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1  cases had a consistent clinical picture and hypoglycorrhachia.
2                  The laboratory signature is hypoglycorrhachia.
3 velopmental delay, acquired microcephaly and hypoglycorrhachia.
4 quired microcephaly, spasticity, ataxia, and hypoglycorrhachia.
5 y more frequently than NMOSD (p < 0.05); CSF hypoglycorrhachia (11%, p = 0.25) and elevated angiotens
6                                              Hypoglycorrhachia and decreased erythrocyte 3-OMG uptake
7  disease or death were preterm birth, severe hypoglycorrhachia, CSF/blood glucose ratio <0.10, and mo
8  the glucose transporter GLUT1, resulting in hypoglycorrhachia, infantile seizures, and developmental
9 G), impaired motor activity, incoordination, hypoglycorrhachia, microencephaly, decreased brain gluco

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