ライフサイエンスコーパス: hypoalbuminemia

1 ht gain, hepatic transaminase elevation, and hypoalbuminemia.

2 models to identify independent predictors of hypoalbuminemia.

3 t which time they had hyperbilirubinemia and hypoalbuminemia.

4 e edema of malnutrition is not solely due to hypoalbuminemia.

5 serum alkaline phosphatase and amylase, and hypoalbuminemia.

6 A total of 27.3% of the RHD patients had hypoalbuminemia.

7 elevated liver transaminases, azotemia, and hypoalbuminemia.

8 d with significant improvement in anemia and hypoalbuminemia.

9 panied by significant weight loss and severe hypoalbuminemia.

10 y toxicity in pregnant women with anemia and hypoalbuminemia.

11 on deficiency along with hypoproteinemia and hypoalbuminemia.

12 ollapse accompanied by hemoconcentration and hypoalbuminemia.

13 k, acute restoration of blood volume, and in hypoalbuminemia.

14 with proteinuria and not as a consequence of hypoalbuminemia.

15 ny cases of hypocalcemia are the artifact of hypoalbuminemia.

16 significant amelioration of proteinuria and hypoalbuminemia.

17 old increase in risk of death independent of hypoalbuminemia.

18 35 g/L as a cutoff, 53% of the subjects had hypoalbuminemia.

19 nd alkaline phosphatase (8% and 18%) levels; hypoalbuminemia (10% and 19%); hyperbilirubinemia (10% a

20 l pain (70%), significant weight loss (92%), hypoalbuminemia (100%; 85% lower than 2.0 g/dL), and ane

21 sus 0.1 +/- 0.1, mutant versus control), and hypoalbuminemia (2.1 +/- 0.4 versus 2.5 +/- 0.2 G/dl, mu

22 ted prothrombin time (18 +/- 3 seconds), and hypoalbuminemia (2.7 +/- 0.6 g/dL).

23 Those with severe (0.5-2.9 g/dL) and mild hypoalbuminemia (3.0-3.6 g/dL) had posttransplant adjust

24 leukocytosis (2.85 [hazard ratio]; P = .02), hypoalbuminemia (3.41; P = .05), and anti-Saccharomyces

25 second prolongation; P < .001), more severe hypoalbuminemia (39% vs. 9% with albumin <3 g/dL; P < .0

26 to ER, LR patients were more likely to have hypoalbuminemia (56% versus 80%), hematocrit <28% (33% v

27 21%) than in the Y90 group (0%; P = .031) or hypoalbuminemia (58% in the cTACE group vs 4% in the Y90

28 Hypoalbuminemia, a biomarker of asparaginase activity, w

29 Patients with GBS may develop hypoalbuminemia after treatment with IVIG, which is rela

30 rs for poor prognosis after PEG include sex, hypoalbuminemia, age, chronic heart failure, and subtota

31 dL (interquartile range, 3.8-4.5 g/dL), with hypoalbuminemia (albumin, <3.5 g/dL) in 20 (12.8%) of 15

32 obal or liver-specific FcRn deletion exhibit hypoalbuminemia, albumin loss into the bile, and increas

33 oratory abnormalities included anemia (all); hypoalbuminemia (all) and thrombocytopenia (6).

34 luded (in decreasing frequency) grade 1 to 2 hypoalbuminemia, aminotransferase elevations, edema, hea

35 oing VRS to explore the relationship between hypoalbuminemia and adverse outcomes and to confirm whet

36 ng ART are low and are influenced as much by hypoalbuminemia and age as by CD4 cell status.

37 Common toxic effects included transient hypoalbuminemia and elevated aminotransferase levels.

38 ever, the associations between eGFR and both hypoalbuminemia and hypertension were NS.

39 disease-related factors are associated with hypoalbuminemia and might be valuable items to include o

40 The association between hypoalbuminemia and posttransplant mortality was stronge

41 entions to slow its progression, predialysis hypoalbuminemia and severe anemia, suboptimal pre-ESRD e

42 In the full mITT population, age, hypoalbuminemia, and cancer were inversely associated wi

43 age, weight loss, anemia, thrombocytopenia, hypoalbuminemia, and excess bone marrow blasts as indepe

44 , malabsorption, protein-losing enteropathy, hypoalbuminemia, and failure to thrive.

45 des, fevers, malaise, anorexia, weight loss, hypoalbuminemia, and gastrointestinal blood loss were co

46 uirement for mechanical circulatory support, hypoalbuminemia, and hepatic dysfunction), intraoperativ

47 is revealed that a nonsurgical intervention, hypoalbuminemia, and higher Acute Physiology and Chronic

48 a lower Karnofsky Performance score, anemia, hypoalbuminemia, and higher C-reactive protein.

