ライフサイエンスコーパス: hypothyroidism

1 een for serum TSH concentration >5 mIUL(-1) (hypothyroidism).

2 had elevated thyroid manganese and developed hypothyroidism.

3 p toward cell-based regenerative therapy for hypothyroidism.

4 role for additional signaling modulators in hypothyroidism.

5 ported a diagnosis of subclinical autoimmune hypothyroidism.

6 ly lost in autoimmune thyroiditis leading to hypothyroidism.

7 adult rodents following prolonged (12 week) hypothyroidism.

8 and 18% in 141 individuals with subclinical hypothyroidism.

9 ith time and dose, likely in compensation to hypothyroidism.

10 ary hypogonadism, adrenal insufficiency, and hypothyroidism.

11 m, proteinuria, reduced kidney function, and hypothyroidism.

12 those affected ultimately develop permanent hypothyroidism.

13 tion, thus creating a state of cell-specific hypothyroidism.

14 n resistance by promoting centrally mediated hypothyroidism.

15 were hearing loss, psychiatric effects, and hypothyroidism.

16 dominantly resulting in blood cytopenias and hypothyroidism.

17 mia, high creatinine, history of stroke, and hypothyroidism.

18 subclinical hyperthyroidism and subclinical hypothyroidism.

19 thyroid hormone levels and of serum PFOA and hypothyroidism.

20 dwh, that rescues hearing despite persistent hypothyroidism.

21 estimate hazard ratios (HRs) and 95% CIs for hypothyroidism.

22 ations in thyroid architecture, and dramatic hypothyroidism.

23 oidism (OR(adj) = 2.3 (95% CI: 1.2, 4.4) and hypothyroidism.

24 1.1, 2.8) was significantly associated with hypothyroidism.

25 mone receptor (TR) during development and in hypothyroidism.

26 eutic targets in the treatment of congenital hypothyroidism.

27 mediates repression by the unliganded TR in hypothyroidism.

28 ression of betaMHC in response to stress and hypothyroidism.

29 ings that accompany both hyperthyroidism and hypothyroidism.

30 g data in patients suffering from congenital hypothyroidism.

31 etabolic differences between mild and severe hypothyroidism.

32 icating that Slc30a10 knock-out mice develop hypothyroidism.

33 yroid participants or those with subclinical hypothyroidism.

34 story was significant for hyperlipidemia and hypothyroidism.

35 t benefits in older persons with subclinical hypothyroidism.

36 ssociated with the development of congenital hypothyroidism.

37 s been used for more than a century to treat hypothyroidism.

38 n diet, thiamine deficiency, alcoholism, and hypothyroidism.

39 Thyroidectomized rats with serum-confirmed hypothyroidism 1 month after surgery were randomized int

40 High-yield tests included thyroid function (hypothyroidism, 10.1%), audiometry (hearing loss, 22.6%)

41 ere were 13 (9.8%) with a prior diagnosis of hypothyroidism, 11 of whom were receiving thyroxine ther

42 eural hearing loss 76%, miscarriage 33%, and hypothyroidism 14%.

43 nic constipation (prevalence 14.2% vs 3.6%), hypothyroidism (14.7% vs 10.4%), obesity (13.0% vs 7.7%)

44 sease (HR 1.93, 95% CI 1.76-2.12; p<0.0001), hypothyroidism (2.31, 2.05-2.60; p<0.0001), and raised t

45 patients (62%) had comorbidities, including hypothyroidism (20%).

46 t common immune-mediated adverse events were hypothyroidism (25 [16%]) and hyperthyroidism (17 [11%])

47 8 [19%]), dysphonia (44 [23%] vs ten [10%]), hypothyroidism (39 [21%] vs seven [7%]), and upper abdom

48 th the known risk factors of diabetes and/or hypothyroidism (46% vs 14% for those without).

49 ne events included pneumonitis (6 [13%]) and hypothyroidism (5 [10%]), mostly </= grade 2.

