ライフサイエンスコーパス: hyperpolarization

1 of neuronal depolarization and downstates of hyperpolarization.

2 nhance glycolysis and increase mitochondrial hyperpolarization.

3 nd in situ quality assurance of the produced hyperpolarization.

4 combined NMR chemosensing with nuclear spin hyperpolarization.

5 RESK protein, resulting in neuronal membrane hyperpolarization.

6 n in GIRK-channel induced pacemaker membrane hyperpolarization.

7 hydrogen without penalty for the achievable hyperpolarization.

8 eurotransmitter release, and induce neuronal hyperpolarization.

9 is inhibition is overcome by pharmacological hyperpolarization.

10 ization, and it recovers only after extended hyperpolarization.

11 r excitability as a result of BPA-induced Vm hyperpolarization.

12 lack of equally efficient tools for membrane hyperpolarization.

13 ent, but are reduced by halorhodopsin-driven hyperpolarization.

14 iring presynaptic activity with postsynaptic hyperpolarization.

15 ept out of the membrane into the axoplasm by hyperpolarization.

16 Na was delayed if the step was preceded by a hyperpolarization.

17 ion in NAG neurons, rather than the expected hyperpolarization.

18 ethods, which recently has been addressed by hyperpolarization.

19 n-selective gating pore current activated by hyperpolarization.

20 ificantly longer delays to spiking following hyperpolarization.

21 .1 channel trafficking, and promote membrane hyperpolarization.

22 ency (fast, F) nerve fibres, consistent with hyperpolarization.

23 including water, on achieving SABRE-promoted hyperpolarization.

24 the plasma membrane, and triggered membrane hyperpolarization.

25 droxyphenylglycine, paired with postsynaptic hyperpolarization.

26 ampen neuronal activity through postsynaptic hyperpolarization.

27 endothelium to generate endothelium-derived hyperpolarization.

28 al increments in light intensity with graded hyperpolarizations.

29 creases glycolysis and induces mitochondrial hyperpolarization, a bax independent anti-apoptotic acti

30 CV hyperpolarization accelerated the rhythm to an extent th

31 ctrical recordings and involved synchronized hyperpolarizations across both MSN subtypes.

32 er cells, identified by immunoreactivity for hyperpolarization activated, cyclic nucleotide-gated cha

33 cAMP, reminiscent of the dual modulation in hyperpolarization-activated and cyclic nucleotide-gated

34 The hyperpolarization-activated and cyclic nucleotide-modula

35 with cysteine residues (S-nitrosylation) on hyperpolarization-activated and nucleotide-gated cation

36 o and in situ to measure currents carried by hyperpolarization-activated and nucleotide-gated cation

37 mutations severely and specifically impaired hyperpolarization-activated cation (Ih) channels.

38 and upper urinary tract involves coexpressed hyperpolarization-activated cation and T-type Ca(2+) cha

39 ubpopulations of BF GABAergic neurons [large hyperpolarization-activated cation current (Ih) and smal

40 c morphology and dendritic expression of the hyperpolarization-activated cation current (Ih).

41 f midbrain DA neurons characterized by small hyperpolarization-activated cation currents (Ih) is spar

42 firing through modulation of both SK and the hyperpolarization-activated cation currents (Ih).

43 aptic input, whereas the remainder exhibited hyperpolarization-activated cation currents and low thre

44 ltage-activated K(+) conductance (gKL) and a hyperpolarization-activated conductance (gh).

45 s to lower heart rate, including sarcolemmal hyperpolarization-activated current (I f) and ryanodine

46 of VTA DA neurons, caused by an up-regulated hyperpolarization-activated current (I(h)).

47 on average, lower input resistance, greater hyperpolarization-activated current (Ih), depolarized re

48 ar cAMP levels, increased the density of the hyperpolarization-activated current and intracellular ca

49 hysical and pharmacological hallmarks of the hyperpolarization-activated current Ih.

