ライフサイエンスコーパス: hypoosmotic

1 ecific function in addition to their role in hypoosmotic adaptation.

2 igher growth rates and were more tolerant of hypoosmotic and high-temperature stress than the wild ty

3 as safety valves preventing cell lysis upon hypoosmotic cell swelling: the channels open under membr

4 After hypoosmotic challenge, OHCs shortened and their diameter

5 discontinuity, exhibits tolerance to extreme hypoosmotic challenge, whereas populations native to bra

6 Osm/kg) induce chromatin condensation, while hypoosmotic conditions (100 mOsm/kg) cause decondensatio

7 ine released from pituicytes under basal and hypoosmotic conditions may act to suppress axon terminal

8 ndings are as follows: brain edema mimicking hypoosmotic conditions stimulates the formation of new O

9 ts, including poor growth and motility under hypoosmotic conditions, and the inability to invade plan

10 In hypoosmotic conditions, which elicit cell edema, OAP for

11 es through the channel opening under extreme hypoosmotic conditions.

12 ory volume decrease in astrocytes exposed to hypoosmotic conditions.

13 f different structural classes as well as by hypoosmotic conditions.

14 The adaptation of rhizobacteria to hypoosmotic environments is also examined in the present

15 ->3),beta-(1-->6)-D-glucans during growth in hypoosmotic environments, and evidence is growing that t

16 s in high-water-permeability membranes under hypoosmotic gradients of different magnitude, as well as

17 mutant strain was only slightly sensitive to hypoosmotic growth conditions compared with the ndvB mut

18 radiotracer assays in cells challenged with hypoosmotic medium (30% reduction in osmolarity).

19 These data suggest that volume expansion by hypoosmotic medium stimulates movement of skAE1 from an

20 ions of taurine significantly decreased with hypoosmotic perfusion, while glutamate, glutamine, and G

21 ated with acute and acclimatory responses to hypoosmotic shock and posits unique mechanisms that enab

22 annels prevent cells from lysing upon sudden hypoosmotic shock by opening and releasing solutes and w

23 s environment in the presence and absence of hypoosmotic shock by reacting a charged sulfhydryl reage

24 ortic and bovine retinal ECs were exposed to hypoosmotic shock for 2 minutes, were allowed to recover

25 n channel required for pollen to survive the hypoosmotic shock of rehydration and for full male ferti

26 When Escherichia coli cells are subject to hypoosmotic shock they are subject to substantial flows

27 "cell integrity" MAPK pathway by heat shock, hypoosmotic shock, and actin perturbation, and we report

28 Furthermore, in response to hyper- and hypoosmotic shock, E. coli cells resumed their preshock

29 owed to react, in the presence or absence of hypoosmotic shock, with cells expressing mechanosensitiv

30 this hypopeak increased with the size of the hypoosmotic shock, with increased water flow.

31 in the protection of bacterial cells against hypoosmotic shock.

32 in the protection of bacterial cells against hypoosmotic shock.

33 ol uptake and glycerol efflux in response to hypoosmotic shock.

34 protect cells from structural damage during hypoosmotic shock.

35 r role in releasing the pressure built up by hypoosmotic shock.

36 ic properties of OHCs, we exposed cells to a hypoosmotic solution for varying durations and then punc

37 are sensitive to prolonged exposure to hyper/hypoosmotic solutions, temperature changes, mechanical m

38 tography analyses demonstrated that in vitro hypoosmotic stimulation (reduction of 40 mOsm/kg) of iso

39 increase in maximal cell volume elicited by hypoosmotic stimulation was significantly smaller in AQP

40 ing in vivo two-photon imaging, we show that hypoosmotic stress (20% reduction in osmolarity) initiat

41 ective in normoxic slices (400 microm) after hypoosmotic stress altered the ECS to mimic ischemia.

42 vis Wilson clone, cells responded quickly to hypoosmotic stress by increasing their brevetoxin cell q

43 We conclude that plastids are under hypoosmotic stress during normal plant growth and dynami

44 se two alpha1 variants were less tolerant of hypoosmotic stress than the wild type and produced CPs w

45 (<1 pg per cell), was unable to balance the hypoosmotic stress, and although brevetoxin production r

46 e of urea conduction in UT-B is increased by hypoosmotic stress, and that the site of osmoregulation

47 of the cell were significantly decreased by hypoosmotic stress, but were unchanged by hyperosmotic s

48 d MSL3 contain leaf epidermal plastids under hypoosmotic stress, even during normal growth and develo

49 -regulate mitochondrial genes in response to hypoosmotic stress.

50 1 could regulate their volume in response to hypoosmotic stress.

51 se valve" that protects bacterial cells from hypoosmotic stress.

52 elief valve", protecting bacteria from acute hypoosmotic stress.

53 e valve," allowing bacteria to survive acute hypoosmotic stress.

54 remodeling of cortical actin in response to hypoosmotic stress.

55 Hypoosmotic swelling markedly reduced microvilli number

56 nal genomic basis of population variation in hypoosmotic tolerance.

57 After a hypoosmotic transfer, there was an additional effect: a

58 After hypoosmotic treatment of isolated mitochondria, mitochon

59 the Arp2/3-depleted cells can be rescued by hypoosmotic treatment.

60 Cellular volume expansion by hypoosmotic volume expansion but not volume expansion by

61 Upon hypoosmotic volume expansion nearly half of the skAE1 is