ライフサイエンスコーパス: hypotension

1 ental benefit or raised safety concerns (ie, hypotension).

2 y higher rates of cardiovascular disease and hypotension.

3 One subject had evidence of orthostatic hypotension.

4 d surgical factors-and perhaps postoperative hypotension.

5 Anaphylactic shock is associated with severe hypotension.

6 ssess the incidence of acetaminophen-induced hypotension.

7 culating aldosterone causes salt wasting and hypotension.

8 oconstriction and venoconstriction result in hypotension.

9 lood pressure, but sepsis is associated with hypotension.

10 een oxygen demand and supply, as with severe hypotension.

11 and SVTs, and an increase in bradycardia and hypotension.

12 imulation may lead to vasopressor-refractory hypotension.

13 red susceptibility for allergen/IgE-mediated hypotension.

14 ptic shock patients and its interaction with hypotension.

15 processes, including neuronal protection and hypotension.

16 troversial because of the risk of stroke and hypotension.

17 bdominal pain, gastrointestinal bleeding and hypotension.

18 suscitation, who lacked an obvious source of hypotension.

19 -based consensus definition of intradialytic hypotension.

20 vation, but unlike the C1 cell group, not by hypotension.

21 e considered to have early postresuscitation hypotension.

22 but was associated with an increased rate of hypotension.

23 ring total IHVR without deleterious systemic hypotension.

24 sified as having symptomatic or asymptomatic hypotension.

25 over 12 months than those without nocturnal hypotension.

26 n patients (56%) had early postresuscitation hypotension.

27 void possible patient harm from drug-induced hypotension.

28 atheter ablation for AF but resulted in more hypotension.

29 erized by higher inflammatory biomarkers and hypotension.

30 tial loss-of-function PLD2 polymorphism with hypotension.

31 anaphylaxis could prevent the development of hypotension.

32 dently associated with acetaminophen-induced hypotension.

33 cific transient abdominal pain and transient hypotension.

34 nditions of severe hemolysis after prolonged hypotension.

35 Toxicities included fever, tachycardia, and hypotension.

36 risk of developing in-hospital occurrence of hypotension.

37 Sepsis was characterized by hypotension (~12 mm Hg), increased heart rate (~80 beats

38 .69), desaturation (8% vs 4%; p = 0.27), and hypotension (13% vs 11%; p = 0.64).

39 < 0.05) higher rates of DGF, 32% versus 19%; hypotension, 14% versus 4%; acute myocardial infarction,

40 Hypotension (1615 [40%] patients) and dehydration (1536

41 n the placebo group had clinically important hypotension (2385 patients [47.6%] vs. 1854 patients [37

42 es of data, such as excluding the absence of hypotension (276 of 491 policies [56.2%]) and hypothermi

43 Among patients with acetaminophen-induced hypotension, 29 (34.9%) required therapeutic interventio

44 1.09-4.16), constipation (2.24, 2.04-2.46), hypotension (3.23, 1.85-5.52), erectile dysfunction (1.3

45 o 56 100 (95% CI, 50 800-61 400) episodes of hypotension, 34 400 (95% CI, 31 200-37 600) episodes of

46 than that of patients who did not experience hypotension (496 min vs 253 min, P = 0.000).

47 The prevalence of hypotension (57% vs 48%), atrial fibrillation (50% vs 40

48 febrile neutropenia (18% [six patients]) and hypotension (6% [two patients]).

49 optera sting-induced anaphylaxis, documented hypotension, absence of urticaria pigmentosa, and normal

50 ts (51.9%) experienced acetaminophen-induced hypotension according to our definition.

51 the early use of ultrasound in patients with hypotension accurately guides diagnosis, significantly r

52 e and baseline heart failure, renal failure, hypotension, acute kidney injury, altered gas exchange,

53 Such outcomes include hypotension, acute urinary retention, and the neurolepti

54 score), patient age, prehospital or arrival hypotension, admission from a long-term care facility, m

55 hreatening kidney disease featuring arterial hypotension along with electrolyte abnormalities.

56 rsening of mismatch by episodic hypoxemia or hypotension also reproducibly triggers PIDs.

57 d no interaction on the relationship between hypotension and 30-day outcomes (interaction P=0.874 for

58 were used to assess the association between hypotension and 30-day outcomes.

59 enal denervation was associated with greater hypotension and a loss of the initial diuresis, but no s

60 0 mL/kg of blood was removed yielding marked hypotension and a rise in plasma lactate.