50 The hyperpolarization-activated current, If, plays an import

51 component of the underlying mechanism is the hyperpolarization-activated current, Ih, as pharmacologi

52 nstants of up to 225 ms, and small-amplitude hyperpolarization-activated currents (IH), characteristi

53 s, which, combined with weaker expression of hyperpolarization-activated currents, lengthened hyperpo

54 to and functions as an auxiliary subunit of hyperpolarization-activated cyclic nucleotide (HCN)-gate

55 ng gene variants with cosegregation, a novel hyperpolarization-activated cyclic nucleotide channel 4

56 ulation of the pacemaking ion channel, HCN4 (hyperpolarization-activated cyclic nucleotide gated chan

57 afferent by altering ion permeation through hyperpolarization-activated cyclic nucleotide-gated (HCN

58 Hyperpolarization-activated cyclic nucleotide-gated (HCN

59 ose of this study was to examine whether the hyperpolarization-activated cyclic nucleotide-gated (HCN

60 ic plasticity and spatial memory through the hyperpolarization-activated cyclic nucleotide-gated (HCN

61 Surprisingly, the distribution of hyperpolarization-activated cyclic nucleotide-gated (HCN

62 The cationic current Ih mediated by hyperpolarization-activated cyclic nucleotide-gated (HCN

63 Hyperpolarization-activated cyclic nucleotide-gated (HCN

64 Hyperpolarization-activated cyclic nucleotide-gated (HCN

65 ntify an interneuron-specific attenuation of hyperpolarization-activated cyclic nucleotide-gated (HCN

66 A and is expressed via a mechanism involving hyperpolarization-activated cyclic nucleotide-gated (HCN

67 ic neuron by altering ion permeation through hyperpolarization-activated cyclic nucleotide-gated (HCN

68 rking memory by increasing the open state of hyperpolarization-activated cyclic nucleotide-gated (HCN

69 ,5'-cyclic adenosine monophosphate (cAMP) to hyperpolarization-activated cyclic nucleotide-gated (HCN

70 The hyperpolarization-activated cyclic nucleotide-gated (HCN

71 In the presence of the hyperpolarization-activated cyclic nucleotide-gated (HCN

72 (+) flux through presynaptic plasma membrane hyperpolarization-activated cyclic nucleotide-gated (HCN

73 In this study, we asked whether the hyperpolarization-activated cyclic nucleotide-gated (HCN

74 Hyperpolarization-activated cyclic nucleotide-gated (HCN

75 TRIP8b, an accessory subunit of hyperpolarization-activated cyclic nucleotide-gated (HCN

76 The hyperpolarization-activated cyclic nucleotide-gated (HCN

77 Hyperpolarization-activated cyclic nucleotide-gated (HCN

78 underlie regulation of structurally related hyperpolarization-activated cyclic nucleotide-gated (HCN

79 SHANK3 protein interacted with hyperpolarization-activated cyclic nucleotide-gated chan

80 One locus had a single QTG, Hcn1 (hyperpolarization-activated cyclic nucleotide-gated chan

81 The gene, hyperpolarization-activated cyclic nucleotide-gated chan

82 axon initial segment and markedly depends on hyperpolarization-activated cyclic nucleotide-gated chan

83 atory effect results from an augmentation of hyperpolarization-activated cyclic nucleotide-gated chan

84 near the plexus showed immunoreactivity for hyperpolarization-activated cyclic nucleotide-gated chan

85 vioral results support an important role for hyperpolarization-activated cyclic nucleotide-gated chan

86 , Gnai1, protein kinase C gamma (Prkcc), and hyperpolarization-activated cyclic nucleotide-gated chan

87 Overexpression of a hyperpolarization-activated cyclic nucleotide-gated ion

88 the subcellular distribution and function of hyperpolarization-activated cyclic nucleotide-gated nons

