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1 nal biochemical phenotype that exerts potent hypotensive effects.
2 ation related through negative inotropic and hypotensive effects.
7 ned by activation of vagal afferents and the hypotensive effect may be secondary to a reduction of ca
8 d mice, O-1918 dose-dependently inhibits the hypotensive effect of abn-cbd but not the hypotensive ef
11 on of icatibant significantly attenuated the hypotensive effect of captopril (maximal decrease in mea
12 uggest a link between the estrogen-dependent hypotensive effect of chronically administered ethanol a
14 aneous pellet, 14.2 microg/day) restored the hypotensive effect of ethanol to a level similar to that
18 B2-receptor antagonist, had no effect on the hypotensive effect of kallistatin yet it abolished the b
21 n and counteracted the clinically beneficial hypotensive effect of stimulating alpha2a receptors in t
22 reported findings that ethanol abolishes the hypotensive effect of the alpha(2)-adrenoceptor agonist
23 he hypotensive effect of abn-cbd but not the hypotensive effect of the CB(1) receptor agonist (-)-11-
27 c receptor (alpha2A-AR) is necessary for the hypotensive effects of clonidine and other sympathoinhib
28 irs the diuretic and natriuretic but not the hypotensive effects of D(1)-like agonist stimulation.
30 hypothesis that a greater attenuation of the hypotensive effects of losartan would be observed in rat
31 ons were independently lesioned, the chronic hypotensive effects of the AT(1) receptor blocker losart
32 the LHA or PAG with lidocaine attenuates the hypotensive effects produced by electrical stimulation o
33 no greater attenuation of losartan's chronic hypotensive effects than animals with lesion of either t
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