50 ing hormone deficiency (9.2%), and thyroidal hypothyroidism (5.8%).

51 oration (4%-9%), photosensitivity (25%-75%), hypothyroidism (6%), hyperthyroidism (0.9%-2%), pulmonar

52 Hypothyroidism, a metabolic disease characterized by low

53 cellular carcinoma, severe anemia, untreated hypothyroidism, Addison syndrome and major depression be

54 a 21% excess risk of developing subclinical hypothyroidism (adjusted HR 1.21; 95% CI 1.03-1.42).

55 a (adjusted OR = 1.5; 95% CI = 1.4-1.6), and hypothyroidism (adjusted OR = 1.2; 95% CI = 1.1-1.4).

56 These societies argue that subclinical hypothyroidism adversely affects cardiovascular outcomes

57 ecific NEMO knock-out mice gradually develop hypothyroidism after birth, which leads to reduced body

58 he odds of MCI with clinical and subclinical hypothyroidism among men was 1.02 (95% CI, 0.57-1.82) an

59 f interest, thyroid dysfunction (10 cases of hypothyroidism and 1 of Graves disease) developed in 11

60 s of follow-up), 1958 (6.5%) had subclinical hypothyroidism and 2574 individuals (8.6%) developed AF

61 90 participants, 2068 (8.1%) had subclinical hypothyroidism and 648 (2.6%) had subclinical hyperthyro

62 ansaminitis and hypophysitis with documented hypothyroidism and adrenal insufficiency.

63 ng development results in maternal and fetal hypothyroidism and associated serious adverse health eff

64 Maternal hypothyroidism and asthma in the children were defined b

65 t HDAC inhibitors are clearly beneficial for hypothyroidism and could be therapeutics for treatment.

66 1/5(dKO)) mice displayed growth retardation, hypothyroidism and defective follicular architecture.

67 Hypothyroidism and Hashimoto's thyroiditis are more comm

68 ignificant elevated risk association between hypothyroidism and HCC in women that was independent of

69 We investigated the association between hypothyroidism and HCC risk in men and women in a case-c

70 indicated a significant association between hypothyroidism and HCC, with an approximate two-fold to

71 ween ever use of organochlorines and risk of hypothyroidism and hyperthyroidism among female spouses

72 As a consequence, both hypothyroidism and hyperthyroidism associate with clinic

73 eatment of subclinical and undiagnosed overt hypothyroidism and hyperthyroidism in adults without goi

74 Both hypothyroidism and hyperthyroidism lead to increased AF

75 Hypothyroidism and hyperthyroidism produced opposite ele

76 thyroid disease was 12.5%, and prevalence of hypothyroidism and hyperthyroidism was 6.9% and 2.1%, re

77 dence suggests that milder forms of maternal hypothyroidism and hypothyroxinemia during pregnancy are

78 ever, its use can cause side effects such as hypothyroidism and increased triglyceride concentrations

79 Hypothyroidism and its presenting signs and symptoms are

80 evant molecular mechanisms linked to central hypothyroidism and macroorchidism in IGSF1 deficiency ar

81 e investigated a common pituitary origin for hypothyroidism and macroorchidism, and the role of IGSF1

82 in Superfamily 1) gene defects cause central hypothyroidism and macroorchidism.

83 association between clinical or subclinical hypothyroidism and MCI (odds ratio [OR], 0.99 [95% CI, 0

84 onstrated an association between subclinical hypothyroidism and metabolic syndrome and selected compo

85 of important co-morbidities (e.g., diabetes, hypothyroidism and metabolic syndrome).

86 ntiating transient from permanent congenital hypothyroidism and predicting the response of patients t

87 sm by which GLIS3 deficiency causes neonatal hypothyroidism and prevents goiter development.

