ライフサイエンスコーパス: hypertensive

1 d increase for antiepileptics to 13-fold for hypertensives).

2 cantly advanced bone ages and are oftentimes hypertensive.

3 3 times more sodium than controls and became hypertensive.

4 renal abnormalities, and 35% of adults were hypertensive.

5 glaucoma suspects, and 108 eyes were ocular hypertensives.

6 emales compared to AA normotensives or white hypertensives.

7 were women, 25% were diabetic, and 91% were hypertensive; 104 started intervention, and 92 completed

8 s; 41% were black; 55% were female; 59% were hypertensive; 22% were diabetic; and 15% were current sm

9 Hypertensive 2K1C rats showed a profound elevation in AB

10 had proteinuria >300 mg/day; 589 (20%) were hypertensive; 495 (18%) obese; 9 (0.3%) were diabetic wh

11 riants are associated with kidney disease in hypertensive AAs.

12 KEY POINTS: In hypertensive adults (HTN), cardiovascular risk increases

13 neurovascular function during cold stress in hypertensive adults (HTN).

14 ltiple 24-h urine samples collected from pre-hypertensive adults 30 to 54 years of age during the tri

15 nd control were defined, respectively, among hypertensive adults as a self-reported diagnosis of hype

16 Our analysis included 17279 Chinese hypertensive adults comprising 6590 men and 10689 women

17 nificantly associated with LE-PAD in Chinese hypertensive adults.

18 tive impairment and reduce blood pressure in hypertensive adults.

19 to calculate regional and global numbers of hypertensive adults.

20 ated to diabetes among normal weight and non-hypertensive adults.

21 sive infiltrative cardiomyopathy that mimics hypertensive and hypertrophic heart disease and often go

22 ion efforts in this population should target hypertensive and metabolic disease.

23 alue of DSE was similar for patients who had hypertensive and normal BP responses (69% versus 73%; P=

24 tion of CCL5 to renal damage and fibrosis in hypertensive and normotensive models of RAS stimulation.

25 rocedures were younger and less likely to be hypertensive and smokers.

26 sion differences between AA and white female hypertensives and pinpointed novel mRNA-microRNA pairs d

27 d excluded patients with ischemic, valvular, hypertensive, and congenital heart disease.

28 er therapeutic classes, like antiepileptics, hypertensives, and gastric and ulcer drugs also showed a

29 infiltrate into the kidney of salt-sensitive hypertensive animals.

30 in 29,985 postmenopausal women who were not hypertensive at baseline.

31 Ten donors were hypertensive before donation.

32 indings confirm clinical trial evidence that hypertensive blacks have poorer outcomes than whites whe

33 well as renalase expression, in genetically hypertensive blood pressure high and genetically hypoten

34 e 153 patients, 145 had prior infarct, 8 had hypertensive brain hemorrhage, and 164 admissions for PS

35 In summary, CSD in 2K1C rats reduces the hypertensive burden and improves renal function.

36 nerate both tonic drive and hyperreflexia in hypertensive (but not normotensive) rats, and both pheno

37 Forty-two patients (24%) were classified as hypertensive by ABPM criteria and 29 (17%) by clinic blo

38 Forty-five patients (26%) were classified as hypertensive by the daytime criterion (>135/85 mm Hg) an

39 emonstrated to have therapeutic potential in hypertensive cardiomyopathy when administered via osmoti

40 We imposed repeated hypertensive challenges using either N(omega)-nitro-L-ar

41 in sheep with normotension (control) or with hypertensive chronic kidney disease (CKD).

42 In hypertensive CKD sheep, RDN reduced blood pressure and i

43 es conducted in the general population or in hypertensive cohorts have shown that ambulatory BP provi

44 erosene users, 8.8% of control subjects were hypertensive compared with 1.8% of ethanol users (P = 0.

45 31), healthy control subjects (n = 51), and hypertensive control subjects (HTN) (n = 14), using CMR

46 A total of 22 hypertensive control subjects and 98 HFpEF subjects unde

47 d during exercise in subjects with HFpEF and hypertensive control subjects to examine their relations

48 -HTN; n = 17), normotensive (NC; n = 17) and hypertensive controls (HTN; n = 16).

49 tion were higher in amyloid patients than in hypertensive controls.

50 entiate between cardiac amyloid patients and hypertensive controls.

