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1 aumatic brain injury, and eight patients had hypoxic ischemic encephalopathy.
2 r disability at 18 months among infants with hypoxic-ischemic encephalopathy.
3 ity during the critical period for perinatal hypoxic-ischemic encephalopathy.
4 g/L who died had a cause of death other than hypoxic-ischemic encephalopathy.
5 in young children who did not have neonatal hypoxic-ischemic encephalopathy.
6 sing therapeutic target in infants suffering hypoxic-ischemic encephalopathy.
7 isability in infants with moderate or severe hypoxic-ischemic encephalopathy.
8 s a window of therapeutic opportunity during hypoxic-ischemic encephalopathy.
9 izures, and received a clinical diagnosis of hypoxic-ischemic encephalopathy.
10 hemorrhage (IPH), extraaxial hemorrhage, and hypoxic-ischemic encephalopathy.
11 disability at age 18 months in infants with hypoxic-ischemic encephalopathy.
12 confidence interval) were calculated: adult hypoxic-ischemic encephalopathy: absent 0% (0%-1%), abno
13 stem (adjusted HR, 46.4; 95% CI, 42.2-51.0), hypoxic ischemic encephalopathy (adjusted HR, 23.6; 95%
14 0 days) were compared with severity of acute hypoxic-ischemic encephalopathy and long-term clinical o
15 atal cerebral sinovenous thrombosis, 11 with hypoxic ischemic encephalopathy, and 5 with neonatal art
16 al in neonates (>/=36 weeks' gestation) with hypoxic-ischemic encephalopathy at 18 US centers in the
17 reduces death or disability for infants with hypoxic-ischemic encephalopathy at 36 weeks' or later ge
18 Among term neonates with moderate or severe hypoxic-ischemic encephalopathy, cooling for longer than
19 ' or later gestation with moderate or severe hypoxic-ischemic encephalopathy enrolled at 6 to 24 hour
21 Epo treatment for term infants with moderate hypoxic-ischemic encephalopathy found reduced disability
23 NFIA expression patterns in human neonatal hypoxic-ischemic encephalopathy (HIE) and MS as well as
32 less than or equal to 17 mug/L argue against hypoxic-ischemic encephalopathy incompatible with reawak
33 s who were full-term with moderate or severe hypoxic ischemic encephalopathy, longer cooling, deeper
35 separately analyze the hypoxic component of hypoxic-ischemic encephalopathy, rats were prepared such
36 at 33.5 degrees C for 72 hours for neonatal hypoxic ischemic encephalopathy reduces death or disabil
37 for 72 hours at 33.5 degrees C for neonatal hypoxic-ischemic encephalopathy reduces death or disabil
38 rophylactic barbiturate therapy for neonatal hypoxic-ischemic encephalopathy showed no significant ef
39 trial of whole-body hypothermia for neonatal hypoxic-ischemic encephalopathy showing a significant re
40 18 girls; age range, 2-12 days) with severe hypoxic-ischemic encephalopathy were examined during the
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