ライフサイエンスコーパス: hypothalamus

1 s, amygdala, hippocampus, septal region, and hypothalamus).

2 of letrozole into the median eminence of the hypothalamus.

3 the dorsal telencephalon, preoptic area and hypothalamus.

4 stria terminalis, and densely to the lateral hypothalamus.

5 in the paraventricular nucleus (PVN) of the hypothalamus.

6 tory fibers was observed in the striatum and hypothalamus.

7 tomics of the paraventricular nucleus of the hypothalamus.

8 unting serotonergic signaling in the lateral hypothalamus.

9 ensity in the paraventricular nucleus of the hypothalamus.

10 icroglia in the arcuate nucleus (ARC) of the hypothalamus.

11 citatory synaptic function in neurons in the hypothalamus.

12 anced leptin-induced STAT3 activation in the hypothalamus.

13 ergic input to rRPa originates caudal to the hypothalamus.

14 onmotor areas of the neocortex, and with the hypothalamus.

15 iour, the primary region of interest was the hypothalamus.

16 cific gene expression in the hippocampus and hypothalamus.

17 ite suppression on downstream targets in the hypothalamus.

18 teriorly from cortex/hippocampus to thalamus/hypothalamus.

19 impaired regulation of energy metabolism in hypothalamus.

20 rom the paraventricular nucleus (PVN) of the hypothalamus.

21 hese processes to build the tuberal/anterior hypothalamus.

22 fewer oxytocin immunoreactive neurons in the hypothalamus.

23 stress in the paraventricular nucleus of the hypothalamus.

24 is the suprachiasmatic nucleus (SCN) of the hypothalamus.

25 ays were impaired in CCR5 and CCL5 deficient hypothalamus.

26 the axons known to emanate from the lateral hypothalamus.

27 us and cingulate cortex, and hippocampus and hypothalamus.

28 perfusion and no change in perfusion in the hypothalamus.

29 notably the prefrontal cortex, amygdala, and hypothalamus.

30 ts to the ventral tegmental area and lateral hypothalamus.

31 DR within the paraventricular nucleus of the hypothalamus.

32 he amygdala, the hippocampus and the ventral hypothalamus.

33 nuclei as well as in the lateral area of the hypothalamus.

34 lvement of transcriptional regulation in the hypothalamus.

35 rganization of functional cell groups in the hypothalamus.

36 e amygdala, hippocampus, frontal cortex, and hypothalamus.

37 of energy homeostasis and food intake in the hypothalamus.

38 ough the induction of galn expression in the hypothalamus.

39 iomelanocortin (POMC) gene expression in the hypothalamus.

40 eurons of the paraventricular nucleus of the hypothalamus.

41 d neurobiotin-labeled neurons in the lateral hypothalamus, 11 were immunohistochemically identified a

42 (85%), thalamus (73%), cerebellum (54%), and hypothalamus (49%) in HSE rats than in control rats, whe

43 (DEGs) (82), followed by the neocortex (76), hypothalamus (63), and cerebellum (26).

44 Here, we show that the hypothalamus, a brain area regulating physiological stat

45 expressed in neural progenitor cells of the hypothalamus, a conserved region of the vertebrate brain

46 dance in mice, thus revealing an unsuspected hypothalamus-accumbens interplay in action selection.

47 metabolic organs and tissues, including the hypothalamus, adipose tissue, and skeletal muscle.

48 wo separate but adjacent nuclei in the basal hypothalamus: an oblique band of aromatase-positive (AR+

49 aventricular and ventromedial neurons of the hypothalamus and activates an MC4R-dependent anorexigeni

50 al region encompassing the ventral striatum, hypothalamus and anterior thalamus.

51 is specifically expressed in neurons of the hypothalamus and appears to be a central neural regulato

52 ogy in raphe nuclei, striatum, thalamus, and hypothalamus and associations with aging, PD progression

53 bilateral anterior cingulate cortices, right hypothalamus and bilateral amygdala).

