ライフサイエンスコーパス: hypertonic solution

1 expressed hBest1 in that it was decreased by hypertonic solution.

2 to over 80% by superfusing the synapses with hypertonic solution.

3 but with the addition of sucrose to obtain a hypertonic solution.

4 m and were filled with either an isotonic or hypertonic solution.

5 rocytes exposed to an EtOH-free hyperosmotic/hypertonic solution.

6 by the immersion of the biological tissue in hypertonic solutions.

7 ial, lateral, and central-in physiologic and hypertonic solutions.

8 ar action on AP-independent EPSPs induced by hypertonic solutions.

9 in fibres maintained in and then fixed using hypertonic solutions.

10 ld be elicited by the action potential or by hypertonic solutions.

11 state model to synaptic responses induced by hypertonic solutions.

12 Intracellular perfusion with hypertonic solutions activated currents carried by anion

13 tal volume of crystalloid, and other colloid/hypertonic solutions administered (odds ratio, 1.8; 95%

14 er train stimulation and dual application of hypertonic solutions) also reveal no abnormalities.

15 This component was increased 33% by a 33% hypertonic solution and was decreased 38% by a 33% hypot

16 ial cells to volume regulate when exposed to hypertonic solutions, and furthermore to identify the io

17 mic acid (300 microM), DIDS (200 microM) and hypertonic solutions, and was virtually abolished in the

18 le to no detectable epithelial damage, while hypertonic solutions caused significant damage, includin

19 lular to the intracellular compartment while hypertonic solutions caused water to move from the intra

20 eral osmolarity or the presence of an apical hypertonic solution decreased the diffusional water perm

21 Hypotonic solutions increased and hypertonic solutions decreased IOP.

22 Hypertonic solutions did increase Lp, however, after we

23 We found that hypertonic solutions did not increase the hydraulic perm

24 Hypertonic solution (HS) was applied by local superfusio

25 rease in RRP employing local applications of hypertonic solutions (HS).

26 ster stimulation indeed affects responses to hypertonic solution in a supralinear manner.

27 nerve-muscle preparations were treated with hypertonic solution in which sodium gluconate was the ma

28 We also found that a hypertonic solution in which the anion was aspartate inc

29 - or 4-fold in preparations pre-treated in a hypertonic solution in which the anion was gluconate.

30 regulatory volume increase after exposure to hypertonic solutions in endothelial cells, did not chang

31 to increase microvessel Lp after exposure to hypertonic solutions in microvessels where EC-ECM attach

32 ydraulic permeability (Lp) after exposure to hypertonic solutions in single perfused mesenteric micro

33 tions abolished the increase in Lp caused by hypertonic solutions in the microvessels pretreated with

34 fter albumin was removed from the perfusate, hypertonic solutions increased Lp of microvessels and th

35 rfusate may reduce EC-ECM attachment because hypertonic solutions increased the Lp of microvessels ab

36 were rescued when the mutant was grown in a hypertonic solution, indicating that FTT_0924 is require

37 A 2400-mOsm/L hypertonic solution (isosal) with a lower sodium content

38 Because of the potential benefits of hypertonic solutions, it is hypothesized that hydroxyeth

39 aken together, these data suggest that these hypertonic solutions may prove useful as anti-inflammato

40 Finally, we found that treatment with a hypertonic solution mimicked the effect we observed with

41 The hypertonic solution produced a positive flux into the ch

42 After exposure to hypotonic and hypertonic solutions, reversible changes in cell height

43 erformed with filter-sterilized isotonic and hypertonic solutions that contained glucose or mannitol

44 nd a significant increase in the response to hypertonic solutions that measure the size of the readil

45 dulation: the larger range of enhancement by hypertonic solutions, the relative lack of effect of osm

46 control by T-tubular DHPR-voltage sensors in hypertonic solutions, thereby permitting CICR to operate

47 Cells shrank when superfused with hypertonic solutions to a minimum relative cell volume o

48 Despite the daily use of hypertonic solutions to remove fluid from patients throu

49 we measured microvessel Lp after exposure to hypertonic solutions under experimental conditions that