61 Occurrence of hypotension and bradycardia did not differ between group

62 Severe hypotension and bradycardia occur at similar prevalence

63 e breathing unassisted, the adjusted odds of hypotension and cardiac arrest were 12.6 (95% confidence

64 the identification of an accurate cause for hypotension and change in physicians' diagnostic uncerta

65 to two groups according to the occurrence of hypotension and compared demographic characteristics, cl

66 evere heart failure, which is accompanied by hypotension and cyanosis, pericardial effusion, low volt

67 dodrine is prescribed to prevent symptomatic hypotension and decrease complications associated with h

68 d cardiogenic shock, acutely manifested with hypotension and dyspnea.

69 n problematic, with development of prolonged hypotension and edema.

70 ETCO2 declined precipitously, followed by hypotension and EKG changes.

71 pain, hypoxia, hypoglycemia, infection, and hypotension and elicit cardiorespiratory stimulation, ad

72 pressure was normal in the NA group; severe hypotension and high mortality were observed in controls

73 d a higher prevalence of vasodilatation with hypotension and higher cardiac outputs necessitating gre

74 shock (higher serum lactate level, combined hypotension and hyperlactatemia, or higher predicted ris

75 nable analysis of the role of these cells in hypotension and hypertension, and may suggest novel ther

76 l role in sepsis, and it is characterized by hypotension and hyporesponsiveness to vasoconstrictors.

77 sults in other physiological effects such as hypotension and hypothermia.

78 y end points included tolerability, systemic hypotension and intracranial hypertension.

79 observed that S1P administration ameliorated hypotension and microvascular leakage following combined

80 hemorrhagic shock and resuscitation-induced hypotension and microvascular leakage.

81 Tumour necrosis factor (TNF)-induced hypotension and mortality are preserved in apo-sGC mice,

82 apture the association between intradialytic hypotension and mortality.

83 r, increase the risk of clinically important hypotension and nonfatal cardiac arrest.

84 roup had higher proportions of patients with hypotension and nonserious angioedema but lower proporti

85 e most serious manifestations being postural hypotension and paradoxical supine hypertension.

86 the renal graft (DGF), which can result from hypotension and pressor use related to the liver transpl

87 omes and the association between in-hospital hypotension and renal function at hospital discharge.

88 assessed factors associated with in-hospital hypotension and subsequent 30-day outcomes.

89 Neurogenic orthostatic hypotension and supine hypertension are common manifesta

90 ients with coexistent neurogenic orthostatic hypotension and supine hypertension, clinicians need to

91 ients with coexistent neurogenic orthostatic hypotension and supine hypertension.

92 ients with coexistent neurogenic orthostatic hypotension and supine hypertension; and the prevalence,

93 fits of treatment for neurogenic orthostatic hypotension and the long-term risks of supine hypertensi

94 There was no association between duration of hypotension and time to discharge.

95 cell toxicities, with early intervention for hypotension and treatment of concurrent infections being

96 t developed clinical symptoms: 3 experienced hypotension and/or bradycardia, 2 experienced abnormal c

97 (VSMCs) led to reduced arterial elasticity, hypotension, and an impaired arterial response to angiot

98 of the consequences of acetaminophen-induced hypotension, and assess the pathophysiologic mechanisms

99 tal MI but increases risks of stroke, death, hypotension, and bradycardia.

100 ncluded symptomatic bradycardia, symptomatic hypotension, and cardiogenic shock.

101 etermine factors associated with in-hospital hypotension, and Cox proportional hazards models were us

102 od exposure to typical symptoms of vomiting, hypotension, and diarrhea has lagged far behind our unde

103 h cytokine-release syndrome including fever, hypotension, and dyspnea.

104 side effects such as peripheral neuropathy, hypotension, and hypersensitivity.

105 and renal failure, with absence of fever or hypotension, and in inpatient-presenting sepsis.

106 paired BP stabilization, initial orthostatic hypotension, and orthostatic hypotension are reported.

107 sure (BP) stabilization, initial orthostatic hypotension, and orthostatic hypotension based on beat-t

108 ng injury had fever, tachycardia, tachypnea, hypotension, and prolonged hypoxemia compared with contr

109 have mild cognitive impairment, orthostatic hypotension, and RBD at baseline, and at prospective fol

110 D for mild cognitive impairment, orthostatic hypotension, and RBD even at baseline visits.

111 ary infiltrates, pleuropericardial effusion, hypotension, and renal failure.

