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1 estosterone or luteinizing hormone (LH) into hypophysectomized and hpg/hpg mice restored Pem expressi
2 at adrenal chromaffin tissue from normal and hypophysectomized animals.
3 able in liver nuclear extracts prepared from hypophysectomized ERalpha-deficient females, the regulat
4                                   A study in hypophysectomized female rats demonstrated that the estr
5                      In addition, adolescent hypophysectomized (hypox) male rats served as controls i
6              GH infusion (female pattern) in hypophysectomized male and female rats decreased ST1E2 m
7                                    Last, the hypophysectomized male rat receiving no growth hormone r
8 mal masculine plasma growth hormone pulse in hypophysectomized male rats did indeed rapidly activate
9 eceptor-deficient little (lit/lit) mice, and hypophysectomized mice were used with replacement of sex
10 , as demonstrated by a lack of expression in hypophysectomized mice, gonadotrophin-deficient hypogona
11  active feedback is absent, the prepuberally hypophysectomized mouse.
12                                              Hypophysectomized P(4)-treated pregnant hamsters also sh
13 tability in chromaffin cells from normal and hypophysectomized (pituitary-ablated) rats.
14 e liver genes, CYP2A2 is highly expressed in hypophysectomized rat liver, despite the absence of GH s
15 by injecting them daily or every 6 days into hypophysectomized rats and determining the effects on bo
16 esterone, and testosterone administration to hypophysectomized rats did not have marked effects on he
17                                           In hypophysectomized rats, in which the ADH protein increas
18 on was restored at various concentrations to hypophysectomized, thyroxine-supplemented female rats to

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