51 (2.16+/-0.69 in HFpEF versus 2.54+/-0.80 in hypertensive controls; P<0.02 and 2.89+/-0.70 in normote

52 analyzed, including 47 with the diagnosis of hypertensive crisis and 47 with normal blood pressure at

53 in the increased sensitivity of offspring to hypertensive damage.

54 n reuptake inhibitor is also associated with hypertensive disease of pregnancy and cesarean delivery.

55 ischemic heart disease (42% of CVD deaths), hypertensive diseases (27%), and cerebrovascular disease

56 n reuptake inhibitor use was associated with hypertensive diseases of pregnancy (OR, 2.82; 95% CI, 1.

57 eading cause in Ragh, and were mainly due to hypertensive diseases of pregnancy.

58 thers: preterm birth, cesarean delivery, and hypertensive diseases of pregnancy.

59 and 53 of 1000 additional women experienced hypertensive diseases of pregnancy.

60 pe 2 diabetes mellitus, atherosclerotic, and hypertensive diseases were also genetically correlated w

61 ibute to the burden of hospitalizations from hypertensive diseases, diabetes, and arrhythmia.

62 roke were 1.80 (95% CI, 1.49-2.18) after any hypertensive disorder in pregnancy (HDP) (gestational hy

63 A hypertensive disorder of pregnancy (severe or moderate p

64 Preeclampsia is a hypertensive disorder of pregnancy in which patients dev

65 entiation can result in preeclampsia (PE), a hypertensive disorder of pregnancy with significant morb

66 Preeclampsia (PE), a serious hypertensive disorder of pregnancy, remains a leading ca

67 eriod, 76,108 of which were complicated by a hypertensive disorder of pregnancy.

68 s of maternal blood pressure development and hypertensive disorders during pregnancy with microvascul

69 is commonly used to direct the management of hypertensive disorders in medical patients, but its appl

70 in part, why Asian women are at low risk of hypertensive disorders in pregnancy.

71 Gestational hypertensive disorders may lead to vascular changes in t

72 k of gestational diabetes mellitus (GDM) and hypertensive disorders of pregnancy (HDP) remains unclea

73 whose first pregnancy was not complicated by hypertensive disorders of pregnancy (HR, 2.01; 95% CI, 1

74 cts are associated with an increased risk of hypertensive disorders of pregnancy and whether the mech

75 Whether hypertensive disorders of pregnancy are also associated

76 [95% CI, 7.3-8.2]), women with a history of hypertensive disorders of pregnancy had significantly in

77 pathy in women with and without a history of hypertensive disorders of pregnancy in a cohort of 1,075

78 ng fetuses with heart defects and women with hypertensive disorders of pregnancy often exhibit angiog

79 spring congenital heart defects and maternal hypertensive disorders of pregnancy overall and for spec

80 Women with a history of hypertensive disorders of pregnancy, compared with women

81 Women with hypertensive disorders of pregnancy, preeclampsia in par

82 y events occurred in women with a history of hypertensive disorders of pregnancy.

83 Children of mothers with gestational hypertensive disorders tended to have narrower retinal a

84 fferent periods of pregnancy and gestational hypertensive disorders.

85 on the heart We analysed the effect of a non-hypertensive dose of leptin on cardiac function, [Ca(2+)

86 Although several anti-hypertensive drugs have been developed as AT(1)R blocker

87 ypertensive injuries without influencing the hypertensive effect of angiotensin II.

88 The hypertensive effects of cediranib were unaffected by los

89 The hypertensive effects of cediranib were unaffected by los

90 for the treatment of ocular GVHD without the hypertensive effects of topical corticosteroids.

91 pathy syndrome (PRES), also called the acute hypertensive encephalopathy and reversible posterior leu

92 can reduce the severity of BP elevation and hypertensive end-organ damage in several animal models.

93 nces in immunity and hypertension as well as hypertensive end-organ damage.

94 6 cohorts; n=4232 fistulas), and 0.03 venous hypertensive events (1 cohort; n=350 fistulas).

