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1 when using their unaffected arm (directional hypokinesia).
2 find the anatomical correlate of directional hypokinesia.
3 on of D2 receptor responses that manifest in hypokinesia.
4  balance disorders but unexpectedly improved hypokinesia.
5           The most common adverse effect was hypokinesia (3 patients in the lorazepam + haloperidol g
6 eration that is associated behaviorally with hypokinesia and rigidity.
7  MPTP: they developed severe DOPA-responsive hypokinesia and tremor together with unresponsive gait a
8 ed that 5% of participants (n=140) had focal hypokinesia, and 1.5% (n=42) had WM abnormalities.
9 d DA tone in direct pathway neurons, whereas hypokinesia at later stages could result from reduced in
10 ts experience loss of normal motor function (hypokinesia), but can develop uncontrollable movements k
11 ) neurons in Parkinson's disease (PD) causes hypokinesia, but DA replacement therapy can elicit exagg
12 induces a dual effect with an improvement of hypokinesia contrasting with a worsening of DOPA-unrespo
13                                             'Hypokinesia', defined as <50% of control group's mean am
14 ly, none of the patients without directional hypokinesia had a lesion in the same region.
15      We found that patients with directional hypokinesia had a lesion involving the ventral lateral p
16 t over-expressed D1 receptors likely mediate hypokinesia in the D1 transgenic animals.
17            The loss of this ability leads to hypokinesia, known PD motor deficits characterized by a
18                             Occlusion led to hypokinesia of the anterior or anterolateral segments of
19 ade of D2 receptors on D1 agonist-stimulated hypokinesia of the D1 over-expressing animals were inves
20 kinsonism was defined as the (1) presence of hypokinesia or bradykinesia plus at least 1 other cardin
21 jects, five developed marked RV dilation and hypokinesia, paradoxic septal motion, pulmonary hyperten
22 ivo, AM404 caused a mild and slow-developing hypokinesia that was significant 60 min after intracereb
23                         The main finding was hypokinesia without decrement in patients with progressi
24 palsy have a specific finger tap pattern of 'hypokinesia without decrement' and they do not have crit

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