112 orphine, midazolam), mechanical ventilation, hypotension, and surgeries.

113 ved IV injections of acetaminophen developed hypotension, and up to one third of the observed episode

114 to determine whether patients with nocturnal hypotension are at greater risk for visual field (VF) lo

115 ial orthostatic hypotension, and orthostatic hypotension are reported.

116 lues greater than 4 mmol/L, with and without hypotension, are significantly associated with in-hospit

117 alues > 4 mmol/L) and presence or absence of hypotension as a marker of clinical outcome.

118 f event in hospital, extracorporeal CPR, and hypotension as the cause of arrest), as well as variable

119 ease in Glasgow Coma Scale, or in those with hypotension as the reason for the call.

120 activator Vav2 exclusively in vSMCs leads to hypotension as well as the elimination of the hypertensi

121 achycardia/bradycardia at 33% (327/977), and hypotension at 27% (261/977) in the delayed group.

122 ial orthostatic hypotension, and orthostatic hypotension based on beat-to-beat blood pressure methods

123 roup was withdrawn from the study because of hypotension before receiving treatment.

124 min; CI, -159 to -125; p < 0.001), baseline hypotension (beta, -39 min; CI, -48 to -32; p < 0.001),

125 s associated with propranolol (hypoglycemia, hypotension, bradycardia, and bronchospasm) occurred inf

126 ty of life, cardiopulmonary morbidity (e.g., hypotension, bradycardia, bronchospasm, and/or congestiv

127 converting enzyme before sepsis worsened the hypotension but enhanced skeletal muscle adenosine triph

128 Intracranial hypotension can mimic other conditions such as aseptic m

129 Orthostatic hypotension can reflect altered activity of this neural

130 ecal ligation and puncture animals developed hypotension, cardiac depression, and vascular hyporespon

131 al hypertension and spontaneous intracranial hypotension, changes in the overall diagnostic strategy

132 rtality among Zambian adults with sepsis and hypotension compared with usual care.

133 efficacious for advance warning of impending hypotension, compared with a basic hypotension threshold

134 Hypotension compromises local tissue perfusion, thereby

135 The degree of hypotension correlated with percentage of ArchT-transduc

136 Intradialytic cerebral ischemia, but not hypotension, correlated with decreased executive cogniti

137 associations of commonly used intradialytic hypotension definitions and mortality.

138 Intradialytic hypotension definitions that considered symptoms, interv

139 Intradialytic hypotension definitions were selected a priori on the ba

140 Acute toxicities including fever, hypotension, delirium, and other neurologic toxicities o

141 AHD was defined as persistent hypotension despite vasopressors and requiring mechanica

142 Intraoperative hypotension does not seem to be a clinically important p

143 e inhibitors are associated with deleterious hypotension during anesthesia and shock.

144 n and decrease complications associated with hypotension during dialysis.

145 th the control group had a greater degree of hypotension during sepsis (68 vs 81 mm Hg; p = 0.003) an

146 e unit and 30-day mortality) and the rate of hypotension during study drug infusion.

147 first presentation, 40 patients experienced hypotension during their ED stay.

148 A 56-year-old man who developed hypotension, dyspnea, hypoxia, and pulseless electrical

149 mmon adverse events including fever, chills, hypotension, edema, hypoalbuminemia, thrombocytopenia, a

150 therapies are often limited by tolerability, hypotension, electrolyte disturbances, and renal dysfunc

151 dataset, 102-of-215 ICU stays experienced >1 hypotension episode (median of 2.5 episodes per day in t

152 es, we analyzed characteristics of sustained hypotension episodes (>15 min) and then developed a logi

153 loped a logistic regression model to predict hypotension episodes using input features related to BP

154 tiffness), autonomic features (constipation, hypotension, erectile dysfunction, urinary dysfunction,

155 for translation, RLN did not induce systemic hypotension even in advanced cirrhosis models.

156 bness, weakness, vertigo, syncope, diplopia, hypotension, floaters, other).

157 wenty-two patients who sustained prehospital hypotension following blunt trauma (15 males and 7 femal

158 radialytic exposure to cerebral ischemia and hypotension for each patient, and entered these values i

159 threshold of less than 90 mm Hg for treating hypotension for individuals 10 years and older based on

160 fference [95% CI], 4 [3-5]; p<0.0001) and by hypotension (from 27% to 43%; difference [95% CI], 15 [1

161 f NCC cause renal salt wasting with arterial hypotension (Gitelman syndrome).

