ライフサイエンスコーパス: hypoxemia

1 tely and died by 4 months of age with severe hypoxemia.

2 he venular endothelium to the same extent as hypoxemia.

3 nal endothelial cell injury) associated with hypoxemia.

4 e control group received HFOV for refractory hypoxemia.

5 ble coughing, urticaria, edema, wheezing and hypoxemia.

6 copy following hypoventilation, may decrease hypoxemia.

7 in patients with severe ARDS and refractory hypoxemia.

8 e development of life-threatening refractory hypoxemia.

9 ked by impaired gas exchange and significant hypoxemia.

10 esterol levels, compared to subjects without hypoxemia.

11 me oxygen values, have only mild to moderate hypoxemia.

12 dyspnea and oxygen for short-term relief of hypoxemia.

13 ems accurate, especially for moderate-severe hypoxemia.

14 and body mass index influenced ictal-related hypoxemia.

15 management of silent infarcts and nocturnal hypoxemia.

16 ation in critically ill patients with severe hypoxemia.

17 o are oxygen non-responsive with intractable hypoxemia.

18 ntially aggravating systemic hypercarbia and hypoxemia.

19 pulmonary disease (COPD) and severe resting hypoxemia.

20 BCs is impaired in diseases characterized by hypoxemia.

21 e lung injury, patchy alveolar flooding, and hypoxemia.

22 ion and pulmonary arterial hypertension with hypoxemia.

23 t-to-left shunt pathophysiology and systemic hypoxemia.

24 and concerns over hypercapnia, acidosis, and hypoxemia.

25 ake at anaerobic threshold, and 54 developed hypoxemia.

26 ce of acute lung injury and resultant severe hypoxemia.

27 of infant human and rabbit hearts to chronic hypoxemia.

28 aryngoscopy (apneic oxygenation) may prevent hypoxemia.

29 d for oxygen and is especially vulnerable to hypoxemia.

30 cardia did not alter the prognostic value of hypoxemia.

31 mes were similarly increased after prolonged hypoxemia.

32 to safety process measures, and frequency of hypoxemia.

33 dent sympathetic activation and/or degree of hypoxemia.

34 ochemical indicators of vitamin A status and hypoxemia.

35 atients is frequently associated with severe hypoxemia.

36 fetus as growth restriction and progressive hypoxemia.

37 la may offer an alternative in patients with hypoxemia.

38 including the management of HPS with severe hypoxemia.

39 are similar to those of SCD mice but without hypoxemia.

40 ts was associated with a lower prevalence of hypoxemia (10.1% vs. 17.4%, p = .022) and a lower preval

41 in more than one patient included transient hypoxemia (19%), radiation pneumonitis (11%), and fatigu

42 sk group experienced more episodes of severe hypoxemia (2% vs 14%, p = 0.03).

43 in the presence of respiratory distress and hypoxemia (2C), use of physical examination therapeutic

44 subjects with severe COPD and only moderate hypoxemia; (3) efficacy of nocturnal O(2) supplementatio

45 e likely than those with normal CXRs to have hypoxemia (45% vs 26%), crackles (69% vs 62%), tachypnea

46 transfusion, higher fluid balance, obesity, hypoxemia, acidosis, tobacco use, emergent hematoma evac

47 Randomized clinical trial of patients with hypoxemia after cardiac surgery at a single ICU in Brazi

48 Among patients with hypoxemia after cardiac surgery, the use of an intensive

49 bation and improve outcomes of patients with hypoxemia after cardiothoracic surgery.

50 ere 67% and 52% (P = .02), and for recurrent hypoxemia after oxygen supplementation were 38% and 18%

51 The hypoxemia after sildenafil administration in group B imp

52 ium further than either hemorrhagic shock or hypoxemia alone did.

53 Hypoxemia also triggered leukocytes adhesion to the venu

54 Nocturnal hypoxemia, an important pathophysiological feature of OS

55 t the immediate implementation of permissive hypoxemia and a comprehensive evaluation of its value in

56 ficant correlations noted between CYP2E1 and hypoxemia and beta-OH butyrate suggest that these factor

57 To examine whether patients with acute hypoxemia and bilateral opacities treated with high-flow

58 n is influenced by the severity of nocturnal hypoxemia and comorbid obesity has not been determined.

59 of dyspnea and fatigue, partially related to hypoxemia and compromised oxygen delivery.

60 dema) in the alveolar airspaces and leads to hypoxemia and death if not corrected.

