ライフサイエンスコーパス: hyperthyroidism

1 ct suppressed levels of TSH (<0.4 mIU L(-1); hyperthyroidism).

2 requent cause is Graves' disease (autoimmune hyperthyroidism).

3 to the fetus and can cause fetal or neonatal hyperthyroidism.

4 erican patients treated with radioiodine for hyperthyroidism.

5 hyroidism, and 1.5% (n = 47) had subclinical hyperthyroidism.

6 dermatitis, vitiligo, panhypopituitarism and hyperthyroidism.

7 roid adenomas (ATAs) are a frequent cause of hyperthyroidism.

8 to the thyrotropin receptor (TSHR) and cause hyperthyroidism.

9 nal treatments for the three common forms of hyperthyroidism.

10 ity in patients treated with radioiodine for hyperthyroidism.

11 ceptor and carbimazole-responsive autoimmune hyperthyroidism.

12 ay activated in cancer cachexia, sepsis, and hyperthyroidism.

13 tios (SMRs) after 3 treatment modalities for hyperthyroidism.

14 all, (131)I appears to be a safe therapy for hyperthyroidism.

15 3 yr developed enlargement of the nodule and hyperthyroidism.

16 mune hyperthyroidism and sporadic congenital hyperthyroidism.

17 itoring or offered alternative treatment for hyperthyroidism.

18 o the elevated serum ferritin levels seen in hyperthyroidism.

19 ute to 131I therapy failure in patients with hyperthyroidism.

20 nctions such as Graves disease and hypo- and hyperthyroidism.

21 and surgical interventions for management of hyperthyroidism.

22 argets for therapeutic treatment of hypo- or hyperthyroidism.

23 ve been reported in elderly individuals with hyperthyroidism.

24 vity, and the occurrence of tissue damage in hyperthyroidism.

25 cy, neonatal thyrotoxicosis, and post-partum hyperthyroidism.

26 IU L(-1) indicating clinical significance of hyperthyroidism.

27 is the preferred choice for relapsed Graves' hyperthyroidism.

28 ypothyroidism and 648 (2.6%) had subclinical hyperthyroidism.

29 and resolve with treatment of the underlying hyperthyroidism.

30 Atrial fibrillation frequently complicates hyperthyroidism.

31 ce would facilitate AF in autoimmune Graves' hyperthyroidism.

32 nesis of atrial fibrillation (AF) in Graves' hyperthyroidism.

33 osensitivity (25%-75%), hypothyroidism (6%), hyperthyroidism (0.9%-2%), pulmonary toxicity (1%-17%),

34 ism (1), therapeutic abortion (1), worsening hyperthyroidism (1), congestive heart failure (1), and w

35 on (1.43, 1.21-1.69; p<0.0001), but not with hyperthyroidism (1.22, 0.96-1.55; p=0.1010) or raised ad

36 3,512 singleton deliveries of which 0.2% had hyperthyroidism, 1.4% primary and 0.1% iatrogenic hypoth

37 se events were hypothyroidism (25 [16%]) and hyperthyroidism (17 [11%]).

38 Graves' hyperthyroidism, a common autoimmune disease caused by p

39 Treatment of hyperthyroidism, a common clinical condition that can ha

40 Hyperthyroidism, a condition associated with excess TH,

41 When present in patients with Graves' hyperthyroidism, AAbeta1AR and AAM2R facilitate developm

42 Graves' disease is the leading cause of hyperthyroidism affecting 1.0-1.6% of the population.

43 hyroidectomy 32 yr ago developed accelerated hyperthyroidism after injection of iodinated contrast me

44 tions with antibody-positive hypothyroidism, hyperthyroidism, AIT, or elevated ATPO.

45 ganochlorines and risk of hypothyroidism and hyperthyroidism among female spouses (n = 16,529) in Iow

46 pairment, diabetes, obesity, hypothyroidism, hyperthyroidism, anaemia, respiratory disease, liver dis

47 data show an association between subclinical hyperthyroidism and development of atrial fibrillation b

48 Hashimoto's thyroiditis are more common than hyperthyroidism and Graves' disease (strong evidence).

49 Hyperthyroidism and hypothyroidism often cause opposing

50 nd blocking Abs in the sera of patients with hyperthyroidism and hypothyroidism, respectively.

51 ically relevant findings that accompany both hyperthyroidism and hypothyroidism.