88 Thirty percent of HMTs developed hypothyroidism and required long-term T4 supplementation

89 These mice provide a new model of central hypothyroidism and reveal a critical role for D3 in the

90 ll increase in the prevalence of subclinical hypothyroidism and thyroid autoimmunity; whether these i

91 , isolated cleft lip and cleft palate (CLP), hypothyroidism and thyroid cancer all map to the FOXE1 l

92 etermine the mechanisms of manganese-induced hypothyroidism and understand how SLC30A10 and SLC39A14

93 Switching on BRAF(V600E) rapidly induced hypothyroidism and virtually abolished thyroid-specific

94 chemotherapy with cediranib, and diarrhoea, hypothyroidism and voice changes were more common during

95 thyroidism, 1.4% primary and 0.1% iatrogenic hypothyroidism, and 1.3% other/unspecified thyroid disea

96 d function, 17% in 313 persons with clinical hypothyroidism, and 18% in 141 individuals with subclini

97 be caused by medications use, renal failure, hypothyroidism, and by prolactinoma - PRL secreting tumo

98 than 50% of women with previously diagnosed hypothyroidism, and can be managed by increasing the lev

99 reatinine and creatine phosphokinase values, hypothyroidism, and cardiac involvement.

100 s mellitus, myopathy, pulmonary dysfunction, hypothyroidism, and death increased as time off cysteami

101 okers, have a history of hemorrhagic stroke, hypothyroidism, and dementia, and less likely to be trea

102 scabies, chronic mucocutaneous candidiasis, hypothyroidism, and esophageal squamous cell carcinoma.

103 one case each of grade 3 pemphigoid, grade 3 hypothyroidism, and grade 3 peripheral sensory neuropath

104 associated with a decline in renal function, hypothyroidism, and hypercalcaemia.

105 e studied 3 sisters with primary amenorrhea, hypothyroidism, and hypergonadotropic hypogonadism.

106 of human kindreds suffering from congenital hypothyroidism, and in the cog congenital goiter mouse a

107 of CD40 on TECs, development of fibrosis and hypothyroidism, and increased expression of proinflammat

108 hypothyroidism is more common than is overt hypothyroidism, and is usually defined as a serum thyroi

109 matic mutation carriers at risk of recurrent hypothyroidism, and provides a rationale for adjunct iod

110 en were found with autoimmune thyroiditis or hypothyroidism, and replacement therapy was initiated in

111 ase, obesity, hyperlipidemia, renal disease, hypothyroidism, and the number of outpatient visits for

112 ction within the normal range or subclinical hypothyroidism are also associated with AF.

113 Constipation, obesity, and hypothyroidism are associated comorbidities.

114 of the genomic changes mediated by the TR in hypothyroidism are independent of NCoR1, suggesting a ro

115 mechanism by which GLIS3 dysfunction causes hypothyroidism are unknown.

116 eases, type 1 diabetes, hyperthyroidism, and hypothyroidism, are the result of autoimmunity.

117 -subclinical hyperthyroidism and subclinical hypothyroidism--are common clinical entities that encomp

118 Some authors have defined subclinical hypothyroidism as a serum TSH between 5 and 10 mIU/L, an

119 serum TSH between 5 and 10 mIU/L, and overt hypothyroidism as a serum TSH greater than 10 mIU/L, but

120 ding hypertension and indicate developmental hypothyroidism as an epigenetic risk factor for cardiova

121 ne (TSH) 0.45 to 4.49 mIU/L, and subclinical hypothyroidism as TSH 4.5 to 19.9 mIU/L with free thyrox

122 ed as TSH of 0.45 to 4.49 mIU/L, subclinical hypothyroidism as TSH of 4.5 to 19.9 mIU/L, and subclini

123 of developing autoimmune diseases, including hypothyroidism, as seen with cancer patients undergoing

124 d CD had increased rates of osteoporosis and hypothyroidism, as well as lower body mass index and lev

125 Additionally, the first case of hypothyroidism associated with a collodion baby was repo

126 pecific TRbeta1-KO mice, we demonstrate that hypothyroidism-associated changes in gene expression and

127 orticotropic hormone deficiency, and primary hypothyroidism at 4 years from diagnosis was 93% +/- 4%,

128 rium at 9q22 near FOXE1 were associated with hypothyroidism at genome-wide significance, the stronges

129 glucose/galactose malabsorption, congenital hypothyroidism, Bartter's syndrome, epilepsy, depression

130 For subclinical hypothyroidism (based on thyroid-stimulating hormone lev

131 Severe iodine deficiency causes goitre and hypothyroidism because, despite an increase in thyroid a

132 s in pubertal timing, development of primary hypothyroidism, breast cancer as a second malignant neop

133 ironmental 131I exposure during childhood on hypothyroidism, but not other thyroid outcomes.

