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1                                 In addition, i.n. vaccination with PspA and IL-12 provided increased
2 onger Th2 responses following the additional i.n. administration of CT compared to i.n. or i.m. gB DN
3                                Additionally, i.n. immunization elicited a stronger neutralizing serum
4 f Y. pestis in mice when it was administered i.n. but actually reduced the 50% lethal dose (LD(50)) b
5 ach peptide with LT(R192G) were administered i.n. to BALB/c mice.
6 g rNV VLPs are immunogenic when administered i.n. in the absence of adjuvant, and addition of adjuvan
7 ed of alveolar macrophages when administered i.n. P. aeruginosa.
8                           In addition, after i.n. challenge with type 14 pneumococci, vaccinated mice
9 tion of antigen-specific B and T cells after i.n. immunization, antibody-secreting cells and antigen-
10 found especially in central CLN 2 days after i.n. immunization and persisted for up to 6 months, wher
11 nd then from the brain for 3 to 4 days after i.n. immunization.
12 2(-/-) mice died significantly earlier after i.n. infection.
13 presence of bacteria in blood 1 to 3 h after i.n. inoculation was sometimes observed, this was infreq
14 prolonged the survival of treated mice after i.n. F. tularensis challenge relative to mock treated an
15  a mucosal IgA immune response in mice after i.n. immunization supports their use as subunit vaccine
16  B-cell-deficient than in C57BL/6 mice after i.n. inoculation.
17 increased survival of neutropenic mice after i.n. P. aeruginosa inoculation.
18 intranasally (i.n.) with S. pneumoniae after i.n. administration of IL-12.
19 significant OVA-specific CTL responses after i.n. coadministration of LT with OVA or OVA257-264.
20 significantly protected (100% survival after i.n. immunization).
21 ter p.o. immunization was greater than after i.n. administration (means, 6.0 and 1.02 ng/ml, respecti
22                             Therefore, after i.n. immunization, superficial and central CLN represent
23 testinal and systemic lymphoid tissues after i.n. inoculation with two or three doses of 2/6-VLPs wit
24                           Protection against i.n. challenge of C57BL/6 mice was evaluated by vaccinat
25                                     Although i.n. DNA immunization was an effective means of inducing
26                        Furthermore, although i.n. and i.v.-immunized mice had comparable levels of ch
27 mmunodeficient (SCID) C.B-17 mice against an i.n. challenge.
28                      In unimmunized mice, an i.n. challenge with vSC8 caused a significant but self-l
29 ollowing vaccination, each group received an i.n. challenge of P. multocida.
30 the MVA prime/i.n. boost regimen received an i.n./i.m. combined C.1086 gp120 boost.
31 t with anti-CD3epsilon-specific antibody and i.n. proinsulin peptide can reverse recent-onset diabete
32 generated by intramuscular (i.m.) gB DNA and i.n. live HSV administration.
33 ference between the responses after o.g. and i.n. administration was even more remarkable.
34 th of time between influenza A infection and i.n. exposure to OVA was crucial, because mice exposed t
35 host resistance to primary LVS infection and i.n. secondary challenge.
36                   In addition, both i.m. and i.n. plasmid immunizations failed to generate an immune
37 d a greater IgG Ab response in both i.p. and i.n. immunized mice and a greater IgA Ab response in muc
38 sal (i.n.) at a low dose and low volume, and i.n. at a high dose and high volume.
39        Rabbits vaccinated with both antigens i.n. or s.c. had a 100% survival rate, few or no bacteri
40 efficacy of administration of these antigens i.n. versus s.c.
41  detected beginning at 3 days p.i. from both i.n.- and i.p.-challenged animals.
42 ) routes are challenged with the allergen by i.n. administration.
43  and these responses were further boosted by i.n. delivery of M.85A.
44  s.c. inoculation but no virulence change by i.n. inoculation in mice.
