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1 motic stabilizer and compatible solute under hypertonic stress.
2 ase in AQP1 luciferase-driven activity under hypertonic stress.
3 lationship also occurs in kidney cells under hypertonic stress.
4 et unknown to be up-regulated in response to hypertonic stress.
5 response shared by all eukaryotes exposed to hypertonic stress.
6 ooperate to induce COX2 expression following hypertonic stress.
7  and NFkappaB was greatly enhanced following hypertonic stress.
8  osmolytes restored their ability to survive hypertonic stress.
9  only PGI2 enhanced RMIC viability following hypertonic stress.
10 died the recovery of human kidney cells from hypertonic stress.
11 r channel protein expression is increased by hypertonic stress.
12 n, but it blocked NRG release in response to hypertonic stress.
13 o increased levels of AQP1 expression during hypertonic stress.
14 etween medullary COX1 and COX2 expression in hypertonic stress.
15 G) pathway functions to sense and respond to hypertonic stress.
16 ctors, phorbol ester, calcium ionophore, and hypertonic stress.
17 found for the zinc-finger protein ZAC1 under hypertonic stress (219-fold, p < 0.001).
18 8-10 h following exposure to acute sublethal hypertonic stress (550 mosmol/kg H(2)O).
19                                 We find that hypertonic stress allows MEN mutants to exit from mitosi
20  activation of TNF gene transcription during hypertonic stress alone.
21                                              Hypertonic stress also increases system A activity as a
22 st that Nup88 is up-regulated in response to hypertonic stress and acts to retain TonEBP in the nucle
23                       p38 mRNA is induced by hypertonic stress and is attenuated with p38 kinase inhi
24 ngly suggest that ZAC1 is up-regulated under hypertonic stress and negatively regulates expression of
25 s study demonstrates that AQP5 is induced by hypertonic stress and that induction requires activation
26 ssive accumulation of betaine may counteract hypertonic stress and thus attenuate hypertonicity-induc
27 was selectively activated in MLE-15 cells by hypertonic stress, and inhibition of ERK activation with
28 inhibitors increased RMIC survival following hypertonic stress, and transduction of RMICs with a cons
29 mal growth conditions, but when subjected to hypertonic stress, become spheroidal in shape and growth
30 ter (P(gpdh-1)::GFP) is detected only during hypertonic stress but is not induced by other stressors.
31 at coordinates the intracellular response to hypertonic stress but was not previously implicated in t
32 HOG1 does prevent Clb2p-Cdc28p inhibition by hypertonic stress, but does not block Cdc28p phosphoryla
33 pithelium in the medulla of the kidney under hypertonic stress by correctly localizing Cldn4 to the t
34 urthermore, lens epithelial cells respond to hypertonic stress by raising taurine transport activity.
35 ys a key role in protecting renal cells from hypertonic stress by stimulating transcription of specif
36                                              Hypertonic stress caused mRNA levels, and primarily that
37                                              Hypertonic stress causes a decrease in CLB2 mRNA, phosph
38                  A novel technique involving hypertonic stress causes membrane 'blebbing' of the Xeno
39 issociation of mRNA stress granules (SGs) in hypertonic-stressed cells and the role of compatible osm
40 t induction of active p53 in mIMCD3 cells by hypertonic stress contributes to cell survival.
41 ion of the G(2) delay in IME cells after the hypertonic stress created by adding NaCl.
42                 In contrast, the response to hypertonic stress does not involve eukaryotic initiation
43                                   A 450 mOsm hypertonic stress elicited 2-fold Ca2+ transients that w
44                                              Hypertonic stress-elicited TRPV1 channel stimulation med
45 ia rapidly induced c-fos expression, whereas hypertonic stress had no effect.
46                                              Hypertonic stress (HS) can alter the function of mammali
47 ntribute to proper nuclear segregation after hypertonic stress in cells that lack Swe1p.
48 responsive enhancer, which was refractory to hypertonic stress in fibroblasts lacking NFAT5, establis
49          We showed that WNK1 is activated by hypertonic stress in kidney epithelial cells and in brea
50 in vivo and reduce their ability to tolerate hypertonic stress in vitro.
51 tion was higher during oxidative stress than hypertonic stress, in agreement with a dramatic decrease
52                                              Hypertonic stress increased PGI2 synthesis 330% above ba
53                     We previously found that hypertonic stress increases PIP(2) by selectively activa
54            Exposure of C2C12 muscle cells to hypertonic stress induced an increase in cell content of
55                                              Hypertonic stress induced in inner medullary (IMCD3) cel
56                                              Hypertonic stress induced the eIF2alpha phosphorylation-
57                      These results suggest a hypertonic stress-induced cell cycle delay in G2 phase t
58 cal PKC was required for TPA-induced but not hypertonic stress-induced cleavage of all EGF family lig
59                                              Hypertonic stress-induced dephosphorylation is blocked b
60  Thus, deletion of SWE1 does not prevent the hypertonic stress-induced inhibition of Clb2p-Cdc28p kin
61 isotonic conditions involves CRM-1 but under hypertonic stress is CRM1-independent.
62 ay, in addition to its role in responding to hypertonic stress, is required at a basal level for the
63                                 For example, hypertonic stress led to strong cleavage of HB-EGF and N
64  mimic of diacylglycerol and PKC activator), hypertonic stress, lysophosphatidic acid (LPA)-induced G
65     Under conditions of nutrient limitation, hypertonic stress or elevated temperature, spm1 delta ce
66 ompare tRNA cleavage patterns in response to hypertonic stress, oxidative stress (arsenite), and trea
67 hysiological stimuli of ectodomain cleavage--hypertonic stress, phorbol ester, or activation of G-pro
68 ropose that eIF2alpha phosphorylation during hypertonic stress promotes apoptosis by sequestration of
69 ys a key role in the mechanism through which hypertonic stress regulates the function of T cells.
70 ralized signaling principle in the mammalian hypertonic stress response relevant to aquaporin express
71 stent with calcineurin-mediated induction of hypertonic stress-response genes, and they suggest that
72 ddition of bacterial sphingomyelinase, or by hypertonic stress, S358 is rapidly dephosphorylated.
73 rbitol from glucose during hyperglycemic and hypertonic stress, the aldose-reducing property of AR ha
74  recruited to membranes and was activated by hypertonic stress through Ser/Thr dephosphorylation.
75                                              Hypertonic stress triggers a cell cycle delay in G2 phas
76 that GSK 3beta inhibitors protect RMICs from hypertonic stress via induction of NFkappaB-COX 2-depend
77 the transcriptional activation of aqp1 under hypertonic stress, we examined the role of the transcrip
78 igating the relationship between insulin and hypertonic stress, we have discovered that osmotic shock
79 f system A activity and SNAT2 mRNA levels by hypertonic stress were independent of eIF2alpha phosphor

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