134 Hypothyroidism can have a variety of presentations.

135 Hypothyroidism can lead to depressed breathing.

136 Vision and hearing disorders as well as hypothyroidism can negatively impact cognitive functioni

137 evothyroxine, which is usually used to treat hypothyroidism, can be considered in such situations.

138 Using a mouse model of hypothyroidism caused by a dominant negative TRalpha1PV

139 Overview of congenital hypothyroidism caused by thyroid hormone synthesis defec

140 Central congenital hypothyroidism (CCH) is more prevalent in children born

141 ype mice, the bacterial components induced a hypothyroidism characterized by elements of both hypotha

142 st thyroglobulin, and development of primary hypothyroidism (decreased levels of serum thyroxin, and

143 before 20 weeks of gestation for subclinical hypothyroidism, defined as a thyrotropin level of 4.00 m

144 e-treated patients with residual symptoms of hypothyroidism despite normalization of the serum TSH ha

145 models where autoimmune thyroid disease and hypothyroidism develop in most mice by 4 mo of age.

146 1.02-1.24) for children born to mothers with hypothyroidism diagnosed before and after delivery, comp

147 ren, 3524 children were born to mothers with hypothyroidism diagnosed before delivery and 4664 diagno

148 observed among children born to mothers with hypothyroidism diagnosed before delivery who did not rec

149 his study while accounting for the timing of hypothyroidism diagnosis.

150 This NTI hypothyroidism did not develop in mice lacking the MyD88

151 12 weeks 3 days) and maternal treatment for hypothyroidism did not result in improved cognitive func

152 adequate evidence that treating subclinical hypothyroidism does not provide clinically meaningful im

153 a non-functional protein lead to congenital hypothyroidism due to I(-) transport defect (ITD).

154 Congenital hypothyroidism due to thyroid dyshormonogenesis is a het

155 Identification of hypothyroidism during pregnancy continues to be challeng

156 Kcne2-deficient mice had hypothyroidism, dwarfism, alopecia, goiter and cardiac a

157 Our findings suggest that maternal hypothyroidism, especially when it is untreated, increas

158 and surrounding tissues leading to profound hypothyroidism, findings consistent with our in vitro re

159 thyroidism in pregnancy, and consequences of hypothyroidism for the fetus and child.

160 Hypothyroidism frequently occurs after treatment for ped

161 ropin and gonadotropin deficiencies, primary hypothyroidism, gonadal failure and obesity.

162 each had grade 3 rhabdomyolysis and grade 2 hypothyroidism; grade 3 iridocyclitis, grade 1 erythema

163 we describe a child with classic features of hypothyroidism (growth retardation, developmental retard

164 A long-term history of hypothyroidism (>10 years) was associated with a statist

165 Specifically, individuals with subclinical hypothyroidism had an adjusted hazard ratio of 1.07 (95%

166 However, in some patients, hypothyroidism has been ameliorated by unusually large a

167 Hypothyroidism has been associated with nonalcoholic ste

168 Maternal hypothyroidism has previously been shown to increase ris

169 rs7850258 allele (G) associated with CLP and hypothyroidism has significantly greater enhancer activi

170 he effects of antiepileptic drugs in central hypothyroidism have not yet been studied, substantial ev

171 ed with survivors not so exposed for primary hypothyroidism (hazard ratio [HR], 6.6; 95% CI, 5.6 to 7

172 For participants with subclinical hypothyroidism, HRs from higher-quality studies were 1.1

173 ipheral neuropathy were evaluated: diabetes, hypothyroidism, hypercholesterolemia, hypertension, vari

174 At least half of the patients had hypothyroidism, hypergonadotropic hypogonadism (in men),

175 sk of reduced urinary concentrating ability, hypothyroidism, hyperparathyroidism, and weight gain.