52 ctive strategies exist for the management of hyperthyroidism and hypothyroidism; these should be tail

53 Hyperthyroidism and primary hypothyroidism were associat

54 ce or amplify the immune response leading to hyperthyroidism and provide new insight into the etiolog

55 ns have been found in familial nonautoimmune hyperthyroidism and sporadic congenital hyperthyroidism.

56 Subclinical thyroid diseases--subclinical hyperthyroidism and subclinical hypothyroidism--are comm

57 alth outcomes, and management of subclinical hyperthyroidism and subclinical hypothyroidism.

58 mmon abnormality with other abnormalities of hyperthyroidism and thyroiditis.

59 Overall, hyperthyroidism and use of thyroid hormone to suppress T

60 an increased odds ratio for hypothyroidism, hyperthyroidism, and antithyroid antibodies.

61 common endocrine diseases, type 1 diabetes, hyperthyroidism, and hypothyroidism, are the result of a

62 , childbirth or breast feeding, a history of hyperthyroidism, and progestin use were not associated w

63 any catabolic states (including denervation, hyperthyroidism, and sepsis) is due to a proteasome-depe

64 of the signs and symptoms of hypothyroidism, hyperthyroidism, and thyroid nodules, as well as the gen

65 diabetes mellitus, hyperparathyroidism, and hyperthyroidism are considered within the context of bot

66 Subclinical hypothyroidism and hyperthyroidism are diagnoses based on laboratory evalua

67 low serum TSH levels, indicating physiologic hyperthyroidism, are at increased risk for new hip and v

68 as TSH of 4.5 to 19.9 mIU/L, and subclinical hyperthyroidism as TSH <0.45 mIU/L, the last two with no

69 of untreated subclinical hypothyroidism and hyperthyroidism, as well as the benefit of initiating tr

70 As a consequence, both hypothyroidism and hyperthyroidism associate with clinically important alte

71 cross-sectional analysis of hypothyroidism, hyperthyroidism, autoimmune thyroiditis (AIT), serum con

72 isease is an autoimmune disorder that causes hyperthyroidism because of autoantibodies that bind to t

73 y studies evaluated treatment of subclinical hyperthyroidism but examined intermediate outcomes.

74 sed increasingly as first-line treatment for hyperthyroidism, but concerns remain about subsequent ri

75 The risk of AF is increased in subclinical hyperthyroidism, but it is uncertain whether variations

76 injecting TSHR A-subunit protein attenuated hyperthyroidism by diverting pathogenic TSHR Abs to a no

77 Fetal hyperthyroidism can be life-threatening, and needs to be

78 Hyperthyroidism causes increased energy intake and expen

79 crease in basal metabolic rate is not due to hyperthyroidism, compensation by the widely expressed un

80 o-moderate intensity, including two cases of hyperthyroidism consistent with autoimmune thyroiditis.

81 elevations due to low TSH signaling in human hyperthyroidism contribute to the bone loss that has tra

82 Acute hyperthyroidism did not affect graft function, but acute

83 Hyperthyroidism did not significantly alter MDA-protein

84 classic clinical triad of Graves' disease is hyperthyroidism, diffuse goiter, and exophthalmos.

85 main, found in a male infant with congenital hyperthyroidism due to a toxic adenoma.

86 Hyperthyroidism due to thyroid-stimulating hormone-secre

87 Other important causes include toxic nodular hyperthyroidism, due to the presence of one or more auto

88 On the other hand, hyperthyroidism during pregnancy has been associated wit

89 n pregnancy, the diagnosis and management of hyperthyroidism during pregnancy, severe life-threatenin

90 ore prevalent in children born to women with hyperthyroidism during pregnancy, suggesting a role for

91 sm in pregnancy can be caused by any type of hyperthyroidism--eg, toxic multinodular goitre or solita

92 o thyroid function test results: subclinical hyperthyroidism, euthyroidism, subclinical hypothyroidis

93 enzyme activity are elevated in the liver in hyperthyroidism, fasting, and diabetes.

94 healthy and sick cats diagnosed with Feline Hyperthyroidism (FH).

95 therapy is a routine procedure of treatment hyperthyroidism for over 50 years.