54 mesolimbic dopamine circuit from the lateral hypothalamus and dorsal raphe nucleus and defined a disc

55 forebrain situated beneath the floor of the hypothalamus and extending through the nasal cavity to e

56 1 is essential for normal development of the hypothalamus and for the correct functioning of the HPA/

57 ide VF (NPVF) is expressed by neurons in the hypothalamus and has been implicated in nociception, but

58 ain circuits to the feeding circuitry in the hypothalamus and hindbrain remains unclear.

59 hindbrain, while vglut3 is restricted to the hypothalamus and hindbrain.

60 Fractional anisotropy was increased in the hypothalamus and hippocampal CA3.

61 Reducing Cadm1 expression in the hypothalamus and hippocampus promoted a negative energy

62 nsported to the brain and accumulated in the hypothalamus and hippocampus to a greater extent than th

63 d in the suprachiasmatic nuclei (SCN) of the hypothalamus and it regulates circadian oscillators in t

64 mRNA level of Rfrp in the dorso/ventromedial hypothalamus and Kiss1, Pomc, and Somatostatin in the ar

65 he dorsal raphe that project to the anterior hypothalamus and may mediate the spatiotemporal release

66 We also found that CIC activity in the hypothalamus and medial amygdala modulates social intera

67 y some specific projections from the lateral hypothalamus and midbrain, we analyzed the distribution

68 and greater GM volume in posterior thalamus, hypothalamus and midbrain.

69 cortex areas, perhaps all, as well as of the hypothalamus and of the limbic system.

70 nalling in the integrated development of the hypothalamus and pituitary.

71 ssed in the arcuate nucleus of the mammalian hypothalamus and plays a key role in regulating food con

72 ginating from paraventricular nucleus of the hypothalamus and presenting as increased sympathetic dri

73 ypocretin and GABAergic cells in the lateral hypothalamus and receive inputs from multiple sleep-wake

74 both homeostatic feeding circuits within the hypothalamus and reward circuits within the ventral tegm

75 regulates ghrelin receptor signalling in the hypothalamus and starvation sensing in mice.

76 ciated with regional brain insulin action in hypothalamus and striatum.

77 the mechanism of synaptic plasticity in the hypothalamus and suggests new strategies to treat neurog

78 content and sulfation levels in the lateral hypothalamus and that HS contributes to the regulation o

79 (HIF-1alpha) controls glucose uptake in the hypothalamus and that it is upregulated in conditions of

80 llar circuit, bidirectionally connecting the hypothalamus and the cerebellum, has been detected, bein

81 e many physiologic functions mediated by the hypothalamus and their genetic regulation.

82 tor output from the amygdala to the anterior hypothalamus and then the lateral septum to modulate agg

83 arcuate nucleus), projecting throughout the hypothalamus and towards the preoptic area.

84 though these T cells readily infiltrated the hypothalamus and triggered local inflammation, they did

85 gamma (30-90 Hz) oscillations in the lateral hypothalamus and upstream brain regions organize food-se

86 Aberrant embryonic development of the hypothalamus and/or pituitary gland in humans results in

87 optic commissure nucleus, dorsal and ventral hypothalamus) and caudal diencephalon, confirming result

88 rtical limbic areas (e.g., amygdala, lateral hypothalamus), and influenced functional connectivity am

89 so found CARTp-ir cells and terminals in the hypothalamus, and a large number of CARTp-ir terminals i

90 rsal telencephalon, habenula, preoptic area, hypothalamus, and cerebellum.

91 erved expression of CARTp in the subpallium, hypothalamus, and dorsal vagal complex of birds suggests

92 following a brain transection caudal to the hypothalamus, and during the blockade of glutamate recep

93 in the postnatal mouse ventral forebrain and hypothalamus, and found that the highest rates of cell d

94 in ependymal cells (tanycytes) of the fetal hypothalamus, and hence neuroendocrine output.

95 , habenula, periaqueductal gray, cerebellum, hypothalamus, and hippocampal CA3.

