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1 opsy revealed extrinsic allergic alveolitis (hypersensitivity pneumonitis).
2 ffect on the TH1/TH17-mediated lung disease, hypersensitivity pneumonitis.
3 , chronic obstructive pulmonary disease, and hypersensitivity pneumonitis.
4 Saccharopolyspora rectivirgula in a model of hypersensitivity pneumonitis.
5 the pathogenesis of fibrosis in experimental hypersensitivity pneumonitis.
6 ntify occult antigen exposure known to cause hypersensitivity pneumonitis.
7 predominant source of IL-17A in experimental hypersensitivity pneumonitis.
8 specific interstitial pneumonia, and chronic hypersensitivity pneumonitis.
9 ffective way to control microbial Ag-induced hypersensitivity pneumonitis.
10 in this murine model of B. subtilis-induced hypersensitivity pneumonitis.
11 and reduced survival in people with chronic hypersensitivity pneumonitis.
12 a domiciliana was the cause of the patient's hypersensitivity pneumonitis.
13 IFN-gamma is essential for the expression of hypersensitivity pneumonitis.
14 inst S. rectivirgula in this murine model of hypersensitivity pneumonitis and pulmonary fibrosis.
15 opment of pulmonary fibrosis in experimental hypersensitivity pneumonitis and to determine the main i
17 centre with recognised expertise in chronic hypersensitivity pneumonitis, and further research and s
18 ria were subsequently diagnosed with chronic hypersensitivity pneumonitis, and most of these cases we
20 underlie the differential susceptibility to hypersensitivity pneumonitis between C57BL/6 and DBA/2 m
22 nflammation in murine models of experimental hypersensitivity pneumonitis, but its role in the develo
23 opathic pulmonary fibrosis (IPF) and chronic hypersensitivity pneumonitis can be indistinguishable; t
25 play a regulatory role in the development of hypersensitivity pneumonitis caused by microbial Ags and
26 critical review of the current knowledge on hypersensitivity pneumonitis caused by the occupational
29 with nosocomial infections and occupational hypersensitivity pneumonitis due to metalworking fluid (
30 s non-specific interstitial pneumonia, 16 as hypersensitivity pneumonitis, four as sarcoidosis, four
31 riod, and 16 of the 20 patients with chronic hypersensitivity pneumonitis had histopathological featu
32 type(s) that produce IL-17A in experimental hypersensitivity pneumonitis have not yet been identifie
33 Farmers lung disease is a common form of hypersensitivity pneumonitis (HP) and is characterized b
41 ospectively compare in patients with chronic hypersensitivity pneumonitis (HP) the computed tomograph
49 sciplinary team agreement for a diagnosis of hypersensitivity pneumonitis is low, highlighting an urg
50 NSIP; kappaw=0.42 [0.37-0.49]); and fair for hypersensitivity pneumonitis (kappaw=0.29 [0.24-0.40]).
51 [range, 23-86 years]), and 192 with chronic hypersensitivity pneumonitis (men, 76; women, 116; media
52 with idiopathic pulmonary fibrosis (n = 5), hypersensitivity pneumonitis (n = 5), and normal lung (n
53 elines had a subsequent diagnosis of chronic hypersensitivity pneumonitis: nine patients had positive
55 independent cohorts of patients with chronic hypersensitivity pneumonitis, one from the University of
56 urgical lung biopsy that was consistent with hypersensitivity pneumonitis; one was IgG positive plus
58 everal interstitial lung diseases, including hypersensitivity pneumonitis, respiratory bronchiolitis-
60 and assessed their associations with chronic hypersensitivity pneumonitis risk, survival, and clinica
61 sy showing a pattern consistent with chronic hypersensitivity pneumonitis); seven were IgG positive p
63 nd reduced survival in patients with chronic hypersensitivity pneumonitis suggest shared pathobiology
64 or MUC5B rs35705950 in patients with chronic hypersensitivity pneumonitis than in healthy controls (2
69 asthma model, we found that the severity of hypersensitivity pneumonitis was unaffected by vancomyci
70 Using a well-established murine model of hypersensitivity pneumonitis, we repeatedly exposed C57B
71 athic nonspecific interstitial pneumonia and hypersensitivity pneumonitis, were reclassified as IPF.
72 lmonary fibrosis (IPF) or chronic (fibrotic) hypersensitivity pneumonitis, which suggests these disor
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