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1 leepiness Scale (ESS, which assesses daytime hypersomnolence).
2 sential for the proinflammatory response and hypersomnolence.
3 of superoxide in the oxidation injury and in hypersomnolence.
4 ysis was used to assess the risk factors for hypersomnolence.
5 stages, was also an independent predictor of hypersomnolence.
6 d slow wave sleep (SWS) were protective from hypersomnolence.
7 o significant association with the degree of hypersomnolence.
8 option in patients with treatment-refractory hypersomnolence.
9  decreased sleep efficiency, without daytime hypersomnolence.
10  obstructive sleep apnea, results in lasting hypersomnolence and is associated with nitration and oxi
11 itical role for NADPH oxidase in the lasting hypersomnolence and oxidative and proinflammatory respon
12               A positive association between hypersomnolence and oxyhemoglobin desaturation (DeltaSaO
13  to not only long-term hypoxia/reoxygenation hypersomnolence but also to carbonylation, lipid peroxid
14 cterize the prevalence of persistent daytime hypersomnolence, difficulties initiating and maintaining
15 ntiation with CSF from patients with central hypersomnolence disorders, with no significant differenc
16  and narcolepsy, a disorder characterized by hypersomnolence during normal wakefulness.
17 olysomnographic parameters and the degree of hypersomnolence in 741 patients with SDB (apnea-hypopnea
18 ry responses, oxidative injury, and residual hypersomnolence in obstructive sleep apnea.
19 ragmentation are independent determinants of hypersomnolence in SDB.
20  and activity monitoring revealed a profound hypersomnolence in these mice.
21                           Persistent daytime hypersomnolence is associated with significant morbidity
22 nal hypoxemia were independent predictors of hypersomnolence (MSLT < 10 min).
23 ols (9 males and 6 females) with unspecified hypersomnolence (n = 7) and miscellaneous neurological c
24 derate-severe sleep apnea, result in lasting hypersomnolence, oxidative injury, and proinflammatory r
25 g with breakfast and lunch, in patients with hypersomnolence syndromes (excluding narcolepsy with cat
26                                 Some central hypersomnolence syndromes are associated with a positive
27                                              Hypersomnolence was quantified with the multiple sleep l
28 crease in stage 2 or SWS the adjusted RR for hypersomnolence were 0.93 and 0.79, respectively.
29          The adjusted relative risks (RR) of hypersomnolence were 1.00, 1.30, and 1.65 for patients w

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