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1 xpressed in the yolk syncytial layer and the hypochord.
2 to be specified as floorplate, notochord or hypochord.
3 ochord lies medially, flanked by presumptive hypochord and both overlie the deep region of the presum
4 cial cartilage, ear, notochord, floor plate, hypochord and fins in a pattern similar to the expressio
5 dorsal midline, prechordal plate, notochord, hypochord and floor plate share a common embryonic origi
6 dline structures, including notochord and/or hypochord and neural floorplate, regulate Xnr-1 expressi
7 inhibiting notochord formation and promoting hypochord and possibly floor plate development, but the
8 sion resolves to the neural tube floorplate, hypochord, and (transiently) the notochord anlage, and i
9 eginning of gastrulation caused expansion of hypochord at the expense of notochord, but floor plate w
12 We demonstrate here that floor plate and hypochord cells arise from distinct regions of the zebra
14 mbryos had reduced numbers of floorplate and hypochord cells; these cells lie above and beneath the n
15 neurulation leads to the complete failure of hypochord development and to the elimination of expressi
18 ate during early neural stages, and that the hypochord does not depend on further notochord signals f
19 ells in the underlying endoderm to take on a hypochord fate during early neural stages, and that the
20 Notch signaling promotes floor plate and hypochord fates over notochord, but has variable effects
21 tants have significantly stronger defects in hypochord formation but not in somitogenesis or hindbrai
25 , continues to act in the tailbud to specify hypochord from a notochord/hypochord bipotential cell po
26 dorsal midline (floor plate, notochord, and hypochord) has been an area of classical research and de
27 both paraxial and axial mesoderm (including hypochord), in similar patterns and amounts in both spec
36 aling during gastrulation entirely prevented hypochord specification but only reduced the number of f
37 hord are critical for the development of the hypochord, which is a transient, endodermally derived st
38 diffusible form of VEGF is expressed by the hypochord, which lies at the embryonic midline immediate
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