96 The other patient manifested hyperthyroidism from stimulation of the tumors by thyroi

97 ifferences were seen between the subclinical hyperthyroidism group and euthyroidism group for inciden

98 l fibrillation, individuals with subclinical hyperthyroidism had a greater incidence of atrial fibril

99 The osteoporosis associated with human hyperthyroidism has traditionally been attributed to ele

100 or recurrent thyroid cancer, or therapy for hyperthyroidism have been calculated and summarized in t

101 No trials of treatment of subclinical hyperthyroidism have been done.

102 on bone and suggested that the bone loss in hyperthyroidism, hitherto attributed solely to elevated

103 hazard ratio [HR], 6.6; 95% CI, 5.6 to 7.8), hyperthyroidism (HR, 1.8; 95% CI, 1.2 to 2.8), thyroid n

104 s in a further increase in the prevalence of hyperthyroidism if iodine intake is subsequently increas

105 Compared to euthyroid rats, hyperthyroidism in 4-month-old rats was associated with

106 These mice showed severe hyperthyroidism in a manner very similar to that describ

107 Identification of hyperthyroidism in a pregnant woman is important because

108 olonged course leads to remission of Graves' hyperthyroidism in about a third of cases.

109 es that 131I treatment for thyroid cancer or hyperthyroidism in adult women confers negligible risk o

110 cal and undiagnosed overt hypothyroidism and hyperthyroidism in adults without goiter or thyroid nodu

111 sease is the most common cause of persistent hyperthyroidism in adults.

112 thyrotropin receptor Abs are responsible for hyperthyroidism in GD.

113 HR) (TSAbs) that induce a sustained state of hyperthyroidism in patients.

114 n the prevalence of toxic nodular goitre and hyperthyroidism in populations.

115 Thyroid storm and hyperthyroidism in pregnancy and during the post-partum

116 utoimmune in nature, is the usual cause; but hyperthyroidism in pregnancy can be caused by any type o

117 Antithyroid drug treatment of hyperthyroidism in pregnant women is controversial becau

118 The more unusual causes of hyperthyroidism, including struma ovarii, thyrotropin-se

119 Although it is established that hyperthyroidism increases AF incidence, the effect of hy

120 Hyperthyroidism increases heart rate, contractility, car

121 Specifically, hyperthyroidism increases the ex vivo activity of pyruva

122 Hyperthyroidism induced by injection of normal rats with

123 Hyperthyroidism induced by T3 treatment caused up-regula

124 Hyperthyroidism is a pathological syndrome in which tiss

125 Hyperthyroidism is associated with increased metabolic r

126 Prolonged hyperthyroidism is best avoided.

127 Hyperthyroidism is characterised by increased thyroid ho

128 normal serum FT4 concentration; subclinical hyperthyroidism is defined as a decrease in serum TSH be

129 The diagnosis of hyperthyroidism is generally straightforward, with raise

130 The most common cause of hyperthyroidism is Graves' disease, followed by toxic no

131 Because Graves' hyperthyroidism is preferentially induced in BALB/c mice

132 The osteoporosis associated with hyperthyroidism is traditionally viewed as a secondary c

133 Both hypothyroidism and hyperthyroidism lead to increased AF vulnerability in a

134 Biochemical evidence of hyperthyroidism may be associated with low bone mass, pa

135 or recurrent thyroid cancer, or therapy for hyperthyroidism may be treated with 7400 MBq (200 mCi) 1

136 At the other end of the spectrum, hyperthyroidism may result from gain of function mutatio

137 arly step in a pathologic cascade leading to hyperthyroidism, metabolic bone disease, vascular calcif

138 Subclinical hyperthyroidism might be associated with an increased ri

139 ne concentrations that are characteristic of hyperthyroidism must be distinguished from physiological

140 Neither hyperthyroidism nor (131)I treatment resulted in a signi

141 pproximately one to two cases of gestational hyperthyroidism occur per 1000 pregnancies.

142 el of Graves disease to show that goiter and hyperthyroidism occur to a much greater extent when the

143 rmone levels, ranging from hypothyroidism to hyperthyroidism on AF inducibility in thyroidectomized r

144 assess the long-term effects of subclinical hyperthyroidism on mortality.

145 tropin receptor (TSHR), the cause of Graves' hyperthyroidism, only develop in humans.

