ライフサイエンスコーパス: hypotensive

1 f intravenous normal saline when they became hypotensive.

2 of lactate values less than 2 mmol/L and not hypotensive.

3 en patients who received procainamide became hypotensive.

4 Male KO mice, but not female KO mice, were hypotensive.

5 contrast, W/W(v) mouse LES was significantly hypotensive (11 +/- 2 mm Hg; N = 6; P < 0.05) with norma

6 Of 97 severe reactions 45 (46%) were hypotensive, 23 (24%) were hypoxemic, and 29 (30%) were

7 lgorithm on data from 563 Chinese women, 206 hypotensive, 357 hypertensive, with information on ethni

8 ow pathway play a central role in the ocular hypotensive action of adenosine A(1) agonists.

9 ed peripherally located CB1 receptors in the hypotensive action of cannabinoids.

10 study was designed to determine whether the hypotensive action of clonidine resulted from its stimul

11 rs and is in part responsible for the ocular hypotensive action of epinephrine.

12 that a significant proportion of the ocular hypotensive action of PGF2alpha in cats is mediated by E

13 establish how this effect is involved in the hypotensive action of prostaglandin analogues in vivo.

14 r, the mechanisms responsible for the ocular hypotensive action of this compound are not completely u

15 This hypotensive action results from an early reduction in aq

16 ion of c-fos expression in brainstem and its hypotensive action, and provide the first evidence that

17 eurons that are likely to be involved in the hypotensive actions of 5-HT(1A) agonists are unclear.

18 These receptors may mediate the ocular hypotensive actions of 5HT2A agonists.

19 r-regulatory actions that buffer the extreme hypotensive actions of the peptides when released in sep

20 oncerning both the affinity for I1Rs and the hypotensive activity of 2-aryliminopyrrolidines.

21 nhuman primates, and its mechanism of ocular hypotensive activity was investigated.

22 s for a substantial proportion of the ocular hypotensive activity.

23 The hypotensive adrenergic antagonist timolol, propranolol,

24 Septic rats became hypotensive after 12 hrs, with a 24-hr mortality of 51.7

25 In patients who are hypotensive after blunt abdominal trauma and not hemodyn

26 Although 59% potentially had an ocular hypotensive agent at 12 months, only 10% had such medica

27 ty with prostaglandin F(2 alpha), the ocular hypotensive agent bimatoprost (Lumigan; Allergan, Inc.,

28 show that 17-phenyl trinor PGE2 is an ocular hypotensive agent in cats.

29 Bimatoprost is a widely used ocular hypotensive agent to treat glaucoma.

30 who were using a single bottle of an ocular hypotensive agent, and objectively measured adherence wi

31 ug delivery device loaded with a proprietary hypotensive agent, DE-117, reduced intraocular pressure

32 st (BMP), which is a highly effective ocular hypotensive agent, is a PGF(2)(alpha) analogue that inhi

33 trong candidate for development as an ocular hypotensive agent.

34 icacious and generally well-tolerated ocular hypotensive agent.

35 y was to assess level of adherence to ocular hypotensive agents and to identify factors affecting adh

36 Good adherence to ocular hypotensive agents is important to control intraocular p

37 s of three pharmacologically distinct ocular hypotensive agents on Cyr61 and CTGF gene expression.

38 The mean (SD) number of hypotensive agents per eye had decreased from 0.7 (1.1)

39 the adherence level to the prescribed ocular hypotensive agents to be sub-optimal and is influenced b

40 of-of-concept study for this class of ocular hypotensive agents.

41 ts, 42.6 % were adherent to their prescribed hypotensive agents.

42 Hypotensive and bradycardic responses to electrical stim

43 However, they were hypotensive and bradycardic when compared with heterozyg

44 ocannabinoids are novel lipid mediators with hypotensive and cardiodepressor activity.

45 Furthermore, AKAP150-/- mice were hypotensive and did not develop angiotensin II-induced h

46 Transgenic mice were hypotensive and exhibited a decreased pressor response.

47 uretic and diuretic peptide that is markedly hypotensive and functions via a separate guanylyl cyclas

48 A, an optically active tropane alkaloid with hypotensive and miotic activity isolated from the Chines

49 ine induced by gamma interferon/CXCL9 in the hypotensive and normotensive patients, respectively, aff

50 all Multiple Organ Dysfunction score between hypotensive and normotensive patients.