61 Hypoxemia and enhanced ventilatory demands result, altho

62 vel to altitude, but this may expose them to hypoxemia and exacerbation of sleep apnea.

63 The association of hypoxemia and hemorrhagic shock did not amplify leukocyt

64 The association of hypoxemia and hemorrhagic shock did not further amplify

65 Ictal hypoxemia and hypercapnia may contribute to SUDEP.

66 These effects, as well as hypoxemia and hypotension, were prevented by 3-aminobenz

67 tein-rich pulmonary edema that causes severe hypoxemia and impaired carbon dioxide excretion.

68 atous optic neuropathy by creating transient hypoxemia and increasing vascular resistance.

69 ng transplant and is characterized by severe hypoxemia and infiltrates in the allograft.

70 ciation was partially explained by overnight hypoxemia and less so by sleep duration.

71 ion of the great arteries is associated with hypoxemia and longer time to surgery.

72 lps detect extracardiac pathology leading to hypoxemia and may be used to guide fluid resuscitation a

73 ificantly associated with the level of fetal hypoxemia and metabolic stress.

74 ease are at risk for respiratory failure and hypoxemia and need to be screened for hepatopulmonary sy

75 is associated with a decreased prevalence of hypoxemia and reduced rate of procedure-related complica

76 physiologic factors, such as propensity for hypoxemia and respiratory arousability.

77 main patent and subject patients to arterial hypoxemia and risk for paradoxical emboli.

78 l life support can lead to rapid reversal of hypoxemia and shock; however, it can also result in vary

79 long-term survival relative to the degree of hypoxemia and the era in which LT was conducted.

80 he severity of lung injury and the degree of hypoxemia and/or the effects of ventilator settings on g

81 Refractory hypoxemia and/or uncompensated hypercapnia despite optim

82 a, and hypomagnesemia; 14 patients (52%) had hypoxemia, and 9 (33%) had oliguria, of whom 5 had anuri

83 ATP content of bronchoalveolar lavage fluid, hypoxemia, and altered nasal potential difference.

84 cular outflow tract obstruction, unexplained hypoxemia, and aortic dissection, among others, can all

85 reducing the frequency of hypoxemia, severe hypoxemia, and apnea.

86 rapy were associated significantly with LRD, hypoxemia, and death.

87 o mobilization were hemodynamic instability, hypoxemia, and dependency on venovenous extracorporeal m

88 ermeability pulmonary edema, severe arterial hypoxemia, and impaired carbon dioxide excretion.

89 rs appear to be exaggerated polycythemia and hypoxemia, and lower and sluggish CBF compared to CMS pa

90 ed sympathetic activity, hemodynamic stress, hypoxemia, and oxidative stress.

91 rioration, sepsis, hyperleukocytosis, severe hypoxemia, and prolonged mechanical ventilation.

92 1 had lower respiratory tract disease (LRD), hypoxemia, and prolonged viral shedding compared with se

93 infected mice from weight loss, hypothermia, hypoxemia, and respiratory compromise.

94 >/=90% [moderate hypoxemia] or <90% [severe hypoxemia]) and 13 obese control subjects.

95 ry end points were the occurrences of severe hypoxemia, apnea, and oxygen supplementation.

96 Hypoxemia appears to be an important factor that underli

97 Obstructive sleep apnea (OSA) and nocturnal hypoxemia are associated with chronic kidney disease and

98 ce rate of ICU-acquired infection and severe hypoxemia are expected outcomes from RBC transfusion tha

99 determine the effect of postoperative IS on hypoxemia, arterial oxygen saturation (Sao2) level, and

100 ntrol of arterial oxygenation and permissive hypoxemia as candidate management strategies in hypoxemi

101 in subsets of patients with mild and severe hypoxemia as defined by PaO(2)/Fio(2).

102 harm will aid the development of permissive hypoxemia as viable clinical strategy.

103 acute myocardial infarction who do not have hypoxemia at baseline is uncertain.

104 We postulate that arterial hypoxemia at high altitude poses a risk of faster diseas

105 More than 40% had hypoxemia at rest (13%) or on exercise (29%), with pulmo

106 rtality, ICU-acquired infections, and severe hypoxemia) at day 30, we used marginal structural models

107 yndrome in acute liver failure are scant and hypoxemia being a commonly encountered systemic complica

108 nt differences in frequency of postoperative hypoxemia between the control and test groups were found

109 horacentesis may be complicated by transient hypoxemia, bleeding, patient discomfort, reexpansion pul

110 Failure to recognize spurious hypoxemia can lead to unnecessary diagnostic tests and t

111 rtality, which was accompanied by attenuated hypoxemia, cardiopulmonary dysfunction, and pulmonary ed

112 e the clearance of Abeta from the brain, and hypoxemia characteristic of sleep-disordered breathing i

113 gue muscles are coactivated in conditions of hypoxemia comparable to that experienced by adults with

114 ardia, tachypnea, hypotension, and prolonged hypoxemia compared with controls.

