ライフサイエンスコーパス: hypervascularity

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1 ors were heterogeneous and depicted areas of hypervascularity.

2 poxia reversed the polycythemia and cerebral hypervascularity.

3 o the etiology of Peters' anomaly and ocular hypervascularity.

4 eatures of the adenomas included evidence of hypervascularity (63%), intratumoral fat (50% of patient

5 atment of ovarian disorders characterized by hypervascularity and hyperplasia.

6 duration, allowing for detection of arterial hypervascularity and portal venous washout.

7 y blocked the cellular infiltrative pattern, hypervascularity, and cell proliferation in vivo, and de

8 en, and led to mesenchymal cell hyperplasia, hypervascularity, and inflammatory cell infiltration.

9 Color Doppler US-depicted hypervascularity correlated with biologically aggressive

10 c lesions around the mediastinum testis with hypervascularity dispersing in ten patients and hypovasc

11 Despite marked induction of hypervascularity, HIF-1alpha did not induce edema, infla

12 creased rCBV in lymphomas is probably due to hypervascularity in foci of active tumor growth; and dec

13 ger than 2 cm shows typical features of HCC (hypervascularity in the arterial phase and washout in th

14 essential role in extensive infiltration and hypervascularity in the xenograft model.

15 Hypervascularity induced by HIF-1alpha could improve the

16 This hypervascularity is evident by both gross and microscopi

17 tumors), propensity for lung metastases, and hypervascularity may make these tumors valuable models f

18 Hyperplasia and hypervascularity of the ovarian theca interna and stroma

19 n in vascularity was the measure of success, hypervascularity was a strong indicator of success (P <.

20 The synovial lining layer thickness, but not hypervascularity, was significantly reduced.

21 Methotrexate did not reduce hypervascularity, which is a prominent differentiating f

22 lay a role in the ability of HIF-1 to induce hypervascularity without excessive permeability.

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