ライフサイエンスコーパス: hypocotyl

1 pylar endosperm) and RAD (radicle plus lower hypocotyl).

2 ght cues (e.g., positive phototropism in the hypocotyl).

3 ed the site of light perception to the upper hypocotyl.

4 tic lesions were observed at the base of the hypocotyl.

5 ous and occurs more prominently in the basal hypocotyl.

6 m, phloem, and primary xylem in the stem and hypocotyl.

7 l controls of cell elongation in Arabidopsis hypocotyl.

8 cell elongation in different regions of the hypocotyl.

9 owth phenotype, deetiolation of the seedling hypocotyl.

10 ontrolled elongation of the seedling stem or hypocotyl.

11 em, unable to flow into the vasculature of a hypocotyl.

12 ross the epidermis below the meristem in the hypocotyl.

13 levels as well as in chloroplast size in the hypocotyl.

14 along the length of the Arabidopsis thaliana hypocotyl.

15 tability are aberrant in etiolated xxt1 xxt2 hypocotyls.

16 that promotes cell elongation in Arabidopsis hypocotyls.

17 veral candidate regulators in the elongating hypocotyls.

18 growth, and, in etiolated seedlings, shorter hypocotyls.

19 issect their trafficking routes in etiolated hypocotyls.

20 growth of Arabidopsis (Arabidopsis thaliana) hypocotyls.

21 resulting in low activity of PIF3 and short hypocotyls.

22 rlapping patterns of expression in etiolated hypocotyls.

23 transcript and late flowering and elongated hypocotyls.

24 psi1-1 seedlings have shorter roots and hypocotyls.

25 in the cotyledon tissue but not meristems or hypocotyls.

26 extent of axial cell expansion in dark-grown hypocotyls.

27 elongated cell types of roots and dark-grown hypocotyls.

28 duced by excising roots from low-light-grown hypocotyls.

29 R sensitivity in leaves and petioles but not hypocotyls.

30 h in rapidly elongating roots and dark-grown hypocotyls.

31 genes are defective in stomata formation in hypocotyls.

32 It was shown that LONG HYPOCOTYL 2 (HY2) is the only FDBR in flowering plants p

33 otropic signalling component Non-Phototropic Hypocotyl 3 (NPH3).

34 ith the signalling component Non-Phototropic Hypocotyl 3 (NPH3).

35 The impact ethylene has on hypocotyl 3D cell anisotropy identified the preferential

36 n of the organ boundary gene LIGHT-SENSITIVE HYPOCOTYL 4 restored RZ function and stem growth in the

37 f5 (pifq) mutants; the dynamics of ELONGATED HYPOCOTYL 5 (HY5) and LONG HYPOCOTYL IN FAR-RED (HFR1) p

38 ion of the transcription activator ELONGATED HYPOCOTYL 5 (HY5) that is associated with chromatins of

39 Whereas the bZIP proteins, HY5 (elongated hypocotyl 5) and HYH (HY5 homologue), are degraded by CO

40 latory and two inhibitory modules, while for hypocotyls, a single inhibitory module is sufficient.

41 (IAA) transport and its accumulation in the hypocotyl above the point of excision where adventitious

42 clarify cell-specific auxin function in the hypocotyl and highlight the complexity of cell type inte

43 hows that the mechanisms underlying rhythmic hypocotyl and leaf growth differ.

44 OP1 and the four SPA genes are essential for hypocotyl and leaf petiole elongation in response to low

45 reduction of GA4 causes severe inhibition of hypocotyl and root elongation, which can be rescued by e

46 g2 double mutants show defects in fertility, hypocotyl and root growth, and responses to light and su

47 duced genome-wide gene expression changes in hypocotyls and cotyledons separately.

48 hemicellulose to the cell wall in dark-grown hypocotyls and in secretory cells of the seed coat.