95 istinguish the role of passive stiffness and hypertensive exercise response with impaired active rela

96 Nighttime IOPs were higher in ocular hypertensive eyes compared to daytime IOPs.

97 In ocular hypertensive eyes, 28 of the 79 eyes (35.4%) classified

98 e expression differences were observed in AA hypertensive females compared to AA normotensives or whi

99 lear cells from AA or white, normotensive or hypertensive females identified thousands of mRNAs diffe

100 While many features of human hypertensive glaucoma are replicated in this model, stru

101 ciated with postpartum hypertension (de novo hypertensive group: odds ratio, 2.25; 95% confidence int

102 terval, 1.19-4.25; P=0.01; in the persistent hypertensive group: odds ratio, 2.61; 95% confidence int

103 ed in isolated myocardium from patients with hypertensive heart disease (HHD) and heart failure with

104 These effects surpass those of hypertensive heart disease alone, supporting a direct ro

105 ased, whereas the proportion associated with hypertensive heart disease and idiopathic fibrosis has i

106 nced higher mortality rates for ischemic and hypertensive heart disease compared with other subgroups

107 Proportion of SCDs associated with hypertensive heart disease with left ventricular hypertr

108 isease (371266 coronary heart disease, 35019 hypertensive heart disease, and 99815 other cardiovascul

109 isease, ischemic stroke, hemorrhagic stroke, hypertensive heart disease, cardiomyopathy, atrial fibri

110 a predominance of nonatherosclerotic stroke, hypertensive heart disease, nonischemic and Chagas cardi

111 ifferentiate between cardiac amyloidosis and hypertensive heart disease.

112 ommunicable disease such as hypertension and hypertensive heart disease.

113 5 amyloid transthyretin, and 4 control with hypertensive heart disease.

114 .5 vs 5.1 deaths per 100000 persons) to 4.2 (hypertensive heart disease: 4.3 vs 17.9 deaths per 10000

115 oups of participants across the continuum of hypertensive heart diseases.

116 in obese diabetic Zucker fatty/Spontaneously hypertensive heart failure F1 hybrid (ZSF1) rats.

117 In early-stage hypertensive HFpEF, LA cardiomyocyte hypertrophy, titin

118 e mutations have been found predominantly in hypertensive human subjects.

119 nd physiological studies in normotensive and hypertensive humans (n=259).

120 vasoconstriction in arterioles from mice and hypertensive humans, an effect dependent on smooth muscl

121 METHODS AND A total of 8796 hypertensive individuals >/=18 years of age were identif

122 tive-controlled clinical trial that enrolled hypertensive individuals >/=55 years of age with at leas

123 ancestry and hypertension (P = 0.037), with hypertensive individuals experiencing a greater increase

124 otassium on serum potassium concentration in hypertensive individuals with normal renal function trea

125 thologies of inflammatory, hypoxic, ischemic/hypertensive, infectious and thrombotic etiologies were

126 s a global protective role during glomerular hypertensive injuries without influencing the hypertensi

127 atory phenotypes, protecting the kidney from hypertensive injury and fibrosis.

128 corticosterone acetate salt)-induced chronic hypertensive kidney disease mouse models.

129 s associated with death and renal failure in hypertensive kidney disease, even among those without ac

130 ays an important role in the pathogenesis of hypertensive kidney disease.

131 litate a potential noninvasive detection for hypertensive kidney injury.

132 oRNA (miRNA) as a non-invasive biomarker for hypertensive kidney injury.

133 glomerular damage and during AngII-mediated hypertensive kidney injury.

134 Hypertensive left ventricular hypertrophy (HTN-LVH) is a

135 opposite of those observed in AngII-induced hypertensive male mice and may be ascribed in part to b

136 opposite of those observed in AngII-induced hypertensive male mice and may be ascribed in part to ba

137 ations will respond to certain types of anti-hypertensive medication.

138 Adoptively transferred T(EM) cells from hypertensive mice homed to the bone marrow and spleen an

139 ast, adoptive transfer of wild-type MDSCs to hypertensive mice reduced blood pressure, whereas the tr

140 iRNA expressions in the kidneys and urine of hypertensive mice with kidney injury induced by deoxycor

141 ere more abundant in colonized and in portal hypertensive mice, as compared to GF and sham-operated m

142 in control and AngII infused (490 ng/kg/min) hypertensive mice.

143 f C1 neurons during specific behaviors or in hypertensive models.

144 ery (PCA) occlusion in old, atherosclerotic, hypertensive monkeys to that in young monkeys.

145 hesus monkeys and 8 of old, atherosclerotic, hypertensive monkeys.

146 young healthy and the old, atherosclerotic, hypertensive monkeys.

147 actor complex, during angiotensin II-induced hypertensive nephropathy provided novel insights into FS

148 ts (GON and normal visual field), and ocular hypertensives (normal disc, normal visual field, and int

149 ve free (PF) Timolol 0.1% eyedrops in ocular hypertensive (OH) and in primary open-angle glaucoma (PO

150 ne and BP measures, and were not known to be hypertensive or have BP>140/90 mmHg.