45 ificantly elevated compared with controls by i.n. delivery of 100 microgram sIL-4R; i.p. delivery of
46 ite neutralizing antibody can be elicited by i.n. immunization with a flagellin-modified P. falciparu
47 ent influenza A virus infection, followed by i.n. inoculation with S. pneumoniae.
48            The immune responses generated by i.n. administration of gB DNA with or without cholera to
49 , mucosal and systemic antibodies induced by i.n. immunization persisted for at least 12 months.
50               To test whether CTL induced by i.n. immunization with OVA peptide and CT were functiona
51 unable to establish respiratory infection by i.n. inoculation.
52  are the dominant T cells induced in NALT by i.n. infections.
53 low volume and was completely preventable by i.n. vaccination of an attenuated virus at a low dose an
54 cosal immune responses, lung DC targeting by i.n. immunization induced protective immunity against en
55  1 h before or after Schu 4 or LVS (100 CFU) i.n. challenge showed that poly(I:C) treatment significa
56  the last immunization, mice were challenged i.n. with 10(4) inclusion-forming units (IFU) of C. muri
57  the last immunization, mice were challenged i.n. with 10(4) inclusion-forming units (IFU) of the C.
58                                     Combined i.n.-parenteral immunization of ferrets with maternal an
59 ory sequence oligodeoxynucleotide conjugate, i.n. and i.d. IT delivery were similarly effective in mo
60                                 In contrast, i.n. immunization was substantially more effective at in
61 concerned that Salmonella bacteria delivered i.n. might access the brain.
62                Immunization by OMV delivered i.n. was the only regimen that resulted in a serum bacte
63 portantly, mice immunized with LVS DeltacapB i.n. or i.d. and then challenged 6 weeks later by aeroso
64            Mice immunized with LVS DeltacapB i.n. or intradermally (i.d.) developed humoral and cellu
65 duced higher titers (1.63 +/- 0.25) than did i.n. immunization with NYVAC-HF (0.88 +/- 0.36; n = 9) a
66 ansmission, was modeled accurately by direct i.n. inoculation of Sendai virus at a low dose and low v
67 at received two immunizations with low doses i.n. (10 or 25 microg) of rNV VLPs and the majority of m
68  was aspirated into the lungs of mice during i.n. immunization and resulted in an acute inflammatory
69 tide conjugate proved to be a more effective i.n. IT reagent for protecting allergic mice from airway
70 .1-100 microgram) was administered by either i.n. or i.p. routes before OVA challenge in OVA-sensitiz
71      Furthermore, sIL-4R treatment by either i.n. or i.p. routes did not reduce airway hyperreactivit
72 c influenza virus infection following either i.n. or intraperitoneal inoculation.
73                   CRs boosted with Ad5.RSV-F i.n. 28 days after an i.m. dose also had significant inc
74                                     Finally, i.n. immunization of C57BL/6 mice with either a recombin
75                                    Following i.n. challenge with nonlethal doses of H10407 and B7A, t
76                                    Following i.n. immunization, CD1, CD2, and CD4 induced significant
77                                    Following i.n. inoculation, latent DeltaM33B(T2) viral DNA was sig
78 s colonization and caries activity following i.n. immunization with GLU or Thio-GLU are attributed to
79  CD4+ T cells home to mouse brains following i.n. infection.
80 mmune responses to liposomal C-GTF following i.n. immunization.
81 t enhanced morbidity and mortality following i.n. MHV-1 infection compared to wild-type C3H/HeN mice.