89 Hyperpolarization-activated cyclic nucleotide-modulated

90 The hyperpolarization-activated cyclic nucleotide-modulated

91 However, unlike eukaryote hyperpolarization-activated cyclic nucleotide-modulated

92 Cyclic nucleotide-gated (CNG) and hyperpolarization-activated cyclic nucleotide-regulated

93 Here, we assessed the role of hyperpolarization-activated cyclic-nucleotide-gated (HCN

94 we demonstrate that dendritically expressed hyperpolarization-activated cyclic-nucleotide-gated (HCN

95 aneuronal map of spectral tuning mediated by hyperpolarization-activated cyclic-nucleotide-gated nons

96 Hyperpolarization-activated, cyclic nucleotide-gated (HC

97 Hyperpolarization-activated, cyclic nucleotide-gated (HC

98 at N-methyl-D-aspartate receptor (NMDAR) and hyperpolarization-activated, cyclic nucleotide-gated (HC

99 Hyperpolarization-activated, cyclic nucleotide-gated cat

100 -dimensional model, based on animal HCN (for Hyperpolarization-activated, cyclic nucleotide-gated K(+

101 CLC-2 is a hyperpolarization-activated, inwardly rectifying chlorid

102 lations of neurons share a large, fast after-hyperpolarization (AHP).

103 n (ADP); (iii) reduced fast and medium after-hyperpolarizations (AHPs); (iv) strongly enhanced burst

104 e enhanced NMR sensitivity provided by SABRE hyperpolarization allows DOSY analysis of low-micromolar

105 y transmembrane elements that sense membrane hyperpolarization, although cAMP reduces the voltage req

106 cells was accompanied by reductions in after-hyperpolarization amplitudes and a left-shift in the fre

107 The Mcl-1 ASOs also induced mitochondrial hyperpolarization and a consequent increase in mitochond

108 was accompanied by a reduction in the after-hyperpolarization and a decrease in the functional activ

109 +) waves, small-conductance K+ (SK)-mediated hyperpolarization and a decrease of transient receptor p

110 tripartite neuronal circuit involving a fast hyperpolarization and a dual regulation of synaptic inpu

111 ed a dual muscarinic action in the form of a hyperpolarization and an increase in excitability uncove

112 significantly reduced GABA-induced membrane hyperpolarization and caused a depolarizing shift in GAB

113 Both hyperpolarization and Cav3.x channel blockade reduce ste

114 inaural auditory gerbil neurons to show that hyperpolarization and cyclic-nucleotide-gated (HCN) chan

115 lation of L6 in vivo results in a mixture of hyperpolarization and depolarization in the thalamic tar

116 est instrumentation demonstrated all-optical hyperpolarization and detection of (129)Xe gas.

117 assium (IK and SK) channels, thereby causing hyperpolarization and endothelium-dependent vasodilatati

118 o excitatory network inputs by both membrane hyperpolarization and enhanced de-inactivation of an A-c

119 olateral TMN neurons but produced a membrane hyperpolarization and increased inhibitory synaptic inpu

120 and the soma and is independent of membrane hyperpolarization and instead likely mediated by lowerin

121 Here, we combined (13)C hyperpolarization and isotopomer analysis to quantify mu

122 ygen species (ROS) generation, mitochondrial hyperpolarization and lipid peroxidation in neuronal cel

123 , and OFF periods, characterized by membrane hyperpolarization and neuronal silence.

124 vide highly sensitive and efficient membrane hyperpolarization and neuronal silencing through light-g

125 A)Rs) in the subependymal zone (SEZ) induces hyperpolarization and osmotic swelling in precursors, th

126 r the first time and establish mitochondrial hyperpolarization and oxidant generation as potential pa

127 ngly, optogenetic inactivation of wS1 caused hyperpolarization and reduced firing in wM1, together wi

128 asic responses in colonic muscles: transient hyperpolarization and relaxation followed by repolarizat

129 y oxygen sensing by triggering mitochondrial hyperpolarization and release of mitochondrial superoxid

130 Presynaptic hyperpolarization and selective block of Cav3.x channels

131 egulated K(+) channels that produce membrane hyperpolarization and shape neurologic, epithelial, card

132 assium (GIRK) channels that mediate membrane hyperpolarization and synaptic inhibition in the brain.

