xclusive categories, including normal colon, hyperplastic polyp, nonadvanced adenoma, and advanced ad

71 mages of neoplastic polyps and 681 images of hyperplastic polyps, obtained from the picture archiving

72 a (odds ratio: 1.60; 95% CI: 1.12, 2.29) and hyperplastic polyps (odds ratio: 1.85; 95% CI: 1.09, 3.1

73 ase of gastric adenocarcinoma arising from a hyperplastic polyp of the fundis of a patient with AAPC.

74 as cloned as a frequently methylated gene in hyperplastic polyps of the colon.

75 fter the following 6 results on colonoscopy: hyperplastic polyp, one 6-mm tubular adenoma, two 6-mm t

76 ases with adenomatous polyps only (n = 639), hyperplastic polyps only (n = 294), and both types of po

77 valuation in 5-10 years, whereas people with hyperplastic polyps only should have a 10-year follow-up

78 for adenoma only, 0.2 (95% CI: 0.1, 0.3) for hyperplastic polyps only, and 0.2 (95% CI: 0.2, 0.4) for

79 tous polyps only, 5.0 (95% CI: 3.3, 7.3) for hyperplastic polyps only, and 6.9 (95% CI: 4.4, 11.1) fo

80 ines, especially if the colonoscopy showed a hyperplastic polyp or a single small adenoma.

81 mas and 34% of adenomatous polyps but not in hyperplastic polyps or normal or transitional mucosa.

82 ory of body mass index had a higher risk for hyperplastic polyps (OR, 4.50; 95% CI, 1.84-10.97).

83 The specificity in individuals with normal, hyperplastic polyps, or nonadvanced adenomas was 82.0% (

84 Hyperplastic polyps retained this normal compartmentaliz

85 nderestimated precursor lesion, the proximal hyperplastic polyp-serrated adenoma pathway.

86 SPs comprise hyperplastic polyps, sessile serrated adenomas/polyps (S

87 Hyperplastic polyps share common lifestyle risk factors

88 itivity 0.94; 95% CI 0.90-0.97) and 55 of 62 hyperplastic polyps (specificity 0.89; 0.78-0.95), with

89 s of cigarette smoking and were stronger for hyperplastic polyps than for adenoma.

90 aging system in the identification of distal hyperplastic polyps that do not need resection, as well

91 sh patients with adenomatous polyps, but not hyperplastic polyps.The I1307K mutation represents a nov

92 Among patients with distal hyperplastic polyps, those with distal tubular adenomas,

93 idual crypts from six colonic adenomas and a hyperplastic polyp were microdissected and characterized

94 ant, adenomatous polyps were present in 25%, hyperplastic polyps were present in 10%, and synchronous

95 ant, adenomatous polyps were present in 16%, hyperplastic polyps were present in 22%, and synchronous

96 the precursor to colorectal cancer, whereas hyperplastic polyps were viewed as innocuous lesions wit

97 st set, the DNN-CAD identified neoplastic or hyperplastic polyps with 96.3% sensitivity, 78.1% specif