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1 low and hemodynamics were unaffected by this hyperpnea.
2 velopment of airway obstruction that follows hyperpnea.
3 g upper airway patency during both apnea and hyperpnea.
4 airway than in parenchymal hysteresis after hyperpnea.
5 ves before and 10, 30, 60, and 120 min after hyperpnea.
6 creased from baseline for at least 1 h after hyperpnea.
7 FEV1 was measured before and after hyperpnea.
8 ardiopulmonary reflexes may trigger exercise hyperpnea.
9 Vasovagal syncope is preceded by a period of hyperpnea and cardiorespiratory decoupling followed by a
12 llary blood volume does not change following hyperpnea, and therefore that changes in pulmonary blood
13 he diaphragm reliably occurs after voluntary hyperpnea at levels sufficient to induce task failure.
14 veness to methacholine or eucapnic voluntary hyperpnea challenge correlated with these inflammatory a
15 ement, methacholine test, eucapnic voluntary hyperpnea challenge, allergy skin prick tests, and bronc
16 aving a significant fall in FEV(1) following hyperpnea compared with control subjects (DeltaFEV(1) =
18 zed exercise challenge or eucapnic voluntary hyperpnea (EVH); and the efficacy of nonpharmaceutical i
20 noxide (DL(CO)) before and after a period of hyperpnea in 13 subjects with asthma with HIB and 10 con
21 y significant falls in specific conductance, hyperpnea in asthmatics, but not in normal subjects, res
22 narrowing that occurs following exercise or hyperpnea in subjects with asthma with hyperpnea-induced
25 ed increases in end-expiratory volume during hyperpnea: maximum 412 +/- 112 (SE) ml (range 75-1,543 m
26 one), during unloaded cycling with isocapnic hyperpnea (muscle and ventilatory pump), during Ex plus
31 ix normal subjects before and after 5 min of hyperpnea: specific conductance, upstream resistance, st
34 We used a canine model of exercise-induced hyperpnea to examine the effects of repeated hyperventil
37 inally, left atrial hypertension and induced hyperpnea were combined in sheep at rest, and the result
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