133 between thalamus and cortex, due to neuronal hyperpolarization and the initiation of low-threshold ca

134 ne cardiac muscle cell line (HL-1) increased hyperpolarization and upstroke velocity of the action po

135 Ca(2+) events result in endothelium-derived hyperpolarization (and SMC relaxation), with magnitude t

136 ated by depolarization and de-inactivated by hyperpolarization, and accounts for the prolonged delay.

137 ce Ca(2+)-activated K(+) channel activation, hyperpolarization, and attenuation of cerebral arterial

138 tion technologies, new approaches to nuclear hyperpolarization, and progress in sample preparation.

139 ivity-dependent, TTX- and ouabain-sensitive, hyperpolarization ( approximately 5 mV), which is mediat

140 n the AP, possibly via AP membrane potential hyperpolarization, are the dominant mechanism of adenosi

141 M 34 attenuated ROV, implicating endothelial hyperpolarization as the underlying signal.

142 nd IKCa channels attenuated ROV, implicating hyperpolarization as the underlying signal.

143 , producing no decrease in CB1-mediated peak hyperpolarization at concentrations up to 10 muM but enh

144 c neurons (opposite trend) instead exhibited hyperpolarization at high repetition frequency but susta

145 mic slow oscillations at low arousal, stable hyperpolarization at intermediate arousal, and depolariz

146 embrane potential to spontaneously jump from hyperpolarization at the first zero-current potential to

147 ion driven by both pathways, with pronounced hyperpolarizations at light offset.

148 ward rectifier potassium (GIRK) channels and hyperpolarization, but in response to continued signalin

149 We have recently demonstrated how hyperpolarization by dissolution dynamic nuclear polariz

150 Hyperpolarization by dissolution dynamic nuclear polariz

151 Here, hyperpolarization by irradiation with microwaves in the

152 reporter ligand in a single scan when using hyperpolarization by means of dissolution dynamic nuclea

153 zation pattern and neural patterning; forced hyperpolarization by misexpression of specific ion chann

154 , CK2 inhibition diminished the medium after hyperpolarization by suppressing the M-current.

155 We suggest that this motoneuron hyperpolarization can bias motor output to left or right

156 Mitochondrial hyperpolarization caused by IL-6 is uncoupled from the p

157 mapping between distinct environments, while hyperpolarization did not.

158 mitochondrial energy production and neuronal hyperpolarization during cellular stress conditions, suc

159 and expanded arbors following halorhodopsin hyperpolarization during development.

160 al models has identified endothelium-derived hyperpolarization (EDH) as a potential mechanism capable

161 striking contrast to the endothelium-derived hyperpolarization (EDH) characteristic of arteries, LECs

162 ther study the role of endothelium-dependent hyperpolarization (EDH), ACh trials were repeated with c

163 pact of the disease on endothelium-dependent hyperpolarization (EDH)-mediated vasorelaxation; 2) incr

164 bited bradykinin-induced endothelium-derived hyperpolarization (EDH)-type relaxation in U46619-precon

165 enetic gate restoration through granule cell hyperpolarization efficiently stopped spontaneous seizur

166 In mature AIIs, membrane hyperpolarization elicits bursting behavior.

167 The sensitivity gains obtained with hyperpolarization enable the real-time observation of (1

168 Specifically, dopamine inhibition and direct hyperpolarization enabled the generation of low-threshol

169 nal gain of 3 orders of magnitude from (13)C hyperpolarization enabled the real time observation of c

170 We confirmed that such hyperpolarization enhances RGC action potential firing i

171 entiviral PDK2 knockdown prevented DeltaPsim hyperpolarization, facilitated apoptosis and reduced myo

172 ly biphasic flash responses, with an initial hyperpolarization followed by a depolarization beyond th

173 f their unprecedented efficiency of membrane hyperpolarization for optogenetic neuron silencing.

