ライフサイエンスコーパス: hypocotyl growth

1 4), a key transcription factor that promotes hypocotyl growth.

2 (PIF3), a key transcription factor promoting hypocotyl growth.

3 esponsible for a component of ploidy-related hypocotyl growth.

4 lly understood how the phytochromes modulate hypocotyl growth.

5 that HMR acts upstream of PIFs in regulating hypocotyl growth.

6 duced an auxin-like swelling response but no hypocotyl growth.

7 apyrases at least as rapidly as it inhibited hypocotyl growth.

8 ion of genes involved in cell elongation and hypocotyl growth.

9 within the molecular framework driving rapid hypocotyl growth.

10 idopsis thaliana, high-resolution studies of hypocotyl growth accomplished by computer-assisted elect

11 to root elongation, but cytokinin effects on hypocotyl growth and ethylene synthesis in these seedlin

12 ga/spy/gal-3 is almost insensitive to GA for hypocotyl growth and its bolting stem is taller than the

13 epistatic to hrb1 under blue light for both hypocotyl growth and light-regulated gene expression res

14 nment, this dual action will strongly retard hypocotyl growth and promote cotyledon opening and expan

15 ions in phyA largely suppress the randomized-hypocotyl growth and the short-hypocotyl phenotype of th

16 ses of gene expression, cotyledon unfolding, hypocotyl growth, and greening observed in the phyA muta

17 tyledon unhooking, unfolding, and expansion, hypocotyl growth, and the accumulation of chlorophylls a

18 the circadian clock, and we review seedling hypocotyl growth as a paradigm of PIFs acting at the int

19 ype seed and mutant seedlings have decreased hypocotyl growth as compared to wildtype seedlings when

20 Hypocotyl growth assays in monochromatic light and micro

21 Together with hypocotyl growth assays showing that the sensitivity and

22 red the effects of a reduced PMF on root and hypocotyl growth, ATP-induced skewed root growth, and ra

23 e changes of single hypocotyl protoplasts or hypocotyl growth, both at high temporal resolution.

24 is both necessary and sufficient to initiate hypocotyl growth, but we also provide evidence for the f

25 We present a model for regulation of hypocotyl growth by specific molecules found in this pat

26 ool in 5ptase11 mutants, we correlated these hypocotyl growth changes with a small increase in the 5P

27 Maximal hypocotyl growth coincides with the phase during which t

28 The xct mutation also causes sugar-specific hypocotyl growth defects, in which mutants are short in

29 g elements and their relationship underlying hypocotyl growth direction.

30 ences the auxin transport process to control hypocotyl growth during de-etiolation.

31 with the negative regulatory role of HOS1 in hypocotyl growth, HOS1-defective mutants exhibited elong

32 ATAF2 modulates hypocotyl growth in a light-dependent manner, with the p

33 night temperature difference [-DIF]) inhibit hypocotyl growth in Arabidopsis (Arabidopsis thaliana).

34 luence-rate blue light (BL) rapidly inhibits hypocotyl growth in Arabidopsis, as in other species, af

35 r basis for the phyB-mediated suppression of hypocotyl growth in Arabidopsis.

36 f red light and a hypersensitive response in hypocotyl growth in continuous red light of higher fluen

37 , defects in cell-cell adhesion, and reduced hypocotyl growth in etiolated seedlings.

38 Previous studies have shown that hypocotyl growth in low red to far-red shade is largely

39 det1 did not show significant inhibition of hypocotyl growth in response to UV-B, while det2 was str

40 g is required in many cell types for correct hypocotyl growth in shade, with a key role for the epide

41 xpression, coinciding with the initiation of hypocotyl growth in the early evening, is positively cor

42 the molecular basis for circadian gating of hypocotyl growth in the early evening.

43 brief heat shocks enhance the inhibition of hypocotyl growth induced by light perceived by phytochro

44 In wild-type Arabidopsis seedlings, hypocotyl growth inhibition and cotyledon expansion were

45 uced AtPP7 expression levels exhibit loss of hypocotyl growth inhibition and display limited cotyledo

46 Phototropism and hypocotyl growth inhibition are modulated by the coactio

47 nterference of phytochrome A (phyA)-mediated hypocotyl growth inhibition in far-red (FR) light.

48 Kinetic analyses of hypocotyl growth inhibition in response to ethylene and

49 hat SOB3 and ESC act redundantly to modulate hypocotyl growth inhibition in response to light.

50 on the order of minutes, that phyA initiated hypocotyl growth inhibition upon the onset of continuous

51 esses the phytochrome-modulated responses of hypocotyl growth inhibition, sucrose-stimulated anthocya

52 onse to photoperiod and a blue light-induced hypocotyl growth inhibition.

53 This suggests that hypocotyl growth is elicited by both local and distal au

54 etic pigments are promoted by light, whereas hypocotyl growth is inhibited.

55 onditions studied, from UV to far-red, early hypocotyl growth is rapidly and robustly suppressed with

56 In contrast to vastly studied hypocotyl growth, little is known about diel regulation

57 Hypocotyl growth of B19OE seedlings in red light was ver

58 We compared root and hypocotyl growth of the single, double, and triple hydro

59 cessary for light-dependent randomization of hypocotyl growth orientation.

60 B transgene complements the phyB-1 red light hypocotyl growth phenotype completely, the PB-phyD and P

61 Loss of ABCB19 partially suppressed the cry1 hypocotyl growth phenotype in blue light.

62 s phenotype is the opposite of the increased hypocotyl growth phenotype previously described for othe

63 ream modules participate in diurnal rhythmic hypocotyl growth: PIF4 and/or PIF5 modulation of auxin-r

64 g converge to influence the transcription of hypocotyl growth-promoting SAUR19 subfamily members.

65 es, we found that SPA1 caused an increase in hypocotyl growth rate after approximately 2 h of continu

66 ight resulted in automatic quantification of hypocotyl growth rate, apical hook opening, and phototro

67 Auxin signaling and ABCB19 protein levels, hypocotyl growth rates, and apical hook opening were mea

68 me and root growth; control of cotyledon and hypocotyl growth requires simultaneous phyA activity in

69 lue-light photoreceptors to coordinate these hypocotyl growth responses is still unclear.

70 alpha3 triple mutants also displayed reduced hypocotyl growth, smaller cotyledon size and a reduced n

71 hypocotyl, which reduced the sensitivity of hypocotyl growth specifically to blue light in long-term

72 Hypocotyl growth suppression by blue light was assessed

73 ion in the regulation of gene expression and hypocotyl growth suppression in Arabidopsis.

74 ng; instead, it showed auxin activity in the hypocotyl growth test.

75 g establishment, blue and red light suppress hypocotyl growth through the cryptochrome 1 (cry1) and p

76 and the conditional use of GA-ATHB5-mediated hypocotyl growth under optimal conditions may be used to

77 HsfB2b is also involved in the regulation of hypocotyl growth under warm, short days.

78 erotrimeric G protein in R and FR control of hypocotyl growth using a loss-of-function approach.

79 ic diurnal variation in Arabidopsis thaliana hypocotyl growth, we found that cellulose synthesis and

80 ing phenotypes, including increased stem and hypocotyl growth, which increases the likelihood of outg