146 the hypothesis that unrecognized subclinical hyperthyroidism or subclinical hypothyroidism is associa

147 ion of the receptor, resulting in congenital hyperthyroidism or the development of actively secreting

148 Women with a history of hyperthyroidism or TSH suppression by thyroid hormone sh

149 was the only pesticide associated with both hyperthyroidism (OR(adj) = 2.3 (95% CI: 1.2, 4.4) and hy

150 ardiomyopathy, significant valvular disease, hyperthyroidism, or hypertension that preceded the onset

151 bunit adenovirus failed to develop TSHR Abs, hyperthyroidism, or splenocyte responses to TSHR Ag.

152 Hypothyroidism and hyperthyroidism produced opposite electrophysiological c

153 Of the covariates, only hyperthyroidism prolonged by at least 2.5 mo was signifi

154 uated interactions of 6 covariates-prolonged hyperthyroidism, prolonged hypothyroidism, smoking, trea

155 apnea, obesity, excessive alcohol, smoking, hyperthyroidism, pulmonary disease, air pollution, and p

156 apnea, obesity, excessive alcohol, smoking, hyperthyroidism, pulmonary disease, air pollution, heart

157 en in 4 patients (grade 3 arthritis, grade 2 hyperthyroidism, recurrent grade 4 pneumonitis).

158 Appropriate treatment of hyperthyroidism relies on identification of the underlyi

159 affects 50% to 60% of patients with Graves' hyperthyroidism, resulting in exophthalmos, periorbital

160 fter adjustment for age, history of previous hyperthyroidism, self-rated health, and use of estrogen

161 Individuals with hypo- or hyperthyroidism showed various changes in electrocardiog

162 TSH level may contribute to the bone loss of hyperthyroidism that has been attributed traditionally t

163 diabetes mellitus, hyperparathyroidism, and hyperthyroidism), they should be considered in the diffe

164 patients with subclinical hypothyroidism and hyperthyroidism through 5 case scenarios that apply the

165 ls compared risks of primary hypothyroidism, hyperthyroidism, thyroid neoplasms, hypopituitarism, obe

166 assessment, and treatment of hypothyroidism, hyperthyroidism, thyroid nodules, and thyroid cancer in

167 In an important study of Graves hyperthyroidism, treatment with radioactive iodine was m

168 Rates of new onset hyperthyroidism (valproate HR 0.24; 95% CI 0.09-0.61; p

169 atios (HRs) of participants with subclinical hyperthyroidism versus euthyrodism were 1.38 (95% CI, 0.

170 oping postthyroidectomy hypocalcemia include hyperthyroidism, vitamin D deficiency, female sex, subst

171 12.5%, and prevalence of hypothyroidism and hyperthyroidism was 6.9% and 2.1%, respectively.

172 Hyperthyroidism was also associated with rare outcomes (

173 After adjustment for TSH level, a history of hyperthyroidism was associated with a twofold increase i

174 It was assumed the hyperthyroidism was caused by evolving toxicity in the a

175 et containing 0.15% propyl-2-thiouracil, and hyperthyroidism was generated by addition of L-thyroxine

176 Hyperthyroidism was induced by daily injection of l-3,5,

177 Hyperthyroidism was induced in 18 male Wistar rats with

178 A history of hyperthyroidism was not significantly related to HCC (OR

179 the nodule had undergone infarction and the hyperthyroidism was secondary to Graves' disease.

180 ergy homeostasis in response to experimental hyperthyroidism, we administered 200 mug tri-iodothyroni

181 er low TSH levels contribute to bone loss in hyperthyroidism, we compared the skeletal phenotypes of

182 d, the HRs for participants with subclinical hyperthyroidism were 2.16 (CI, 0.87 to 5.37) for hip fra

183 th subclinical hypothyroidism or subclinical hyperthyroidism were also included (n=23).

184 atrial fibrillation and progression to overt hyperthyroidism were rated as good, but no data supporte

185 lin G purified from 38 patients with Graves' hyperthyroidism with AF (n=17) or sinus rhythm (n=21) an

186 ease and the inability to cure toxic nodular hyperthyroidism with antithyroid drugs alone, radioiodin

187 incidence and mortality in those treated for hyperthyroidism with radioiodine is reassuring.

188 en reported after treatment of patients with hyperthyroidism with radioiodine.

189 These mice developed severe hyperthyroidism with significant elevations in both tetr

190 is the treatment of choice in most cases of hyperthyroidism, with a standard 7,000-cGy (rad) thyroid

191 Cardiac hypertrophy is another effect of hyperthyroidism, with an increase in the abundance of mi