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1 100 mmol/L glucose, or no additional solute (hypotonic solution).
2 howed impaired regulatory volume decrease in hypotonic solution.
3 nd blocked cell swelling normally induced by hypotonic solution.
4 ed ciliary epithelial (NPCE) cells by a 23 % hypotonic solution.
5 onic solution and was decreased 38% by a 33% hypotonic solution.
6 volume decrease during sustained exposure to hypotonic solution.
7 rtonic media but with a steeper slope in the hypotonic solutions.
8 cells secreted insulin when challenged with hypotonic solutions.
9 in cRNA by measuring the rate of swelling in hypotonic solutions.
10 also observed in cells following exposure to hypotonic solutions.
11 t sympathetic neurones during application of hypotonic solutions.
12 esicles followed by washes with isotonic and hypotonic solutions.
13 ha-phorbol 12,13-didecanoate (4alphaPDD) and hypotonic solutions.
14 esponses to the TRPV4 agonists 4alphaPDD and hypotonic solutions.
18 o single-fiber recordings in rat showed that hypotonic solution activated 54% of C-fibers, an effect
22 al membrane is permeable when replicating in hypotonic solution and within macrophages, resulting in
23 anges in relative cell volume in response to hypotonic solutions and glucose were studied in single i
26 could be repeatedly activated by exposure to hypotonic solution but when ATP was omitted the current
27 cell and perforated patch recording methods, hypotonic solution caused cell swelling and the activati
30 and water content were combined to show that hypotonic solutions caused water to move from the extrac
31 icantly attenuated cell volume regulation in hypotonic solutions, consistent with VRAC inhibition.
38 ngs, cell swelling induced by 1) exposure to hypotonic solution or 2) intracellular perfusion with hy
39 owing cell swelling induced by exposure to a hypotonic solution or in response to calcium-mobilizing
42 ic acid and, to a lesser extent, DIDS to the hypotonic solution potentiated swelling and blocked the
43 sodium absorption greater than control with hypotonic solution represented sodium absorption stimula
44 by glucose, greater than that noted with the hypotonic solution, represented sodium absorption stimul
48 ed with isomolar amounts of sucrose, whereas hypotonic solution was the same as isotonic solution wit
50 ls, we show that Ca2+ influx is triggered by hypotonic solutions, which can be partially blocked by G
51 currents inactivated during the exposure to hypotonic solution with time constants of 59 and 304 s f
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