ライフサイエンスコーパス: hypocotyl elongation

1 r Phytochrome Interacting Factor 4 (PIF4) on hypocotyl elongation.

2 ive regulator of phyB-mediated inhibition of hypocotyl elongation.

3 lowering, circadian cotyledon movements, and hypocotyl elongation.

4 utamate and glycine synergism also regulates hypocotyl elongation.

5 oreceptor mediating blue light inhibition of hypocotyl elongation.

6 shoot initiation, and brassinolide-mediated hypocotyl elongation.

7 mental processes including de-etiolation and hypocotyl elongation.

8 diating the blue light-induced inhibition of hypocotyl elongation.

9 toreceptor, cryptochrome (CRY1), involved in hypocotyl elongation.

10 as observed in light-dependent inhibition of hypocotyl elongation.

11 ectable consequences for the photocontrol of hypocotyl elongation.

12 rease in auxin levels resulting in increased hypocotyl elongation.

13 ce by DELLAs correlates closely with reduced hypocotyl elongation.

14 dopsis thaliana which mediates inhibition of hypocotyl elongation.

15 although it has no significant effect on the hypocotyl elongation.

16 altered responses to NPA in root curling and hypocotyl elongation.

17 of vegetative photomorphogenesis, including hypocotyl elongation.

18 ngs have opposite BR-response phenotypes for hypocotyl elongation.

19 HXK1)-mediated pathway to regulate etiolated hypocotyl elongation.

20 eviously shown to be important regulators of hypocotyl elongation.

21 ular anisotropy driving Arabidopsis thaliana hypocotyl elongation.

22 ding seed germination, stomatal closure, and hypocotyl elongation.

23 how that cotyledon-generated auxin regulates hypocotyl elongation.

24 curately describes wild-type root growth and hypocotyl elongation.

25 nced resistance to DELLA accumulation during hypocotyl elongation.

26 mote the known shade-induced acceleration of hypocotyl elongation.

27 NG FACTOR4 (PIF4) and PIF5 in the control of hypocotyl elongation.

28 h no reduction in postgermination radical or hypocotyl elongation.

29 ponents acting downstream of phyA to inhibit hypocotyl elongation.

30 oral initiation and blue light inhibition of hypocotyl elongation.

31 re and is required for temperature-dependent hypocotyl elongation.

32 fore abrogate PIF3-mediated light control of hypocotyl elongation.

33 acid [IAA], 2,4-D, 1-NAA) have no effect on hypocotyl elongation.

34 Unlike 2,4-D alone, this pro-2,4-D enhanced hypocotyl elongation.

35 gs and identified 100 compounds that promote hypocotyl elongation.

36 opmental defects, including reduced root and hypocotyl elongation.

37 rfi2-1 mutation in the expression of CO and hypocotyl elongation.

38 iting a thermotolerance defect as assayed by hypocotyl elongation, 10-day-old hot1 seedlings were als

39 High-temperature-mediated hypocotyl elongation additionally involves localized cha

40 umpkin hypocotyls during the period of rapid hypocotyl elongation after which mRNA levels declined, w

41 with SPY acting in the GA pathway to inhibit hypocotyl elongation and also acting as a light-regulate

42 cryptochrome), which mediates inhibition of hypocotyl elongation and anthocyanin biosynthesis, has r

43 s concluded that CRY1-mediated inhibition of hypocotyl elongation and anthocyanin production requires

44 ns similar to cry1 and include inhibition of hypocotyl elongation and blue light-dependent anthocyani

45 wth medium greatly enhances the reduction in hypocotyl elongation and cellulose content of shv3svl1 T

46 s to specifically mediate SHB1 regulation of hypocotyl elongation and CHLOROPHYLL a/b BINDING PROTEIN

47 B and cry2 in the control of flowering time, hypocotyl elongation and circadian period by the clock.

48 s show only small defects in photocontrol of hypocotyl elongation and cotyledon opening under prolong

49 motion of cotyledon expansion, repression of hypocotyl elongation and flowering time in addition to o

50 endent circadian period effects, and altered hypocotyl elongation and flowering time.

51 ariety of physiological processes, including hypocotyl elongation and flowering time.

