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1 ult mouse beta-cell caused hyperglycemia and hypoinsulinemia.
2 g, deficits in DAT function that result from hypoinsulinemia.
3 eks, mice on HFD exhibited hyperglycemia and hypoinsulinemia.
4 tal diabetes, evidenced by hyperglycemia and hypoinsulinemia.
5 demia, hepatic steatosis, hyperglycemia, and hypoinsulinemia.
6 alformations, impaired glucose tolerance and hypoinsulinemia.
7 diabetes with insulitis, hyperglycemia, and hypoinsulinemia.
8 topic p53 gene encoding Delta40p53 developed hypoinsulinemia and glucose intolerance by 3 months of a
10 glycemia and glucose intolerance with severe hypoinsulinemia and reduced islet insulin content and gl
11 body weight, caused fetal hyperglycemia and hypoinsulinemia, and decreased the relative islet area.
16 e literature on the effect of hyperglycemia, hypoinsulinemia, and ketoacidosis, as well as the role o
17 l fat deposition, and induced hyperglycemia, hypoinsulinemia, and low-density lipoprotein hypercholes
18 ad reduced beta-cell proliferation and mass, hypoinsulinemia, and mild diabetes, phenotypes rescued b
19 isplayed higher blood glucose levels, marked hypoinsulinemia, and reduced beta-cell mass compared wit
21 ocol I, hyperglycemic (approximately 10 mM) -hypoinsulinemia (approximately 40 pM); protocol II, eugl
22 breakdown, an acute gluconeogenic effect of hypoinsulinemia becomes manifest, whereas inhibition of
23 ssue, hypoadiponectinemia, hypoglycemia, and hypoinsulinemia but with relatively normal glucose dispo
24 pha deletion, mice develop hyperglycemia and hypoinsulinemia, due to defective beta cell function tha
25 stricted feeding (RF), immediately creates a hypoinsulinemia during the active phase, which initiates
26 eome of 120-d-old OVE26 transgenic mice with hypoinsulinemia, hyperglycemia, hyperlipidemia, and prot
27 lpha(-/-) mice, the p85beta(-/-) mice showed hypoinsulinemia, hypoglycemia, and improved insulin sens
28 We show that Asna1(beta-/-) mice develop hypoinsulinemia, impaired insulin secretion, and glucose
31 ensitivity of a brain reward system and that hypoinsulinemia may be the common factor in food restric
32 of a euglycemic clamp study, indicating that hypoinsulinemia promotes insulin sensitivity in chronica
33 in-induced hyperglycemia, which results from hypoinsulinemia, reduced expression of renal insulin rec
34 -AL, ALS/Lt mice exhibited hyperglycemia and hypoinsulinemia, whereas ALR/Lt mice maintained normal p
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