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3 out asthma to assess if asthmatic cells were hypersecretory and determined whether the underlying mec
4 cells cultured from subjects with asthma are hypersecretory compared with cells from healthy donors a
7 osal gland hypertrophy are shared with other hypersecretory diseases, such as chronic obstructive pul
9 h should lead to the rational design of anti-hypersecretory drugs for treatment of airway mucus hyper
10 ingly, human CLCA1 is a critical mediator of hypersecretory lung diseases, such as asthma, chronic ob
14 or Zollinger-Ellison syndrome and other acid hypersecretory states had interval histories taken every
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