49 ABP1 on transcriptomic changes in dark-grown hypocotyls and investigated the consequences of gene exp

50 PGX1(AT) plants, PGX2(AT) plants have longer hypocotyls and larger rosette leaves, but they also uniq

51 esolved gravitropism measurements of atlazy1 hypocotyls and primary inflorescence stems showed a sign

52 angles and gravitropic behavior of seedling hypocotyls and primary roots.

53 ers of the SAUR19-24 subfamily exhibit short hypocotyls and reduced leaf size.

54 detected in the epidermal layers of leaves, hypocotyls and roots; in the root, it was predominantly

55 l than did wild-type seedlings, and had wavy hypocotyls and twisted inflorescence stems.

56 ar interactions in the plant embryonic stem (hypocotyl), and analyzing these using quantitative netwo

57 root initiation in the Arabidopsis thaliana hypocotyl, and we demonstrate that they act by modulatin

58 vascular cells of cotyledons, leaves, roots, hypocotyls, and anthers.

59 use light-dependent arrhythmicity, elongated hypocotyls, and early flowering.

60 tosis to endoreplication was lower in abcb19 hypocotyls, and fluorescence microscopy showed the CCS52

61 ithin the vasculature of Arabidopsis leaves, hypocotyls, and roots.

62 of IBA is much lower than IAA in Arabidopsis hypocotyls, and the transport mechanism is distinct from

63 al targeting approach therefore excludes the hypocotyl apex as the site for light perception for phot

64 -GFP (P1-GFP) expression was targeted to the hypocotyl apex of the phot-deficient mutant using the pr

65 te in more detail the functional role of the hypocotyl apex, and the regions surrounding it, in estab

66 n of CUC3::P1-GFP was clearly visible at the hypocotyl apex, with weaker expression in the cotyledons

67 In the hypocotyl assays, the basal halves of APY-suppressed hyp

68 Phototropic hypocotyl bending in response to blue light excitation i

69 In Arabidopsis thaliana, phototropic hypocotyl bending is initiated by the blue light recepto

70 Here, we show that phototropic hypocotyl bending is strongly dependent on the activity

71 ocotyl is a prerequisite for phot1-dependent hypocotyl bending.

72 ds expression of Bn-FAE1.1 into the axis and hypocotyl but also acts negatively to repress expression

73 omain B-class GATA genes, most strikingly in hypocotyls but also in cotyledons.

74 to far-red shade by the cotyledons triggers hypocotyl cell elongation and auxin target gene expressi

75 Quantitative analysis of hypocotyl cell growth in the nek6-1 mutant demonstrated

76 ng growth by modulating DNA accessibility of hypocotyl cell size regulatory genes.

77 The two proteins were identified in hypocotyl cell wall extracts by proteomics.

78 fying the major array pattern classes in the hypocotyl cell.

79 over, we demonstrate that Golgi transport in hypocotyl cells can be accurately predicted from the act

80 in microtubule dynamics in spr1 eb1b mutant hypocotyl cells correlated well with the severity of gro

81 n cytoskeleton in both growing and elongated hypocotyl cells has structural properties facilitating e

82 ar cortical microtubule arrays in dark-grown hypocotyl cells organize into a transverse coaligned pat

83 induces approximately 80% of the light-grown hypocotyl cells to form transverse arrays over a 2-h per

84 the hypocotyl where it induces elongation of hypocotyl cells.

85 tensive transcriptome reconfiguration in the hypocotyls compared with the cotyledons.

86 protein to be lower in the nuclei of abcb19 hypocotyls compared with wild type.

87 l assays, the basal halves of APY-suppressed hypocotyls contained considerably lower free indole-3-ac

88 n mtp8-2 mutant, Mn no longer accumulates in hypocotyl cortex cells and sub-epidermal cells of the em

89 ls, followed by the root apical meristem and hypocotyl, cotyledons, and shoot apical meristem.

90 The elongated cells of the hypocotyl create a variety of microtubule array patterns

91 or instance, secondary growth of Arabidopsis hypocotyls creates a radial pattern of highly specialize

92 We acquired hypocotyl cross-sections from tiled high-resolution imag

93 While hypocotyls did not accumulate lipophilic pigments during

94 clustered stomata in the leaves, whereas the hypocotyls did not have any stomata.