82 ivated in the cervical lymph nodes following i.n. inoculation and then differentiated into Th17 cells
83         Transmission was more efficient from i.n.- than from i.p.-challenged gps, with 17% versus 83%
84                               CVD 1203 given i.n. elicited high titers of antilipopolysaccharide (ant
85                                     However, i.n. inoculation of gnotobiotic pigs with 2/6-VLPs did n
86                                 Immunization i.n. with pneumococcal surface protein A (PspA) and IL-1
87                            Animals immunized i.n. on days 0, 28, and 76 with bacterial vectors carryi
88                            Animals immunized i.n. with OVA and CT were protected against tumor develo
89             Positive controls were immunized i.n. with C. trachomatis MoPn elementary bodies (EB).
90  we identified an attenuated and immunogenic i.n. vaccine candidate expressing GP from the pre-N posi
91 levels of IgG2a were four- sixfold higher in i.n.-immunized mice than in any of the other groups.
92 greater IgA Ab response in mucosal washes in i.n. immunized mice compared with SHIV VLPs.
93                     MyD88(-/-) mice infected i.n. had higher numbers of CFU in the lungs as well as h
94                                  The initial i.n. dose-response with bacterial vectors alone identifi
95 o, only 14% (3/21) of the animals inoculated i.n. with Chlamydia had positive vaginal cultures.
96 unogenic in gnotobiotic pigs when inoculated i.n. and that the adjuvant mLT enhanced their immunogeni
97                                  Intranasal (i.n.) administration of a single dose of the H7N7 NL/03
98                                  Intranasal (i.n.) immunization of BALB/c mice with ntPEpilinPAK gene
99                                  Intranasal (i.n.) immunization of C57BL/6 mice with CT, CTA1-DD, LT,
100                                  Intranasal (i.n.) immunization of mice with CAT showed significantly
101                                  Intranasal (i.n.) immunization with bacterial protein antigens coupl
102                                  Intranasal (i.n.) infection of A/J mice with the CoV mouse hepatitis
103                                  Intranasal (i.n.) infections preferentially generate Th17 cells.
104                                  Intranasal (i.n.) inoculation of mice represents an experimental app
105                                  Intranasal (i.n.) inoculation of mice with the attenuated EHV-1 stra
106 ith wild-type C57BL/6 mice after intranasal (i.n.) and intravenous (i.v.) infection with live C. neof
107 les virus (MV) replication after intranasal (i.n.) challenge, this model can be used to assess the ef
108 rts of GI T cell responses after intranasal (i.n.) delivery of antigens that do not directly target t
109 body responses are induced after intranasal (i.n.) immunization of rHagB and if monophosphoryl lipid
110 rotection (80% survival) against intranasal (i.n.) challenge with ~240 median lethal doses (LD50) (2.
111 ignificant protection against an intranasal (i.n.) challenge as determined by the change in body weig
112 ce protection in mice against an intranasal (i.n.) challenge.
113 m of EBOV glycoprotein GP, as an intranasal (i.n.) EBOV vaccine.
114 tive immune responses through an intranasal (i.n.) route in mice.
115 ) not exposed to detergent as an intranasal (i.n.) vaccine.
116  both intraperitoneal (i.p.) and intranasal (i.n.) inoculation, M33 was attenuated for infection of t
117 ce sensitized to OVA by i.p. and intranasal (i.n.) routes are challenged with the allergen by i.n. ad
118 YR) and its Fab fragment (VN) by intranasal (i.n.) administration to infected SCID mice.
119 vaccination protocol followed by intranasal (i.n.) challenge of C57BL/6 mice with an equal number of
120 s type 1 (HIV-1) isolate 89.6 by intranasal (i.n.) immunization of mice with gp120 and gp140 together
121 cant immune responses in mice by intranasal (i.n.) immunization.
122 nicity induced in BALB/c mice by intranasal (i.n.) inoculation of enterotoxigenic Escherichia coli (E
123                     In contrast, intranasal (i.n.) rAd immunization led to similarly robust local tra
124    Here, we found that following intranasal (i.n.) challenge, titers of virus in the lungs of the imm
125  the adult mouse model following intranasal (i.n.) immunization with fragments of VP6 and a subsequen
126 lymphoid tissue (NALT) following intranasal (i.n.) infections is investigated.
