ライフサイエンスコーパス: hypertrophic scar

1 id, significant, and lasting improvements in hypertrophic scar.

2 ore-after cohort study in burn patients with hypertrophic scars.

3 sses a wide spectrum, from chronic wounds to hypertrophic scars.

4 the development and treatment of keloids and hypertrophic scars.

5 p-regulated in skin wounds and in normal and hypertrophic scars.

6 ed to evaluate integrin roles in keloids and hypertrophic scars.

7 hyperproliferating situations, psoriasis and hypertrophic scars.

8 Unlike humans, rats do not develop hypertrophic scars.

9 preciation of its importance in fibrosis and hypertrophic scarring.

10 enotype was typical except that he exhibited hypertrophic scarring.

11 e and surgery to minimise the development of hypertrophic scarring.

12 is higher in keloids (63% +/- 3.6% SEM) and hypertrophic scars (45% +/- 2.7% SEM) than in normal ski

13 Now up to 70% of patients develop hypertrophic scars after burns.

14 have therapeutic value in the prevention of hypertrophic scarring and in the treatment of fibrotic d

15 F-beta1) upregulation has been implicated in hypertrophic scars and keloids, but it is unclear if it

16 nd potentially prevent abnormal healing like hypertrophic scars and keloids.

17 s are histopathologically identical to human hypertrophic scars and persist for more than six months

18 ng scars are structurally identical to human hypertrophic scars and showed dramatic increases in volu

19 rmabrasion, wound healing, safety, scarring, hypertrophic scar, and keloid.

20 Because keratinocytes in psoriasis and hypertrophic scars are activated, we conclude that K15 e

21 Keloids and hypertrophic scars are significant symptomatic clinical

22 w-up analysis; 28 (16.2%) of these developed hypertrophic scars, but the model was otherwise safe.

23 roliferative phase of wound healing produces hypertrophic scars by inhibiting cellular apoptosis thro

24 Hypertrophic scar contracture is considered to be a path

25 In hypertrophic scars, EMT-related genes were elevated alon

26 Preventing the formation of hypertrophic scars, especially those that are a result o

27 These scar fibroblasts (hypertrophic scar fibroblasts (HTSFs)) retain the myofib

28 Complications included hypertrophic scar formation (35), atelectasis (12), pleu

29 cal delivery of S100A12 resulted in a marked hypertrophic scar formation in a validated rabbit hypert

30 To clarify the importance of apoptosis in hypertrophic scar formation, we examine the effects of m

31 f IL-1 signaling was effective in preventing hypertrophic scar formation.

32 and fibrosis than control mice in a model of hypertrophic scar formation.

33 We found that keloids and hypertrophic scars have marked alterations in fibroblast

34 chanisms behind the pathogenesis of postburn hypertrophic scar (HS) remain unclear.

35 Hypertrophic scar (HTS) formation is a frequent postoper

36 aded control wounds in an established murine hypertrophic scar (HTS) model.

37 The genetic determinants of post-burn hypertrophic scarring (HTS) are unknown, and melanocorti

38 Hypertrophic scars (HTS), frequently seen after traumati

39 ity and thus excess scar-tissue formation of hypertrophic scars (HTS).

40 key functions of myofibroblasts derived from hypertrophic scars (HTSF) are constitutively activated b

41 e traumatic or thermal injury to the dermis, hypertrophic scars (HTSs) often develop and these scar f

42 e traumatic or thermal injury to the dermis, hypertrophic scars (HTSs) often develop in humans.

43 Keloids and hypertrophic scars in children are effectively treated w

44 to a healing wound is sufficient to produce hypertrophic scars in mice.

45 trophic scar formation in a validated rabbit hypertrophic scar model compared with saline control.

46 ), dislodged sternal bar (n = 3), and mildly hypertrophic scar (n = 12).

47 ned integrin expression in keloids (n = 11), hypertrophic scars (n = 5), radiation ulcers (n = 2), an

48 Hypertrophic scars occur following cutaneous wounding an

49 TRPC3 is highly expressed in human hypertrophic scar tissue and mechanical stimuli are know

50 ion of wound healing to attenuate or prevent hypertrophic scarring, well-designed trials to confirm t

51 monstrates additional features of keloid and hypertrophic scarring which we were not able to consider