95 High-temperature-mediated hypocotyl elongation additionally involves localized cha

96 wth medium greatly enhances the reduction in hypocotyl elongation and cellulose content of shv3svl1 T

97 ariety of physiological processes, including hypocotyl elongation and flowering time.

98 ole of phyA-dependent CKI1 expression in the hypocotyl elongation and hook development during skotomo

99 functionally implicated in the inhibition of hypocotyl elongation and known to be a direct target of

100 me 2 (CRY2) mediate blue light inhibition of hypocotyl elongation and long-day (LD) promotion of flor

101 t receptor that mediates light inhibition of hypocotyl elongation and long-day promotion of floral in

102 ion, but both were active in AtD14-dependent hypocotyl elongation and secondary shoot growth.

103 Root growth and hypocotyl elongation are convenient downstream physiolog

104 Here, we study hypocotyl elongation as a proxy for shoot elongation and

105 of response from inhibition to promotion of hypocotyl elongation by light.

106 Seedling root growth and hypocotyl elongation can be accurately predicted using a

107 adruple pifq mutant displays clearly reduced hypocotyl elongation compared to wild-type in response t

108 emonstrate that the magnitude of Suc-induced hypocotyl elongation depends on the day length and light

109 light/dark cycles, we found that Suc-induced hypocotyl elongation did not occur in tps1 mutants and o

110 in-signaling machinery to regulate etiolated hypocotyl elongation growth in Arabidopsis.

111 iologically, Glc and BR interact to regulate hypocotyl elongation growth of etiolated Arabidopsis (Ar

112 -6 double mutant displayed severe defects in hypocotyl elongation growth similar to its bri1-6 parent

113 In seedlings, these proteins repress hypocotyl elongation in a daylength- and sucrose-depende

114 ibit folate biosynthesis in plants, restrict hypocotyl elongation in a sugar-dependent fashion.

115 at, when overexpressed, resulted in enhanced hypocotyl elongation in etiolated Arabidopsis thaliana s

116 ediating a signal that underlies Suc-induced hypocotyl elongation in light/dark cycles.

117 nt with ethylene or auxin inhibitors reduced hypocotyl elongation in PIF4 overexpressor (PIF4ox) and

118 gibberellins (GAs) antagonistically regulate hypocotyl elongation in plants.

119 tions of additional AHL genes in suppressing hypocotyl elongation in the light.

120 e fluence rate response where suppression of hypocotyl elongation increases incrementally with light

121 Hypocotyl elongation is a highly coordinated physiologic

122 Under short-day (SD) photocycles, hypocotyl elongation is maximal at dawn, being promoted

123 elicits shade- and high temperature-induced hypocotyl elongation largely independently of 3-IPA-medi

124 As GA signalling directs hypocotyl elongation largely through promoting PIF activ

125 In contrast, the reduced hypocotyl elongation of ethylene biosynthesis and signal

126 Mutation in DET1 changed the sensitivity of hypocotyl elongation of mutant seedlings to GA and paclo

127 articipates positively in the control of the hypocotyl elongation response to plant proximity, a role

128 ophore inactivation and associated disparate hypocotyl elongation responses under far-red (FR) light.

129 Plants lacking PGX1 display reduced hypocotyl elongation that is complemented by transgenic

130 ts in fertility, and enhanced sensitivity of hypocotyl elongation to red but not to far-red or blue l

131 We found that Suc-induced hypocotyl elongation under light/dark cycles does not in

132 ight- and phytochrome-mediated regulation of hypocotyl elongation under red (R) and FR illumination.

133 xin and gibberellin signaling in Suc-induced hypocotyl elongation under short photoperiods.

134 er transcription factor that participates in hypocotyl elongation under short-day conditions.

135 -regulated protein stability drives rhythmic hypocotyl elongation with peak growth at dawn.