127 he lungs of WT animals following intranasal (i.n.) virus inoculation, while STAT1-/- mice developed a
128 al pneumonia, we show that local intranasal (i.n.) or systemic subcutaneous (s.c.) administration of
129  employed: intramuscular (i.m.), intranasal (i.n.) at a low dose and low volume, and i.n. at a high d
130  Following intramuscular (i.m.), intranasal (i.n.), or intravaginal (IVAG) immunization with VEE/SIN-
131             In neutropenic mice, intranasal (i.n.) doses of P. aeruginosa as low as 10 to 100 CFU/mou
132 ccine by either the oral (p.o.), intranasal (i.n.), or rectal route.
133 known concerning the efficacy of intranasal (i.n. ) administration of these antigens in inducing prot
134                  A comparison of intranasal (i.n.) and parenteral immunization of BCG showed that whi
135  we investigated the efficacy of intranasal (i.n.) conjugate vaccine delivery using interleukin-12 (I
136      The protective potential of intranasal (i.n.) immunization with this chimeric immunogen was comp
137 s investigated following oral or intranasal (i.n.) administration of an aqueous adjuvant formulation
138 halitis following i.c., s.c., or intranasal (i.n.) challenge with the virulent VEEV ZPC738 strain (ZP
139 against an intradermal (i.d.) or intranasal (i.n.) challenge with vaccinia virus (vSC8) or a recombin
140 oneal cells after either i.p. or intranasal (i.n.) inoculation.
141 sponses in milk, while MVA prime/intranasal (i.n.) boost induced robust milk Env-specific IgA respons
142 vious study we demonstrated that intranasal (i.n.) vaccination promotes a Th17 biased immune response
143 ministered rLaSota/gp160 via the intranasal (i.n.) or intramuscular (i.m.) route in different prime-b
144 ministered to BALB/c mice by the intranasal (i.n.) route to evaluate the induction of mucosal antibod
145 vant, mice were immunized by the intranasal (i.n.) route with antigen alone or in conjunction with Fl
146 effect of immunizing mice by the intranasal (i.n.) route with Salmonella expressing an insoluble prot
147 ALB/c mice were immunized by the intranasal (i.n.) route with the surface protein adhesin AgI/II of S
148  Adult CD-1 mice infected by the intranasal (i.n.) route, showed that VEEV and WEEV enter the brain t
149  dose of HSV-2 strain 186 by the intranasal (i.n.) route.
150 ized mice, when delivered by the intranasal (i.n.) vs the intradermal (i.d.) route.
151         Mice that received three intranasal (i.n.) immunizations of H3N2 vaccine in the presence of L
152 tolerance induced by exposure to intranasal (i.n.) OVA and the subsequent development of AHR.
153 rotavirus (HRV), followed by two intranasal (i.n.) doses of a rotavirus-like particle (2/6-VLPs) vacc
154 overy and retained virulence via intranasal (i.n.) infection.
155 -G) was administered to mice via intranasal (i.n.), intramuscular (i.m.), and oral inoculation.
156 tivity following repeated weekly intranasal (i.n.) GSK2245035.
157 vaccination routes compared were intranasal (i.n.) and intradermal (i.d.) inoculation of the Francise
158 ; the 50% lethal dose (LD(50)) intranasally (i.n.) is >10,000-fold that of LVS.
159 vere disease when administered intranasally (i.n.) or intraperitoneally (i.p.).
160 ed CS constructs, administered intranasally (i.n.) or subcutaneously (s.c.), developed similar levels
161 livered subcutaneously (s.c.), intranasally (i.n.), i.m., or transcutaneously (t.c.).