174 ce (n gate) showed a longer peak and shallow hyperpolarization; for power-law activation of the sodiu

175 Fluorine-19 NMR and hyperpolarization form a powerful combination for drug s

176 tive in swimming or struggling are raised by hyperpolarization from the activation of fast transient

177 Furthermore, the mode-shift displayed by hyperpolarization-gated cation channels is likely caused

178 Moreover, although hyperpolarization has successfully quantified pyruvate-l

179 Hyperpolarization (HP) of nuclear spins is critical for

180 followed by a prolonged outward current and hyperpolarization in 80% of neurons.

181 ted for their influence on CP55,940-mediated hyperpolarization in AtT20-hCB1 cells.

182 nsistent with a role for endothelium-derived hyperpolarization in functional sympatholysis.

183 e leads to a long-lasting membrane potential hyperpolarization in hippocampal principal cells that is

184 Up4A induced P2Y1R-SK-channel-mediated hyperpolarization in isolated PDGFRalpha(+) cells, which

185 omplex I activity and mitochondrial membrane hyperpolarization in mitochondria from IDD rat pulmonary

186 250 nM and produced an approximately 8.5 mV hyperpolarization in ocular surface potential difference

187 interactions, and may induce the erosion of hyperpolarization in spectroscopy and MRI.

188 Deinactivation of Cav3.x channels by hyperpolarization increases T-type Ca(2+) influx upon re

189 netic control much more efficiently than the hyperpolarization induced by light-activated chloride pu

190 Furthermore, hyperpolarization induced with cromakalim or valinomycin

191 muli to regular spiking, and fully abolished hyperpolarization-induced burst firing.

192 rpolarization-activated currents, lengthened hyperpolarization-induced delays in spiking relative to

193 ressed in Xenopus oocytes, which displayed a hyperpolarization-induced inward current that was enhanc

194 Plasma membrane hyperpolarization is crucial for mammalian sperm to acqu

195 However, once hyperpolarization is imparted on a molecule, the magneti

196 Considering that capacitation-induced hyperpolarization is mediated by SLO3, we evaluated the

197 ad not been demonstrated that SLO3-dependent hyperpolarization is required for Ca(2+) entry through C

198 ose that initiation of endothelium-dependent hyperpolarization is the underlying signalling event nec

199 ns; (iii) reducing the fast and medium after-hyperpolarizations; (iv) broadening action potentials; a

200 rane potential, oxidative stress, or chronic hyperpolarization lead to increased Oma1-mediated proteo

201 Hyperpolarization level is the main determinant of wheth

202 l for in vivo applications because of longer hyperpolarization lifetimes, lack of background signal,

203 BAA potentiation such as under propofol, its hyperpolarization may determine whether a patient experi

204 Advances in hyperpolarization methods improve the applicability of N

205 betes exhibited mitochondrial inner-membrane hyperpolarization (MHP).

206 sed relaxation (human and murine colons) and hyperpolarization (murine colon) that was blocked by the

207 Here we report bulk, room-temperature hyperpolarization of (13)C nuclear spins observed via hi

208 in heterologous cells, (ii) BigLEN-mediated hyperpolarization of BLA pyramidal neurons, and (iii) fe

209 Hyperpolarization of cell membrane induces cell differen

210 that can establish prolonged, physiological hyperpolarization of cells at low light intensities.