51 Our objective was to compare four groups, hypotensive and normotensive resuscitation of hemorrhage

52 quiring PN after CPB were significantly more hypotensive and required more vasopressive drugs during

53 may be reliable alternatives, only to detect hypotensive and therapy-responding patients.

54 been ascribed to Allium extracts, including hypotensive and vasorelaxant activities.

55 The third patient eventually became hypotensive and was treated with repeated sessions of re

56 13/3893 (0.3%) had severe reactions (6 were hypotensive) and 6 reported syncope.

57 , four patients (out of 150) became febrile, hypotensive, and experienced a rapid rise in transaminas

58 in replacement when the ob/ob mice developed hypotensive ARF with a higher dose of LPS (0.5 mg/kg).

59 aring all patients or only patients who were hypotensive at enrollment.

60 r in conscious mice, alpha 1A/C KO mice were hypotensive at rest, with an 8-12% reduction of blood pr

61 uring 10-min normovolemic baseline, bleed to hypotensive baseline, 10-min ITD-assisted breathing, 10

62 No rats were anemic, hypoglycemic, or hypotensive before CCI.

63 l exercise prescriptions remain unclear, but hypotensive benefits have been noted for mild to vigorou

64 There are hypotensive benefits to exercise training found across a

65 rtensive blood pressure high and genetically hypotensive blood pressure low mice.

66 ischemia compared with those with normal or hypotensive BP responses but are not more likely to have

67 e hypertensive BPH (blood pressure high) and hypotensive BPL (blood pressure low).

68 display dramatically enhanced and prolonged hypotensive, bradycardic, and sedative-hypnotic response

69 lternative to norepinephrine in managing the hypotensive brain dead donor.

70 Hemodynamic stress via hypotensive challenge has been shown previously to cause

71 Hypotensive challenge increased LC discharge rates of ad

72 In some rats LC activation by hypotensive challenge was also examined.

73 intracerebroventricularly administered CRF, hypotensive challenge, sciatic nerve stimulation, and ca

74 cleus and LC neurons were still activated by hypotensive challenge.

75 tem would normally be expected to respond to hypotensive challenge; however, inflammation appears to

76 esponses parallel the sympathoinhibitory and hypotensive components of the Bezold-Jarisch reflex.

77 AVP deficiency may contribute to this hypotensive condition.

78 Ps, a protein kinase A inhibitor, attenuated hypotensive dilation (35+/-1 vs. 16+/-2% before and afte

79 hat the released uPA impairs hypercapnic and hypotensive dilation through an LRP- and ERK MAPK depend

80 tion of the gene responsible for orthostatic hypotensive disorder in these families may advance under

81 Familial orthostatic hypotensive disorder is characterized by light-headednes

82 pressor effect of vasopressin replacement in hypotensive donors.

83 ing was found 3 hours after injection with a hypotensive dose of Salmonella enteritidis LPS.

84 h treatment is usually initiated with ocular hypotensive drops, laser trabeculoplasty and surgery may

85 beta-adrenoceptor agonist, isoproterenol, a hypotensive drug that typically produces only water inta

86 e mouse eye that facilitates study of ocular hypotensive drugs and mouse models of glaucoma.

87 therapeutic effects of candidates for ocular hypotensive drugs.

88 leads to the best results in terms of ocular hypotensive effect and safety.

89 the mechanism(s) responsible for this ocular hypotensive effect has not been established.

90 We defined the portal hypotensive effect in rat models of sinusoidal PHT and t

91 ned by activation of vagal afferents and the hypotensive effect may be secondary to a reduction of ca

92 d mice, O-1918 dose-dependently inhibits the hypotensive effect of abn-cbd but not the hypotensive ef

93 The hypotensive effect of alpha2 agonists was completely abs

94 ribution of TRPV(1) receptors to the in vivo hypotensive effect of anandamide is equivocal.

95 on of icatibant significantly attenuated the hypotensive effect of captopril (maximal decrease in mea

96 uggest a link between the estrogen-dependent hypotensive effect of chronically administered ethanol a

97 abolished the bradycardia and attenuated the hypotensive effect of endomorphin 1.

98 aneous pellet, 14.2 microg/day) restored the hypotensive effect of ethanol to a level similar to that

99 action is involved in the estrogen-dependent hypotensive effect of ethanol.

