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1 cases had a consistent clinical picture and hypoglycorrhachia.
2 The laboratory signature is hypoglycorrhachia.
3 velopmental delay, acquired microcephaly and hypoglycorrhachia.
4 quired microcephaly, spasticity, ataxia, and hypoglycorrhachia.
5 y more frequently than NMOSD (p < 0.05); CSF hypoglycorrhachia (11%, p = 0.25) and elevated angiotens
7 disease or death were preterm birth, severe hypoglycorrhachia, CSF/blood glucose ratio <0.10, and mo
8 the glucose transporter GLUT1, resulting in hypoglycorrhachia, infantile seizures, and developmental
9 G), impaired motor activity, incoordination, hypoglycorrhachia, microencephaly, decreased brain gluco
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