1 nide, when simultaneously added with EtOH to
hyponatremic buffer, failed to reverse the EtOH-induced
2 Both groups were studied under
hyponatremic conditions and/or with the addition of p38(
3 otonic media (112 mM NaCl, mimicking in vivo
hyponatremic conditions associated with EtOH withdrawal)
4 One patient had unsuspected
hyponatremic encephalopathy and died of cardiopulmonary
5 Prompt recognition of
hyponatremic encephalopathy and early treatment with hyp
6 eview of articles involving risk factors for
hyponatremic encephalopathy and treatment of hyponatremi
7 In patients with
hyponatremic encephalopathy exhibiting neurologic manife
8 hyponatremic encephalopathy and treatment of
hyponatremic encephalopathy in critical illness.
9 Hyponatremic encephalopathy is a frequently encountered
10 Hyponatremic encephalopathy, symptomatic cerebral edema
11 sure and can be the initial manifestation of
hyponatremic encephalopathy.
12 genic pulmonary edema can be associated with
hyponatremic encephalopathy.
13 Here, we describe a 3-month-old
hyponatremic infant found to have a phenylalanine 229 to
14 iuretic hormone levels increased more in the
hyponatremic patients (P < .02), despite their consisten
15 As
hyponatremic patients have elevated circulating arginine
16 Optimal management of
hyponatremic patients involves weighing the risk for ill
17 and edema and normalized serum sodium in the
hyponatremic patients.
18 actions of Avpr inhibitors used commonly in
hyponatremic patients.
19 The water-loaded rats were
hyponatremic (
plasma Na+, 98 to 122 mmol/L) and manifest
20 , chronically hypernatremic, and chronically
hyponatremic rats received pituitary stalk compression (
21 However, in
hyponatremic rats SC caused degeneration of 97% of the A
22 In both normonatremic and
hyponatremic rats, infusion of hypertonic saline without
23 In
hyponatremic rats, oxytocin and vasopressin cells also r
24 In contrast,
hyponatremic SC rats exhibited peak water intakes of 600