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1 low and hemodynamics were unaffected by this hyperpnea.
2 velopment of airway obstruction that follows hyperpnea.
3 g upper airway patency during both apnea and hyperpnea.
4  airway than in parenchymal hysteresis after hyperpnea.
5 ves before and 10, 30, 60, and 120 min after hyperpnea.
6 creased from baseline for at least 1 h after hyperpnea.
7           FEV1 was measured before and after hyperpnea.
8 ardiopulmonary reflexes may trigger exercise hyperpnea.
9 Vasovagal syncope is preceded by a period of hyperpnea and cardiorespiratory decoupling followed by a
10  these patients and relates to both exercise hyperpnea and peak exercise oxygen consumption.
11         Breathing 8% CO2 elicited a moderate hyperpnea, and minute ventilation during the final minut
12 llary blood volume does not change following hyperpnea, and therefore that changes in pulmonary blood
13 he diaphragm reliably occurs after voluntary hyperpnea at levels sufficient to induce task failure.
14 veness to methacholine or eucapnic voluntary hyperpnea challenge correlated with these inflammatory a
15 ement, methacholine test, eucapnic voluntary hyperpnea challenge, allergy skin prick tests, and bronc
16 aving a significant fall in FEV(1) following hyperpnea compared with control subjects (DeltaFEV(1) =
17                          To ensure that this hyperpnea did not augment diaphragmatic activity enough
18 zed exercise challenge or eucapnic voluntary hyperpnea (EVH); and the efficacy of nonpharmaceutical i
19                             We conclude that hyperpnea has significant effects on the lung parenchyma
20 noxide (DL(CO)) before and after a period of hyperpnea in 13 subjects with asthma with HIB and 10 con
21 y significant falls in specific conductance, hyperpnea in asthmatics, but not in normal subjects, res
22  narrowing that occurs following exercise or hyperpnea in subjects with asthma with hyperpnea-induced
23 se or hyperpnea in subjects with asthma with hyperpnea-induced bronchospasm (HIB).
24                             We conclude that hyperpnea is a major mechanism of interstitial liquid cl
25 ed increases in end-expiratory volume during hyperpnea: maximum 412 +/- 112 (SE) ml (range 75-1,543 m
26 one), during unloaded cycling with isocapnic hyperpnea (muscle and ventilatory pump), during Ex plus
27 five subjects voluntarily mimicked their CO2 hyperpnea on a separate occasion.
28                               The effects of hyperpnea on parenchymal lung mechanics are unknown, but
29                              After voluntary hyperpnea, seven of the 10 subjects displayed at least a
30                             During voluntary hyperpnea, she could quickly reduce her FET(CO(2)) to 4.
31 ix normal subjects before and after 5 min of hyperpnea: specific conductance, upstream resistance, st
32 rences in edema clearance are related to the hyperpnea that accompanies exercise.
33                                        After hyperpnea, the mean percentage decrease from baseline in
34   We used a canine model of exercise-induced hyperpnea to examine the effects of repeated hyperventil
35 diaphragmatic fatigue occurs after voluntary hyperpnea to task failure.
36      The ventilatory response to 10% oxygen (hyperpnea) was significantly attenuated during quiet sle
37 inally, left atrial hypertension and induced hyperpnea were combined in sheep at rest, and the result
38 ased prevalence of lung disease secondary to hyperpnea with cold air.
39      The percentage fall in twitch Pdi after hyperpnea with the two techniques was not significantly

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