162 nea pigs (gps) were challenged intranasally (i.n.) or intraperitoneally (i.p.) with 10,000 times the
163 ) lumen of a cohort challenged intranasally (i.n.) with S. pneumoniae type 6A, which is predominantly
164 cles (PorB VRP) were delivered intranasally (i.n.) or subcutaneously (s.c.) into the dorsal area or t
165 o Fcgamma receptors (FcgammaR) intranasally (i.n.) enhances immunogenicity and protection against int
166 binant priming delivered first intranasally (i.n.) plus orally and then intratracheally (i.t.), follo
167   When used as a vaccine given intranasally (i.n.), INA-inactivated influenza virus induced immune re
168         Animals were immunized intranasally (i.n.) and/or intramuscularly (i.m.) and subsequently cha
169 ve control group was immunized intranasally (i.n.) with 10(4) inclusion-forming units (IFU) of C. tra
170     BALB/c mice were immunized intranasally (i.n.) with gB DNA or DNA expressing beta-galactosidase (
171 nd C57BL/6 mice were immunized intranasally (i.n.) with peptides corresponding to a known CTL epitope
172 c lung response was similar in intranasally (i.n.) sensitized IL-10-/- and wild-type mice from a diff
173           In vivo, BALB/c mice intranasally (i.n.) treated with poly(I:C) (100 microg/mouse) 1 h befo
174 ested by infecting BALB/c mice intranasally (i.n.) with S. pneumoniae after i.n. administration of IL
175 zed intravaginally (i.vag.) or intranasally (i.n.) with a bacterial protein antigen (AgI/II of Strept
176 fected intradermally (i.d.) or intranasally (i.n.) with LVS succumbed to infection with doses 2 log u
177 ize CD4 KO mice either i.p. or intranasally (i.n.).
178 mice, orogastrically (o.g.) or intranasally (i.n.).
179 ministered to gnotobiotic pigs intranasally (i.n.) with a mutant Escherichia coli heat-labile toxin,
180 a virus infection to show that intranasally (i.n.) primed memory CD8+ T cells possess a unique abilit
181 nfected intravaginally (i.v.), intranasally (i.n.), orally (p.o.), or subcutaneously (s.c.) with C. t
182  antibody that were vaccinated intranasally (i.n.) developed lower neutralizing titers, with NYVAC-HF
183         BALB/c mice vaccinated intranasally (i.n.) with KKF24 and subsequently challenged with wild-t
184 nterleukin-12 (IL-12) delivered intransally (i.n.) as an antiviral respiratory adjuvant.
185 econdary challenge but not high doses of LVS i.n. challenge, independently of the route of vaccinatio
186 4 or 8 weeks later with a lethal dose of LVS i.n., they were 100% protected from illness and death an
187 nt than protein boosting by either the i.m., i.n., or t.c. route, suggesting that this route may be p
188                               Thus, maternal i.n./i.m. combined immunization is a novel strategy to e
189                                      In mice i.n. challenged with B7A, low serum IgG antibody titers
190                                      In mice i.n. challenged with H10407, serum immunoglobulin G (IgG
191 la tularensis (iFT) organisms to FcR in mice i.n., with MAb-iFT immune complexes, enhances F. tularen
192 Ure (25 microg p.o. or rectally or 10 microg i.n.) plus heat-labile toxin from Escherichia coli as th
193 mol, i.n.) prevented iron-induced (4.2 nmol, i.n.) oxidative stress and nigral injury, reflected by a
194 usion of freshly prepared GSNO (0-16.8 nmol, i.n.) prevented iron-induced (4.2 nmol, i.n.) oxidative
195 n vitro, T cells from the superficial CLN of i.n. immunized mice secreted both gamma interferon and i
196    In this study, we examined the effects of i.n. IL-12 treatment on induction of protective humoral
197         This indicated that our procedure of i.n. administration of Ab did not make optimal use of th
198 differentiation of mass spectral profiles of i.n.-inoculated mouse lung tissues from those of i.d.-in
199 resulting in the preferential recruitment of i.n.-primed memory CD8+ T cells to the lung airways.