151 nsion who are normotensive in the clinic but hypertensive outside.

152 Compared with normotensive controls, hypertensive participants demonstrated increased late/ea

153 ults with BMI<24 while significant among non-hypertensive participants, but not in their counterparts

154 140 image pairs from 100 glaucomatous/ocular hypertensive patient eyes using a handheld stereo viewer

155 ing self-monitoring to no self-monitoring in hypertensive patients (June 2016).

156 Interestingly, untreated hypertensive patients (n=20) had a cerebral blood flow s

157 and sex-matched RA-normotensive (n = 13), RA-hypertensive patients (RA-HTN; n = 17), normotensive (NC

158 y of the RI was examined in 256 asymptomatic hypertensive patients and 10 patients with heart failure

159 METHODS AND Cohort study included 92 hypertensive patients and 27 providers in 3 safety-net p

160 nce of this ADAM17-mediated ACE2 shedding in hypertensive patients and further identify the cell type

161 that RI provides an approach for stratifying hypertensive patients and is suitable for testing in oth

162 ge randomized trials that randomly allocated hypertensive patients at high risk for cardiovascular di

163 Moreover, human hypertensive patients have demonstrated increased suscep

164 content, we analyzed samples from 14 mildly hypertensive patients in a crossover study during a high

165 dent predictor of cardiovascular outcomes in hypertensive patients including myocardial infarction, f

166 We randomized 126 hypertensive patients on hemodialysis to a standardized

167 Thirteen pre- and stage-1 hypertensive patients received a single dose of 30 mL of

168 characterized cohort of untreated borderline hypertensive patients suggested that ARHGAP42 genotype h

169 Hypertensive patients underwent multimodal cardiac asses

170 use of amiloride plus hydrochlorothiazide in hypertensive patients who need treatment with a diuretic

171 4 high-quality trials involving 10,857 older hypertensive patients with a mean follow-up of 3.1 years

172 ive myocardial CCL is associated with HHF in hypertensive patients with HF.

173 Hypertensive patients with hyperaldosteronism or normal

174 between the ADC value and systolic strain in hypertensive patients with left ventricular hypertrophy

175 culating biomarkers of heart failure (HF) in hypertensive patients with normal resting echocardiograp

176 h left ventricular hypertrophy (HTN LVH) and hypertensive patients without LVH (HTN non-LVH) using ca

177 Compared with hypertensive patients without LVH (n=191; 74.6%) and tho

178 en 1966 and end 2013 in cohorts with >/= 40% hypertensive patients, and exclusive of trials in acute

179 frican American patients, Hispanic patients, hypertensive patients, and those with hypertrophy.

180 bleeds which predicts cerebral hemorrhage in hypertensive patients, as well as progression to hyperte

181 In older hypertensive patients, intensive BP control (systolic BP

182 DAM17-mediated ACE2 shedding in the brain of hypertensive patients, leading to a loss in compensatory

183 y be considered for primary AF prevention in hypertensive patients, previous studies have yielded con

184 ACE2 activity in the cerebrospinal fluid of hypertensive patients, which was correlated with systoli

185 that ADAM17 was upregulated in the brain of hypertensive patients.

186 on and tested its potential to risk-stratify hypertensive patients.

187 ion with a prevalence of 5-10% in unreferred hypertensive patients.

188 nvasive central BP monitoring in elderly and hypertensive patients.

189 l be effective in reducing blood pressure in hypertensive patients.

190 ary care providers and their black and white hypertensive patients.

191 oves peripheral insulin sensitivity in obese hypertensive patients.

192 wering strategies in older (age >/=65 years) hypertensive patients.

193 Mice lacking RGS2 display a hypertensive phenotype, and several RGS2 missense mutati

194 shGRK2 mice prior to the development of the hypertensive phenotype.

195 atment options, a substantial portion of the hypertensive population has uncontrolled blood pressure.

196 of secondary hypertension, among the general hypertensive population the true prevalence of primary a

197 nd control from each country were applied to hypertensive populations to obtain regional and global e

198 n potassium retention and in both normal and hypertensive populations.