100 The hypotensive effect of inhaled Y-27632 on hypoxic PH was

101 In EP2-/- mice, the characteristic hypotensive effect of intravenous PGE2 infusion was abse

102 B2-receptor antagonist, had no effect on the hypotensive effect of kallistatin yet it abolished the b

103 These data indicate that the early hypotensive effect of latanoprost in the mouse eye is as

104 The initial hypotensive effect of spinal anaesthesia is caused by a

105 reported findings that ethanol abolishes the hypotensive effect of the alpha(2)-adrenoceptor agonist

106 he hypotensive effect of abn-cbd but not the hypotensive effect of the CB(1) receptor agonist (-)-11-

107 SC, at a dose selected for lack of long-term hypotensive effects at baseline).

108 To compare the ocular hypotensive effects of 15-keto latanoprost (KL) with the

109 rdiovascular regulation, which contribute to hypotensive effects of acupuncture.

110 e cardioprotective, bronchoconstrictive, and hypotensive effects of adenosine.

111 c receptor (alpha2A-AR) is necessary for the hypotensive effects of clonidine and other sympathoinhib

112 irs the diuretic and natriuretic but not the hypotensive effects of D(1)-like agonist stimulation.

113 The early and late hypotensive effects of endotoxin were attenuated with 20

114 hypothesis that a greater attenuation of the hypotensive effects of losartan would be observed in rat

115 ons were independently lesioned, the chronic hypotensive effects of the AT(1) receptor blocker losart

116 the LHA or PAG with lidocaine attenuates the hypotensive effects produced by electrical stimulation o

117 no greater attenuation of losartan's chronic hypotensive effects than animals with lesion of either t

118 The hypotensive effects were observed within 3 wk after the

119 ation related through negative inotropic and hypotensive effects.

120 nal biochemical phenotype that exerts potent hypotensive effects.

121 this 28-day study was to evaluate the ocular hypotensive efficacy and safety of AR-13324 ophthalmic s

122 The definition for a hypotensive episode is either a systolic blood pressure

123 n, corrected by renal artery stenting, and 1 hypotensive episode, which resolved with medication adju

124 values collected before, during, and after a hypotensive episode.

125 Many hypotensive episodes in the ICU are drug related and req

126 hundred fifty-eight patients experienced 204 hypotensive episodes that were considered unintentional

127 ons based on dynamic variables surrounding a hypotensive event is a new approach to predicting progno

128 A single, acute hypotensive event lasting 30 mins did not aggravate the

129 ain swelling, pupillary abnormalities, early hypotensive events (before intensive care monitoring), a

130 The duration of hypotensive exposure and development of SSI was assessed

131 logistic regression; the association between hypotensive exposure and duration of hospitalization was

132 olume 7.5% NaCl adenosine, lidocaine, and Mg hypotensive fluid resuscitation from the rat to the pig.

133 o, which were widely used as ingredients for hypotensive food, showed a slight selectivity for the N-

134 ion and 417 of the remaining 1596 (26%) were hypotensive for age.

135 Fixed-combination ocular hypotensives have multiple advantages, but triple-therap

136 were either volume expanded or subjected to hypotensive hemorrhage.

137 +/- 40% of control), but was not changed by hypotensive hemorrhage.

138 Hypotensive hemostasis is usually an effective means to

139 This approach includes hypotensive hemostasis, minimizing fluid resuscitation,

140 Endotoxemia induced an immediate hypotensive, hyperdynamic, tachycardic state with progre

141 The injury induced a hypotensive-hyperdynamic circulation; increases in pulmo

142 nary bypass just before rewarming commenced (hypotensive, hypothermic), after rewarming (hypotensive,

143 whether naloxone would augment tolerance to hypotensive hypovolemic stress (lower body negative pres

144 subjects with UARS (7.2 +/- 0.84 mm Hg), or hypotensive insomniacs (7.4 +/- 1.1 mm Hg) (p < 0.01).

145 In contrast, W/W(v) mice have hypotensive LES with unimpaired relaxation, suggesting t

146 betes, uveitis, retinitis pigmentosa, ocular hypotensive lipids, internal limiting membrane peeling,

147 geal manometry showed a high prevalence of a hypotensive lower esophageal sphincter (55%) and impaire

148 and ICU mortality (p = 0.76) associated with hypotensive mean arterial pressure readings (</=60 mm Hg

149 lial nitric oxide synthase and production of hypotensive mediators, nitric oxide and cyclic guanosine

150 nsion may respond to specific antagonists to hypotensive mediators.