162 The ED stay of the hypotension group was significantly longer than that of

163 evaluation of patients with undifferentiated hypotension has been proposed in several protocols.

164 esponses to physical stressors (for example, hypotension, hemorrhage and presence of lipopolysacchari

165 sedation, lightheadedness, tachycardia, and hypotension; however, we were able to minimize these eff

166 n the collecting duct of mouse kidney caused hypotension, hypokalemia, and metabolic alkalosis, an ex

167 Infection, hypotension, hyponatremia, insomnia or stress, and benzo

168 s were characterized as having intradialytic hypotension if they met the corresponding definition in

169 Intracranial hypotension (IH) is an uncommon, benign, and usually sel

170 Hypertension was present in 64 (65.3%), hypotension in 67 (68.4%), hyperglycemia in 17 (18.1%),

171 The immediate cause of arrest was hypotension in 67%, respiratory decompensation in 44%, a

172 Vasopressor infusion (VPI) is used to treat hypotension in an ICU.

173 cant mortality increase with the presence of hypotension in conjunction with serum lactate elevation

174 interdialytic weight gain and intradialytic hypotension in ESRD.

175 5 in UMICs vs 492 [39%] of 1260 in HICs) and hypotension in HICs (564 [45%] of 1260 vs 611 [38%%] of

176 eatment of persistent vasopressor-refractory hypotension in patients with septic shock would be worth

177 tions were moderate dehydration in one, mild hypotension in seven, and mild to moderate electrolyte d

178 luded in the study if they had an episode of hypotension in the 24 hours prior to the clinical pharma

179 and cardiopulmonary arrest characteristics, hypotension in the first 6 hours following return of spo

180 adverse events (AEs) included hemorrhage or hypotension; incidence of common hemorrhagic AEs (includ

181 Factors strongly associated with in-hospital hypotension included randomization to nesiritide (odds r

182 Intradialytic hypotension is a serious and frequent complication of he

183 Perioperative hypotension is associated with an increase in postoperat

184 t evaluation of the effects of intradialytic hypotension is difficult.

185 d flow, abnormal blood coagulation, systemic hypotension, ischemic vascular disorders, and autoimmune

186 greater than or equal to 55 years, systolic hypotension less than or equal to 70 mm Hg, transfusion

187 p < 0.0001); hospital length of stay before hypotension: less than 3 days (+50 min; p < 0.0001), bet

188 ecting one or both ventricles accompanied by hypotension, low cardiac output, and high filling pressu

189 (0.81, 1.17) for a 5-minute increase in MAP hypotension < 55 mm Hg time (P = 0.71).

190 here was no adjusted association between SBP hypotension < 80 mm Hg and SSI, with an estimated odds r

191 , 0-3 points, with 1 point each for systolic hypotension [</=100 mm Hg], tachypnea [>/=22/min], or al

192 nical studies suggest that postresuscitation hypotension may be harmful, and laboratory studies sugge

193 Furthermore, clinically meaningful hypotension may not be as low as current guidelines sugg

194 come of this study was a composite of severe hypotension (mean arterial pressure < 60 mm Hg) and brad

195 nonstroke neurological event, major trauma, hypotension, metabolic or electrolyte abnormalities, ren

196 severe reactions to Hymenoptera stings with hypotension might represent the most relevant factor in

197 Hypotension might thus be expected to promote infection,

198 ster solution found was based on orthostatic hypotension, mild cognitive impairment, rapid eye moveme

199 Among adults with sepsis and hypotension, most of whom were positive for HIV, in a re

200 esthesia), and cardiopulmonary outcomes (ie, hypotension, myocardial infarction, stroke), adjusted fo

201 occurred in four patients (13%) at 6 months: hypotension (n=2), worsening hypertension (n=1), intermi

202 penia (n=12 [11%]), dyspnoea (n=3 [3%]), and hypotension (n=3 [3%]) in the BAT group.

203 ract infections (n=9 [11%]), and orthostatic hypotension (n=8 [10%]).

