1 rnative theory predicts that ASL is normally
hyposmotic.
2 During
hyposmotic (
170 mosmol kg-1) exposure there were large,
3 roM) completely protected both isosmotic and
hyposmotic (
233 mOsm) slices against hypoxic damage.
4 Cell swelling induced by
hyposmotic (
250 osmol (l solution)(-1)) bath solution st
5 %, 35%, and 13% recovery rate in slices made
hyposmotic (
273, 253, and 233 mOsm, respectively).
6 Cell swelling induced by
hyposmotic bath solution stimulated Cl(-) currents in ar
7 Hyposmotic cell swelling caused a marked increase in den
8 e inhibitors (calyculin A and okadaic acid),
hyposmotic cell swelling failed to further up-regulate I
9 currents were elicited by both isosmotic and
hyposmotic cell swelling.
10 A 50 mosmol l-1
hyposmotic challenge (300 to 250 mosmol l-1) constricted
11 Cell depolarization or
hyposmotic challenge shorten the cell and reduce membran
12 for channel activation were not affected by
hyposmotic challenge.
13 ding failed to arise during large (100 mOsm)
hyposmotic challenges, underscoring that neuronal swelli
14 lites and synthetic agonists, in addition to
hyposmotic challenges.
15 ssion for the ratio of I- permeability under
hyposmotic condition to that under isosmotic condition (
16 ignificantly diminished in slices exposed to
hyposmotic conditions as 57% of control (isosmotic) slic
17 enopus laevis expressing TcAQP swelled under
hyposmotic conditions indicating water permeability, whi
18 We studied the effect of
hyposmotic conditions on three colonic (Caco2, HRT18, HT
19 (Em) was -58 and -32 mV under isosmotic and
hyposmotic conditions, respectively.
20 influx, together with Em under isosmotic and
hyposmotic conditions, were used to calculate ER.
21 slightly in response to cell swelling under
hyposmotic conditions.
22 I- pulses under isosmotic and, subsequently,
hyposmotic conditions.
23 ase in activation of the NMDA receptor under
hyposmotic conditions.
24 hole-cell configuration in the presence of a
hyposmotic gradient.
25 Exposure to a
hyposmotic/
hypotonic solution also led to a significant
26 to prevent ingested ice from nucleating the
hyposmotic intestinal fluids.
27 after swelling induced by 60 min exposure to
hyposmotic media (170 mosmol kg-1) relative to isosmotic
28 or the volume increase of cancer cells under
hyposmotic pressure.
29 In addition, P-gp decreased the magnitude of
hyposmotic shock required to activate the channels and t
30 ctivated Cl- channels with ER = 4.9 when the
hyposmotic shock was 110 +/- 10 mosM.
31 when cells were exposed to a smaller (10 %)
hyposmotic shock.
32 Cd2+ had no effect upon RVD following a 30 %
hyposmotic shock.
33 VD) within 16 min when exposed to 30 or 10 %
hyposmotic shocks.
34 e) while affording substantial protection to
hyposmotic slices (233 mOsm), as 54% of the treated slic
35 usly increased the volume of normal cells in
hyposmotic solutions by up to 7%, but 9AC had no effect
36 eously reduced the volume of normal cells in
hyposmotic solutions by up to approximately 10%, but Gd3
37 ulatory volume decrease (RVD) in response to
hyposmotic solutions.
38 e, amiloride, or HOE 694 are consistent with
hyposmotic stimulation of apical NHE3 activity and provi
39 ng a greater than 9-fold increase during the
hyposmotic stimulus.
40 Mild
hyposmotic stress (0.9T) resulted in significant cell sw
41 Hyposmotic stress induced significant releases of aspart
42 Therefore,
hyposmotic stress inhibits cell growth and, depending on
43 cells to correct their volume in response to
hyposmotic stress via the efflux of inorganic and organi
44 pases and protein kinases in the response to
hyposmotic stress were undertaken using an in vivo rat c
45 azoxide eliminated volume change due to mild
hyposmotic stress, similar to that previously noted with
46 nted Cdk and cyclin degradation and reversed
hyposmotic stress-induced growth arrest, whereas calpain
47 each other when the cells were submitted to
hyposmotic stress.
48 the first evidence that NHE3 is regulated by
hyposmotic stress.
49 y a delayed regulatory volume decrease after
hyposmotic stress.
50 to I- influx were significantly faster under
hyposmotic than under isosmotic conditions.