200                            The resistance of i.n.-immunized mice (and to some extent the i.v.-exposed
201   Our studies demonstrate the superiority of i.n. versus i.m. vaccination in protection against both
202 ype 3 pneumococci, there was 75% survival of i.n. vaccinated mice compared to 0% survival of unvaccin
203                        Thus, the tendency of i.n. infection to induce Th17 cells is related to cytoki
204 of priming (intraperitoneal) followed by one i.n. challenge we found that IL-10-/- C57BL/6 mice had h
205 parable T cell responses in the spleen, only i.n. delivery elicited specific T cell responses in the
206  profiles in Th2-sensitized mice, while only i.n. IT had significant immunomodulatory activity on B a
207 ither one or both antigens by either s.c. or i.n. administration.
208 ion of CT compared to i.n. or i.m. gB DNA or i.n. live HSV immunization.
209 tears and serum of guinea pigs after o.g. or i.n. immunization, the i.n. route elicited significantly
210 ties distinct from those elicited by i.m. or i.n. rAd immunization.
211  CRs vaccinated with Ad5.RSV-F given i.m. or i.n., and these responses correlated with reduced replic
212 In animal models of type 1 diabetes, oral or i.n. immunization with islet antigens induces Tregs that
213 onorrhoeae recombinant porin B (Ng-rPorB) or i.n. with Eagle's minimal essential medium (MEM-0).
214 and control C3H/HeOuJ mice following i.v. or i.n. challenge with C. neoformans.
215 en administered at higher doses by the oral, i.n., and i.p. routes than the wild-type strain even tho
216                                     Overall, i.n. administration of ntPEpilinPAK induced mucosal and
217 tide inhibitor of PAR2 signalling, pepducin, i.n. before allergen challenges and then assessed AHR an
218 ungs was consistently detected by day 6 post-i.n. challenge for the immunized mice and by day 14 for
219                      At day 7 postinfection, i.n.- and i.v.-immunized mice had high levels of chlamyd
220 mals previously immunized with the MVA prime/i.n. boost regimen received an i.n./i.m. combined C.1086
221 o 10-fold-higher doses of NOMV were required i.n. compared to i.p. to elicit an equivalent bactericid
222  on day 28, a single intranasal challenge (s.i.n.) with either OVA or ragweed.
223                      Eight hours after the s.i.n., BAL fluid was obtained.
224                      Mice receiving a second i.n. immunization with liposomal antigen and MPL-AF had
225                                     A single i.n. inoculation with KyA induced protective immunity ag
226                           Moreover, a single i.n. or IVAG immunization with VEE/SIN-Gag induced a lar
227                                Specifically, i.n. or s.c. administration of c-di-GMP 48 and 24 h prio
228 tended these studies to compare the standard i.n./i.t. regimen with additional mucosal administration
229 ual antigen can be corrected by a subsequent i.n. virus infection.
230 sponse that is protective against subsequent i.n. challenge with the wild-type strain.
231 o elicit mucosal antibody responses and that i.n. immunization resulted in increased total, immunoglo
232   Collectively, the results demonstrate that i.n. vaccination with KKF24 induces a vigorous Th1-type
233                    Our results indicate that i.n. and possibly even oral delivery of live Salmonella
234                  These results indicate that i.n. delivery of meningococcal NOMV in mice is highly ef
235                    This study indicates that i.n. immunization with both PMT and CN induces an effect
236 l and immunohistochemical findings show that i.n.-infected gps display enhanced lung pathology and EB
237 estigations may be the first to suggest that i.n. IT is more effective than i.d. IT for the treatment
238                     The results suggest that i.n.-challenged gps are more infectious to naive animals
239                                          The i.n. administration of 100 microgram sIL-4R before aller
240                                          The i.n. delivery of rNV VLPs was more effective than the or
241                                          The i.n. immunization induced predominantly IgA antibody-sec
242                                          The i.n. inoculation of BALB/c mice with large doses of ETEC
243                                    After the i.n. challenge mice immunized with MOMP, CpG, and alum s
244 l responses of female mice given VLPs by the i.n. and oral routes were also examined.