136 reviously uncharacterized LHE (LIGHT-INDUCED HYPOCOTYL ELONGATION) gene, which we show impacts light-

137 ppressed chlorophyll synthesis, promotion of hypocotyl elongation, and formation of a closed apical h

138 active in KAI2-dependent seed germination or hypocotyl elongation, but both were active in AtD14-depe

139 rphogenesis, as illustrated by inhibition of hypocotyl elongation, cotyledon opening, and leaf greeni

140 induction of seed germination, inhibition of hypocotyl elongation, induction of cotyledon opening, ra

141 e overexpressors has differential effects on hypocotyl elongation, leaf shape, and petiole length, as

142 e of these genes in the control of greening, hypocotyl elongation, phyllotaxy, floral organ initiatio

143 g deoxystrigolactones to inhibit Arabidopsis hypocotyl elongation, regulate seedling gene expression,

144 ure in distinct developmental traits such as hypocotyl elongation, root elongation, and flowering tim

145 lthough only SMAX1 regulates germination and hypocotyl elongation, SMAX1 and SMXL6,7,8 have complemen

146 r Phytochrome Interacting Factor 4 (PIF4) on hypocotyl elongation.

147 ngs have opposite BR-response phenotypes for hypocotyl elongation.

148 HXK1)-mediated pathway to regulate etiolated hypocotyl elongation.

149 eviously shown to be important regulators of hypocotyl elongation.

150 ular anisotropy driving Arabidopsis thaliana hypocotyl elongation.

151 ding seed germination, stomatal closure, and hypocotyl elongation.

152 how that cotyledon-generated auxin regulates hypocotyl elongation.

153 curately describes wild-type root growth and hypocotyl elongation.

154 nced resistance to DELLA accumulation during hypocotyl elongation.

155 ce by DELLAs correlates closely with reduced hypocotyl elongation.

156 tation of Arabidopsis (Arabidopsis thaliana) hypocotyl epidermal cells, dynamic cortical microtubules

157 aliana with transcriptional profiling of the hypocotyl epidermis from Brassica rapa, we show that aux

158 ted with growth symmetry breaking within the hypocotyl epidermis.

159 he expense of IAA-Glu (IAA-glutamate) in the hypocotyl epidermis.

160 localized synthesis of ABCB19 protein after hypocotyl excision leads to enhanced IAA transport and l

161 In the hypocotyl expansion zone, indaziflam caused an atypical

162 comparison, average speed in the A. thaliana hypocotyl expressing GFP-AtCESA6 was 184 +/- 86 nm min(-

163 Hypocotyl extension in the shade and outgrowth of new le

164 In hypocotyls, GA levels were reduced in a phytochrome inte

165 gical significance of BR-mediated changes in hypocotyl graviresponse lies in the fact that BR signali

166 the circadian clock, and we review seedling hypocotyl growth as a paradigm of PIFs acting at the int

167 ATAF2 modulates hypocotyl growth in a light-dependent manner, with the p

168 night temperature difference [-DIF]) inhibit hypocotyl growth in Arabidopsis (Arabidopsis thaliana).

169 r basis for the phyB-mediated suppression of hypocotyl growth in Arabidopsis.

170 Previous studies have shown that hypocotyl growth in low red to far-red shade is largely

171 g is required in many cell types for correct hypocotyl growth in shade, with a key role for the epide

172 xpression, coinciding with the initiation of hypocotyl growth in the early evening, is positively cor

173 brief heat shocks enhance the inhibition of hypocotyl growth induced by light perceived by phytochro

174 This suggests that hypocotyl growth is elicited by both local and distal au

175 etic pigments are promoted by light, whereas hypocotyl growth is inhibited.

176 me and root growth; control of cotyledon and hypocotyl growth requires simultaneous phyA activity in

177 ion in the regulation of gene expression and hypocotyl growth suppression in Arabidopsis.

178 ng; instead, it showed auxin activity in the hypocotyl growth test.

