1                     Vitamin A deficiency and hypervitaminosis A cause disruption of normal cellular h

2                               The finding of hypervitaminosis A in Zambian children was supported by

3 population and could avoid the potential for hypervitaminosis A that was observed with the use of pre

4 ders such as obesity and hypoparathyroidism, hypervitaminosis A, tetracycline use and thyroid replace

5 retinyl ester concentrations are elevated in hypervitaminosis A.

6 osis, closely resemble the manifestations of hypervitaminosis A.

7 ed tumor growth and that induced symptoms of hypervitaminosis A.

8  of total serum vitamin A indicate potential hypervitaminosis, but this cutoff was derived from small

9 uption of the Klotho gene in mice results in hypervitaminosis D and a syndrome resembling accelerated

10 n of Fgf-23 in mice (Fgf-23(-/-)) results in hypervitaminosis D, abnormal mineral metabolism, and red