96 prefrontal cortex (vmPFC), insula, amygdala, hypothalamus, and periaqueductal gray emerge as central

97 -rich extrastriatal brain regions (thalamus, hypothalamus, and pons).

98 gions of the telencephalon, preoptic region, hypothalamus, and thalamus at all stages investigated.

99 od intake (ventral tegmental area, striatum, hypothalamus, and thalamus), we describe how activity of

100 hly enriched in distinct nuclei of the mouse hypothalamus, and that MANF expression in the hypothalam

101 pathways include functionally opposed PFC-->hypothalamus 'appetitive driver' and PFC-->striatum 'app

102 (AgRP) neurons in the arcuate nucleus of the hypothalamus are critical for homeostatic feeding behavi

103 s in the suprachiasmatic nuclei (SCN) of the hypothalamus are described as master pacemaker cells for

104 ligodendrocytes and 12 of the 63 DEGs in the hypothalamus are oligodendrocyte- and myelin-specific ge

105 The arcuate nucleus of the hypothalamus (ARH) is critical for the regulation of hom

106 In the arcuate nucleus of the hypothalamus (ARH) satiety signaling (anorexigenic) pro-

107 wth of axons from the arcuate nucleus of the hypothalamus (ARH) to other regions that control energy

108 d week of life in the arcuate nucleus of the hypothalamus (ARH).

109 hereas the PGE2 synthesizing capacity in the hypothalamus, as reflected in the levels of cyclooxygena

110 LepNPEG5K and activated leptin receptors in hypothalamus at lower dose than native leptin.

111 eptors in the median preoptic nucleus of the hypothalamus, but the origin of the pyrogenic PGE2 has n

112 Here we map neuronal identities in the hypothalamus by single-cell RNA sequencing.

113 recombination detected in the hippocampus or hypothalamus by the LacZ or fluorescent tdTomato reporte

114 species, lesions in the preoptic area of the hypothalamus cause profound sleep impairment, indicating

115 and reveal a sufficient and discrete NTS to hypothalamus circuit controlling appetite.

116 ct demonstration of an obligate brainstem-to-hypothalamus circuit orchestrating general stress excita

117 found that Lin28a is highly expressed in the hypothalamus compared with peripheral tissues.

118 levated serotonin transporter binding in the hypothalamus (compared with controls, individuals with L

119 linical observations suggest that the caudal hypothalamus comprises a key node of the ascending arous

120 In vivo whole-cell recordings in the hypothalamus confirmed near-perfect synaptic integration

121 ceptor desensitization in the brain stem and hypothalamus, consistent with exon 7 involvement in morp

122 The hypothalamus contains the highest diversity of neurons i

123 ergic modulation from the A11 nucleus of the hypothalamus contributes to plasticity in a model of chr

124 ecies have indeed confirmed that the lateral hypothalamus contributes to reward mechanisms.

125 top-down projections from the septum to the hypothalamus control food intake negatively.

126 ls, the suprachiasmatic nucleus (SCN) of the hypothalamus coordinates daily rhythms including sleep-w

127 Ex vivo studies with hypothalamus-derived astrocytes showed that LPL expressi

128 lso demonstrate that distinct regions of the hypothalamus differentially control insulin and glucagon

129 Classical descriptions of the hypothalamus divide it into three rostro-caudal domains

130 vior in rats with lesions in the dorsomedial hypothalamus (DMH) challenged with a shock-inducing dose

131 al allele, in the dorsomedial nucleus of the hypothalamus (DMH) developed obesity and reduced energy

132 arge electrolytic lesions of the dorsomedial hypothalamus (DMH) promoted hypothermia in cold-exposed

133 araventricular nucleus (PVN) and dorsomedial hypothalamus (DMH).

134 ntral part of the dorsomedial nucleus of the hypothalamus (DMHv), and to the medial parvocellular par

135 ere was an increase in neuronal birth in the hypothalamus, dorsal thalamus, posterior tuberculum, and

136 ntrast, many cell groups in the midbrain and hypothalamus exhibit low FMRP levels.

