ライフサイエンスコーパス: hypoxemic

1 neonates who require BAS are typically more hypoxemic.

2 nd in group D, was neither hypertrophied nor hypoxemic.

3 sly as normoxemic or a normoxemic patient as hypoxemic.

4 chanical ventilation in patients who develop hypoxemic acute respiratory failure after abdominal surg

5 outcomes of immunocompromised patients with hypoxemic acute respiratory failure treated with high-fl

6 ompromised patients admitted to the ICU with hypoxemic acute respiratory failure, early noninvasive v

7 In immunocompromised patients with hypoxemic acute respiratory failure, high-flow nasal oxy

8 tality among immunocompromised patients with hypoxemic acute respiratory failure.

9 itically ill immunocompromised patients with hypoxemic acute respiratory failure.

10 itically ill immunocompromised patients with hypoxemic acute respiratory failure.

11 baseline physiological values with low-flow hypoxemic and normoxemic perfusion but not with low-flow

12 ons 45 (46%) were hypotensive, 23 (24%) were hypoxemic, and 29 (30%) were mixed.

13 ved across the bioenergetically more active, hypoxemic, and acidotic femoral circulation (P<0.05 vers

14 The unmatched, severely hypoxemic, and younger ECMO-treated patients had, howeve

15 in groups B and C, was hypertrophied but not hypoxemic; and in group D, was neither hypertrophied nor

16 Patients with hypoxemic ARF in the NPPV group had a significantly lowe

17 may only have a phenotype when the animal is hypoxemic at altitude.

18 uring physical activity but are not severely hypoxemic at rest; (2) efficacy of LTOT in subjects with

19 where the heart is chronically perfused with hypoxemic blood.

20 ventricular free wall, which in group A, was hypoxemic but not hypertrophied; in groups B and C, was

21 t or failed intubation increases the risk of hypoxemic cardiopulmonary arrest and/or pulmonary aspira

22 he hypothalamo-pituitary-adrenal axis during hypoxemic challenges to homeostasis in a fashion similar

23 mproves exercise tolerance of normoxemic and hypoxemic chronic obstructive pulmonary disease (COPD) p

24 exercise testing in 27 patients with severe hypoxemic chronic obstructive pulmonary disease (COPD),

25 tudy in healthy volunteers showed that under hypoxemic conditions hepcidin is repressed and duodenal

26 oxide (NO) in the circulation, especially in hypoxemic conditions.

27 Eleven stable hypoxemic COPD patients (mean +/- SD: FEV1 = 1.00 +/- 0.

28 d dyspnea reductions in patients with severe hypoxemic COPD.

29 oxemia as candidate management strategies in hypoxemic critically ill patients.

30 ed to 36 weeks' postmenstrual age, prolonged hypoxemic episodes during the first 2 to 3 months after

31 t were recorded at 3-minute intervals during hypoxemic episodes lasting 15 minutes to 2 hours.

32 The number of hypoxemic episodes lasting 2 minutes or longer per 100 h

33 sociation was significant only for prolonged hypoxemic episodes lasting at least 1 minute (relative r

34 Hypoxemic episodes were associated with an estimated inc

35 The rate of hypoxemic events continued to decrease (from 8.1 events

36 The numbers of hypoxemic events in the blinded and open arms were 132 a

37 Thirty-five percent of all hypoxemic events occurred with completely normal ventila

38 18 months of 56.5% in the highest decile of hypoxemic exposure vs 36.9% in the lowest decile (modele

39 nrolled patients who were progressively more hypoxemic; exposure to the prone position was extended f

40 Hypoxemic, normoxemic, and AO-induced hyperoxemic blood

41 31 hypoxemic (Pao2 </= 7.3 kPa) and 63 less hypoxemic (Pao2 > 8.0 kPa) patients with COPD (39% vs. 5

42 OPD cohort, PFO prevalence was similar in 31 hypoxemic (Pao2 </= 7.3 kPa) and 63 less hypoxemic (Pao2

43 error in SpO2 was never enough to classify a hypoxemic patient erroneously as normoxemic or a normoxe

44 d has not been evaluated in mild-to-moderate hypoxemic patients for whom high-flow nasal cannula oxyg

45 ophageal echocardiography when investigating hypoxemic patients in the intensive care unit, to assess

46 Acute hypoxemic patients with bilateral infiltrates treated wi

47 py improves submaximal exercise tolerance in hypoxemic patients with chronic obstructive pulmonary di

48 Hypoxemic patients with COPD do not have a higher preval

49 cannula, and a demand flow device in 15 male hypoxemic patients with COPD.

