ライフサイエンスコーパス: i.a.

1 ermined by injection of BK (1 microgram kg-1 i.a.), des-Arg9-BK (1 microgram kg-1 i.a., a specific ki

2 am kg-1 i.a.), des-Arg9-BK (1 microgram kg-1 i.a., a specific kinin B1 receptor agonist), kinin B2 re

3 were measured before and after 0.56 mg kg(-1)i.a. of the selective nNOS inhibitor S-methyl-l-thiocitr

4 A higher dose of amiloride (5 microg kg(-1), i.a.) not only blocked the pressor and cardioaccelerator

5 ound a dose of amiloride (0.5 microg kg(-1); i.a.) that attenuated the pressor and cardioaccelerator

6 ATP (0.01-10 mg kg-1, i.a.) and alpha,beta-methylene-ATP (1-30 microg kg-1, i.

7 alpha,beta-methylene-ATP (1-30 microg kg-1, i.a.) each induced dose-dependent increases in afferent

8 eceptor antagonist (IL-1ra, 1.5 microg kg-1, i.a.) significantly attenuated the increased activity in

9 Conversely, IL-1ra (1.5 microg kg-1, i.a.) significantly attenuated the responses of five of

10 IL-1beta (15 ng kg-1, i.a.) significantly augmented the responses of 13 of 16

11 IL-1beta (15 ng kg-1, i.a.) significantly enhanced the increased activity of 1

12 Also, 2-methylserotonin (100 microg kg-1, i.a.), a 5-HT3 receptor agonist, stimulated eleven of tw

13 ist, alpha-methylserotonin (100 microg kg-1, i.a.), and the 5-HT1 receptor agonist, 5-carboxamidotryp

14 t, 5-carboxamidotryptamine (100 microg kg-1, i.a.), did not alter the impulse activity of these twelv

15 by alpha,beta-methylene-ATP (30 microg kg-1, i.a.).

16 by alpha,beta-methylene-ATP (30 microg kg-1, i.a.).

17 al afferents to histamine (5-10 microg kg-1, i.a.).

18 a (breathing 8% O2 for 5 min) and adenosine (i.a. infusion for 5 min) before and after administration

19 ebro-ventricular (i.c.v.) or intra-arterial (i.a.) administration to pentobarbital-anesthetized rats

20 made dependent following the intra-arterial (i.a.) infusion of increasing concentrations (35-100 mg k

21 Intra-arterial (i.a.) injection of a high dose of IL-1beta (500 ng kg-1)

22 of rat animal model, after intra-articular (i.a.) administration, and it induces systemic effects.

23 Mixed intra-articular (i.a.) fluid and subsynovial fluid were sampled at the en

24 dipeptides permeate the blood-brain barrier; i.a. Gly-Gln was ineffective.

25 by breath retention), and exposure to cold (i.a., immersions in ice cold water).

26 nist CCPA (2-chloro-N6-cyclopentyladenosine; i.a. infusion for 5 min).

27 -Gln) (5 mg/kg) was also effective following i.a. injection and significantly attenuated the fall in

28 WA children were given i.a. steroids within 2 months of diagnosis; the injectio

29 Reflected fractions calculated from HA i.a. accumulation or subsynovial dilution showed the sam

30 N-ethylcarboxamidoadeno sin e hydrochloride; i.a. infusion for 5 min).

31 ces of surgical treatment of obesity include i.a.: intestinal anastomotic leakage, impaired intestina

32 Intraamniotic (i.a.) injection of SP-A caused preterm delivery of fetus

33 Rabbits were treated with intraarticular (i.a.) injections of caspase inhibitors 3 times per week

34 Cyclo(Gly-Gln) (0.5, 5.0 or 50.0 mg/kg i.a.) had no effect on arterial pressure or heart rate w

35 on of chylous lymph; devazepide (100 microg, i.a.) abolished the afferent response to chylous lymph i

36 sion of the antioxidant vitamin C (25 mg/min i.a.) to determine the portion of FDD inhibited by radic

37 Early and continued use of i.a. steroids may be associated with less LLD in young c

38 temic hypoxia (breathing 8% O2 for 5 min) or i.a. infusion for 5 min of adenosine, the NO donor sodiu

39 Twelve WA children had subsequent i.a. steroid injections (mean 3.25 injections per child

40 third eye meditation), breathing techniques (i.a., cyclic hyperventilation followed by breath retenti

41 e to confounding conditions ancillary tests (i.a. imaging studies) may be useful.

42 The i.a. concentration of HA2000 increased substantially rel

43 o and in vivo, after its conjugation and the i.a. injection of HA-sCT did not trigger any systemic ef

44 togenic diet is used as anti-seizure therapy i.a. in tuberous sclerosis patients, but its impact on c

45 d with NC children who were not treated with i.a. steroids.