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1 eridol-treated controls, which were markedly hypokinetic.
2 was similar to that of segments that became hypokinetic (1.06 +/- 0.72 versus 1.02 +/- 0.77 mL.g-1.m
4 nce of surgical therapy for the treatment of hypokinetic and hyperkinetic movement disorders has, how
5 surgical treatment for medication-refractory hypokinetic and hyperkinetic movement disorders, and it
6 nternus (GPi) to treat medically intractable hypokinetic and hyperkinetic movement disorders, the cou
10 their role in movement "vigor," leading to "hypokinetic dysarthria" or "hypophonia." This is an addi
12 mutant is sufficient to elicit age-dependent hypokinetic motor deficits and DA neuron loss in vivo, a
15 d indirect pathways, have been linked to the hypokinetic motor output associated with the disease.
18 rkinson's disease (PD) is the most prevalent hypokinetic movement disorder, and symptomatic PD pathog
21 injected with autologous MSCs into akinetic/hypokinetic myocardial territories not receiving bypass
24 left ventricular ischemia, but a dilated and hypokinetic right ventricle suggested pulmonary patholog
25 re, combining dystonia and chorea, and (2) a hypokinetic score, combining bradykinesia and rigidity.
27 tabolism was negatively correlated only with hypokinetic scores in the cuneus (z score = 3.95, P < .0
28 as only limited by false positive results in hypokinetic segments and not by the test interval or cli
29 criteria for predicting recovery of severely hypokinetic segments on preoperative imaging were tracer
31 A) receptor antagonists could ameliorate the hypokinetic symptoms of advanced Parkinson's disease pat
33 oped atypical parkinsonism, and two a severe hypokinetic syndrome with a tendency to eye closure and
34 ta- and gamma1-synucleins were more severely hypokinetic than animals lacking one or the other synucl
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