211 Early frog embryos exhibit a characteristic hyperpolarization of cells lining the neural tube; disru

212 Arousing dopamine caused transient hyperpolarization of dFB neurons within tens of millisec

213 Thus, ROV of FAs reflects hyperpolarization of downstream arterioles that conducts

214 aptic cleft alkalization during light-evoked hyperpolarization of HCs also involves changes in bicarb

215 The hyperpolarization of heteronuclei via signal amplificati

216 Optogenetic hyperpolarization of interneurons had spatially uniform

217 ediated by siRNA attenuated morphine-induced hyperpolarization of membrane potential in AtT20 cells.

218 atin-resistant cells was correlated with the hyperpolarization of mitochondria in these cells and mit

219 Direct NMR hyperpolarization of naturally abundant (15)N sites in m

220 Hyperpolarization of nuclear spins can dramatically incr

221 el in OX neurons, which was required for the hyperpolarization of OX neurons by leptin.

222 GSK induced hyperpolarization of PDGFRalpha(+) cells and decreased d

223 mes, induce acidification of the cytosol and hyperpolarization of plasma cell membranes.

224 The ERG a-wave is the light-induced hyperpolarization of retinal photoreceptors, and the b-w

225 Hyperpolarization of substrates for magnetic resonance s

226 These effects are because of hyperpolarization of the baseline membrane potential by

227 rtening of the action potential duration and hyperpolarization of the cardiomyocyte resting membrane

228 GH43 ABNs are activated by calcium ions via hyperpolarization of the catalytically relevant histidin

229 rise in intracellular Ca(2+) ([Ca(2+) ]i ), hyperpolarization of the endothelium coordinates vascula

230 Hyperpolarization of the nervous system using a histamin

231 tifying potassium (GIRK) channels leads to a hyperpolarization of the neuron's membrane potential, pr

232 ium-bicarbonate co-transporter, leading to a hyperpolarization of the neuron.

233 rily to cholinergic cells, we observe a slow hyperpolarization of the resting membrane potential and

234 ling to the surface membrane translates to a hyperpolarization of the reversal potential for GABA (EG

235 t amplitude by reducing NKA activity despite hyperpolarization of the RMP.

236 ts are consistent with findings showing that hyperpolarization of the sperm plasma membrane is necess

237 its activity blocks the capacitation-induced hyperpolarization of the sperm plasma membrane without b

238 receptor potential melastatin 1 channels and hyperpolarization of these cells.

239 ion to cell-autonomous effects, we show that hyperpolarization of transmembrane potential (Vmem) in v

240 ate in the ventral SCN at night and enhances hyperpolarization of ventral SCN neurons at this time.

241 ble spin exchange optical pumping (SEOP) and hyperpolarization of xenon-129 gas.

242 The effect of hyperpolarization on [Ca(2+)]i was abolished following r

243 d receptors, we analyzed the effect of their hyperpolarization on respiration in spontaneously breath

244 ct prevented by attenuation of mitochondrial hyperpolarization or by scavenging mitochondrial oxidant

245 ssor of neural induction, impairs the normal hyperpolarization pattern and neural patterning; forced

246 ells, with respect to mitochondrial membrane hyperpolarization, plasma membrane depolarization, and i

247 giant axon, Cole and Moore noted that strong hyperpolarization preceding a depolarizing voltage-clamp

248 olarization promotes Ca wave propagation and hyperpolarization prevents it.

249 e environment to completely control the PHIP hyperpolarization process including remotely triggering

250 embrane properties may also enhance or limit hyperpolarization, raising the question of how intrinsic

251 ces activated platelet-induced mitochondrial hyperpolarization, reactive oxygen species overload and

252 As expected, the opioid-induced hyperpolarization reduced the excitability of the neuron

253 nstead exhibit a period of depolarization or hyperpolarization referred to as an afterpotential.

254 The resulting hyperpolarization renders the neurons less responsive to

255 en and elongate upon cell depolarization and hyperpolarization, respectively.