151 The initiation of ocular hypotensive medication among OHTS participants originall

152 we censored data after initiation of ocular hypotensive medication or glaucoma surgery of any kind.

153 t and second eyes to the same topical ocular hypotensive medication.

154 pnea-hypopnea index [AHI], 35) not receiving hypotensive medication.

155 and by 367 trabeculectomies (86%) including hypotensive medication.

156 g but not more than 21 mm Hg with or without hypotensive medication.

157 C >0.75), from 0.29 to 0.80 in patients with hypotensive medications (3 time points with ICCs >0.75),

158 .75), from -0.07 to 0.60 in patients without hypotensive medications (zero time points with ICC >0.75

159 With the development of potent arterial hypotensive medications for arterial hypertension, arter

160 to determine the response to topical ocular hypotensive medications has been recently debated.

161 Washout of hypotensive medications was repeated at 12 and 24 months

162 93), and the proportion of patients using no hypotensive medications was significantly higher at 24 m

163 up in eyes treated with a mean of 0.9 ocular hypotensive medications, and 14.5 (3.4) mm Hg in the dou

164 up in eyes treated with a mean of 2.3 ocular hypotensive medications, and 25.7 (8.0) mm Hg in the dou

165 eyes and separately in eyes with and without hypotensive medications, and in eyes naive and non-naive

166 cess was stratified according to IOP, use of hypotensive medications, bleb needling, and resuturing/r

167 After a washout of all pre-study ocular hypotensive medications, eligible patients were randomiz

168 d likely improve access to prescribed ocular hypotensive medications.

169 as an initial therapy or in conjunction with hypotensive medications.

170 eful addition to the armamentarium of ocular hypotensive medications.

171 Commercially available topical ocular hypotensive medications.

172 SE than those with normal (n=19 770; 90%) or hypotensive (n=274; 1%) BP responses (32% versus 21% ver

173 those born to infected women were more often hypotensive, needed intubation, had neonatal seizures, a

174 The dorsolateral PAG contains a NO producing hypotensive network.

175 course of their disease, some of the sickest hypotensive newborns become unresponsive to volume and p

176 (hypotensive, hypothermic), after rewarming (hypotensive, normothermic) just before discontinuation o

177 events (pulmonary hypertensive and systemic hypotensive) occurring within 10 min of protamine admini

178 No orthostatic hypotensive or hypertensive reactions were observed.

179 However, SNP did not improve hypotensive PAD after FPI in females and paradoxically c

180 steroid insufficiency should be suspected in hypotensive patients who have responded poorly to fluids

181 ds (sensitivity 100%, specificity 99.3%) and hypotensive patients with blunt abdominal trauma (sensit

182 The study group consisted of 128 hypotensive patients with blunt abdominal trauma who und

183 ation of patients with precordial wounds and hypotensive patients with blunt torso trauma, immediate

184 In hypotensive patients, 73% of respondents (108 of 149) re

185 minutes, from 32+/-32 to 20+/-17 minutes in hypotensive patients, and for craniotomy decreased from

186 base deficit) were significantly altered in hypotensive patients.

187 A possible function for this potent hypotensive peptide in the regulation of intraocular pre

188 Adrenomedullin is a recently discovered hypotensive peptide that is expressed in a variety of ce

189 Adrenomedullin (AM) is a newly discovered hypotensive peptide which is believed to play an importa

190 Adrenomedullin (AM), a potent hypotensive peptide, is produced in numerous tissues inc

191 Two potent hypotensive peptides, adrenomedullin (AM) and proadrenom

192 Indeed, alpha(1D)-AR KO mice display a hypotensive phenotype and are resistant to salt-induced

193 eta2-AdR expression, possibly leading to the hypotensive phenotype during the rest phase.

194 nage from peripheral tissues and thus with a hypotensive phenotype.

195 Phenylephrine decreased impairment of hypotensive pial artery dilation after fluid percussion

196 in spontaneously breathing normotensive and hypotensive pigs to increase blood pressure and enhance

197 impedance threshold device (-12 cm H(2)O) in hypotensive pigs, systolic blood pressure (mean +/- sem)

198 d pressure profiles validated the beneficial hypotensive properties of these prodrugs.

199 CD-NP also demonstrates less hypotensive properties when compared with B-type natriur

200 phrine are equally effective in treating the hypotensive pulmonary donor in this rodent model.