176 positive associations with antibody-positive hypothyroidism, hyperthyroidism, AIT, or elevated ATPO.

177 sterol, renal impairment, diabetes, obesity, hypothyroidism, hyperthyroidism, anaemia, respiratory di

178 associated with an increased odds ratio for hypothyroidism, hyperthyroidism, and antithyroid antibod

179 rral, knowledge of the signs and symptoms of hypothyroidism, hyperthyroidism, and thyroid nodules, as

180 We conducted a cross-sectional analysis of hypothyroidism, hyperthyroidism, autoimmune thyroiditis

181 regression models compared risks of primary hypothyroidism, hyperthyroidism, thyroid neoplasms, hypo

182 n, radiological assessment, and treatment of hypothyroidism, hyperthyroidism, thyroid nodules, and th

183 gan toxicity has been limited to correctable hypothyroidism in 42% of irradiated patients, and one ca

184 ts developing after transplantation included hypothyroidism in 5 subjects and enteritis in 1 subject.

185 r radiotherapy were asymptomatic compensated hypothyroidism in 9 patients (10%), osteonecrosis and mo

186 We report here a case of acquired hypothyroidism in a pediatric patient who first presente

187 common findings that can be associated with hypothyroidism in children.

188 d mice developed multiple autoantibodies and hypothyroidism in contrast to mice treated with bone mar

189 Hypothyroidism in fetal sheep induced by removal of the

190 d mutations in the ChEL domain to congenital hypothyroidism in humans and rodents; these mutations bl

191 1 by TRalpha1 mutants could lead to clinical hypothyroidism in humans.

192 Mutations in DUOX2 produce congenital hypothyroidism in humans.

193 Genetic disruption of KCNQ1-KCNE2 causes hypothyroidism in mice, resulting in cardiac hypertrophy

194 oid hormone receptor alpha gene (THRA) cause hypothyroidism in patients with growth and developmental

195 We tested the hypothesis that the severe hypothyroidism in patients with THRA mutations is due to

196 targets in treating TRalpha1 mutant-mediated hypothyroidism in patients.

197 h has focused on detection and management of hypothyroidism in pregnancy, and consequences of hypothy

198 Early detection and appropriate treatment of hypothyroidism in pregnant women may be an area for poss

199 2320R mutation is responsible for congenital hypothyroidism in rdw/rdw rats, in which a lack of secon

200 presents a spontaneous, inherited recessive hypothyroidism in Sprague-Dawley rats with devastating f

201 The association between 131I dose and hypothyroidism in the Belarusian cohort is consistent wi

202 terest in the possible link between maternal hypothyroidism in the perinatal period and childhood ast

203 Hypothyroidism in the sheep fetus resulted in an asymmet

204 ue the high mortality rate due to congenital hypothyroidism in these mice.

205 omponents, whole immunoglobulins induced NTI hypothyroidism in wild-type mice, but not in those lacki

206 Hypothyroidism increased atrial interstitial fibrosis, b

207 The risk for hypothyroidism increased with the duration of sunitinib

208 genetic complexity of protective effects on hypothyroidism-induced hearing impairment, an F1 intercr

209 hance our understanding of the mechanisms of hypothyroidism-induced hearing impairment.

210 Hypothyroidism-induced NAFLD has generally been attribut

211 Our results show that the pathogenesis of hypothyroidism-induced NAFLD is both intra- and extrahep

212 yndrome, hemolytic anemia, thrombocytopenia, hypothyroidism, inflammatory myositis, Raynaud's disease

213 ge (identical to that causing human goitrous hypothyroidism) inhibits this maturation, although the C

214 in NLGN) mutation responsible for congenital hypothyroidism into NLGN3, we show that mutations in the

215 t we discovered a tertiary form of inherited hypothyroidism involving the hypothalamus.

