ライフサイエンスコーパス: hyperstimulation

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1 s consistently observed early after cerulein hyperstimulation.

2 intraductal bile acid infusion and cerulein hyperstimulation.

3 f acute pancreatitis (AP) based on caerulein hyperstimulation.

4 hominissuis biofilm induces TNF-alpha-driven hyperstimulation and apoptosis of surveilling phagocytes

5 ould explain the etiology of several hormone hyperstimulation and resistance syndromes.

6 luteinizing hormone with subsequent ovarian hyperstimulation as a model to identify mechanisms invol

7 concentration, whereas fragment 1-453 shows hyperstimulation at low ratios of DNA to enzyme, a pheno

8 ormone-overexpressing mouse model of ovarian hyperstimulation developed multifocal mammary tumors in

9 The hyperstimulation effect can be interpreted in terms of a

10 hemokine, mcp-1, were induced after cerulein hyperstimulation in vivo.

11 prevented premature LH surges during ovarian hyperstimulation in women.

12 sulin-like growth factor-I receptor (IGF-IR) hyperstimulation induced hyperproliferation and antiapop

13 aling by Akt1 kinase since striatal dopamine hyperstimulation is associated with psychosis and schizo

14 and lack of mitogen-activated protein kinase hyperstimulation likely are responsible for constraining

15 s, effects of PCP, including a glutamatergic hyperstimulation, may be necessary to account for the ps

16 We used a cerulein hyperstimulation model of acute pancreatitis and measure

17 However, the secretagogue hyperstimulation model of pancreatitis is the most commo

18 pancreatitis outcomes using a mild caerulein hyperstimulation model were similar between IP3R2(-/-) a

19 er addition of free H2A.Z-H2B dimer leads to hyperstimulation of ATPase activity, eviction of nucleos

20 Following optogenetic hyperstimulation of Caenorhabditis elegans motoneurons,

21 damage in blinding retinal diseases in which hyperstimulation of glutamate receptors is implicated as

22 Genetically engineered hyperstimulation of guanosine 3',5'-cyclic monophosphate

23 ngly, while derepression of MdrT resulted in hyperstimulation of IFN-beta, it results in significant

24 t of various cancer cells with AS1411 caused hyperstimulation of macropinocytosis, provoking an incre

25 gain-of-function mutations, leading to MgADP hyperstimulation of the channel, are located in the D-lo

26 ulation of p16INK4 and p27(Kip1) and lack of hyperstimulation of the mitogen-activated protein kinase

27 riptional role of GPS2 as a guardian against hyperstimulation of the TNF-alpha-induced gene program.

28 tely 10 bp; fragments >or=32 bp manifest the hyperstimulation phenomenon.

29 r deficits persisted long after cessation of hyperstimulation, providing evidence for a critical peri

30 on with gonadotropins is followed by Ovarian Hyperstimulation Syndrome (OHSS) in some women.

31 reased lupus activity and 2 (13%) in ovarian hyperstimulation syndrome.

32 eimer's disease, malaria, burns, and ovarian hyperstimulation syndrome.

33 Despite this T-cell "hyperstimulation" using TRICOM vectors, no evidence of a

34 Rates of uterine hyperstimulation were low in both the misoprostol and Fo

35 Controlled ovarian hyperstimulation with gonadotropins is followed by Ovari

36 reatitis observed in Munc18c(+/-) mice after hyperstimulation with the CCK-8 analog caerulein.

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