201 axis cases (9.2%) and were more common after hypotensive reactions and with pre-existing lung disease

202 d with different selectivities by hyper- and hypotensive regions of the PAG.

203 reated with a NO synthase inhibitor remained hypotensive relative to the wild type.

204 lly or specifically in endothelial cells are hypotensive, resistant to angiotensin II-induced hyperte

205 eversed the HS-induced pressor response to a hypotensive response (from 121 +/- 3 to 68 +/- 2 mmHg),

206 e depend on the spleen; (d) PAF mediated the hypotensive response and its action in the spleen; and (

207 This hypotensive response appears to be due to the inhibition

208 irectly influence MAP blocked the MPO evoked hypotensive response in 9/11 cases.

209 tant finding is that statins exert an ocular hypotensive response in an organ-culture perfusion model

210 ulfate (OSCS) derivative that could elicit a hypotensive response in pigs following a single high-dos

211 tive B(1)R agonist produced a dose-dependent hypotensive response in the knockout mice only.

212 duce vasodilation ex vivo, produces an acute hypotensive response in wild-type mice.

213 reduction of IOP in rabbits, with the ocular hypotensive response lasting for more than 48 hrs.

214 these studies suggest that the MPO elicited hypotensive response may involve NO production in PAG ne

215 (2) The MPO elicited hypotensive response may utilize this network.

216 re performed on three patients with systemic hypotensive response pattern on captopril renography.

217 ery stenosis in three patients with systemic hypotensive response pattern on captopril renography.

218 The systemic hypotensive response pattern seen on captopril renograph

219 The hypotensive response to alpha2AR agonists was lost in th

220 716A (3 mg/kg i.v.), which also inhibits the hypotensive response to anandamide or 2-AG.

221 The predominant hypotensive response to bolus intravenous injections of

222 One patient in Group 1 had a hypotensive response to dipyridamole and was exluded fro

223 spite variable effectiveness in blocking the hypotensive response to injected bradykinin.

224 The hypotensive response to isoproterenol, however, is signi

225 MPD decreased the hypotensive response to levodopa.

226 o lower blood pressure because we observe no hypotensive response to moxonidine in alpha(2A)-AR-null

227 nock-in mouse was substantively deficient in hypotensive response to nitroglycerin compared with wild

228 the IC50 values had no effect on the ocular hypotensive response to PGF2alpha.

229 outflow facility associated with the ocular hypotensive response to the adenosine agonist cyclohexyl

230 uction in blood pressure in all fetuses; the hypotensive response was greatest in fetuses studied nea

231 In TRPV(1)(+/+) mice, this hypotensive response was preceded by a transient, profou

232 P, while significantly inhibiting the ocular hypotensive response.

233 ography is described that is due to systemic hypotensive response.

234 role of individual alpha2AR subtypes in the hypotensive response.

235 explainable by an overly exuberant systemic hypotensive response.

236 ADR SPNs (by 2.9+/-0.5 spikes/s; n=19) with hypotensive responses (DeltaMAP=33.2+/-5.3 and 26.4+/-5.

237 , liposome-encapsulated SOD enhanced in vivo hypotensive responses to acetylcholine and in vitro resp

238 ium diet, they show increased but incomplete hypotensive responses to acute treatment an ET(A)-antago

239 ide (NO) in the gracile nucleus modifies the hypotensive responses to electroacupuncture (EA) stimula

240 mpared with placebo, alcohol potentiated the hypotensive responses to LBNP, particularly at -40 mm Hg

241 (2) HA-evoked hypotensive responses were mediated by PAG neuronal acti

242 d Mg (n = 8) or 4 mL/kg 7.5% NaCl (n = 8) at hypotensive resuscitation and 0.9% NaCl +/- adenosine an

243 enosine and lidocaine, adenocaine) and Mg on hypotensive resuscitation and coagulopathy in the rat mo

244 Hg for 90 minutes, followed by 60 minutes of hypotensive resuscitation and infusion of shed blood.

245 Hypotensive resuscitation is gaining clinical acceptance

246 Normotensive and hypotensive resuscitation to mean arterial pressures of

247 Fluid volume infused during hypotensive resuscitation was 40% less in the 7.5% NaCl-

248 At 60 minutes of hypotensive resuscitation, treatment with 7.5% NaCl + ad

249 nction and reduces fluid requirements during hypotensive resuscitation, whereas a second AL infusion

250 pparent when used for either normotensive or hypotensive resuscitation.

