ライフサイエンスコーパス: hypophagic

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1 hepatic steatosis yet are not hyperactive or hypophagic.

2 Reduction in the hypophagic action of 5-HT, possibly acting at 5-HT(1A),

3 type-1 (CB1) receptor antagonists are potent hypophagic agents, but the sites where this acute action

4 tite were also tolerant to three other known hypophagic agents, lipopolysaccharide (LPS), muramyl dip

5 Dopamine-deficient (DD) mice become hypophagic and die of starvation by 3 to 4 weeks of age

6 These mice become hypoactive and hypophagic and die of starvation by 4 weeks of age.

7 cy than wild-type littermates, despite being hypophagic and maintaining normal metabolic rates.

8 n normal but gradually become hypoactive and hypophagic, and die at 3 weeks of age.

9 Pmch-deficient (Pmch-/-) mice are lean, hypophagic, and have an increased metabolic rate.

10 Furthermore, orexin knockout mice are hypophagic compared with weight and age-matched litterma

11 he brain histamine system to fully exert its hypophagic effect and that the oxytocin neuron-rich nucl

12 To dissect the mechanisms underlying the hypophagic effect of CB1 receptor blockade, we combined

13 ition of these neurons may contribute to the hypophagic effect of estrogen.

14 body weight, i.p.) blocked the enterostatin hypophagic effects in these KO mice.

15 deficient, or DD, mice) are resistant to the hypophagic effects of a moderate dose of AMPH (2 microg/

16 lly, blockade of LDTg GLP-1Rs attenuates the hypophagic effects of a systemic GLP-1R agonist.

17 MC4-R, respectively), which may mediate the hypophagic effects of alpha-melanocyte-stimulating hormo

18 med to examine the mechanisms underlying the hypophagic effects of amylin receptor activation in the

19 tly increases food intake and attenuates the hypophagic effects of gastric distension.

20 1R activation contributes to hypothermic and hypophagic effects of hindbrain CART, whereas CART-induc

21 c brain neurons is dispensable for the acute hypophagic effects of rimonabant.

22 t that the PBN critically contributes to the hypophagic effects of systemically delivered GLP-1R agon

23 These hypophagic effects were based on reduced meal size, and

24 ncentrating hormone (MCH)-deficient mice are hypophagic, lean, and do not develop hepatosteatosis whe

25 We identified that PRMT2(-/-) mice are hypophagic, lean, and have significantly reduced serum l

26 it growth retardation and the adult mice are hypophagic, lean, and resistant to high-fat diet-induced

27 c beta cells and hypothalamus produced lean, hypophagic mice with increased physical activity and imp

28 ion of MCH in the hypothalamus with the lean hypophagic phenotype coupled with increased resting meta

29 al loss of AgRP/NPY neurons leads to a lean, hypophagic phenotype.

30 n the amygdala was sufficient to restore the hypophagic property of D-Fen.

31 The hypophagic response of Nckx4 knock-out mice is accompani

32 e hypothesized that it may contribute to the hypophagic response to overfeeding.

33 ity to leptin and confirmed reports that the hypophagic response to third ventricular (i3vt) leptin i

34 found that, as for leptin, ADX enhanced the hypophagic response via a glucocorticoid-dependent mecha

35 P neurons, and reduces both anxiety-like and hypophagic responses to acute stress.

36 coordinating endocrine and behavioral (i.e., hypophagic) responses to stress.

37 These results indicate that CNS MCs mediate hypophagic signaling in response to involuntary overfeed

38 Overweight animals are hypophagic, suggesting that IGFII affects fat metabolism

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