ライフサイエンスコーパス: hypotonic stress

1 eness) and in kidney and heart (resulting in hypotonic stress).

2 cei brucei was reminiscent of swelling under hypotonic stress.

3 response to AVP stimulation and low chloride hypotonic stress.

4 imuli, including ionising radiation (IR) and hypotonic stress.

5 um, mercury, and zinc exposure as well as by hypotonic stress.

6 inhibition of protein synthesis mediated by hypotonic stress.

7 holine, and during mechanical stimulation by hypotonic stress.

8 n basal ATP release or release stimulated by hypotonic stress.

9 l role in allowing the cell to survive acute hypotonic stress.

10 exhibited distinctly different responses to hypotonic stress.

11 icantly decreased ATP release in response to hypotonic stress.

12 ry human amnion cells were also increased by hypotonic stress.

13 C-R cells exhibited enhanced survival during hypotonic stress.

14 HOG1 like kinase (p38) was not activated by hypotonic stress.

15 for efficient ATM signalling in response to hypotonic stress.

16 expression of factors required to respond to hypotonic stress - a mechanism that may allow the cell t

17 In addition, hypotonic stress also led to a modest increase in WNK1 a

18 nd, NCCbeta is not activated by low-chloride hypotonic stress, although the unique Ste20-related prol

19 omplexes and other osmotic stressors, namely hypotonic stress and glutamate, induced transient swelli

20 ls, basal ATM activity and ATM activation by hypotonic stress and inhibition of DNA replication was i

21 One of these kinases, Lyn, was activated by hypotonic stress and phosphorylated TRPV4 in an immune c

22 Both hypotonic stress- and ATP-induced processing were blocke

23 mbrane Cl- currents that can be activated by hypotonic stress are involved in mechanisms controlling

24 Our results identify hypotonic stress as a (previously unrecognized) contribu

25 Insig-2 is not affected by hypotonic stress because of its slower turnover rate.

26 esponse to ATM activation by chloroquine and hypotonic stress, but not after induction of double-stra

27 s together constitute the VRAC pore and that hypotonic stress can activate VRAC through a decrease in

28 Hypotonic stress causes rapid cell swelling and initiate

29 Hypotonic stress, causing tension in the vesicle membran

30 Increasing the hypotonic stress did not result in an increased activati

31 nase is specific for hyperosmotic stress, as hypotonic stress does not activate it.

32 Hypotonic stress (from 280 to 221 mosmol kg(-1)) to roun

33 Hypotonic stress immediately (within 5 s) activated tyro

34 helial cells release less ATP in response to hypotonic stress in an inflammatory environment (IFN-gam

35 The activity induced by hypotonic stress in erythrocyte membranes had the pH dep

36 mammalian hypothalamus, and is activated by hypotonic stress in vitro.

37 al actin filaments, sClC3 contributes to the hypotonic stress-induced VSOACs in NIH/3T3 cells.

38 ults suggest that the signaling mechanism of hypotonic stress is distinct from that of hyperosmolar s

39 eduction of [Cl(-)]i, either by low chloride hypotonic stress or coinjection of oocytes with the solu

40 s are unable to fuse vacuoles in response to hypotonic stress or nutrient starvation.

41 f endothelial cells with fluid shear stress, hypotonic stress, or membrane fluidizing agent leads to

42 rks in two different types of cells, and how hypotonic stress promotes tumor formation when the funct

43 ) polymorphism showed diminished response to hypotonic stress (relative to the wild-type channel) and

44 hways, to signal responses to hypertonic and hypotonic stress, respectively.

45 reporter gene constructs suggested that the hypotonic stress response element maps to the serum resp

46 We observed that hypotonic stress resulted in rapid tyrosine phosphorylat

47 In aggregate, these data indicate that hypotonic stress results in Src family tyrosine kinase-d

48 Here we report that hypotonic stress reverses the sterol-mediated inhibition

49 ls also displayed increased constitutive and hypotonic stress-stimulated release of UDP-GlcNAc.

50 Hypotonic stress to flattened, non-dividing cells activa

51 The effect of hypotonic stress was mimicked by chlorpromazine, which i

52 Actinomycin D blocked COX-2 mRNA increase by hypotonic stress, while cycloheximide enhanced COX-2 mRN