115 oxygenation and reduced prevalence of severe hypoxemia compared with nonrebreathing bag reservoir fac

116 se to AngII was less in patients with severe hypoxemia compared with those with moderate hypoxemia (P

117 agic telangiectasia: iron deficiency impairs hypoxemia compensations by restricting erythropoiesis an

118 During hemorrhagic shock, the occurrence of hypoxemia considerably alters villous intestinal perfusi

119 The impact of hypoxemia correction on liver disease severity warrants

120 Higher levels of hypoxemia correlated with increased volume and thickness

121 case identification that included the milder hypoxemia criterion for ALI would yield incidence number

122 Hypoxemia decreased RBCs velocity in intestinal villi bu

123 The primary outcome was frequency of hypoxemia, defined as an Sao2 level of less than 92% wit

124 ectin-1:mIgG1 and Dectin-1:mIgG2a Fc reduced hypoxemia despite minimal effects on fungal burden in th

125 atic sickle cell disease exacerbated by mild hypoxemia, despite a newborn-screening diagnosis of sick

126 Hypoxemia developed in 62 patients (1.9%) in the oxygen

127 Hypoxemia developed in 69% of patients in the blinded ar

128 that trigger white matter injury, transient hypoxemia disrupted SPN arborization and functional matu

129 rs in sepsis patients with severe refractory hypoxemia due to ARDS (2C); prone positioning in sepsis-

130 ve sleep apnea is primarily characterized by hypoxemia due to frequent apneic episodes and fragmentat

131 of advanced liver disease, characterized by hypoxemia due to intrapulmonary vascular dilatations.

132 Hypoxemia, due to a number of different reasons, is comm

133 cted at limiting the duration or severity of hypoxemia during brain development may mitigate disturba

134 of oxygen delivery and, thus, is at risk for hypoxemia during emergency airway management.

135 warning system for hypoxemia, would decrease hypoxemia during endoscopy.

136 to providing a reasonable safeguard against hypoxemia during laryngoscopy and endotracheal intubatio

137 Treatment for hypoxemia during OLV also needs to be modified.

138 = 0.03) and subjects with any two events of hypoxemia during sedation, maintenance or recovery were

139 Marked hypoxemia during sleep may occur in hepatopulmonary synd

140 ge in body mass index, severity of nocturnal hypoxemia, E/E', and sex were independently associated w

141 hyperalgesia in volunteers, while nocturnal hypoxemia enhanced morphine analgesic potency in childre

142 No systemic hypertension and no arterial hypoxemia existed to explain the PAH.

143 Mean percentages of recorded time with hypoxemia for the least and most affected 10% of infants

144 As hypoxemia frequently occurs simultaneously with hemorrha

145 Mice were randomly assigned to hypoxemia group in which we decreased inspired oxygen fr

146 We now studied if mild prolonged hypoxemia had an adverse effect on the fetal cardiovascu

147 Transplant recipients with more severe hypoxemia had increased risk of death after liver transp

148 Ictal hypoxemia has been reported in small series of cases and

149 EAS) for severe left atrial hypertension and hypoxemia has not been critically evaluated.

150 ; 95% CI, 2.33-3.28; p < 0.01) and of severe hypoxemia (hazard ratio, 1.29; 95% CI, 1.14-1.47; p < 0.

151 , since he developed progressive dyspnea and hypoxemia, he was admitted to our hospital.