251 s to phenylephrine and angiotensin II during hypotensive sepsis in conscious sheep.

252 Administration of clonidine during hypotensive sepsis reduced renal sympathetic nerve activ

253 were measured in fasted healthy (n = 10) and hypotensive septic (n = 6) adults by using a 6-h constan

254 vivo arginine and NO kinetics are lacking in hypotensive septic adults.

255 pressure underestimates central pressure in hypotensive septic patients receiving high-dose vasopres

256 n and the intravascular NO synthesis rate in hypotensive septic patients.

257 Conversely, hypotensive Sgk1 KO mice exhibited low WNK1 expression a

258 order characterized by transient episodes of hypotensive shock and anasarca thought to arise from rev

259 Refractory hypotensive shock was fatal in 55 of 115 patients treate

260 se ingesting other pesticides and often from hypotensive shock.

261 the prevalence of vasopressin deficiency in hypotensive solid organ donors without clinical evidence

262 ng 60% of the blood volume and maintaining a hypotensive state.

263 essed by applying rapid pulses of hyper- and hypotensive stimuli to the neck via a customised collar.

264 During a hypotensive stimulus, cardiovascular homeostasis is main

265 between sexes and menstrual phases during a hypotensive stimulus.

266 s at rest and during a nitroprusside-induced hypotensive stimulus.

267 sensitivity and the sympathetic response to hypotensive stress are attenuated after antecedent hypog

268 -anesthetized rats before, during, and after hypotensive stress elicited by intravenous nitroprusside

269 50488 also attenuated tonic LC activation by hypotensive stress, an effect mediated by CRF afferents.

270 electively engaged during the termination of hypotensive stress.

271 ardiac events (1 myocardial infarction and 1 hypotensive supraventricular tachyarrhythmia), neither o

272 There was a higher risk of hypotensive symptoms in the combination-therapy group th

273 Our results indicated that hypotensive systolic noninvasive blood pressure readings

274 SETTING, AND PATIENTS: The 316 patients were hypotensive (systolic blood pressure=81 mm Hg) and tachy

275 While lacking renal actions, CNP is less hypotensive than the cardiac peptides atrial natriuretic

276 Persistency with ocular hypotensive therapies has been found to be poor.

277 has been observed commonly with other ocular hypotensive therapies in this population.

278 n or of initiation or augmentation of ocular hypotensive therapy by 3 years, after repeated ranibizum

279 ut the potential risks of intensive arterial hypotensive therapy, particularly that administered in t

280 or the initiation or augmentation of ocular hypotensive therapy, through 3 years of follow-up.

281 CT, using BCC to improve adherence to ocular hypotensive therapy, to our knowledge is the first withi

282 inimizing sensory stimulation and hypoxic or hypotensive transients in stroke and brain injury would

283 rabecular meshwork outflow may be the future hypotensive treatment for glaucoma patients.

284 gical treatment is not feasible then medical hypotensive treatment may be a viable alternative.

285 Two animals in the hypotensive treatment protocols died during the second a

286 and higher occurrences of deaths only in the hypotensive treatment protocols suggest that resuscitati

287 scontinuation and washout of existing ocular hypotensive treatment, patients who had intraocular pres

288 e significantly higher with normotensive vs. hypotensive treatment.

289 stained IOP elevation or the need for ocular hypotensive treatment.

290 h lactate levels (>10 mmol) with both of the hypotensive treatments and also with the HS90 treatment,

291 s. LR when compared with the normotensive or hypotensive treatments.

292 al levels of SNOs in red blood cells and are hypotensive under anesthesia.

293 undred seventy-three patients with recurrent hypotensive ventricular tachyarrhythmias refractory to l

294 response rate) survived 24 h without another hypotensive ventricular tachyarrhythmic event while bein

295 Thus, the clinical picture was that of hypotensive ventricular tachycardia (VT).

296 ated over the first 7 days postinjury in the hypotensive versus normotensive patients.

297 ther felt nor visible on the ECG presents as hypotensive VT.

298 ere we report that P311-/- mice are markedly hypotensive with accompanying defects in vascular tone a

299 Septic rats became unwell and hypotensive, with a mortality of 64% at 48 hrs (0% in co

300 hypertensive rats with sodium retention were hypotensive, with decreases in total peripheral resistan