137 hippocampus, and amygdala) and homeostatic (hypothalamus) food motivation and hyperactivation in cog

138 Gamma-rhythmic input to the lateral hypothalamus from somatostatin-positive lateral septum c

139 ion data with the chromatin landscape in the hypothalamus, frontal cortex, and amygdala of socially c

140 o knock down Glp1r expression throughout the hypothalamus (GLP-1RKD(DeltaNkx2.1cre)).

141 The hypothalamus has a vital role in controlling food intake

142 The suprachiasmatic nucleus (SCN) of the hypothalamus has an essential role in orchestrating circ

143 or wake-promoting cell groups in the lateral hypothalamus (Hcrt), tuberomammillary nuclei (histamine;

144 It has been proposed that the hypothalamus helps to control ageing, but the mechanisms

145 tterning and neuronal differentiation of the hypothalamus, highlighting recent evidence that illustra

146 ns in zebrafish larvae, which project to the hypothalamus, hindbrain, and spinal cord, including regi

147 ero)phosphate in brain regions including the hypothalamus, hippocampus, and cerebellum.

148 lin signaling in skeletal muscle, brown fat, hypothalamus, hippocampus, and prefrontal cortex but not

149 yzed the transcriptome of the C57BL/6J mouse hypothalamus, hippocampus, neocortex, and cerebellum to

150 glucose-sensing neurons in the ventromedial hypothalamus in glucokinase-Cre mice, which express Cre

151 Our comparative analysis reveals that the hypothalamus in mammals and teleosts has evolved in a di

152 In hypothalamus in vitro, reduced glutamatergic neurotransm

153 hibition of HDAC5 activity in the mediobasal hypothalamus increases food intake and modulates pathway

154 leptin treatment in three brain areas (i.e., hypothalamus, insula/superior temporal gyrus, medial pre

155 Neural pathways, especially those in the hypothalamus, integrate multiple nutritional, hormonal,

156 ior-regulatory nuclei and the neuroendocrine hypothalamus, integrating social and anxiety-related beh

157 NOS was largely restricted to regions of the hypothalamus involved in the control of bodily functions

158 ased from the paraventricular nucleus of the hypothalamus is a major regulator of the HPA axis.

159 The hypothalamus is an evolutionarily conserved brain struct

160 or within the paraventricular nucleus of the hypothalamus is an important central component of hypoth

161 The lateral hypothalamus is known to drive food consumption during p

162 ffects of oxytocin on neural circuits in the hypothalamus is needed to establish the utility of targe

163 ypothalamus, and that MANF expression in the hypothalamus is upregulated in response to fasting.

164 t neuroestradiol, locally synthesized in the hypothalamus, is a part of estrogen's positive feedback

165 cular hypothalamus, Kiss1(AVPV), and arcuate hypothalamus, Kiss1(ARH)), which drive the pulsatile act

166 located in the anteroventral periventricular hypothalamus, Kiss1(AVPV), and arcuate hypothalamus, Kis

167 e posterior thalamic nucleus and the lateral hypothalamus (lateral torus, lateral recess nucleus, hyp

168 Damage to the lateral hypothalamus (LH) causes profound physical inactivity in

169 The lateral hypothalamus (LH) controls energy balance.

170 Electrical stimulation of the lateral hypothalamus (LH) has two motivational effects: long tra

171 However, the lateral hypothalamus (LH) is also a key reward-control locus in

172 shown that increased activity in the lateral hypothalamus (LH) promotes feeding.

173 Projections from the lateral hypothalamus (LH) to the ventral tegmental area (VTA), c

174 ding the lateral habenula (LHb), the lateral hypothalamus (LH), and the midbrain are not only recipro

175 e VLPO-projecting neurons within the lateral hypothalamus (LH), as well as their function in the arou

176 erations in sleep, we focused on the lateral hypothalamus (LH).

177 e feeding via its projections to the lateral hypothalamus (LH).

