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1 tic target, not only for asthma but also for hypersecretory airway diseases.
2 ade may provide effective new treatments for hypersecretory airway diseases.
3 out asthma to assess if asthmatic cells were hypersecretory and determined whether the underlying mec
4 cells cultured from subjects with asthma are hypersecretory compared with cells from healthy donors a
5 roles for inhibitors of the EGF-R cascade in hypersecretory diseases of airways.
6 plasia is a critical pathological feature in hypersecretory diseases of airways.
7 osal gland hypertrophy are shared with other hypersecretory diseases, such as chronic obstructive pul
8 provide new therapeutic approaches in airway hypersecretory diseases.
9 h should lead to the rational design of anti-hypersecretory drugs for treatment of airway mucus hyper
10 ingly, human CLCA1 is a critical mediator of hypersecretory lung diseases, such as asthma, chronic ob
11 hophysiology of asthma impact upon the mucus hypersecretory phenotype.
12 thermore, asthma might have a specific mucus hypersecretory phenotype.
13 te in a positive feedback loop involving the hypersecretory response.
14 or Zollinger-Ellison syndrome and other acid hypersecretory states had interval histories taken every

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