179 and the conditional use of GA-ATHB5-mediated hypocotyl growth under optimal conditions may be used to

180 HsfB2b is also involved in the regulation of hypocotyl growth under warm, short days.

181 ses of gene expression, cotyledon unfolding, hypocotyl growth, and greening observed in the phyA muta

182 e changes of single hypocotyl protoplasts or hypocotyl growth, both at high temporal resolution.

183 is both necessary and sufficient to initiate hypocotyl growth, but we also provide evidence for the f

184 with the negative regulatory role of HOS1 in hypocotyl growth, HOS1-defective mutants exhibited elong

185 In contrast to vastly studied hypocotyl growth, little is known about diel regulation

186 ic diurnal variation in Arabidopsis thaliana hypocotyl growth, we found that cellulose synthesis and

187 ing phenotypes, including increased stem and hypocotyl growth, which increases the likelihood of outg

188 g converge to influence the transcription of hypocotyl growth-promoting SAUR19 subfamily members.

189 lly understood how the phytochromes modulate hypocotyl growth.

190 that HMR acts upstream of PIFs in regulating hypocotyl growth.

191 duced an auxin-like swelling response but no hypocotyl growth.

192 ion of genes involved in cell elongation and hypocotyl growth.

193 within the molecular framework driving rapid hypocotyl growth.

194 (PIF3), a key transcription factor promoting hypocotyl growth.

195 4), a key transcription factor that promotes hypocotyl growth.

196 esponsible for a component of ploidy-related hypocotyl growth.

197 In Arabidopsis, the seedling hypocotyl has emerged as an exemplar model system to stu

198 The middle and upper hypocotyl have a greater requirement for GA to promote c

199 UV-B also stabilizes the bHLH protein LONG HYPOCOTYL IN FAR RED (HFR1), which can bind to and inhib

200 date a previously unidentified role for long hypocotyl in far red 1, a negative regulator of the PIFs

201 levels of the transcriptional cofactor LONG HYPOCOTYL IN FAR RED1, which also binds to PIF1 and othe

202 This latter regulation requires LONG HYPOCOTYL IN FAR RED1/SLENDER IN CANOPY SHADE1 and phyto

203 mics of ELONGATED HYPOCOTYL 5 (HY5) and LONG HYPOCOTYL IN FAR-RED (HFR1) proteins; and the epistatic

204 In addition, the long hypocotyl in far-red light phenotype of the laf6 mutant

205 lves the COP1/SPA ubiquitination target LONG HYPOCOTYL IN FR LIGHT1 but not ELONGATED HYPOCOTYL5.

206 anced activity in the vascular region of the hypocotyl in response to cotreatment of Suc and sulfonam

207 Arabidopsis (Arabidopsis thaliana) Short Hypocotyl in White Light1 (SHW1) encodes a Ser-Arg-Asp-r

208 During secondary growth of hypocotyls in Arabidopsis thaliana, the xylem undergoes

209 of free auxin in specialized organs such as hypocotyls in response to shade and high temperature.

210 ugar-sensing mechanisms in the elongation of hypocotyls in response to Suc.

211 , HOS1-defective mutants exhibited elongated hypocotyls in the light.

212 tivity in specific regions of both roots and hypocotyls, in good correlation with transcriptomic data

213 ile constitutive expression of PCH1 shortens hypocotyls independent of day length.

214 in transport and that auxin transport in the hypocotyl is a prerequisite for phot1-dependent hypocoty

215 The Arabidopsis thaliana hypocotyl is a robust system for studying the interplay

216 IF- and light-regulated stomata formation in hypocotyls is critically dependent on LLM-domain B-GATA

217 One of these, LATE ELONGATED HYPOCOTYL, is known in A. thaliana to regulate many stre

218 e with Sl-MMPs in the apoplast of the tomato hypocotyl, it exhibited increased stability in transgeni

219 ent and aboveground plant tissues, including hypocotyls, leaves, and stems.

