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1 elium-dependent vasodilatation, and enhanced hypoxic pulmonary vasoconstriction.
2 er increases in [Ca(2+)](i) that cause acute hypoxic pulmonary vasoconstriction.
3 exacerbated by endotoxin-mediated failure of hypoxic pulmonary vasoconstriction.
4 l, to the pulmonary selectivity required for hypoxic pulmonary vasoconstriction.
5 ity in acute hypoxia, consistent with uneven hypoxic pulmonary vasoconstriction.
6 ponsible for the modulatory effects of pH on hypoxic pulmonary vasoconstriction.
7  arterial resistance is most consistent with hypoxic pulmonary vasoconstriction.
8 ient mice, sildenafil markedly blunted acute hypoxic pulmonary vasoconstriction.
9 tes for the K(+) channel isoforms initiating hypoxic pulmonary vasoconstriction.
10  receptor activity of epinephrine attenuated hypoxic pulmonary vasoconstriction.
11  decreases in Pao2/Fio2 ratio, and a loss of hypoxic pulmonary vasoconstriction (48 hrs, -90.5% vs. b
12                                              Hypoxic pulmonary vasoconstriction, a protective mechani
13                     In Group "E+OA" (n = 5), hypoxic pulmonary vasoconstriction after ALI was blocked
14                             We conclude that hypoxic pulmonary vasoconstriction and a profound catech
15          We studied the effects of sepsis on hypoxic pulmonary vasoconstriction and attempted to dete
16 that pergolide might have similar effects on hypoxic pulmonary vasoconstriction and Kv channels.
17      Therapy with supplemental oxygen alters hypoxic pulmonary vasoconstriction and modulates the Hal
18 mitochondrial metabolism by hypoxia to acute hypoxic pulmonary vasoconstriction and progression of pu
19                                              Hypoxic pulmonary vasoconstriction and responses to exog
20 by HbFe(II)NO-bearing RBCs may contribute to hypoxic pulmonary vasoconstriction and the rebound effec
21           The cellular mechanisms underlying hypoxic pulmonary vasoconstriction are not fully underst
22 ondria function as the O2 sensors underlying hypoxic pulmonary vasoconstriction by releasing reactive
23 lmonary microvessels and is the mechanism of hypoxic pulmonary vasoconstriction failure associated wi
24 provides evidence for a severe impairment of hypoxic pulmonary vasoconstriction following combined bu
25                                       Uneven hypoxic pulmonary vasoconstriction has been proposed to
26 sponsible for sensing hypoxia and initiating hypoxic pulmonary vasoconstriction (HPV) are unclear.
27  hemoglobin (Hb) and red blood cells augment hypoxic pulmonary vasoconstriction (HPV) by scavenging n
28                                              Hypoxic pulmonary vasoconstriction (HPV) directs blood f
29                                              Hypoxic pulmonary vasoconstriction (HPV) is a physiologi
30                                              Hypoxic pulmonary vasoconstriction (HPV) is a physiologi
31                                              Hypoxic pulmonary vasoconstriction (HPV) is driven by th
32                                              Hypoxic pulmonary vasoconstriction (HPV) is inhibited by
33                    It has been proposed that hypoxic pulmonary vasoconstriction (HPV) is mediated via
34                                              Hypoxic pulmonary vasoconstriction (HPV) is unique to pu
35                                              Hypoxic pulmonary vasoconstriction (HPV) maintains blood
36                                              Hypoxic pulmonary vasoconstriction (HPV) normally optimi
37                                              Hypoxic pulmonary vasoconstriction (HPV) optimizes pulmo
38 binoid anandamide (AEA) is a key mediator of hypoxic pulmonary vasoconstriction (HPV) via fatty acid
39 tial to avoid life-threatening hypoxemia via hypoxic pulmonary vasoconstriction (HPV) which matches p
40                    This phenomenon is called hypoxic pulmonary vasoconstriction (HPV), and is respons
41  a potential site of O(2) sensing underlying hypoxic pulmonary vasoconstriction (HPV), but 2 disparat
42                Free hemoglobin (Hb) augments hypoxic pulmonary vasoconstriction (HPV), ostensibly by
43                             This phenomenon, hypoxic pulmonary vasoconstriction (HPV), preserves the
44                Sepsis and endotoxemia impair hypoxic pulmonary vasoconstriction (HPV), thereby reduci
45                Sepsis and endotoxemia impair hypoxic pulmonary vasoconstriction (HPV), thereby reduci
46             To assess the role of cPLA(2) in hypoxic pulmonary vasoconstriction (HPV), we measured th
47  perfusion, which is proposed to result from hypoxic pulmonary vasoconstriction (HPV).