49 urvived for at least 12 months, proteinuria, hypoalbuminemia, and hypercholesterolemia improved durin

50 pain, watery diarrhea, fever, leukocytosis, hypoalbuminemia, and hypovolemia.

51 rum transaminase levels, hyperbilirubinemia, hypoalbuminemia, and prolongation of clotting times.

52 ting with severe protein-losing enteropathy, hypoalbuminemia, and proximal myopathy who had not respo

53 tead, analyses revealed hepatic dysfunction, hypoalbuminemia, and vascular/oxygenation insufficiency.

54 Anemia or hypoalbuminemia are useful clues to NSAID enteropathy.

55 knowledge, the utility of proteinuria and/or hypoalbuminemia as biomarkers of thrombotic risk remains

56 Hypoalbuminemia, ascribed to malnutrition, has been one

57 Our data suggest that hypoalbuminemia at listing reveals a vulnerable populati

58 Hypoalbuminemia at listing was a significant predictor o

59 Anemia and hypoalbuminemia at presentation were independently assoc

60 antibodies or peptide mimetics, which cause hypoalbuminemia, biliary loss of albumin, and increased

61 ration, and clinical features of AH, such as hypoalbuminemia, bilirubinemia, and splenomegaly.

62 severe but reversible hemoconcentration and hypoalbuminemia caused by leakage of fluids and macromol

63 (in malnourished patients and in those with hypoalbuminemia, cholangitis or long-term jaundice; with

64 Most of them had impaired renal function, hypoalbuminemia, concurrent infection, and/or concomitan

65 The almost ubiquitous hypoalbuminemia confounded the interpretation of acid-ba

66 Our results suggest that proteinuria and/or hypoalbuminemia could be developed as clinically meaning

67 Individuals with hypoalbuminemia (defined as a serum albumin concentratio

68 , and a vascular leak syndrome (hypotension, hypoalbuminemia, edema).

69 ) represents the association of proteinuria, hypoalbuminemia, edema, and hyperlipidemia.

70 oteinuria in excess of 3.5 g/24 h along with hypoalbuminemia, edema, hyperlipidemia (hypertriglycerid

71 s of nephrotic syndrome include proteinuria, hypoalbuminemia, edema, hyperlipidemia and lipiduria.

72 ated mice exhibited progressive proteinuria, hypoalbuminemia, elevated blood urea nitrogen (BUN) leve

73 Untreated NS rats showed heavy proteinuria; hypoalbuminemia; elevated plasma cholesterol, triglyceri

74 city is vascular leak syndrome manifested by hypoalbuminemia, fluid retention, hypotension and, in on

75 Stroke patients with hypoalbuminemia had a greater risk of infective complica

76 The high prevalence of pre-ESRD hypoalbuminemia, hematocrit <28%, and lack of EPO use su

77 and geographic factors, the odds ratios for hypoalbuminemia, hematocrit <28%, and lack of EPO use we

78 e trials involving surgery or trauma, burns, hypoalbuminemia, high-risk neonates, ascites, and other

79 arterial pressure, low stroke volume index, hypoalbuminemia, history of cerebral vascular disease, e

80 ling pressures, lower cardiac index, anemia, hypoalbuminemia, hyperbilirubinemia, cognitive impairmen

81 gnificantly correlated with age, alcoholism, hypoalbuminemia, hyperbilirubinemia, renal insufficiency

82 io with anemia, acidosis, hyperphosphatemia, hypoalbuminemia, hyperparathyroidism, and hypertension a

83 ine ratios associated with anemia, acidosis, hypoalbuminemia, hyperparathyroidism, and hypertension b

84 boratory examinations showed the presence of hypoalbuminemia, hypogammaglobulinemia, and an elevated

85 the predominant findings included diarrhea, hypoalbuminemia, hyponatremia, hypokalemia, hypocalcemia

86 860 mg/m2, one of seven patients had grade 3 hypoalbuminemia/hypophosphatemia.

87 lth characteristics that are associated with hypoalbuminemia in community-dwelling older persons, we

88 Hypoalbuminemia in dialysis patients is primarily a cons

89 y volume expansion plays any role in causing hypoalbuminemia in ESRD patients.

90 The profound loss of albumin led to hypoalbuminemia in some diabetic animals.

91 forts to improve nutrition, as it may affect hypoalbuminemia in SPK recipients, may be one strategy f

92 ine the incidence and clinical correlates of hypoalbuminemia in SPK recipients.