178 The lateral hypothalamus (LHA) integrates reward and appetitive beha

179 dentified.SIGNIFICANCE STATEMENT The lateral hypothalamus (LHA) regulates motivated feeding behavior

180 A new study suggests that the lateral hypothalamus may also participate in the formation and s

181 iferation and neuroinflammation in the adult hypothalamus may contribute to the pathogenesis of obesi

182 The mediobasal hypothalamus (MBH) contains neurons capable of directly

183 ates microglial reactivity in the mediobasal hypothalamus (MBH) in association with decreased number

184 he current study, we examined the mediobasal hypothalamus (MBH) of 57 obese human subjects and 54 age

185 nant influence on developmentally hard-wired hypothalamus-mediated male territorial aggression.

186 regulation of multiple GPCRs throughout the hypothalamus.Melanocortin receptor accessory protein 2 (

187 eactive fibers were found in the subpallium, hypothalamus, midbrain, and brainstem.

188 stained increases in the amygdala, striatum, hypothalamus, midbrain, right insula, and right dorsolat

189 examined in the serum (protein), spleen, and hypothalamus (mRNA).

190 septum enable separate signalling by lateral hypothalamus neurons according to their feeding-related

191 Within the hypothalamus, neurons that express agouti-related protei

192 increased CADM1 and CADM2 expression in the hypothalamus of human subjects.

193 e PGE2 synthesizing capacity in the anterior hypothalamus of mice lacking such capacity with a lentiv

194 in the suprachiasmatic nucleus (SCN) of the hypothalamus of mice that displayed contagious scratchin

195 sopressin is differentially expressed in the hypothalamus of the two species, with increased levels a

196 te-putamen, nucleus accumbens, thalamus, and hypothalamus) of BAC aldh1l1-translational ribosome affi

197 ells profusely innervated the preoptic area, hypothalamus, optic tectum, semicircular torus, and caud

198 not depend on the activity of neurons in the hypothalamus or on glutamate receptor activation in rRPa

199 The suprachiasmatic nucleus (SCN) of the hypothalamus orchestrates daily rhythms of physiology an

200 d by oxytocin neurons in the paraventricular hypothalamus (Oxt(PVH) neurons), which mildly attenuated

201 uronal activation) in multiple nuclei in the hypothalamus (paraventricular, dorsomedial, and lateral

202 area; bed nuclei of terminal stria; anterior hypothalamus; paraventricular, arcuate, and dorsomedial

203 g expression of Foxa2 mRNA in the developing hypothalamus, pituitary, pancreas, lungs and oesophagus

204 on (HCC) is a promising measure of long-term hypothalamus-pituitary-adrenal (HPA) axis activity.

205 o innervations from adrenergic input and the hypothalamus-pituitary-adrenal (HPA) axis.

206 Instead of consecutive hypothalamus-pituitary-adrenal axis activation, we repor

207 The hypothalamus-pituitary-adrenal axis is sensitive to chan

208 vation and excess glucocorticoid release via hypothalamus-pituitary-adrenal axis stimulation.

209 tions in the neuroendocrine system along the hypothalamus-pituitary-gonadal axis; however, most studi

210 nput to a mathematical model of the hormonal hypothalamus-pituitary-ovarian control of ovulation in w

211 l means due to participation of cells in the hypothalamus-pituitary-ovary feedback control loop.

212 thematical model of the female rainbow trout hypothalamus-pituitary-ovary-liver axis to use as a tool

213 e that contaminant-related activation of the hypothalamus-pituitary-thyroid axis in the kestrels incr

214 eostasis by the feedback loops involving the hypothalamus-pituitary-thyroid axis is disrupted by thes

215 in the paraventricular nucleus (PVN) of the hypothalamus play a large role in the regulation of hepa

216 essing neurons in the arcuate nucleus of the hypothalamus play a pivotal role in the regulation of en

217 tor (NMDAR)-mediated excitatory drive in the hypothalamus plays a critical role in increased sympathe

218 ead transcriptional factor O1 (FoxO1) in the hypothalamus plays a crucial role in mediation of leptin

219 s found to be expressed predominantly in the hypothalamus, pons, and medulla of posthatch chick brain

220 rter binding in the striatum, brainstem, and hypothalamus, possibly reflecting compensatory changes i

221 pecially from cortex/hippocampus to thalamus/hypothalamus posteriorly.