52 ole of phyA-dependent CKI1 expression in the hypocotyl elongation and hook development during skotomo

53 ings, BMAA caused a 2- to 3-fold increase in hypocotyl elongation and inhibited cotyledon opening dur

54 functionally implicated in the inhibition of hypocotyl elongation and known to be a direct target of

55 f auxin-regulated growth processes including hypocotyl elongation and lateral root formation.

56 hypersensitivity to red light inhibition of hypocotyl elongation and light-regulated gene expression

57 me 2 (CRY2) mediate blue light inhibition of hypocotyl elongation and long-day (LD) promotion of flor

58 t receptor that mediates light inhibition of hypocotyl elongation and long-day promotion of floral in

59 ving a unique role in regulating FR-mediated hypocotyl elongation and meristem- and/or leaf primordia

60 However, ethylene-induced inhibition of hypocotyl elongation and petiole epinasty are normal in

61 e 2 (CRY2) mediates blue light inhibition of hypocotyl elongation and photoperiodic control of floral

62 ight receptor regulating light inhibition of hypocotyl elongation and photoperiodic flowering in Arab

63 many aspects of plant development, including hypocotyl elongation and photoperiodic induction of flow

64 that CRY2 mediates blue light inhibition of hypocotyl elongation and photoperiodic promotion of flor

65 etween light and auxin signaling pathways in hypocotyl elongation and phototropism responses.

66 bri1, did not respond to brassinosteroids in hypocotyl elongation and primary root inhibition assays,

67 t photomorphogenesis, such as suppression of hypocotyl elongation and promotion of cotyledon expansio

68 ion, but both were active in AtD14-dependent hypocotyl elongation and secondary shoot growth.

69 assic glucose signaling responses, including hypocotyl elongation and seed germination, with exposure

70 lines, including FR-dependent inhibition of hypocotyl elongation and stimulation of anthocyanin accu

71 mediates blue light-dependent inhibition of hypocotyl elongation and stimulation of cotyledon openin

72 seedling development, such as inhibition of hypocotyl elongation and the promotion of greening, acti

73 vibrational stimulation, with an increase in hypocotyl elongation and up-regulation of TCH gene expre

74 t of an end-of-day pulse of far-red light on hypocotyl elongation, and a decrease in the number of ro

75 ppressed chlorophyll synthesis, promotion of hypocotyl elongation, and formation of a closed apical h

76 erellic acid (GA) promotes germination, stem/hypocotyl elongation, and leaf expansion during seedling

77 Root growth and hypocotyl elongation are convenient downstream physiolog

78 Here, we study hypocotyl elongation as a proxy for shoot elongation and

79 However, hypocotyl elongation assays indicated that suppression o

80 on of root growth, lateral root development, hypocotyl elongation at high temperature, and apical dom

81 nd class of IAA precursor, displayed reduced hypocotyl elongation but normal cotyledon size and only

82 active in KAI2-dependent seed germination or hypocotyl elongation, but both were active in AtD14-depe

83 ty of the inhibitory effect of blue light on hypocotyl elongation, but phytochrome photoreceptors als

84 by increasing GA biosynthesis, but inhibits hypocotyl elongation by decreasing the responsiveness to

85 of response from inhibition to promotion of hypocotyl elongation by light.

86 The extent of inhibition of hypocotyl elongation by NPA increased in a fluence-rate-

87 ased upon induction of R-hypersensitivity of hypocotyl elongation by overexpression of the apoprotein

88 Seedling root growth and hypocotyl elongation can be accurately predicted using a

89 adruple pifq mutant displays clearly reduced hypocotyl elongation compared to wild-type in response t

90 esC), Arabidopsis seedlings exhibit dramatic hypocotyl elongation compared with seedlings grown at 20

91 tomorphogenic responses in plants, including hypocotyl elongation, cotyledon expansion, and control o

92 rphogenesis, as illustrated by inhibition of hypocotyl elongation, cotyledon opening, and leaf greeni

93 We show that the hypocotyl elongation defect in the det3 mutant is condit

94 ry and coding sequences of RCN1, whereas the hypocotyl elongation defect of rcn1 plants can be comple

95 emonstrate that the magnitude of Suc-induced hypocotyl elongation depends on the day length and light

96 light/dark cycles, we found that Suc-induced hypocotyl elongation did not occur in tps1 mutants and o