152 Obstructive sleep apnea causes intermittent hypoxemia, hemodynamic fluctuations, and sleep fragmenta

153 ressure level of 40 mm Hg during 30 minutes, hypoxemia-hemorrhagic shock group in which PaO2 was decr

154 opped in a synergistic manner (69% +/- 3% in hypoxemia-hemorrhagic shock group vs 94 +/- 2 in hemorrh

155 igations in animals suggest that concomitant hypoxemia hinders resuscitation attempts, and that epine

156 Carotid body chemoreceptors sense hypoxemia, hypercapnia, and acidosis and play an importa

157 in total lung capacity, resulting in chronic hypoxemia, hypercapnia, and increased erythropoietin syn

158 e high risk for death associated with severe hypoxemia, hypercapnia, or both not responding to maximi

159 e high risk for death associated with severe hypoxemia, hypercapnia, or both not responding to maximi

160 ologic (Glasgow Coma Scale) and respiratory (hypoxemia, hypercapnia, or nursing requirements for comp

161 physiologically detrimental conditions (e.g. hypoxemia, hypercarbia, acidosis, hypothermia, hypervole

162 Every blood gas derangement (hypoxemia, hyperoxemia, hypocapnia, hypercapnia, and aci

163 ery are reviewed, relative to the threats of hypoxemia, hyperoxia and mechanical lung injury.

164 in adults, the initial severity of arterial hypoxemia in children correlates well with mortality.

165 fetuses enhanced the vasodilator response to hypoxemia in circulations in which blood flow is known t

166 The management of hypoxemia in critically ill patients is challenging.

167 vel related strategies for the management of hypoxemia in critically ill patients.

168 ation-perfusion mismatch is a major cause of hypoxemia in cystic fibrosis.

169 on (PAH) is commonly associated with chronic hypoxemia in disorders such as chronic obstructive pulmo

170 ve signals all noninvasively-measured during hypoxemia in healthy volunteers include four signals mea

171 ere selected that explored the physiology of hypoxemia in healthy volunteers or critically ill patien

172 corporeal membrane oxygenation) to alleviate hypoxemia in patients unable to maintain reasonable oxyg

173 oes not appear to lead to increased rates of hypoxemia in patients undergoing ambulatory upper endosc

174 etermine the incidence and severity of ictal hypoxemia in patients with localization-related epilepsy

175 med on these 25 subjects to examine cerebral hypoxemia in specific regions (periventricular white mat

176 Nocturnal hypoxemia in subjects at high risk for OSA was associate

177 ol metabolism in erythrocytes, and implicate hypoxemia in the pathobiology of erythrocyte-based vascu

178 ric results, whereas there was more profound hypoxemia in the PH group.

179 esponses to stress and diminished arousal to hypoxemia, in part because of failures in genetically de

180 entrations; each 10% increase in duration of hypoxemia increased BNP by 9.6% (95% confidence interval

181 ociation of HD with severe SDB and nocturnal hypoxemia independent of age, BMI, and the higher preval

182 cally derived apnea-hypopnea index (AHI) and hypoxemia index (percent of sleep time with oxyhemoglobi

183 using the apnea-hypopnea index (AHI) and the hypoxemia index (percent sleep time below 90% oxyhemoglo

184 number of apneas/hypopneas per hour) and the hypoxemia index (percentage of time below 90% O2 saturat

185 s significantly associated with both AHI and hypoxemia index after adjustment for age, sex, ethnicity

186 ckness was significantly associated with the hypoxemia index but not with AHI.

187 A higher AHI and higher hypoxemia index were also associated with larger LV dias

188 For categories of increasing severity of the hypoxemia index, the corresponding relative hazards were

189 The severity of nocturnal hypoxemia influences the magnitude of renal, but not the

190 inority of patients with ARDS and refractory hypoxemia, institution of these therapies may be conside

191 Refractory hypoxemia is a common finding in these patients and can

192 Permissive hypoxemia is a concept that is untested clinically and r

193 Hypoxemia is a feared complication of acute liver failur

194 Hypoxemia is a risk factor for long-term neuropsychologi

195 to many pathophysiological causes; avoiding hypoxemia is an important objective during neonatal anes

196 Arterial hypoxemia is associated with cerebral and retinal venous

197 Hypoxemia is common during endotracheal intubation of cr

198 Hypoxemia is common in diabetes, and reflex responses to

199 Tissue hypoxemia is common in several pathological diseases, in

200 Hypoxemia is due to many pathophysiological causes; avoi

201 are consistent with an injury model in which hypoxemia is exacerbated by endotoxin-mediated failure o

202 Hypoxemia is mainly due to intrapulmonary shunt, whereas

203 The aim of permissive hypoxemia is to minimize the possible harms caused by re

204 current arousals from sleep and intermittent hypoxemia) is common among older adults.

205 clude species that tolerate acute or chronic hypoxemia like deep-diving mammals and high-altitude inh

206 d in reduced carotid arterial O2 saturation (hypoxemia), lung pathology, pulmonary edema, reduced lun

207 Lipoplexes induced no weight loss, hypoxemia, lung dysfunction, pulmonary edema, or pulmona

208 fits of attempting to fully reverse arterial hypoxemia may be outweighed by the harms associated with

209 Since intermittent hypoxemia may underlie cardiovascular sequelae of sleep

210 r other interventions targeted at mitigating hypoxemia, may be inadequately appreciated.

