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1 elium-dependent vasodilatation, and enhanced hypoxic pulmonary vasoconstriction.
2 er increases in [Ca(2+)](i) that cause acute hypoxic pulmonary vasoconstriction.
3 exacerbated by endotoxin-mediated failure of hypoxic pulmonary vasoconstriction.
4 l, to the pulmonary selectivity required for hypoxic pulmonary vasoconstriction.
5 ity in acute hypoxia, consistent with uneven hypoxic pulmonary vasoconstriction.
6 ponsible for the modulatory effects of pH on hypoxic pulmonary vasoconstriction.
7 arterial resistance is most consistent with hypoxic pulmonary vasoconstriction.
8 ient mice, sildenafil markedly blunted acute hypoxic pulmonary vasoconstriction.
9 tes for the K(+) channel isoforms initiating hypoxic pulmonary vasoconstriction.
10 receptor activity of epinephrine attenuated hypoxic pulmonary vasoconstriction.
11 decreases in Pao2/Fio2 ratio, and a loss of hypoxic pulmonary vasoconstriction (48 hrs, -90.5% vs. b
18 mitochondrial metabolism by hypoxia to acute hypoxic pulmonary vasoconstriction and progression of pu
20 by HbFe(II)NO-bearing RBCs may contribute to hypoxic pulmonary vasoconstriction and the rebound effec
22 ondria function as the O2 sensors underlying hypoxic pulmonary vasoconstriction by releasing reactive
23 lmonary microvessels and is the mechanism of hypoxic pulmonary vasoconstriction failure associated wi
24 provides evidence for a severe impairment of hypoxic pulmonary vasoconstriction following combined bu
26 sponsible for sensing hypoxia and initiating hypoxic pulmonary vasoconstriction (HPV) are unclear.
27 hemoglobin (Hb) and red blood cells augment hypoxic pulmonary vasoconstriction (HPV) by scavenging n
38 binoid anandamide (AEA) is a key mediator of hypoxic pulmonary vasoconstriction (HPV) via fatty acid
39 tial to avoid life-threatening hypoxemia via hypoxic pulmonary vasoconstriction (HPV) which matches p
41 a potential site of O(2) sensing underlying hypoxic pulmonary vasoconstriction (HPV), but 2 disparat
53 ry vessels, which may explain the failure of hypoxic pulmonary vasoconstriction in acute respiratory
54 3) dipyridamole blunts the severity of acute hypoxic pulmonary vasoconstriction in children with exag
55 of the proximal region of the ETC attenuates hypoxic pulmonary vasoconstriction in rat lungs and bloc
58 tude include hyperventilation, polycythemia, hypoxic pulmonary vasoconstriction-increased intracellul
63 thesis of the etiology of HAPE suggests that hypoxic pulmonary vasoconstriction is extensive but not
64 striction and attempted to determine whether hypoxic pulmonary vasoconstriction is influenced by pyri
67 addition to airway obstruction, the loss of hypoxic pulmonary vasoconstriction may help to explain w
68 hanism also may be an important component of hypoxic pulmonary vasoconstriction, memory, and circadia
71 n polyoxyethylene conjugate had no effect on hypoxic pulmonary vasoconstriction or pulmonary shunt.
72 We found that dasatinib treatment attenuated hypoxic pulmonary vasoconstriction responses and increas
73 shunt or may worsen oxygenation by reversing hypoxic pulmonary vasoconstriction, therapy increasing v
74 arterial smooth muscle cells responsible for hypoxic pulmonary vasoconstriction to limit perfusion of
78 redictions of a computer lung model in which hypoxic pulmonary vasoconstriction was suppressed by end
79 Inhaled nitric oxide was able to abolish hypoxic pulmonary vasoconstriction when insufflated into
80 lation of ENO provided immediate relief from hypoxic pulmonary vasoconstriction without affecting sys
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