216 Hypothyroidism is a common condition of thyroid hormone

217 Recent studies suggest that long-term hypothyroidism is associated with human hepatocellular c

218 The definition of hypothyroidism is based on statistical reference ranges

219 he use of levothyroxine to treat subclinical hypothyroidism is controversial.

220 Once overt hypothyroidism is diagnosed, treatment with levothyroxin

221 ociated with HLA SNP variation and show that hypothyroidism is genetically correlated with Type I dia

222 ormone accelerates energy expenditure; thus, hypothyroidism is intuitively associated with obesity.

223 Subclinical hypothyroidism is known to be associated with increased

224 The current treatment for patients with hypothyroidism is levothyroxine (L-T4) along with normal

225 Subclinical hypothyroidism is more common than is overt hypothyroidi

226 (4) to T(3) in the elderly, in whom marginal hypothyroidism is relatively common.

227 Congenital hypothyroidism is the most common neonatal endocrine dis

228 Hypothyroidism is the most common pregnancy-related thyr

229 Because hypothyroidism leads to growth defects and premature dea

230 ologic disorders, chronic pulmonary disease, hypothyroidism, liver disease, AIDS, coagulopathy, defic

231 cant positive associations of 131I dose with hypothyroidism (mainly subclinical and antibody-negative

232 n, these findings suggest that developmental hypothyroidism may be the cause of cardiovascular disord

233 iodine deficiency-related maternal and child hypothyroidism may cause severe mental retardation, rece

234 Evidence indicates that cardiac hypothyroidism may contribute to heart failure progressi

235 Mutations causing hypothyroidism might induce solely local/regional misfol

236 mic immune-mediated complications, including hypothyroidism (n = 3), colitis (n = 2), and hepatitis (

237 The odds ratio for hypothyroidism (n = 39) was 1.54 [95% confidence interva

238 naemia (n=2), fatigue (n=1), rash (n=1), and hypothyroidism (n=1).

239 syndrome of neonatal diabetes and congenital hypothyroidism (NDH).

240 ted in a syndrome with neonatal diabetes and hypothyroidism (NDH).

241 We also report novel SNP associations for hypothyroidism near HLA-DQA1/HLA-DQB1 at rs6906021 (comb

242 ther features, including ichthyosis, primary hypothyroidism, neurological symptoms, and cryptorchidis

243 SUV correlated neither with the degree of hypothyroidism nor with the titer of TPO antibodies.

244 Grade 2 hypothyroidism occurred in 43% of patients.

245 Sedation, weight increase, and hypothyroidism occurred more frequently in the quetiapin

246 sease was associated with increased rates of hypothyroidism (odds ratio, 2.2; P < .01) and a lower th

247 oidism increases AF incidence, the effect of hypothyroidism on AF is unclear.

248 l will be useful for studying the effects of hypothyroidism on multiple organ systems.

249 Sixty-three (47.4%) had a prior diagnosis of hypothyroidism or autoimmune thyroiditis, of whom 56 wer

250 Treatment for subclinical hypothyroidism or hypothyroxinemia beginning between 8 a

251 men who are identified as having subclinical hypothyroidism or hypothyroxinemia during pregnancy impr

252 eening of all pregnant women for subclinical hypothyroidism or thyroid autoimmunity is not recommende

253 ; however, no symptoms or signs of potential hypothyroidism or thyrotoxicosis were seen.

254 The strongest association in the PheWAS was hypothyroidism (OR = 0.76, p = 2.7 x 10(-13)), which had

255 .12; (95% CI: 0.83, 1.52) (n=105 cases); for hypothyroidism, OR=1.35 (95% CI: 0.86, 2.13) (n=49 cases

256 ted with endocrine or metabolic abnormality (hypothyroidism, panhypopituitarism and renal rickets).