211 Acute interventions specifically targeting hypoxemia, metabolic acidosis, hyperglycemia, and cardio

212 lusive categories of ARDS based on degree of hypoxemia: mild (200 mm Hg < PaO2/FIO2 </= 300 mm Hg)

213 The severe hypoxemia, moderate volume loss, and perfusion patterns

214 sk intolerance (n = 11, 30%), and refractory hypoxemia (n = 1, 2.7%).

215 ratory distress syndrome use some measure of hypoxemia, neither the Berlin definition nor recently pr

216 Ictal hypoxemia occurs often in patients with localization-rel

217 rotective factor of the occurrence of severe hypoxemia (odds ratio, 0.146; 95% CI, 0.01-0.90; p = 0.0

218 -venous extracorporeal membrane oxygenation, hypoxemia (odds ratio, 1.68; 95% CI, 1.09-2.57) and mode

219 In conclusion, hypoxemia of HPS is frequently progressive.

220 gy to withstand episodes of subsequent acute hypoxemia of the type that may occur during labor and de

221 improve survival among patients with severe hypoxemia on conventional mechanical ventilation.

222 fetuses were exposed to 2 episodes of acute hypoxemia, on separate days, during infusion with either

223 preterm infants may experience intermittent hypoxemia or bradycardia for many weeks after birth.

224 n to intestinal venules compared with either hypoxemia or hemorrhagic shock alone.

225 transient worsening of mismatch by episodic hypoxemia or hypotension also reproducibly triggers PIDs

226 n of the uterine vasculature during maternal hypoxemia or hypotension makes this goal a significant c

227 depression or airway obstruction leading to hypoxemia or hypoventilation.

228 anagement of isolated fast breathing without hypoxemia or other clinical signs of illness in term you

229 exposing children to recurrent intermittent hypoxemia or oxidative stress, may amplify the adverse e

230 tatus (mean nocturnal SaO2, >/=90% [moderate hypoxemia] or <90% [severe hypoxemia]) and 13 obese cont

231 The presence of moderate hypoxemia (oxygen saturation </=96%; LR, 2.8 [95% CI, 2.

232 fference in coronary flow was accentuated by hypoxemia (P < 0.001).

233 , however, depending on baseline severity of hypoxemia (P = 0.0003), with harm increasing with PaO2/F

234 hypoxemia compared with those with moderate hypoxemia (P = 0.001) and obese control subjects after 3

235 n patients with COPD who have severe resting hypoxemia (Pao(2) </=55 mm Hg or Spo(2) </=88%) (Grade:

236 The relationships between hypoxemia (PaO2 < 60mm Hg), normoxia (PaO2 60-100mm Hg),

237 en therapy in patients with COPD and resting hypoxemia (Pao2 < or =55 mm Hg).

238 ated patients admitted to the ICU with acute hypoxemia (PaO2/FIO2 </= 300) and bilateral opacities.

239 44 [54%] vs 135 [72%]; p=0.006), more severe hypoxemia (PaO2/FIO2: 165+/-73 mm Hg vs 199+/-79 mm Hg;

240 Severe hypoxemia percentages in the blinded and open arms were

241 bariatric surgery, the severity of nocturnal hypoxemia predicted Angptl4 levels in subcutaneous adipo

242 CI, 1.13-4.56), but not obstructive apnea or hypoxemia, predicted incident atrial fibrillation.

243 Hypoxemia, pulmonary edema, and levels of BALF alveolar

244 and critical care medicine, including apnea, hypoxemia, pulmonary hypertension, asthma, cystic fibros

245 Episodes of hypoxemia (pulse oximeter oxygen saturation <80%) or bra

246 vels correlated with the degree of nocturnal hypoxemia (r=0.68, P=0.001).

247 CPAP corrected OSA and hypoxemia (RDI: 42 +/- 4 vs. 4 +/- 1 h(-1), P < 0.001; d

248 emental approach to the management of severe hypoxemia requires implementation of the strategies revi

249 unit patients) were characterized by severe hypoxemia (requiring high levels of inspired oxygen and

250 Hypoxemia resolved after the institution of chemotherapy

251 Tachypnea and hypoxemia resolved faster in older children (P = 0.0001)

252 re indicated in cases of fluid retention and hypoxemia, respectively.

