ライフサイエンスコーパス: hypoactivity

1 with severe mood dysregulation demonstrated hypoactivity.

2 ross imaging methods and primarily reflected hypoactivity.

3 disease (AD) has been linked to cholinergic hypoactivity.

4 ive of absence epilepsy, chronic ataxia, and hypoactivity.

5 mines along with losses of motoric function, hypoactivity and abbreviated life-span.

6 Treatments targeting amygdala hypoactivity and blunted salience during positive autobi

7 Both hypoactivity and hyperactivity in the amygdala are assoc

8 and experimental PD, the GPe and STN exhibit hypoactivity and hyperactivity, respectively, and abnorm

9 MA, and deficiencies in Ox signaling lead to hypoactivity and hypophagia.

10 d short-term synaptic plasticity, behavioral hypoactivity and impaired recognition memory.

11 nd 12-14 (MA) month-old Hdc(-/-) mice showed hypoactivity and increased measures of anxiety in the op

12 Mice lacking NgR are viable but display hypoactivity and motor impairment.

13 ons, which are mostly within the MeA, caused hypoactivity and obesity in both male and female mice fe

14 aggregant Tau causes neuronal and astrocytic hypoactivity and presynaptic dysfunction instead.

15 eus accumbens (NAc) that results in neuronal hypoactivity and thereby enhances behavioral cocaine res

16 ce have trace brain dopamine content, severe hypoactivity, and aphagia, and they die without interven

17 ayed increased sensorimotor gating, anxiety, hypoactivity, and decreased motor coordination, compared

18 kness behavior symptoms, including anorexia, hypoactivity, and hyperthermia.

19 d in neurons, and this leads to hyperphagia, hypoactivity, and increased fat mass.

20 mated open-field test, IDUA(-/-) mice showed hypoactivity as early as 2 months of age and altered anx

21 nd muscle temperatures, true hypothermia and hypoactivity as well as clearly diminished locomotor and

22 strated anxiety-like avoidance of open arms, hypoactivity, as well as unaltered within-trial and betw

23 ctivity are reliable markers for cholinergic hypoactivity associated with cognitive function deficit

24 y and rearing at 1 month of age, followed by hypoactivity at 4 months and gait anomalies at 1 year.

25 tive starting at 5 months, later changing to hypoactivity before early mortality.

26 They demonstrate that area 32 hypoactivity causes behavioral generalization relevant t

27 associated with amygdala and anterior insula hypoactivity during a complex affective processing task

28 milar to the depressed group, while amygdala hypoactivity during positive autobiographical recall is

29 der animals with diabetes exhibited detrusor hypoactivity, findings consistent with clinical features

30 cute biphasic locomotor effects of morphine (hypoactivity followed by hyperactivity) were examined.

31 prefrontal cortex (VmPFC), marked by initial hypoactivity followed by increased VmPFC activation, poi

32 es to dopamine receptor stimulation, showing hypoactivity following injection of d-amphetamine or met

33 mice show features similar to RS, including hypoactivity, forelimb stereotypies, breathing irregular

34 NMDAR hypoactivity has been implicated in the pathophysiology

35 lic brain inhibition as the primary cause of hypoactivity, hypothermia and hyporesponsiveness.

36 The ability of CP 55,940 to produce motor hypoactivity, hypothermia and immobility was reduced 163

37 thalamus are suggestive of the NMDA receptor hypoactivity hypothesis of schizophrenia and are consist

38 for the aetiology of the posterior cingulate hypoactivity in Alzheimer's disease, but also show how d

39 Moreover, hypoactivity in hippocampus and cortex among EL offsprin

40 pletes monoamines, and causes depression and hypoactivity in humans and rodents.

41 ctivity is a necessary event for cholinergic hypoactivity in PC12 cells.

42 P < .05) during executive functioning tasks; hypoactivity in posterior insula (P < .005) during posit

43 ence suggests depression is characterized by hypoactivity in the dorsal anterior cingulate, whereas h

44 nterior insula and bilateral cerebellum, and hypoactivity in the dorsal medial prefrontal cortex (mPF

45 They showed relative hypoactivity in the left occipital cortex for the low sp

46 ted that injury results in abnormal neuronal hypoactivity in the non-injured primary somatosensory co

47 Furthermore, DJ-1(-/-) mice displayed hypoactivity in the open field.

48 The pooled meta-analysis showed hypoactivity in the posterior insula, superior temporal,

49 in association with prefrontal and striatal hypoactivity in the schizophrenia group.

50 SHR in the residential figure-eight maze and hypoactivity in the SD in the running wheels.

51 PTSD is associated with hypoactivity in the ventromedial prefrontal cortex (vmPF

52 uitry models of these disorders propose that hypoactivity in the vmPFC engenders disinhibited activit

53 features of RTT: tremors, motor impairments, hypoactivity, increased anxiety-related behavior, seizur

54 Yet, genetically conferred MAOA or 5-HTT hypoactivity is associated with altered aggression and i

55 Thus, cholinergic hypoactivity is thought to be important in cognitive dys

56 One cause of this hypoactivity may be defective corticocortical or thalamo

57 The role of Par-4 in inducing cholinergic hypoactivity may have significant implications in the un

58 Amygdala hypoactivity may represent an intermediate phenotype, of

59 luN2 phosphorylation, we postulated that Src hypoactivity may result from convergent alterations of v

60 The high-trauma-exposed group displayed hypoactivity of cerebellar regions in response to neutra

61 of explicit (more than implicit) memory and hypoactivity of cholinergic projections to the hippocamp

62 The hypoactivity of dorsolateral prefrontal cortex in schizo

63 Hypoactivity of excitatory NMDAR-mediated neurotransmiss

64 observations suggest that the hypophagia and hypoactivity of mutants result not only because of the a

65 Hypoactivity of NRF2 in Krt16-/- footpad skin correlated

66 Finally, our data show that hyper- or hypoactivity of PP5 mutants increases Hsp90 binding to i

67 Relative hyperactivity and hypoactivity of regional cerebral blood flow in brain re

68 ed neurotransmission accompanying functional hypoactivity of the frontal lobes.

69 Hyperserotonergic state and hypoactivity of the hypothalamic-pituitary-adrenal axis

70 either overactivity of the arousal systems, hypoactivity of the sleep-inducing systems, or both.

71 Furthermore, hypoactivity of the spinal cannabinoid system results in

72 ic polymorphism rs16147 may contribute to IL hypoactivity, resulting in impaired extinction memory an

73 As with the nocturnal hypoactivity, this effect was observed only during the f

74 motional traits are associated with amygdala hypoactivity to consciously perceived fear, while low le

75 Dopamine agonist-related ventral striatal hypoactivity to risk is consistent with impaired risk ev

76 l cholinergic responses such as hypothermia, hypoactivity, tremor, and salivation were enhanced in GR

77 We additionally report hypoactivity, unrelated to anxiety or motoric function,

78 gnitive control load, however, Abeta-related hypoactivity was found in the right inferior frontal cor

79 ingulate, pre-supplementary motor and insula hypoactivity was observed for both successful NOGOs and

80 In contrast, inflammation-induced hypoactivity was unaffected, demonstrating the physiolog

81 To test genomic underpinnings for Src hypoactivity, we examined genome-wide association study

82 ctions (effective connectivity), rather than hypoactivity within individual brain regions, may contri