222 ative photoreceptors) are distributed in the hypothalamus (postoptic commissure nucleus, dorsal and v

223 over, quantifications of BDNF protein in the hypothalamus, prefrontal cortex, and hippocampus reveale

224 Similar to the hippocampus, the hypothalamus presents constitutive and stimulated neurog

225 nic receptor type-1 (Chrm1) signaling in the hypothalamus promotes G-CSF-elicited HSC mobilization vi

226 h in the diet and injected directly into the hypothalamus, PUFAs were capable of increasing hypothala

227 ssing satiety neurons in the paraventricular hypothalamus (PVH(MC4R) neurons).

228 mainly in the paraventricular nucleus of the hypothalamus (PVH) but preserved Crh expression in other

229 ularly in the paraventricular nucleus of the hypothalamus (PVH), play an essential role in blood pres

230 vation of the paraventricular nucleus of the hypothalamus (PVH), with fibers and varicosities in clos

231 HPA) effector neurons in the paraventricular hypothalamus (PVH).

232 ctions to the paraventricular nucleus of the hypothalamus (PVN), which is involved in the regulation

233 utside of the paraventricular nucleus of the hypothalamus (PVN).

234 populate the paraventricular nucleus of the hypothalamus (PVN).

235 The hypothalamus receives strong descending inputs from the

236 r, and similarly to native hormone activated hypothalamus receptors after direct injection into brain

237 rier (BBB) to the brain where it acts on the hypothalamus receptors to control appetite and thermogen

238 NAcSh, medial prefrontal cortex, and lateral hypothalamus) recruited by activation of glutamatergic a

239 Also, the inhibition of CaMKII in the hypothalamus reduces elevated blood pressure and sympath

240 As the hypothalamus regulates multiple functional outputs, it i

241 k in the suprachiasmatic nuclei (SCN) of the hypothalamus regulates physiology and behavior across th

242 arious brain regions, their phenotype in the hypothalamus remains largely unknown.

243 ration of T rapidly potentiates amygdala and hypothalamus responses to angry facial expressions.

244 conditional deletion of Shh in the anterior hypothalamus results in a fully penetrant phenotype char

245 transporter, ASBT-expressing neurons in the hypothalamus, revealed a significant decrease following

246 regulation of emotional behavior within the hypothalamus.SIGNIFICANCE STATEMENT Given the rising sci

247 Here we report that hypothalamus-specific Crh knockout mice (Sim1CrhKO mice,

248 rbital cortex is reciprocally connected with hypothalamus, structural abnormalities in these regions

249 veral brain areas both in and outside of the hypothalamus, such as autonomic regions of the brain ste

250 exist between AgRP1 and AgRP2 neurons in the hypothalamus, suggesting communication and coordination

251 s (paraventricular, dorsomedial, and lateral hypothalamus), thalamus (paraventricular and centromedia

252 dies have identified brain areas outside the hypothalamus that are activated under these 'non-homeost

253 citatory vasopressin neurons in the anterior hypothalamus that may gate corticotropin-releasing facto

254 urons of the dorsal raphe nuclei and lateral hypothalamus that project to the mesolimbic dopamine cir

255 ll number of neurons in the larval zebrafish hypothalamus that project widely in the brain.