256 Hyperpolarization responses are conducted to SMCs via ga

257 neurons and suggested to mediate purinergic hyperpolarization responses in smooth muscles of the gas

258 However, the relaxation and hyperpolarization responses to Up4A are not mediated by

259 The activation of IKir by large hyperpolarizations resulted in significant attenuation o

260 The hyperpolarization signal also synergizes with canonical

261 ization technique that uses para-hydrogen as hyperpolarization source.

262 By combining relaxation measurements with hyperpolarization, spins on the surface of the nanodiamo

263 t activity in FLCs was spontaneous transient hyperpolarizations (STHs), with maximum amplitudes above

264 f14(-/-) Purkinje neurons, by prior membrane hyperpolarization, suggesting that the iFGF14-mediated r

265 SABRE is a nuclear spin hyperpolarization technique based on the reversible asso

266 is an inexpensive, fast, and even continuous hyperpolarization technique that uses para-hydrogen as h

267 We also describe how modern hyperpolarization techniques like dynamic nuclear polari

268 Spine Ca(2+) signals were less sensitive to hyperpolarization than shaft synapses, suggesting amplif

269 to produce a rapidly propagating retrograde hyperpolarization that causes upstream arteriolar dilati

270 Tetanic contraction initiates hyperpolarization that conducts along endothelium into F

271 abinoid production leads to a cell-intrinsic hyperpolarization that controls self activity.

272 K(+) channel (CNGK) mediates a cGMP-induced hyperpolarization that evokes Ca(2+) influx.

273 This suggests a warming-induced hyperpolarization that is reduced by blocking electroneu

274 Taking advantage of hyperpolarization, the ligand concentration in the excha

275 se was always the same but after significant hyperpolarization there was a long lag before the rise b

276 SHP-1, inhibited sodium channels and caused hyperpolarization through activation of TREK2 K(+) chann

277 e sensor that might be activated by membrane hyperpolarization, thus increasing the voltage sensitivi

278 mately -40 mV) via exposure to 10 muM NS309 (hyperpolarization to approximately -80 mV), via equilibr

279 Particularly tantalizing is future use of hyperpolarization to increase sensitivity by orders of m

280 ted to efficiently transfer its nuclear spin hyperpolarization to nitrogen-15 in pyridine and nicotin

281 e arteries integrates Ca(2+) signalling with hyperpolarization to promote relaxation of smooth muscle

282 ies revealed that the mutant required longer hyperpolarization to recover from fast inactivation, whi

283 nce of RhoA, increased Cdc42 activity and MK hyperpolarization triggers GPIb-dependent transmigration

284 Hyperpolarization turns typically weak NMR and MRI respo

285 oncentration ([Ca(2+)]i) to endothelial cell hyperpolarization underlies smooth muscle cell relaxatio

286 citatory input to CA1, while the post-ripple hyperpolarization varies proportionately.

287 Hyperpolarization via dynamic nuclear polarization (DNP)

288 f molecular structures and sites amenable to hyperpolarization via low-cost parahydrogen-based method

289 Hyperpolarization via optical pumping/dynamic nuclear po

290 per channels is limited by Ca(2+)-controlled hyperpolarization via Slo3.

291 The ability to perform continuous in situ hyperpolarization via the Overhauser mechanism, in combi

292 uppress GABA release and induce postsynaptic hyperpolarization was unaffected.

293 After providing an overview of MR and hyperpolarization, we will discuss the latest advances i

294 Mitochondrial hyperpolarization, which can promote mitochondrial super

295 The hyperpolarization, which is specific to CA3 and CA2 pyra

296 epithelial cells stimulates K(+) efflux and hyperpolarization, which lead to NLRP3 inflammasome acti

297 for the transport and application of nuclear hyperpolarization, which leads to NMR and MRI signals en

298 action potentials or by pairing postsynaptic hyperpolarization with activation of group I metabotropi

299 ated Ca channel inhibitor, nifedipine, or by hyperpolarization with diazoxide.

300 combining a magnetic enhancement technique (hyperpolarization) with cellular glycolytic activity.