97 tifies apical hook opening and inhibition of hypocotyl elongation during photomorphogenesis of Arabid

98 16 functions to promote seed germination and hypocotyl elongation during the early stages of Arabidop

99 lue light (BL) rapidly and strongly inhibits hypocotyl elongation during the photomorphogenic respons

100 ly mediate light-induced inhibition of stem (hypocotyl) elongation during the development of photoaut

101 and differentiation, and the retardation in hypocotyl elongation enables growth and development in d

102 nsport, growth, and gravitropism, while rcn1 hypocotyl elongation exhibited enhanced ethylene respons

103 st few seconds of blue light and to suppress hypocotyl elongation for at least 120 min.

104 We developed a screen, based on hypocotyl elongation, for mutants of Arabidopsis thalian

105 reviously uncharacterized LHE (LIGHT-INDUCED HYPOCOTYL ELONGATION) gene, which we show impacts light-

106 in-signaling machinery to regulate etiolated hypocotyl elongation growth in Arabidopsis.

107 iologically, Glc and BR interact to regulate hypocotyl elongation growth of etiolated Arabidopsis (Ar

108 -6 double mutant displayed severe defects in hypocotyl elongation growth similar to its bri1-6 parent

109 of four loci required for thermotolerance of hypocotyl elongation, hot1-1, hot2-1, hot3-1, and hot4-1

110 In seedlings, these proteins repress hypocotyl elongation in a daylength- and sucrose-depende

111 ibit folate biosynthesis in plants, restrict hypocotyl elongation in a sugar-dependent fashion.

112 The pattern of hypocotyl elongation in constant light includes a daily

113 ve, photoreceptor imposing the inhibition of hypocotyl elongation in deetiolating seedlings in respon

114 ecific, as the bim409 mutant exhibits normal hypocotyl elongation in etiolated (dark grown) plants (+

115 at, when overexpressed, resulted in enhanced hypocotyl elongation in etiolated Arabidopsis thaliana s

116 ke other BR-deficient mutants, the defect of hypocotyl elongation in fk-J79 cannot be corrected by ex

117 d mutants that exhibit reduced inhibition of hypocotyl elongation in FRc.

118 results lead us to the view that the rate of hypocotyl elongation in light is determined by at least

119 ediating a signal that underlies Suc-induced hypocotyl elongation in light/dark cycles.

120 showed significantly enhanced inhibition of hypocotyl elongation in low-white, red, far-red, blue, a

121 nt with ethylene or auxin inhibitors reduced hypocotyl elongation in PIF4 overexpressor (PIF4ox) and

122 gibberellins (GAs) antagonistically regulate hypocotyl elongation in plants.

123 essing phyC displayed enhanced inhibition of hypocotyl elongation in Rc, but were unchanged in respon

124 A similar additive effect on hypocotyl elongation in red and blue light is also obser

125 to modulate the phyB-mediated inhibition of hypocotyl elongation in red light and to function togeth

126 We measured a decrease in hypocotyl elongation in response to acidic pH in the tra

127 ACTING FACTOR 4 (PIF4), is also required for hypocotyl elongation in response to high temperature.

128 verexpression of AtRALF23 impairs BL-induced hypocotyl elongation in seedlings, and mature overexpres

129 In contrast, hypocotyl elongation in the dark was decreased independe

130 Arabidopsis thaliana developmental response, hypocotyl elongation in the dark, to detail the underpin

131 e oil and cannot utilize applied sucrose for hypocotyl elongation in the dark.

132 egulated genes in the dark, and HY5 inhibits hypocotyl elongation in the light.

133 tions of additional AHL genes in suppressing hypocotyl elongation in the light.

134 By contrast, inhibition of hypocotyl elongation in xct is hyposensitive to red ligh

135 e fluence rate response where suppression of hypocotyl elongation increases incrementally with light

136 induction of seed germination, inhibition of hypocotyl elongation, induction of cotyledon opening, ra

137 Hypocotyl elongation is a highly coordinated physiologic

138 ive to floral development is accelerated and hypocotyl elongation is accentuated in these mutants und

139 ansition from darkness to light, the rate of hypocotyl elongation is determined from the integration

140 The effect of BMAA on hypocotyl elongation is light specific.

141 Under short-day (SD) photocycles, hypocotyl elongation is maximal at dawn, being promoted

142 elicits shade- and high temperature-induced hypocotyl elongation largely independently of 3-IPA-medi

