ライフサイエンスコーパス: hypokalemic

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1 rized by polyhydramnios, premature delivery, hypokalemic alkalosis and hypercalciuria.

2 lbumin intravenously; it was associated with hypokalemic alkalosis and Pitressin-resistant impairment

3 bule cause Gitelman's syndrome, an inherited hypokalemic alkalosis with hypomagnesemia and hypocalciu

4 ls, and Bartter's syndrome, characterized by hypokalemic alkalosis, hypercalciuria, increased serum a

5 rtension, hyporeninemia, hypoaldosteronemia, hypokalemic alkalosis, low birth weight, failure to thri

6 East, who display profound hypertension and hypokalemic alkalosis.

7 ized CLDN10 mutations in two patients with a hypokalemic-alkalotic salt-losing nephropathy.

8 ized by a non-BS, non-GS autosomal recessive hypokalemic-alkalotic salt-losing phenotype.

9 One hypokalemic, arsenic trioxide-treated patient with relap

10 termine its effect on potassium retention in hypokalemic, critically ill patients.

11 le syndrome is an autosomal dominant form of hypokalemic hypertension due to mutations in the beta- o

12 he proband was referred because of resistant hypokalemic hypertension, suppressed renin and aldostero

13 (BS) is a family of disorders manifested by hypokalemic hypochloremic metabolic alkalosis with normo

14 syndrome is a channelopathy consisting of a hypokalemic, hypomagnesemic, metabolic alkalosis associa

15 ty to acidify urine, variable hyperchloremic hypokalemic metabolic acidosis, nephrocalcinosis, and ne

16 ystem in patients with genetically confirmed hypokalemic periodic paralysis (Cav1.1-R1239H mutation,

17 cks resemble those of patients with familial hypokalemic periodic paralysis (hypoKPP) and resolve wit

18 Hypokalemic periodic paralysis (hypoKPP) is characterize

19 sover trials, one involving 42 subjects with hypokalemic periodic paralysis (HypoPP) and the other in

20 Hypokalemic periodic paralysis (HypoPP) is a familial sk

21 Hypokalemic periodic paralysis (HypoPP) is an ion channe

22 Mutations in this channel cause hypokalemic periodic paralysis (HypoPP), a human autosom

23 nother disorder of sarcolemmal excitability, hypokalemic periodic paralysis (HypoPP), which is usuall

24 Thyrotoxic hypokalemic periodic paralysis (TPP) is characterized by

25 ta may help explain the mechanism underlying hypokalemic periodic paralysis and the patient's worseni

26 ith alterations in channel subunits, such as hypokalemic periodic paralysis in humans and the weaver

27 A novel mutation in a family with hypokalemic periodic paralysis is described.

28 897S) or CaV1.1-R2 (R900S, R1239H) linked to hypokalemic periodic paralysis type 1 and of CaV1.3-R3 (

29 Two patients with genetically confirmed hypokalemic periodic paralysis underwent calf muscle ima

30 In patients with hypokalemic periodic paralysis versus healthy volunteers

31 ge-gated calcium channel of skeletal muscle (hypokalemic periodic paralysis), the neuronal P/Q-type v

32 m channel (CaV1.1) have been associated with hypokalemic periodic paralysis, but how the pathogenesis

33 In the case of hypokalemic periodic paralysis, mutations of one of the

34 utations confer susceptibility to thyrotoxic hypokalemic periodic paralysis.

35 the primary pathophysiological mechanism in hypokalemic periodic paralysis.

36 However, in hypokalemic rats a major component (40%) of uptake could

37 .9 +/- 0.5 microl/g per min in plexuses from hypokalemic rats compared to 4.5 +/- 0.5 microl/g/min in

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