204 Initial orthostatic hypotension occurred in 32.9% (95% CI, 31.2%-34.6%) of t

205 Hypotension occurred in 85 (26%) patients in the fenoldo

206 Concurrently, progressive systemic hypotension occurred with a fall in left atrial pressure

207 tio, 1.59; 95% CI, 1.02-2.47), nonpersistent hypotension (odds ratio, 6.24; 95% CI, 3.58-10.86), band

208 5 of 7141 (21.8%) patients had an episode of hypotension, of which 73.1% were asymptomatic and 26.9%

209 arrier function to ameliorate trauma-induced hypotension, offering a novel therapeutic opportunity fo

210 and non-motor features including orthostatic hypotension (OH) and cognitive impairment.

211 Orthostatic hypotension (OH) is a common cardiovascular disorder, wi

212 Orthostatic hypotension (OH) is a common cause of transient cerebral

213 esults, we divided patients into orthostatic hypotension (OH), postural tachycardia syndrome (POTS),

214 primary outcome was the absence of postural hypotension on postoperative day 1, defined as a decreas

215 The primary outcome, absence of postural hypotension on postoperative day 1, occurred in 95% of t

216 Patients with orthostatic hypotension or absent sympathetic skin response at palms

217 abdominal surgery, the incidence of postural hypotension or adrenal insufficiency is similar among th

218 s has been described in patients with severe hypotension or anaphylaxis.

219 symptoms in 164 patients in the presence of hypotension or bradycardia (method of symptoms).

220 should similarly consider the likelihood of hypotension or bradycardia before starting either sedati

221 s could be found in the prevalence of severe hypotension or bradycardia in either the unmatched or ma

222 ceiving assisted ventilation who experienced hypotension or cardiac arrest developed these signs with

223 ndan group and the placebo group in rates of hypotension or cardiac arrhythmias.

224 sia to thoracic epidural without the risk of hypotension or epidural hematoma.

225 KNO3 did not lead to clinically significant hypotension or methemoglobinemia.

226 f blood products, not operative case length, hypotension, or vasopressor use, was associated with pos

227 risk of developing in-hospital occurrence of hypotension: OR, 0.254 [95% CI, 0.091-0.706].

228 ely had angioedema/urticaria associated with hypotension (P = .004).

229 97 (0.81, 1.17) per 5-minute increase in SBP hypotension (P = 0.54).

230 excessive sedation (p = 0.31), or new-onset hypotension (p = 1.0) that resulted in study drug discon

231 Whereas neurogenic orthostatic hypotension poses risks for falls and can be associated

232 horts suggest that an episode of prehospital hypotension post trauma leads to early, dynamic reprogra

233 Cumulative nocturnal hypotension predicted VF loss in this cohort.

234 al hypertension and spontaneous intracranial hypotension produce highly disabling headaches, and thre

235 uncertainty before and after the ultrasound hypotension protocol (1.85-1.34; -0.51 [95% CI, -0.41 to

236 , the leading diagnosis after the ultrasound hypotension protocol demonstrated excellent concordance

237 sought to assess the impact of an ultrasound hypotension protocol on physicians' diagnostic certainty

238 rasound on each patient using a standardized hypotension protocol.

239 significantly increased the risk of arterial hypotension, pruritus, urinary retention, and motor bloc

240 s Sepsis-3 septic shock if they demonstrated hypotension, received vasopressors, and exhibited a lact

241 ization of DCD organs is limited by hypoxia, hypotension, reduced--then absent--organ perfusion, and

242 al hypertension and spontaneous intracranial hypotension remain common.

243 There was a higher rate of hypotension requiring medication adjustment in the aggre

244 cases (15%), including pulmonary hemorrhage, hypotension requiring vasoactive support, conduit disrup

245 be identified using the clinical criteria of hypotension requiring vasopressor therapy to maintain me

246 erence [RD], -0.02 [95% CI, -0.09 to 0.04]), hypotension requiring volume expanders (RR, 0.71 [95% CI

247 was only 1 adverse effect recorded (infusion hypotension) requiring cessation of rituximab.

248 type 2 presentation, defined as progressive hypotension responsive to treatment, was found in 20/33

249 Drug use was also associated with hypotension (RR, 1.91 [CI, 1.60 to 2.28]), acute urinary

250 tal stroke (RR: 1.76; 95% CI: 1.07 to 2.91), hypotension (RR: 1.47; 95% CI: 1.34 to 1.60), and bradyc

251 atal stroke (RR: 1.76; 95% CI:1.07 to 2.91), hypotension (RR: 1.47; 95% CI: 1.34 to 1.60), and bradyc

252 The Delphi process identified hypotension, serum lactate level, and vasopressor therap

253 nfidence interval [CI], 1.00-1.06; P=0.044), hypotension/shock (OR, 6.43; 95% CI, 2.88-18.98; P=0.001

254 s greater than 4 mmol/L, in conjunction with hypotension, significantly increased mortality when comp

255 tients who were defined as having refractory hypotension, signs of organ hypoperfusion, or both.

