ライフサイエンスコーパス: hypersusceptibility

1 s also isolated in our screen for Ub-peptide hypersusceptibility.

2 ycline binding site that correlates with its hypersusceptibility.

3 transient periods of hyperinfectiousness or hypersusceptibility.

4 these mutations partially ablate amprenavir hypersusceptibility.

5 with cystic fibrosis (CF) have a pronounced hypersusceptibility (80 to 90%) to Pseudomonas aeruginos

6 in some of these subjects induces amprenavir hypersusceptibility and a reduction of fitness and repli

7 Here, we provide a rationale for this "hypersusceptibility" based on a comparative analysis of

8 V type 1(NL4-3) (HIV-1(NL4-3)) to ritonavir (hypersusceptibility [HS]).

9 and APV binding to I50L variants, leading to hypersusceptibility in these two cases.

10 lta4h mutations confer hypersusceptibility independent of LTB(4) reduction, by

11 The organic solvent hypersusceptibility is a newly described phenotype for a

12 rain (pXO1+ pXO2-) of B. anthracis exhibited hypersusceptibility not only to protamine but also to al

13 of P. aeruginosa may be a critical factor in hypersusceptibility of CF patients to chronic lung infec

14 The hypersusceptibility of fur-deficient Salmonella to the c

15 The hypersusceptibility of gshA and gshB mutants lacking gam

16 een speculated to contribute directly to the hypersusceptibility of specific granule deficiency (SGD)

17 The hypersusceptibility of the autophagy mutants was associa

18 The hypersusceptibility of the DeltaoxyRS mutant was attenua

19 -induced defense gene expression, leading to hypersusceptibility of the ERF6-EAR transgenic plants to

20 Hypersusceptibility of welders to pneumococcal pneumonia

21 Judged by the hypersusceptibility phenotype of acrA mutants, the AcrAB

22 s demonstrated by the complementation of the hypersusceptibility phenotype of the acrA mutant.

23 e screened 10,100 Mtb transposon mutants for hypersusceptibility to acidified nitrite.

24 this reaction in CF patients underlies their hypersusceptibility to chronic P. aeruginosa infection.

25 The alphaB-crystallin mutants exhibited hypersusceptibility to develop pericardial edema when ch

26 utant mitochondrial ribosome associated with hypersusceptibility to drug-induced ototoxicity.

27 Baseline HIV-1 hypersusceptibility to efavirenz (< or = 0.4-fold differ

28 f 2 PIs, being naive to NNRTIs, and baseline hypersusceptibility to efavirenz were associated with a

29 ia coli acrAB multidrug efflux genes, caused hypersusceptibility to erythromycin, rifampin, novobioci

30 le mutant analyses revealed that only rhd1-4 hypersusceptibility to H. schachtii and increased root h

31 s correlated with DeltagraS or DeltaEpsilonL hypersusceptibility to hNP-1 or RP-1 (but not hBD-2) at

32 ntation of exogenous antigen; it also caused hypersusceptibility to infection by mouse cytomegaloviru

33 sts new insights into the pathophysiology of hypersusceptibility to infection may lead to novel poten

34 one day following IAV challenge resulted in hypersusceptibility to lethality.

35 of the cytotoxic all-trans retinaldehyde and hypersusceptibility to light-induced photoreceptor degen

36 ivity of AtFAAH, hypersensitivity to ABA and hypersusceptibility to nonhost pathogens are independent

37 and oxyR, and this phenotype correlated with hypersusceptibility to OHPs, suggesting overlapping or c

38 TLR4 deficiency causes hypersusceptibility to oxidant-induced injury.

39 tonomous expression of HYR1 complemented the hypersusceptibility to phagocyte-mediated killing of a b

40 onclude that PAFr plays an important role in hypersusceptibility to pneumococcal infection in sickle

41 of Salmonella typhimurium conferred specific hypersusceptibility to S-nitrosothiol NO-donor compounds

42 th yield increase and displays a substantial hypersusceptibility to several antibiotics.

43 eplacements in motif B confer broad-spectrum hypersusceptibility to substrate analog inhibitors.

44 , causes root-specific phenotypes, including hypersusceptibility to the cyst nematode Heterodera scha

45 gly, our data show that I132M confers marked hypersusceptibility to the nucleoside analogs lamivudine

46 omycin and telavancin), but also resulted in hypersusceptibility to these antibiotics and to a variet

47 Dicer-related helicase 1 (DRH-1), to display hypersusceptibility to VSV infection as evidenced by ele

48 on conferred resistance to lamivudine and/or hypersusceptibility to zidovudine (AZT).

49 etravirine (ETV; 1.9-4.7-fold) and decreased hypersusceptibility to zidovudine (AZT; 1.4-2.2-fold).

50 transient kinetic analyses showed that this hypersusceptibility was due to I132M decreasing the enzy

51 Gag-processing efficiency, while amprenavir hypersusceptibility was further diminished.

52 Importantly, however, this hypersusceptibility was largely reversed in the presence

53 ly display what is known in the HIV field as hypersusceptibility--when the nematodes become resistant