48 eculated to contribute to Ca2+ influx during hypoxic pulmonary vasoconstriction (HPV).
49 ndria function as the O(2) sensor underlying hypoxic pulmonary vasoconstriction (HPV).
50  Red blood cells (RBCs) are known to augment hypoxic pulmonary vasoconstriction (HPV).
51 result in membrane depolarization leading to hypoxic pulmonary vasoconstriction (HPV).
52  cells (PASMCs) has been proposed to mediate hypoxic pulmonary vasoconstriction (HPV).
53 ry vessels, which may explain the failure of hypoxic pulmonary vasoconstriction in acute respiratory
54 3) dipyridamole blunts the severity of acute hypoxic pulmonary vasoconstriction in children with exag
55 of the proximal region of the ETC attenuates hypoxic pulmonary vasoconstriction in rat lungs and bloc
56                           Pergolide enhanced hypoxic pulmonary vasoconstriction in the isolated perfu
57            Anorexigens were shown to enhance hypoxic pulmonary vasoconstriction, in part by inhibitin
58 tude include hyperventilation, polycythemia, hypoxic pulmonary vasoconstriction-increased intracellul
59                                              Hypoxic pulmonary vasoconstriction inhibition by Escheri
60                                              Hypoxic pulmonary vasoconstriction is a local reflex in
61                                              Hypoxic pulmonary vasoconstriction is blunted during sep
62                                              Hypoxic pulmonary vasoconstriction is correlated with pu
63 thesis of the etiology of HAPE suggests that hypoxic pulmonary vasoconstriction is extensive but not
64 striction and attempted to determine whether hypoxic pulmonary vasoconstriction is influenced by pyri
65                                              Hypoxic pulmonary vasoconstriction is initiated by inhib
66                                              Hypoxic pulmonary vasoconstriction is the primary physio
67  addition to airway obstruction, the loss of hypoxic pulmonary vasoconstriction may help to explain w
68 hanism also may be an important component of hypoxic pulmonary vasoconstriction, memory, and circadia
69                                              Hypoxic pulmonary vasoconstriction occurred following WL
70                                 For example, hypoxic pulmonary vasoconstriction optimizes ventilation
71 n polyoxyethylene conjugate had no effect on hypoxic pulmonary vasoconstriction or pulmonary shunt.
72 We found that dasatinib treatment attenuated hypoxic pulmonary vasoconstriction responses and increas
73 shunt or may worsen oxygenation by reversing hypoxic pulmonary vasoconstriction, therapy increasing v
74 arterial smooth muscle cells responsible for hypoxic pulmonary vasoconstriction to limit perfusion of
75                                              Hypoxic pulmonary vasoconstriction was assessed as chang
76                                              Hypoxic pulmonary vasoconstriction was assessed as the c
77                                              Hypoxic pulmonary vasoconstriction was repeatedly tested
78 redictions of a computer lung model in which hypoxic pulmonary vasoconstriction was suppressed by end
79     Inhaled nitric oxide was able to abolish hypoxic pulmonary vasoconstriction when insufflated into
80 lation of ENO provided immediate relief from hypoxic pulmonary vasoconstriction without affecting sys

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