253 opulmonary resuscitation results in profound hypoxemia, respiratory acidosis, and significantly worse

254 eatening situations associated with profound hypoxemia, respiratory acidosis, or hemodynamic compromi

255 markers of obstructive sleep apnea severity (hypoxemia, respiratory disturbances, and sleep fragmenta

256 ertrophy and thickening were associated with hypoxemia, respiratory disturbances, and sleep fragmenta

257 on to lower respiratory tract disease (LRD), hypoxemia, respiratory failure, and overall and influenz

258 After WLS, there was rapid onset of profound hypoxemia resulting in acute pulmonary hypertension and

259 s of overwhelming pulmonary inflammation and hypoxemia, resulting in high morbidity and mortality.

260 Intriguingly, postnatal hypoxemia, ROS scavenging, or inhibition of DDR all prol

261 bance index [RDI] > 15 h(-1)) with nocturnal hypoxemia (SaO2 < 90% for >12% of the night) were studie

262 not demonstrate any effect on postoperative hypoxemia, Sao2 level, or postoperative pulmonary compli

263 The primary end point was the occurrence of hypoxemia; secondary end points were the occurrences of

264 e presence of terminal illness, tachypnea or hypoxemia, septic shock, platelet count <150,000 cells/m

265 ective ERCP/EUS by reducing the frequency of hypoxemia, severe hypoxemia, and apnea.

266 OSA) were higher in OSA and correlated with hypoxemia severity.

267 adherence, major complications, and rates of hypoxemia (SpO2 <90%).

268 25 h(-1)), stratified according to nocturnal hypoxemia status (mean nocturnal SaO2, >/=90% [moderate

269 management) were similar in mild and severe hypoxemia subsets as defined by PaO(2)/Fio(2) ratios.

270 ients with critical illness characterized by hypoxemia such as acute respiratory distress syndrome.

271 of severe respiratory failure and refractory hypoxemia such as that seen in patients with severe acut

272 blood flow is known to increase during acute hypoxemia, such as the umbilical vascular bed.

273 However, the burden of hypoxemia (the time spent with oxygen saturation <90%) s

274 In patients with moderate-to-severe hypoxemia, the expired tidal volume above 9.5 mL/kg pred

275 After 5 days of chronic hypoxemia, the umbilical cord was completely occluded fo

276 Secondly, we describe permissive hypoxemia: the acceptance of levels of arterial oxygenat

277 epatopulmonary syndrome leads to progressive hypoxemia through diffuse vasodilatation of the pulmonar

278 ral vasoconstriction during subsequent acute hypoxemia through elevated nitric oxide (NO) activity.

279 Some measures of hypoxemia to guide 'need' for transfusion have potential

280 o evaluate adherence to process measures and hypoxemia trends over time.

281 Finally, we chemically induce hypoxemia via hemolytic anemia resulting in HIF stabiliz

282 nsing is essential to avoid life-threatening hypoxemia via hypoxic pulmonary vasoconstriction (HPV) w

283 Sleep-related hypoxemia was also associated with glucose intolerance i

284 rease in Kupffer cell phagocytosis following hypoxemia was also prevented by Wortmannin or YC-1 treat

285 When hypoxemia was associated to hemorrhagic shock, it decrea

286 Hypoxemia was associated with an increased mortality in

287 Worsening hypoxemia was associated with initiation of rescue thera

288 This level of hypoxemia was insufficient to affect the occurrence of a

289 In vivo, hypoxemia was more severe in Cx40-/- mice than in wild-t

290 Severity of hypoxemia was not associated with the blood pressure or

291 ected myocardial infarction who did not have hypoxemia was not found to reduce 1-year all-cause morta

292 erichia coli endotoxin infusion until severe hypoxemia was obtained.

293 ith hyperleukocytosis are at risk for severe hypoxemia, which may be real or spurious.

294 ent in patients treated with efaproxiral was hypoxemia, which was reversible and effectively managed

295 ivation and accumulation in the lung lead to hypoxemia, widespread tissue damage, and respiratory fai

296 xycycline caused progressive lung injury and hypoxemia with a high mortality rate.

297 ired vasodilation and oxygen delivery during hypoxemia with advancing age.

298 scopy following hypoventilation may decrease hypoxemia without compromising patient tolerance.

299 Thus, an effective and rapid treatment for hypoxemia would be revolutionary.

300 n to function as an early warning system for hypoxemia, would decrease hypoxemia during endoscopy.