245  soluble proteins when coadministered by the i.n. or oral route.
246                        Mice immunized by the i.n. route had higher levels of salivary, plasma, and va
247 ce given AgI/II with LT-IIa or LT-IIb by the i.n. route had significantly higher mucosal and systemic
248  B7-1, B7-2, and B7-1/2 knockout mice by the i.n. route revealed that the ability of FljB to increase
249                               Priming by the i.n. route was more immunogenic than by the i.m. route,
250 .v. 70 days later, animals preexposed by the i.n. route were highly resistant to reinfection, with gr
251                        Mice immunized by the i.n. route with antigen and FljB exhibited significantly
252 ctivity was highest in mice immunized by the i.n. route with antigen formulations containing MPL-AF (
253 unized three times (2-week intervals) by the i.n. route with HagB (20 microg) alone or with MPL (25 m
254 eived phosphate-buffered saline alone by the i.n. route.
255 ea pigs after o.g. or i.n. immunization, the i.n. route elicited significantly higher antibody titers
256 sma IgA and plasma IgG, respectively (in the i.n. immunized groups).
257 urvival was also significantly better in the i.n.-parenteral group (3 of 9) than in the other HF-vacc
258              These studies indicate that the i.n. challenge of BALB/c mice with ETEC strains may prov
259 sease, we found similar results by using the i.n. and intraperitoneal (i.p.) routes of inoculation fo
260            Importantly, immunization via the i.n. but not the s.c. route elicited sporozoite neutrali
261           Two or three immunizations via the i.n. or i.m. route induced a more potent systemic and mu
262 or 100% protection when administered via the i.n. route.
263                                   Therefore, i.n. immunization is a potential delivery route of choic
264                                        Three i.n. doses of gB DNA over a 3-week period resulted in a
265 doses of attenuated Wa HRV (AttHRV3x), three i.n. doses of 2/6-VLPs plus mLT (VLP3x), three i.n. dose
266 n. doses of 2/6-VLPs plus mLT (VLP3x), three i.n. doses of purified double-layered inactivated Wa HRV
267 tional i.n. administration of CT compared to i.n. or i.m. gB DNA or i.n. live HSV immunization.
268  to OVA was crucial, because mice exposed to i.n. OVA 15-30 days after viral inoculation developed ne
269 A abrogated tolerance induced by exposure to i.n. OVA, and instead led to the development of AHR acco
270             i.m. inoculation was inferior to i.n. inoculation at inducing antibody responses and prot
271 t that infant ferrets are less responsive to i.n. vaccination than are older ferrets and raises quest
272 nd subsequently challenged with RSV/A/Tracy (i.n.) to assess protection.
273 wing aerosol challenge with M. tuberculosis, i.n. boosting of BCG with either BCG or M.85A afforded u
274   For the second regimen (VLP2x/AttHRV), two i.n. doses of 2/6-VLPs+mLT were given, followed by one o
275                    Of the animals vaccinated i.n. with the Chlamydia, 81% (17/21) had embryos in both
276                           Rabbits vaccinated i.n. had significant nasal and bronchoalveolar lavage Ig
277 n vaccinated mice challenged with ZPC738 via i.n. or i.c. route, we regularly detected high levels of
278                       High-dose, high-volume i.n. inoculation resulted in the highest levels of antib
279                         Low-dose, low-volume i.n. inoculation afforded complete protection from conta
280                                       Weekly i.n. GSK2245035 20 ng was well tolerated and reduced all
281                               When mice were i.n. challenged 4 months after the last boost, titers of
282                               Mice that were i.n. administered H3N2 vaccine alone, without LT(R192G),
283  lungs and the CLN of animals immunized with i.n. administered beta-Gal DNA.
284 n G (IgG) and Fab cleared the infection with i.n. 50% effective doses (ED(50)s) of 16 and 90 pmol, re

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