199 The long-term effects of hypertensive pregnancies on brain structure and cognitiv

200 e critical insight as to the contribution of hypertensive pregnancies to risk of cognitive decline an

201 nd microvascular differences in offspring of hypertensive pregnancies, and there is interest in wheth

202 A history of a hypertensive pregnancy disorder was obtained by a self-r

203 were used to assess the association between hypertensive pregnancy disorders and cognition, adjustin

204 Hypertensive pregnancy disorders are associated with wor

205 Hypertensive pregnancy disorders have been associated wi

206 Women with histories of hypertensive pregnancy disorders performed worse on all

207 sposition or persistent damage caused by the hypertensive pregnancy.

208 white-matter lesions 5 to 10 years after the hypertensive pregnancy.

209 ovided therapeutic benefit in a rat model of hypertensive proteinuric kidney disease.

210 dventitial fibroblasts from human and bovine hypertensive pulmonary arterial walls (PH-Fibs) that exh

211 These hypertensive QTL alleles were then merged to systematica

212 he prevalence of children with BP within the hypertensive range (>90th percentile) were compared betw

213 reduced the odds of having an SBP within the hypertensive range at 7 y of age (OR: 0.32; 95% CI: 0.11

214 in genetic hypertension in the spontaneously hypertensive rat (SHR) has not been investigated.

215 Previous studies in the spontaneously hypertensive rat (SHR) support a role for leukotriene B4

216 Previous studies in the spontaneously hypertensive rat (SHR) support a role for leukotriene B4

217 all pathology were examined in spontaneously hypertensive rat and chronic angiotensin II infusion rat

218 all pathology were examined in spontaneously hypertensive rat and chronic angiotensin II infusion rat

219 ells are dominant in the (SHR) spontaneously hypertensive rat and expand in response to nicotinic cho

220 At advanced stages of FSGS, fawn-hooded hypertensive rat kidneys exhibited distinctly increased

221 We have used a spontaneously hypertensive rat model to investigate the antihypertensi

222 increase in blood pressure in spontaneously hypertensive rat was associated with gut pathology that

223 lity of 5- to 6-month-old male spontaneously hypertensive rats (SHR) and age/sex-matched normotensive

224 Spontaneously hypertensive rats (SHR) and normotensive Wistar-Kyoto ra

225 Spontaneously hypertensive rats (SHR) are the most widely used animal

226 Losartan, and placebo- treated Spontaneously Hypertensive Rats (SHR) by both noninvasive and invasive

227 to suppress blood pressure in spontaneously hypertensive rats (SHR).

228 rve effects of BSJYD on LVH in spontaneously hypertensive rats (SHRs) and explore its possible mechan

229 d from the stellate ganglia of spontaneously hypertensive rats (SHRs) and their normotensive controls

230 In spontaneously hypertensive rats (SHRs), high ABP is associated with en

231 ese effects are exaggerated in spontaneously hypertensive rats (SHRs), resulting in an augmented CO2

232 presympathetic neurons in male spontaneously hypertensive rats (SHRs).

233 In hypertensive rats and old mice, gene transfer of longevi

234 oduce greater vascular resistance changes in hypertensive rats because the system is saturated as a c

235 l function in the paraventricular nucleus of hypertensive rats by promoting mitochondrial biogenesis

236 (ii) Spontaneously hypertensive rats have more orexin neurons and more CO2

237 Spontaneously hypertensive rats received either bilateral RF-RDN or sh

238 optic nucleus and paraventricular nucleus of hypertensive rats that contributes to neurohumoral activ

239 or sham-RDN (n=14) treatment, spontaneously hypertensive rats were subjected to 30 minutes of transi

240 We found that chronic treatment of hypertensive rats with pomegranate extract significantly

241 e cerebral artery occlusion in spontaneously hypertensive rats, a strain representative of the transi

242 ar in magnitude between normotensive and CIH hypertensive rats, but basal arterial pressure in CIH ra

243 terial pressure in both normotensive and CIH hypertensive rats, but they are not involved in the enha

244 flammation in the paraventricular nucleus of hypertensive rats.

245 not change hemodynamic parameters in portal hypertensive rats.

246 gulated compared with sham-RDN spontaneously hypertensive rats.

247 e and increased firing activity of MNCs from hypertensive rats.

248 nerve discharges in normotensive, but not in hypertensive, rats.

249 active LV relaxation (p = 0.01) and a marked hypertensive reaction to exercise due to pathologic arte

250 CI: 0.90, 1.19) if they were normotensive or hypertensive, respectively (P-interaction = 0.003).

251 ad 46% and 52% higher odds of being obese or hypertensive, respectively.