256 that neural representations within the mouse hypothalamus, that underlie innate social behaviours, ar

257 s are located in the ventral periventricular hypothalamus (the equivalent of the mammalian arcuate nu

258 eeding relies on distinct cell groups in the hypothalamus, the activity of which also changes in anti

259 In the developing hypothalamus, the fat-derived hormone leptin stimulates

260 eurons of the paraventricular nucleus of the hypothalamus, the goal being to determine the extent of

261 sponse is in part mediated by neurons in the hypothalamus, the role of specific cell types in other b

262 lts suggesting that a specific region of the hypothalamus, the rostral posterior hypothalamic nucleus

263 In the arcuate nucleus of the hypothalamus, these changes are needed for lordosis beha

264 en serotonergic control of CRF levels in the hypothalamus to DRN-driven serotonergic control of CRF l

265 to the suprachiasmatic nucleus (SCN) of the hypothalamus to entrain circadian rhythms that are gener

266 atic regulation of body weight by mainly the hypothalamus to include hedonic controls of appetite by

267 e that TCF7L1 is required in the prospective hypothalamus to maintain normal expression of the hypoth

268 ne secretagogue receptor 1a (GHSR1a), in the hypothalamus to signal hunger and promote food intake.

269 pping of axonal projections leading from the hypothalamus to the brainstem and spinal cord.

270 of developmental factors in the amygdala and hypothalamus, underpinned by a systems-wide shift in exp

271 nNOS in the postnatal and adult female mouse hypothalamus using immunohistochemistry.

272 caused by IL-1beta-mediated signaling to the hypothalamus via the vagus nerve.

273 The ventromedial hypothalamus (VMH) and the brain melanocortin system bot

274 The ventromedial hypothalamus (VMH) and the central melanocortin system b

275 AAF) and impaired activation of ventromedial hypothalamus (VMH) glucose-inhibited (GI) neurons by low

276 tergic neurotransmission in the ventromedial hypothalamus (VMH) in response to hypoglycemia and to el

277 The ventromedial nucleus of the hypothalamus (VMH) plays a critical role in regulating s

278 (PR)-expressing neurons in the ventromedial hypothalamus (VMH) that are critical for male territoria

279 entrolateral subdivision of the ventromedial hypothalamus (VMHvl) control mating and fighting in rode

280 n the ventrolateral part of the ventromedial hypothalamus (VMHvl) that express estrogen receptor-alph

281 t the ventrolateral part of the ventromedial hypothalamus (VMHvl), an area with a known role in attac

282 d the ventrolateral part of the ventromedial hypothalamus (VMHvl)-a region required for male mouse ag

283 entrolateral subdivision of the ventromedial hypothalamus (VMHvl).

284 ose-inhibitory neurons from the ventromedial hypothalamus was impaired in BG4KO mice.

285 rthermore, the leptin signal transduction in hypothalamus was improved by salidroside.

286 cifically in the ventromedial nucleus of the hypothalamus was sufficient to alleviate ER stress and t

287 In this study, D1R derived from rat hypothalamus was used as a template and thus self-organi

288 -HT2A) and 5-HT2C serotonin receptors in the hypothalamus were altered by GLP-1R activation.

289 striatal (123)I-FP-CIT binding ratios in the hypothalamus were significantly lower in PSP than in MSA

290 striatal (123)I-FP-CIT binding ratios in the hypothalamus were significantly lower in PSP than in MSA

291 in-coupled receptor expressed in the brain's hypothalamus where it regulates energy homeostasis.

292 rom the paraventricular nucleus (PVN) of the hypothalamus, where loss of OGT was associated with impa

293 deep in the brain parenchyma, such as in the hypothalamus, whereas larger vessels, and particularly t

294 d by the suprachiasmatic nuclei (SCN) in the hypothalamus, which are primarily entrained by light det

295 production in deep brain areas, such as the hypothalamus, which is the site of thermoregulatory neur

296 triggered increased ceramide content in the hypothalamus, which may contribute to hypothalamic insul

297 tyric-acid)-releasing neurons of the lateral hypothalamus, which promote the transition to wakefulnes

298 stria terminalis, dorsal raphe, and lateral hypothalamus, which regulate primitive, yet fundamental

299 The hypothalamus, which regulates fundamental aspects of phy

300 pression in the pituitary as compared to the hypothalamus, with multiple genes more highly expressed