ライフサイエンスコーパス: hypersomnia

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1 large number of neurons activated during PS hypersomnia.

2 depressive episodes, including insomnia and hypersomnia.

3 atypical patients experience hyperphagia and hypersomnia.

4 mportant diagnostic tool in the diagnosis of hypersomnias.

5 Common symptoms were hypersomnia (100%), cognitive changes (96%, including a

6 aracterized by atypical symptoms, especially hypersomnia, afternoon or evening slump, reverse diurnal

7 erity and reversed neurovegetative symptoms (hypersomnia and either hyperphagia or weight gain).

8 episode, in addition to atypical features of hypersomnia and hyperphagia.

9 ing only the reversed vegetative symptoms of hypersomnia and hyperphagia.

10 y information on 42 subjects with idiopathic hypersomnia and obtained detailed follow-up evaluations

11 large number of neurons activated during PS hypersomnia and projecting to the VLPAG/dDpMe.

12 In patients with central hypersomnia and thus a high pretest probability for narc

13 ay also aid more accurate phenotyping of the hypersomnias and in particular clarify heterogeneity amo

14 ted experiencing significantly more fatigue, hypersomnia, and psychomotor retardation during the most

15 h other sleep disorders, patients with other hypersomnias, and patients with narcolepsy with normal h

16 Narcolepsy and idiopathic hypersomnia are chronic brain disorders with an onset at

17 The features of idiopathic hypersomnia are not well defined.

18 e, failures were associated with fatigue and hypersomnia, but less so with sadness, anhedonia, and ap

19 disorder with symptoms that include periodic hypersomnia, cognitive and behavioural disturbances.

20 acterized by relapsing-remitting episodes of hypersomnia, cognitive disturbances, and behavioral dist

21 During episodes, all patients had hypersomnia, cognitive impairment, and derealization; 66

22 e therapy are recommended for narcolepsy and hypersomnia; continuous positive airway pressure, weight

23 , but was characterized by increased eating, hypersomnia, frequent, relatively short episodes, and a

24 depressive disorder include mood reactivity, hypersomnia, hyperphagia, leaden paralysis, and rejectio

25 ve mood plus at least 2 additional symptoms: hypersomnia, hyperphagia, leaden paralysis, or lifetime

26 The pathophysiology of idiopathic hypersomnia (IH) remains unclear.

27 alences were 23.1% for insomnia and 6.7% for hypersomnia in 1994.

28 16 years.SUMMARY: Awareness of the extent of hypersomnia in children will allow physicians to effecti

29 Idiopathic hypersomnia is a rare syndrome in which clinical heterog

30 tions for children with narcolepsy and other hypersomnias of central origin in order to raise awarene

31 more sleep disturbances including insomnia, hypersomnia, or nightmares than nonsuicidal patients.

32 Hypersomnia, rapid eye movement sleep disorder and/or na

33 The clinical usefulness of CSF testing in hypersomnia that is symptomatic of a neurological disord

34 sease characterized by recurrent episodes of hypersomnia with behavioral and cognitive disturbances.

35 without cataplexy (N-C), 21 with idiopathic hypersomnia with long sleep time (IHL), 20 with BIISS an

36 nly 29% of subjects had 'classic' idiopathic hypersomnia with non-imperative sleepiness, long unrefre

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