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1 eridol-treated controls, which were markedly hypokinetic.
2  was similar to that of segments that became hypokinetic (1.06 +/- 0.72 versus 1.02 +/- 0.77 mL.g-1.m
3                                         Most hypokinetic and 40% of akinetic/dyskinetic myocardial se
4 nce of surgical therapy for the treatment of hypokinetic and hyperkinetic movement disorders has, how
5 surgical treatment for medication-refractory hypokinetic and hyperkinetic movement disorders, and it
6 nternus (GPi) to treat medically intractable hypokinetic and hyperkinetic movement disorders, the cou
7  marrow transplantation partially attenuated hypokinetic and motor deficits in HD mice.
8 of viable segments that recover to normal or hypokinetic contractility.
9 vel therapeutic strategies for patients with hypokinetic disorders, such as Parkinson's disease.
10  their role in movement "vigor," leading to "hypokinetic dysarthria" or "hypophonia." This is an addi
11 oregistered with contrast enhancement in the hypokinetic infarct on MRI.
12 mutant is sufficient to elicit age-dependent hypokinetic motor deficits and DA neuron loss in vivo, a
13 which is significantly correlated with their hypokinetic motor deficits.
14  exhibit multiple late-onset and progressive hypokinetic motor deficits.
15 d indirect pathways, have been linked to the hypokinetic motor output associated with the disease.
16 uency band, is believed to be related to the hypokinetic motor symptoms of the disorder.
17 kinsonism (n=4), or a mixed hyperkinetic and hypokinetic movement disorder (n=2).
18 rkinson's disease (PD) is the most prevalent hypokinetic movement disorder, and symptomatic PD pathog
19         Parkinson's disease, the most common hypokinetic movement disorder, has received much attenti
20        The presence of both hyperkinetic and hypokinetic movement disorders such as ataxia, tremor, a
21  injected with autologous MSCs into akinetic/hypokinetic myocardial territories not receiving bypass
22                                     Severely hypokinetic or akinetic segments showed a 2.4-fold incre
23 triatal brain regions and show a paradoxical hypokinetic response to D1 agonist.
24 left ventricular ischemia, but a dilated and hypokinetic right ventricle suggested pulmonary patholog
25 re, combining dystonia and chorea, and (2) a hypokinetic score, combining bradykinesia and rigidity.
26  graded each LV segment as normal (score=1), hypokinetic (score=2), or akinetic (score=3).
27 tabolism was negatively correlated only with hypokinetic scores in the cuneus (z score = 3.95, P < .0
28 as only limited by false positive results in hypokinetic segments and not by the test interval or cli
29 criteria for predicting recovery of severely hypokinetic segments on preoperative imaging were tracer
30 .001) due to false positive results (16%) in hypokinetic segments.
31 A) receptor antagonists could ameliorate the hypokinetic symptoms of advanced Parkinson's disease pat
32 efect conceivably has a role in parkinsonian hypokinetic symptoms.
33 oped atypical parkinsonism, and two a severe hypokinetic syndrome with a tendency to eye closure and
34 ta- and gamma1-synucleins were more severely hypokinetic than animals lacking one or the other synucl

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