252 Patients with hypertensive response (n=1905; 9%) were more likely to h

253 =70% stenosis) was lower in patients who had hypertensive response compared with those who had normal

254 Patients with hypertensive response during DSE are more likely to have

255 females have lower BP before age 60, blunted hypertensive response to angiotensin II, and a leftward

256 ble effects on renal sodium handling and the hypertensive response, accruing from the functions of th

257 Micu2 in regulating angiotensin II-mediated hypertensive responses that are critical in protecting t

258 A pro-hypertensive role for 20-HETE was implicated by normaliz

259 rd currents in MNCs of sham and renovascular hypertensive (RVH) rats.

260 neurosecretory cells (MNCs) in renovascular hypertensive (RVH) rats.

261 al-time PCR in MNCs in sham and renovascular hypertensive (RVH) rats.

262 from sham, but not in MNCs from renovascular hypertensive (RVH) rats.

263 ne users), and 6.4% of control subjects were hypertensive (SBP >/=140 and/or DBP >/=90 mm Hg) versus

264 lood pressure (Normotensive-SBP <140 mmHg or hypertensive-SBP >/= 140 mmHg).

265 also noted in prehypertensive spontaneously hypertensive (SH) rats, which are known to exhibit CB hy

266 to be hyper-responsive in both spontaneously hypertensive (SHR) and Goldblatt hypertensive (two kidne

267 c stellate neurons from normal (WKY) and pro-hypertensive (SHR) rats that are sympathetically hyper-r

268 ed aortic and mesenteric vasoconstriction in hypertensive Sphk1(-/-) mice.

269 Apparent Mineralocorticoid Excess and other hypertensive states is often overlooked and is unresolve

270 matory cytokine and its level is elevated in hypertensive states.

271 n women, and did not differ significantly by hypertensive status.

272 amage that occur on delayed exposure to mild hypertensive stimuli.

273 higher levels of all 10 CV autoantibodies in hypertensive subjects (n = 77) compared with healthy par

274 be used to evaluate autoantibody profiles in hypertensive subjects at risk for heart failure.

275 tary potassium over a 4-wk period is safe in hypertensive subjects who have normal renal function and

276 beta 1-adrenegic receptor best discriminated hypertensive subjects with adverse left ventricular (LV)

277 (LV) remodeling or dysfunction (n = 49) from hypertensive subjects with normal LV structure and funct

278 microarray study, including normotensive and hypertensive subjects, were used to demonstrate an assoc

279 ing the early stages of heart failure and in hypertensive subjects.

280 s well as progression to hypertension in non-hypertensive subjects.

281 h to further lower blood pressure in treated hypertensive subjects.

282 help target treatment strategies in specific hypertensive subpopulations.

283 Interventions to prevent repeated hypertensive surges could attenuate formation of hyperte

284 The hypertensive syndrome of Apparent Mineralocorticoid Exce

285 rmation and context on the intensity of anti-hypertensive therapy in conjunction with the release of

286 nd efficacy of differing intensities of anti-hypertensive therapy in mild to moderate CKD, where SPRI

287 participants recruited from the Diabetes and Hypertensive Treatment Center, Feira de Santana, Bahia,

288 AC) can further guide the allocation of anti-hypertensive treatment intensity.

289 ontaneously hypertensive (SHR) and Goldblatt hypertensive (two kidney one clip; 2K1C) rats.

290 The underlying pulmonary hypertensive vascular disease (PHVD) is characterized by

291 rotein and activity were up-regulated in the hypertensive vasculature.

292 al amyloid angiopathy (lobar structures) and hypertensive vasculopathy (deep brain structures).

293 The anti-hypertensive, vasodilatory, anti-fibrotic, and anti-hype

294 a critical adaptation of fibroblasts in the hypertensive vessel wall that drives proliferative and p

295 comatous eyes, glaucoma suspects, and ocular hypertensives with 24-2 and 10-2 visual fields.

296 AND Genome-wide analysis included 228 white hypertensives with BP determined at baseline and after 9

297 ckness (60.1+/-14.8 years, 85% male), and 20 hypertensives with prominent myocardial remodeling.

298 tion of CCB or ACEi use and breast cancer in hypertensive women aged >/=55 years at 3 sites in the Ka

299 ontrols were frequency-matched 2:1 to 12,513 hypertensive women by age and questionnaire cycle.

300 diuretics (TD) are commonly prescribed anti-hypertensives worldwide.