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1 leepiness Scale (ESS, which assesses daytime hypersomnolence).
2 sential for the proinflammatory response and hypersomnolence.
3 of superoxide in the oxidation injury and in hypersomnolence.
4 ysis was used to assess the risk factors for hypersomnolence.
5 stages, was also an independent predictor of hypersomnolence.
6 d slow wave sleep (SWS) were protective from hypersomnolence.
7 o significant association with the degree of hypersomnolence.
8 option in patients with treatment-refractory hypersomnolence.
9 decreased sleep efficiency, without daytime hypersomnolence.
10 obstructive sleep apnea, results in lasting hypersomnolence and is associated with nitration and oxi
11 itical role for NADPH oxidase in the lasting hypersomnolence and oxidative and proinflammatory respon
13 to not only long-term hypoxia/reoxygenation hypersomnolence but also to carbonylation, lipid peroxid
14 cterize the prevalence of persistent daytime hypersomnolence, difficulties initiating and maintaining
15 ntiation with CSF from patients with central hypersomnolence disorders, with no significant differenc
17 olysomnographic parameters and the degree of hypersomnolence in 741 patients with SDB (apnea-hypopnea
23 ols (9 males and 6 females) with unspecified hypersomnolence (n = 7) and miscellaneous neurological c
24 derate-severe sleep apnea, result in lasting hypersomnolence, oxidative injury, and proinflammatory r
25 g with breakfast and lunch, in patients with hypersomnolence syndromes (excluding narcolepsy with cat
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