50 RBCs from hypoxemic patients with elevated pulmonary arterial pres

51 ial oxygen content and oxygen consumption in hypoxemic patients, identify patient subgroups at higher

52 s desaturation during intubation of severely hypoxemic patients, it does not allow for per-procedure

53 Corticosteroids are an important adjunct for hypoxemic patients.

54 All patients were hypoxemic (PO2 64.2 +/- 8.2 mm Hg), and 5 of 21 (24%) we

55 o improve oxygenation in children with acute hypoxemic respiratory failure (AHRF), but their roles in

56 s receiving this drug for treatment of acute hypoxemic respiratory failure (AHRF), in order to determ

57 outcomes of nonintubated patients with acute hypoxemic respiratory failure (AHRF).

58 ng noninvasive ventilation for de novo acute hypoxemic respiratory failure (i.e., not due to exacerba

59 iratory failure patients (n = 95), and acute hypoxemic respiratory failure (n = 144).

60 s used ECLS for 100 adults with severe acute hypoxemic respiratory failure (n = 94): paO2/FiO2 ratio

61 acute respiratory infection had more severe hypoxemic respiratory failure (PaO2/FIO2: 106 [66, 160]

62 ad undergone abdominal surgery and developed hypoxemic respiratory failure (partial oxygen pressure <

63 d patients without hypercapnia who had acute hypoxemic respiratory failure and a ratio of the partial

64 r the treatment of large patients with acute hypoxemic respiratory failure and asymmetric lung diseas

65 MERS-CoV causes severe acute hypoxemic respiratory failure and considerable extrapulm

66 genation in near-term and term newborns with hypoxemic respiratory failure and persistent pulmonary h

67 support in near-term and term newborns with hypoxemic respiratory failure and persistent pulmonary h

68 brane oxygenation is needed in neonates with hypoxemic respiratory failure and pulmonary hypertension

69 severe end of this spectrum may present with hypoxemic respiratory failure and pulmonary infiltrates,

70 nger, required assisted ventilation, and had hypoxemic respiratory failure as defined by an oxygenati

71 y reveals diffuse bilateral infiltrates, and hypoxemic respiratory failure develops despite appropria

72 Among patients with hypoxemic respiratory failure following abdominal surger

73 cillatory ventilation for treatment of acute hypoxemic respiratory failure in children with diffuse a

74 e of high mortality rate among patients with hypoxemic respiratory failure in the intervention arm (8

75 hould be administered in patients with acute hypoxemic respiratory failure is debated.

76 Although pediatric survivors of acute hypoxemic respiratory failure perceive neither a limitat

77 admission to the intensive care unit due to hypoxemic respiratory failure requiring mechanical venti

78 tions, was able to support the same level of hypoxemic respiratory failure secondary to acute lung in

79 als but in a higher percent of patients with hypoxemic respiratory failure than reported in these tri

80 distress syndrome (ARDS) is a form of severe hypoxemic respiratory failure that is characterized by i

81 ALI is a syndrome of acute hypoxemic respiratory failure that is not primarily card

82 e hospital mortality for patients with acute hypoxemic respiratory failure who failed NPPV was 64%.

83 on, pulmonary hypertension, and severe acute hypoxemic respiratory failure who underwent endotracheal

84 t of eosinophilic lung disease develop acute hypoxemic respiratory failure with a rapid response to t

85 ritical illness syndrome consisting of acute hypoxemic respiratory failure with bilateral pulmonary i

86 ng noninvasive ventilation for de novo acute hypoxemic respiratory failure, and a high expired tidal

87 In 217 children with acute hypoxemic respiratory failure, initial end-tidal alveola

88 ot in patients with post-extubation failure, hypoxemic respiratory failure, or end-stage cancer.

89 In patients with nonhypercapnic acute hypoxemic respiratory failure, treatment with high-flow

90 (n = 51, 82%) was the main etiology of acute hypoxemic respiratory failure.

91 tric oxide (NO) in four patients with severe hypoxemic respiratory failure.

92 of ventilator support in children with acute hypoxemic respiratory failure.

93 -aged patients and is associated with severe hypoxemic respiratory failure.

94 d conditions and presented with acute severe hypoxemic respiratory failure.

95 Mechanical ventilation was begun for hypoxemic respiratory failure.

96 al fibrosis with decreasing lung volumes and hypoxemic respiratory failure.

97 is established therapy for term infants with hypoxemic respiratory failure.

98 th high hospital mortality for patients with hypoxemic respiratory failure.

99 aled nitric oxide in premature newborns with hypoxemic respiratory failure.

100 ailure patients, and 86% in 57 patients with hypoxemic respiratory failure.

101 d nitric oxide in the premature newborn with hypoxemic respiratory failure.

102 nt for intensive care in children with acute hypoxemic respiratory failure.

103 ane oxygenation in term neonates with severe hypoxemic respiratory failure.

104 n clinical outcome in neonatal patients with hypoxemic respiratory failure.

105 onia with pleural effusion and clinically as hypoxemic respiratory insufficiency.

106 S, 25 cirrhotic patients without HPS, and 15 hypoxemic subjects with intrinsic lung disease alone.

107 Fibrin deposition is a salient feature of hypoxemic vasculature and results from induction of tiss