220 ined, these results show that PIF3 regulates hypocotyl length downstream, whereas PIF4 and PIF5 regul

221 ntrast to pif4 and pif5 mutants, the reduced hypocotyl length in pif3 cannot be rescued by either ACC

222 delineate Arabidopsis (Arabidopsis thaliana) hypocotyl length kinetics in response to ethylene and sh

223 h downstream, whereas PIF4 and PIF5 regulate hypocotyl length upstream of an auxin and ethylene casca

224 th AtHY5, which does not cause any change in hypocotyl length when overexpressed in Arabidopsis, the

225 BBX19 expression by RNA interference reduces hypocotyl length, and its constitutive expression promot

226 t factors that impact the photoregulation of hypocotyl length, we conducted comparative gene expressi

227 ted to leaf number and complexity as well as hypocotyl length.

228 ens period, delays flowering time and alters hypocotyl length.

229 the phytochrome-dependent photoregulation of hypocotyl length.

230 yB that have roles in the photoregulation of hypocotyl length.

231 abidopsis plants, resulting in a decrease in hypocotyl length.

232 onfucosylated xyloglucan, rescued dark-grown hypocotyl lengthening of ABP1 knockdown seedlings.

233 e amplitude of the rhythms of late elongated hypocotyl (LHY) and circadian clock associated1 (CCA1) e

234 sic mutant alleles accumulate LATE ELONGATED HYPOCOTYL (LHY) and CIRCADIAN CLOCK ASSOCIATED1 (CCA1) s

235 DIAN CLOCK ASSOCIATED1 (CCA1)/LATE ELONGATED HYPOCOTYL (LHY) and the evening gene TIMING OF CAB EXPRE

236 clock associated 1 (CCA1) and late elongated hypocotyl (LHY) to restrict their expression to near daw

237 ian Clock-associated 1 (CCA1)/late elongated hypocotyl (LHY).

238 CLOCK ASSOCIATED1 (CCA1) and LATE ELONGATED HYPOCOTYL (LHY).

239 rcadian clock gene P. hybrida LATE ELONGATED HYPOCOTYL (LHY; PhLHY) regulates the daily expression pa

240 Finally, the decreased XyG abundance in hypocotyl longitudinal cell walls of germinating embryos

241 pstream regulators we identified a LONG PALE HYPOCOTYL (LPH) gene whose activity is indispensable for

242 early flowering plants with overly elongated hypocotyls mainly in short days.

243 d the phytochrome-chromophore-deficient long hypocotyl mutant hy1.

244 is approach has been used exclusively on the hypocotyl of Arabidopsis thaliana.

245 The hypocotyls of Arabidopsis (Arabidopsis thaliana) also el

246 also disordered the cellular arrangement of hypocotyls of Arabidopsis plants, resulting in a decreas

247 organ (cotyledons) and in rapidly elongating hypocotyls of Arabidopsis thaliana PIFs initiate transcr

248 eduction of basipetal auxin transport in the hypocotyls of d6pk as well as in pin mutants.

249 Hypocotyls of SOB3 mutant seedlings grown in white light

250 Etiolated hypocotyls of the quadruple atlazy1,2,3,4 mutant were es

251 Examination of hypocotyls of these plants revealed normal vasculature i

252 activation of the reporter in the roots and hypocotyls of unwounded seedlings.

253 In Arabidopsis dark-grown hypocotyls, one PME (AtPME3) and one PMEI (AtPMEI7) were

254 n CIRCADIAN CLOCK ASSOCIATED1/LATE ELONGATED HYPOCOTYL or GIGANTEA demonstrated their requirement for

255 ng SAUR63:GFP or SAUR63:GUS fusions had long hypocotyls, petals and stamen filaments, suggesting that

256 OB3/AHL29 and ESC/AHL27 confer a subtle long-hypocotyl phenotype compared with the WT or either singl

257 ssense allele sob3-6 confers a dramatic long-hypocotyl phenotype in the light.