93 , are highly correlated with proteinuria and hypoalbuminemia in the puromycin aminonucleoside and adr

94 We defined hypoalbuminemia in two ways: < 35 g/L (1.2% of the sampl

95 suggested by the progressive development of hypoalbuminemia in wild-type mice transplanted with FcRn

96 .2-4.1 g/dL) (P < .001), and the number with hypoalbuminemia increased to 60 (34.5%) of 174 (P < .001

97 Hypoalbuminemia (IRR, 4.35; 95% CI, 2.78-6.81; P < .001)

98 Hypoalbuminemia is an independent risk factor for death

99 Persistent post-SPK hypoalbuminemia is associated with an increased risk for

100 Hypoalbuminemia is associated with mortality in patients

101 Hypoalbuminemia is associated with poorer outcomes in re

102 nosed by the third method; when adjusted for hypoalbuminemia, it reliably detected the hidden abnorma

103 Studies have suggested that hypoalbuminemia itself impairs delivery of effective amo

104 All but one patient showed hypoalbuminemia (<3.5 g/dL); those with HAT also had sig

105 Viewed in this manner, hypoalbuminemia may offer an opportunity to improve pati

106 is newly described mechanism, in addition to hypoalbuminemia, may contribute to platelet hyperactivit

107 Hyperbilirubinemia and hypoalbuminemia occurred and survival decreased after tr

108 Hypoalbuminemia occurred in 79%, including 15% with grad

109 Dramatic weight loss and hypoalbuminemia often follow acute hospitalization.

110 The effect of albumin level and hypoalbuminemia on the risk of post-SPK events (cardiac

111 There was no hypoalbuminemia or apparent evidence of lupus nephritis

112 d with LR, and whether LR is associated with hypoalbuminemia or late initiation of dialysis.

113 1.1 to 1.7 per 10 years), those with greater hypoalbuminemia (OR: 0.49, 95% CI: 0.31 to 0.76 per mg/d

114 weakness, cognitive impairment, anemia, and hypoalbuminemia outperformed other frailty scales and is

115 Stepwise multivariate analysis identified hypoalbuminemia (p < 0.001) and the burden of comorbid d

116 of delirium including older age (P < 0.001), hypoalbuminemia (P < 0.001), impaired functional status

117 (PFS, 2.95; P = .03; OS, 3.14; P = .03), and hypoalbuminemia (PFS, 2.96; P = .05; OS, 3.64; P = .04).

118 and adverse outcomes and to confirm whether hypoalbuminemia plays a role in risk evaluation.

119 Hypoalbuminemia predicts disability and mortality in pat

120 The nephrotic syndrome is defined by edema, hypoalbuminemia, proteinuria, and hyperlipemia with elev

121 ls: 35-fold; KO: 5400-fold versus baseline), hypoalbuminemia, reduced GFR, and marked glomerular dama

122 Post-SPK hypoalbuminemia resolves over time in many patients.

123 ythmia (RR, 1.51; 95% CI, 1.04 to 2.19), and hypoalbuminemia (RR, 0.56; 95% CI, 0.42 to 0.74 per 1 g/

124 Diabetes can compound hypoalbuminemia's detrimental effects.

125 Hypoalbuminemia (serum albumin level < or =3.5 g/dL) was

126 he prevalence of and factors associated with hypoalbuminemia, severe anemia, and erythropoietin (EPO)

127 me, has been associated with proteinuria and hypoalbuminemia severity.

128 w plasma-cell infiltration greater than 50%, hypoalbuminemia, thrombocytopenia) were predictive of ov

129 h 3 independently significant comorbidities, hypoalbuminemia, thrombocytopenia, and high lactate dehy

130 including fever, chills, hypotension, edema, hypoalbuminemia, thrombocytopenia, and transaminasemia w

131 The addition of hypoalbuminemia to Euro score enhanced net reclassificat

132 to 50 microg/Kg every other day x 3 included hypoalbuminemia, transaminase elevations, fatigue, and e

133 Hypoalbuminemia was also independently associated with t

134 Hypoalbuminemia was an independent predictor of in-hospi

135 These findings indicated that hypoalbuminemia was an independent risk factor for in-ho

136 Hypoalbuminemia was associated with an increased chance

137 SPK-related hypoalbuminemia was associated with an increased risk fo

138 were performed to determine whether post-SPK hypoalbuminemia was associated with pre-SPK variables.

139 Marked hypoalbuminemia was common among the critically ill pati

140 Post-SPK hypoalbuminemia was defined as a serum albumin level < o

141 Anemia and hypoalbuminemia were not detected in any of the patients

142 1 or 2 fatigue, dryness of skin, anemia, and hypoalbuminemia were the most frequent toxicities report