256 ed appropriate antimicrobials after onset of hypotension, source of infection was associated with dea

257 trointestinal tract dysfunction, orthostatic hypotension, sweating abnormalities, or erectile dysfunc

258 or cognitive decline but are associated with hypotension, syncope, and greater medication burden.

259 Rates of serious adverse events of hypotension, syncope, electrolyte abnormalities, and acu

260 CVD death), and (ii) serious adverse events (hypotension, syncope, electrolyte abnormalities, bradyca

261 h intracranial hypertension and intracranial hypotension syndromes, which can be symptomatic or idiop

262 ock (cardiac output 58% +/- 1% of baseline), hypotension (systemic arterial pressure 31 +/- 1 mm Hg),

263 variation less than 12% (normovolemia) with hypotension (systolic blood pressure < 90 mm Hg) were re

264 inflammatory response syndrome criteria) and hypotension (systolic blood pressure </=90 mm Hg or mean

265 E. coli, hyperdynamic sepsis developed with hypotension, tachycardia, increased cardiac output, incr

266 ing one or more affected vital signs (fever, hypotension, tachycardia, or tachypnoea; 96 [44%] of 217

267 irements for managing neurogenic orthostatic hypotension that manifests with falls or cognitive impai

268 In patients with acetaminophen-induced hypotension, the nadir mean arterial pressure was 64 mm

269 tension when treating neurogenic orthostatic hypotension; the effectiveness of nocturnal antihyperten

270 impending hypotension, compared with a basic hypotension threshold alert.

271 ving 3.0 and 1.0 mug/kg per day were grade 3 hypotension, thrombocytopenia, and elevations of ALT and

272 was also no adjusted association between MAP hypotension time and SSI, with estimated odds ratio of 0

273 c autonomic failure (symptomatic orthostatic hypotension, urinary incontinence, or both) at diagnosis

274 eater than 2 mmol/L alone or combinations of hypotension, vasopressors, and serum lactate level 2 mmo

275 The prevalence of orthostatic hypotension was 6.9% (95% CI, 5.9%-7.8%) in the total po

276 Each episode of unintentional hypotension was assessed for suspected drug-related caus

277 In-hospital hypotension was associated with increased hazard of 30-d

278 Hypotension was defined as a decrease in the mean arteri

279 Hypotension was defined as systolic and/or diastolic blo

280 tion from pediatric cardiac arrest, systolic hypotension was documented in 56% and was associated wit

281 Severe hypotension was more frequent in the more intensive trea

282 Hypotension was observed 30 minutes (95% CI, 15-71) afte

283 Development of severe hypotension was responsible for a further increase in an

284 Systemic hypotension was the most frequent complication following

285 e on ligand binding, receptor signaling, and hypotension, we report a series of modified analogues in

286 Absolute rates of hypotension were 2.4% in the intensive treatment group v

287 In conclusion, episodes of hypotension were common during VPI in the ICU.

288 Episodes of hypotension were common, with mean arterial pressure fal

289 ansion, neurologic deterioration, and severe hypotension were not significant.

290 Grade 3 fatigue and hypotension were reported in two patients each (4%).

291 h combined lactate greater than 4 mmol/L and hypotension when compared with 29% mortality in patients

292 udies of monogenic forms of hypertension and hypotension, which identified rare variants that primari

293 Hypotension while hospitalized for acute decompensated h

294 Outcomes associated with episodes of hypotension while hospitalized with acute decompensated

295 al hypertension and spontaneous intracranial hypotension will guide the development of new treatments

296 tolerance to indomethacin (six patients) and hypotension with eplerenone (two patients).

297 toxicities: grade 4 sepsis syndrome, grade 4 hypotension with grade 3 rash and fevers, grade 4 aspart

298 found that there was no relationship between hypotension with normovolemia (stroke volume variation <

299 Angiotensin II reversed sepsis-induced hypotension with systemic and regional hemodynamic effec

300 osis, diabetes insipidus, and salt-sensitive hypotension, with depletion of sodium potassium chloride