258 ruption of BAS1 and SOB7 abolishes the short-hypocotyl phenotype of ATAF2 loss-of-function seedlings

259 background is likely causative for the long hypocotyl phenotype previously attributed to disrupted A

260 Specifically, SOB3 mutant hypocotyl phenotypes, which are readily apparent when th

261 During hypocotyl photomorphogenesis, light signals are sensed b

262 We measured either volume changes of single hypocotyl protoplasts or hypocotyl growth, both at high

263 Elongation of the Arabidopsis hypocotyl pushes the shoot-producing meristem out of the

264 ibuted differently within cotyledons and the hypocotyl/radicle axis in embryos of the oilseed crop Ca

265 odels predicted that the outer cotyledon and hypocotyl/radicle generate the bulk of plastidic reducta

266 is predicted for the outer cotyledon and the hypocotyl/radicle only.

267 lar level with reduced cell expansion in the hypocotyl relative to the wild type.

268 de (BL) insensitive, while the double mutant hypocotyls remain sensitive.

269 In turn, these rhythms gated the hypocotyl response to red light, in part by changing the

270 ls of the unexpanded light-grown Arabidopsis hypocotyl results in a transient burst of anisotropic ce

271 is defective in cell expansion in dark-grown hypocotyls, roots, and adult plants.

272 vement of [(3)H]indole-3-acetic acid in both hypocotyl sections and primary roots of Arabidopsis seed

273 oy a custom image-based method for measuring hypocotyl segment elongation with high resolution and a

274 In contrast, hypocotyl segments overexpressing a PP2C.D phosphatase a

275 sing the mechanical extensibility of excised hypocotyl segments.

276 og BBX24 regulate deetiolation processes and hypocotyl shade avoidance response in an additive manner

277 ed from upper (growing) regions of 3-day-old hypocotyls showed ploidy levels to be lower in abcb19 mu

278 pch1 seedlings have overly elongated hypocotyls specifically under short days while constitut

279 ss of ClpP3 (clpp3-1) leads to arrest at the hypocotyl stage; this developmental arrest can be remove

280 evel of basipetally transported auxin in the hypocotyl than did wild-type seedlings, and had wavy hyp

281 In hypocotyls, the combination of circadian expression of P

282 signaling sensitizes the dark-grown seedling hypocotyl to the presence of obstacles, overriding gravi

283 c stress in roots and ABA transport from the hypocotyl to the shoot and root.

284 ulations available for Arabidopsis etiolated hypocotyls to clarify how auxin is perceived and the dow

285 ncrease the growth of specific organs (e.g., hypocotyls) to enhance access to sunlight.

286 downregulate symplasmic permeability during hypocotyl tropic response.

287 Cell elongation in the basal part of the hypocotyl under -DIF was restored by both 1-aminocyclopr

288 SHORT HYPOCOTYL UNDER BLUE1 (SHB1) is a key regulatory gene of

289 Overexpression of Arabidopsis SHORT HYPOCOTYL UNDER BLUE1::uidA (SHB1:uidA) in canola produc

290 Loss-of-function mutants show an elongated hypocotyl under far-red light and are impaired in other

291 ly suppressed excessive radial growth of the hypocotyl vasculature during secondary growth.

292 We focused on phenotypes of hypocotyl vasculatures caused by double mutation in EREC

293 This newly synthesized auxin moves to the hypocotyl where it induces elongation of hypocotyl cells

294 ngs grow initially through elongation of the hypocotyl, which is regulated by signaling pathways that

295 bidopsis thaliana line with longer etiolated hypocotyls, which overexpresses a gene encoding a polyga

296 expansion; for example, epidermal cells from hypocotyls with reduced CP are longer than wild-type cel

297 -based reporter of mitosis throughout abcb19 hypocotyls without an equivalent effect on mitosis promp

298 Upon floral transition, the hypocotyl xylem gained a competency to respond to GA in

299 ARY WALL THICKENING PROMOTING FACTORs in the hypocotyl xylem.

300 iformly expressed throughout the Arabidopsis hypocotyl, yet decapitation experiments have localized t