1 ving platelet counts in patients with severe hypersplenism.

2                         Twenty-two (34%) had hypersplenism.

3 ients developed gastrointestinal bleeding or hypersplenism.

4      All patients had laboratory evidence of hypersplenism.

5 nts on this therapy need to be monitored for hypersplenism.

6 l splenectomy appears to control symptoms of hypersplenism and splenic sequestration.

7 hildren without HS had decreased symptoms of hypersplenism and splenic sequestration.

8 pectrum of ALPS and its major complications: hypersplenism, autoimmune hemolytic anemia, thrombocytop

9 nt evidence pointing toward a combination of hypersplenism, autoimmunity, and direct viral infection

10       Avoiding splenectomy while controlling hypersplenism by using corticosteroid-sparing treatments

11 aled several significant findings, including hypersplenism, decreased erythrocyte volume, increased n

12                                Patients with hypersplenism had significant increases in platelet and

13 ortal decompression by TIPS would ameliorate hypersplenism in patients with portal hypertension.

14  splenomegaly, and 5 patients developed mild hypersplenism that did not require any specific treatmen

15                           Five children with hypersplenism underwent embolization of 30%-40% of the s

16                  Resolution of pretransplant hypersplenism, with the release of sequestered splenic g