ライフサイエンスコーパス: hypochromic

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1 mutant, frascati (frs), that shows profound hypochromic anaemia and erythroid maturation arrest owin

2 Homozygous mk/mk mice have microcytic, hypochromic anaemia due to severe defects in intestinal

3 Here we show that the hypochromic anaemia in shiraz (sir) zebrafish mutants is

4 ing to identify the gene responsible for the hypochromic anaemia of the zebrafish mutant weissherbst.

5 ish mutant sauternes (sau) has a microcytic, hypochromic anaemia, suggesting that haemoglobin product

6 tation develop moderate to severe microcytic hypochromic anaemia.

7 In most cases, the red cells were hypochromic and microcytic, consistent with abnormalitie

8 continued, weh(Tp85c-/-) zebrafish developed hypochromic anemia and impaired erythroid maturation des

9 Belgrade rats exhibit microcytic, hypochromic anemia and systemic iron deficiency due to a

10 use mutant, which suffers from a microcytic, hypochromic anemia apparently due to defective iron tran

11 autosomal recessively inherited, microcytic, hypochromic anemia associated with abnormal reticulocyte

12 ic deficiency of Mfrn1 results in a profound hypochromic anemia in vertebrate species.

13 cation and characterization of the zebrafish hypochromic anemia mutant, gavi, which exhibits transfer

14 afish have resulted in the identification of hypochromic anemia mutants with a range of mutations aff

15 ron deficiency and eventually the microcytic hypochromic anemia or iron deficiency anemia that is the

16 HNF1A(-/-) mice displayed microcytic hypochromic anemia with reticulocytosis that was partial

17 These mice exhibited microcytic, hypochromic anemia, as did lethally irradiated mice that

18 f3 in Danio rerio resulted in microcytic and hypochromic anemia.

19 Quantification of hypochromic cells is valuable in the differential diagno

20 ast, Fe(III)Blm and HO(2)-Co(III)Blm induced hypochromic effects in the CD spectrum of I and altered

21 DNA base stacking in Pf3, as judged by Raman hypochromic effects, differs significantly from that occ

22 thridinium interactions provide compensatory hypochromic effects.

23 n of DMT1 identified in a female with severe hypochromic microcytic anemia and iron overload.

24 The mutation, zinfandel, has a hypochromic microcytic anemia as an embryo, but later re

25 These animals exhibit hypochromic microcytic anemia due to impaired intestinal

26 Homozygous sublytic mice develop hypochromic microcytic anemia with reduced osmotic fragi

27 e zebrafish chianti (cia) mutant manifests a hypochromic, microcytic anemia after the onset of embryo

28 ependent polycythemia in heterozygotes and a hypochromic, microcytic anemia in homozygotes.

29 antation analyses reveal a mild, congenital, hypochromic, microcytic anemia intrinsic to the hematopo

30 ristics and pathogenesis of a new recessive, hypochromic, microcytic anemia mouse mutant, nm1054.

31 A hypochromic, microcytic anemia was present from birth, a

32 of chardonnay (cdy), a zebrafish mutant with hypochromic, microcytic anemia, and positioned the mutan

33 sis reveals a moderately severe, congenital, hypochromic, microcytic anemia, with an elevated red cel

34 iron acquisition by red blood cells leads to hypochromic, microcytic anemia.

35 to hepcidin induction, iron deficiency and a hypochromic, microcytic anemia.

36 Hypochromic, microcytic anemias are typically the result

37 ysplastic syndrome [ATMDS]) characterized by hypochromic, microcytic, anisopoikilocytic red blood cel

38 Examination of the hypochromic mutant, weissherbst, and adult hypoxia-treat

39 /-) adults have mild anemia characterized by hypochromic red blood cells (RBCs), reticulocytosis, and

40 From the hypochromic responses and the induced emission intensiti

41 The hypochromic shift and Tm for triplex formation increases

42 2 and G2A2G5A2G2 are very shallow with small hypochromic shifts denoting negligible binding at physio

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