257 Those results persist after excluding hypothyroidism patients from the statistical analysis.

258 Thus, the hypothyroidism phenotype of Slc30a10 single knockouts is

259 , we characterize a mouse model of secondary hypothyroidism, pituitary transcription factor 1 (Pit1(d

260 Clinical manifestations of hypothyroidism range from life threatening to no signs o

261 For patients with subclinical hypothyroidism, recommendations for therapy differ betwe

262 perspectives on whether treating subclinical hypothyroidism reduces morbidity and mortality, whether

263 Chronic hypothyroidism resulted in severely blunted basal human

264 -specific iPSCs generated from patients with hypothyroidism resulting from NKX2-1 haploinsufficiency.

265 valuate the relationship between subclinical hypothyroidism (SCH) and all-cause and cardiovascular di

266 Monotherapy Fails to Restore All Markers of Hypothyroidism" should read "new technology is needed to

267 ariates-prolonged hyperthyroidism, prolonged hypothyroidism, smoking, treatment with an antithyroid d

268 ss the epidemiology, causes, and symptoms of hypothyroidism; summarise evidence on diagnosis, long-te

269 those of patients with neonatal diabetes and hypothyroidism syndrome, including the development of di

270 a genome-wide association study for primary hypothyroidism, the most common thyroid disease.

271 rs of age and who had persisting subclinical hypothyroidism (thyrotropin level, 4.60 to 19.99 mIU per

272 fferent thyroid hormone levels, ranging from hypothyroidism to hyperthyroidism on AF inducibility in

273 ataplexy, hypocretin deficiency, and central hypothyroidism, together with brainstem encephalitis ref

274 ism did not affect graft function, but acute hypothyroidism transiently impaired human C-peptide secr

275 In the subclinical hypothyroidism trial, the median IQ score of the childre

276 A total of 677 women with subclinical hypothyroidism underwent randomization at a mean of 16.7

277 The excess odds ratio per 1 Gy for hypothyroidism was 0.34 (95% CI: 0.15, 0.62) and varied

278 rt study found that treatment of subclinical hypothyroidism was associated with decreased risk for co

279 e analyzed, an increased risk of subclinical hypothyroidism was associated with high blood pressure (

280 rly people, neither clinical nor subclinical hypothyroidism was associated with MCI.

281 Iatrogenic hypothyroidism was associated with sepsis and neonatal a

282 Hypothyroidism was generated in SCID-beige mice using an

283 The prevalence of clinical hypothyroidism was increased in patients taking lithium

284 Hypothyroidism was induced by administering 0.04% PTU in

285 Hypothyroidism was reduced in those taking valproate (HR

286 Hypothyroidism was the most common abnormality with othe

287 Clinical and subclinical hypothyroidism were ascertained from a medical records l

288 Hyperthyroidism and primary hypothyroidism were associated with sepsis, respiratory

289 Clinical and subclinical hypothyroidism were associated with worse Global gait th

290 GHD and primary hypothyroidism were diagnosed in a majority of subjects

291 .2 years, the incident rates for subclinical hypothyroidism were substantially higher in participants

292 cases distinguishing between NTIS and severe hypothyroidism, which is a rare primary cause for admiss

293 at a greater risk for developing subclinical hypothyroidism, while its mechanisms and temporal conseq

294 can result in autosomal recessive congenital hypothyroidism with deficient thyroglobulin.

295 There was an increased odds of hypothyroidism with ever use of organochlorine insectici

296 TBMEHP exposure in the rat produced maternal hypothyroidism with markedly decreased serum T3 (3,3 ,5-

297 Association of clinical and subclinical hypothyroidism with MCI.

298 An association of clinical and subclinical hypothyroidism with mild cognitive impairment (MCI) has

299 The patient showed congenital central hypothyroidism with reduced TSH biopotency, over-secreti

300 to adulthood, D3KO mice demonstrate central hypothyroidism, with low serum levels of 3,5,3',5'-tetra