ライフサイエンスコーパス: hypokalemic periodic paralysis

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1 the primary pathophysiological mechanism in hypokalemic periodic paralysis.

2 utations confer susceptibility to thyrotoxic hypokalemic periodic paralysis.

3 ta may help explain the mechanism underlying hypokalemic periodic paralysis and the patient's worseni

4 m channel (CaV1.1) have been associated with hypokalemic periodic paralysis, but how the pathogenesis

5 ystem in patients with genetically confirmed hypokalemic periodic paralysis (Cav1.1-R1239H mutation,

6 cks resemble those of patients with familial hypokalemic periodic paralysis (hypoKPP) and resolve wit

7 Hypokalemic periodic paralysis (hypoKPP) is characterize

8 sover trials, one involving 42 subjects with hypokalemic periodic paralysis (HypoPP) and the other in

9 Hypokalemic periodic paralysis (HypoPP) is a familial sk

10 Hypokalemic periodic paralysis (HypoPP) is an ion channe

11 Mutations in this channel cause hypokalemic periodic paralysis (HypoPP), a human autosom

12 nother disorder of sarcolemmal excitability, hypokalemic periodic paralysis (HypoPP), which is usuall

13 ith alterations in channel subunits, such as hypokalemic periodic paralysis in humans and the weaver

14 A novel mutation in a family with hypokalemic periodic paralysis is described.

15 In the case of hypokalemic periodic paralysis, mutations of one of the

16 ge-gated calcium channel of skeletal muscle (hypokalemic periodic paralysis), the neuronal P/Q-type v

17 Thyrotoxic hypokalemic periodic paralysis (TPP) is characterized by

18 897S) or CaV1.1-R2 (R900S, R1239H) linked to hypokalemic periodic paralysis type 1 and of CaV1.3-R3 (

19 Two patients with genetically confirmed hypokalemic periodic paralysis underwent calf muscle ima

20 In patients with hypokalemic periodic paralysis versus healthy volunteers

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