143 As GA signalling directs hypocotyl elongation largely through promoting PIF activ

144 s throughout the plant life cycle, including hypocotyl elongation, leaf expansion and apical dominanc

145 e overexpressors has differential effects on hypocotyl elongation, leaf shape, and petiole length, as

146 In contrast, the reduced hypocotyl elongation of ethylene biosynthesis and signal

147 Mutation in DET1 changed the sensitivity of hypocotyl elongation of mutant seedlings to GA and paclo

148 that causes a two- to three-fold increase in hypocotyl elongation on Arabidopsis seedlings grown in t

149 e of these genes in the control of greening, hypocotyl elongation, phyllotaxy, floral organ initiatio

150 e decreased response to red light (R) of the hypocotyl elongation rate in these mutants.

151 o far-red, red, and blue-light inhibition of hypocotyl elongation, reduced chlorophyll and anthocyani

152 g deoxystrigolactones to inhibit Arabidopsis hypocotyl elongation, regulate seedling gene expression,

153 prolonged continuous red light (Rc) promotes hypocotyl elongation relative to dark controls, the reci

154 The normal BMAA-induced hypocotyl elongation response observed on wild-type seed

155 ants are shown to lack an end-of-day-far-red hypocotyl elongation response that requires a stable Pfr

156 articipates positively in the control of the hypocotyl elongation response to plant proximity, a role

157 UV-A, and green light for the inhibition of hypocotyl elongation response.

158 h red and far-red light in the inhibition of hypocotyl elongation response; a classic phytochrome phe

159 ophore inactivation and associated disparate hypocotyl elongation responses under far-red (FR) light.

160 ure in distinct developmental traits such as hypocotyl elongation, root elongation, and flowering tim

161 thermotolerance defects in seed germination, hypocotyl elongation, root growth, and seedling survival

162 alterations of red light effects on seedling hypocotyl elongation, rosette leaf morphology, and chlor

163 lthough only SMAX1 regulates germination and hypocotyl elongation, SMAX1 and SMXL6,7,8 have complemen

164 nse was necessary for (a). the inhibition of hypocotyl elongation that develops within minutes of the

165 Plants lacking PGX1 display reduced hypocotyl elongation that is complemented by transgenic

166 ression in fhy1-3 caused an insensitivity of hypocotyl elongation to FR and blue light (B) indistingu

167 characteristics include hypersensitivity of hypocotyl elongation to inhibition by white, blue, red,

168 erring ethylene insensitivity restore normal hypocotyl elongation to rcn1.

169 ts in fertility, and enhanced sensitivity of hypocotyl elongation to red but not to far-red or blue l

170 me-mediated degradation of phytochrome A and hypocotyl elongation under far-red light.

171 f COP1 function, as indicated by exaggerated hypocotyl elongation under light conditions.

172 We found that Suc-induced hypocotyl elongation under light/dark cycles does not in

173 ight- and phytochrome-mediated regulation of hypocotyl elongation under red (R) and FR illumination.

174 the physiological effects of IBA and IAA on hypocotyl elongation under several light conditions were

175 xin and gibberellin signaling in Suc-induced hypocotyl elongation under short photoperiods.

176 er transcription factor that participates in hypocotyl elongation under short-day conditions.

177 retain and exert a dual capacity to modulate hypocotyl elongation under these conditions, by concomit

178 nclude that the RCN1 protein affects overall hypocotyl elongation via negative regulation of ethylene

179 The rhythm of hypocotyl elongation was entrained by light-dark cycles

180 The fluence rate response of hypocotyl elongation was examined and showed a biphasic

181 When inhibition of hypocotyl elongation was measured, elf3 mutant seedlings

182 ediated by glutamate and glycine, as well as hypocotyl elongation, were inhibited by 6,7-dinitroquino

183 y to red and far-red light in the control of hypocotyl elongation, whereas increments in TOC1 gene do

184 display a phenotype of reduced inhibition of hypocotyl elongation, which is specific to far-red light

185 -regulated protein stability drives rhythmic hypocotyl elongation with peak growth at dawn.

186 strated that protein levels increased in the hypocotyl elongation zone when shifted from the dark to